ライフサイエンスコーパス: freckle

1 e effect on beard hair colour, skin type and freckling.

2 interval (CI) 1.3-3.1) was seen for current freckling.

3 cafe-au-lait macules, [2] axillary/inguinal freckling, [3] shortened stature, [4] tibial bone curvat

4 Absence of back nevi, presence of many freckles, a sun-sensitive phenotypic index, and prior am

5 nevi and V600K with increased nevi and less freckling (all P < 0.05).

6 People with pale skin, red hair, freckles and an inability to tan--the 'red hair/fair ski

7 Iris freckles and the IPE cyst did not have intrinsic vascula

8 1R and TYR are associated with pigmentation, freckling and cutaneous sun sensitivity, well-recognized

9 probands with familial CALMs with or without freckling and no other NF1 features, 69 (73%; 95% CI, 63

10 dominant phenotype of CALMs with or without freckling and no other NF1 features.

11 significantly more likely to have noticeable freckling and possibly also Fitzpatrick skin types 1-3.

12 tics, absence of back nevi, presence of many freckles, and a sun-sensitive phenotypic index were inde

13 haracterized by cafe-au-lait spots, skinfold freckling, and cutaneous neurofibromas.

14 with cafe au lait macules (CALMs), axillary freckling, and macrocephaly.

15 d/light brown hair, increased nevi, and less freckling, and NRAS(+) with older age relative to the wi

16 These alleles are also linked to red hair, freckling, and sun sensitivity, all of which are known m

17 nocortin 1 receptor (MC1R) lead to red hair, freckling, and sun sensitivity.

18 population such as that leading to red hair, freckling, and sun-sensitivity.

19 to 13; the most common were nevi, skin type, freckle density, age, hair color, and sunburn history.

20 edictors (age, sex, ancestry, nevus density, freckling density, hair color, tanning ability, adult su

21 ed or worsened during treatment were nausea, freckles, headache, and skin hyperpigmentation.

22 ood and adulthood and for mole frequency and freckling in childhood, suggesting some recall bias.

23 melanomas or benign iris lesions, including freckles, nevi, and an iris pigment epithelial (IPE) cys

24 Associations of phenotypic characteristics (freckles, nevi, phenotypic index) and MC1R status with i

25 own to have an excess of skin tumors, if the freckles of PJS patients are actually small, benign tumo

26 resence of more than 5 CALMs with or without freckling or an NF1-compatible family history.

27 esence of pigment dispersion (P = .70), iris freckles (P = .15), corectopia (P > .999), ectropion (P

28 ssess the number and type of nevi, extent of freckling, solar damage, and skin type.

29 K11 gene cause characteristic hamartomas and freckling to develop in patients with Peutz-Jeghers synd