ライフサイエンスコーパス: free fatty acid

1 glycerols) and Peruvian morwong (most EPA as free fatty acid).

2 mpanied by the increase of phospholipids and free fatty acids.

3 l metabolites and increased levels of plasma free fatty acids.

4 neutral lipids, cholesterol, ceramides, and free fatty acids.

5 inantly triacylglycerols, sterol esters, and free fatty acids.

6 ction profiles but shared elevated levels of free fatty acids.

7 epatic uptake of adipose-derived circulating free fatty acids.

8 tabolites in muscle and elevated circulating free fatty acids.

9 despite normal insulin suppression of plasma free fatty acids.

10 the hydrolysis of TGs to diacylglycerols and free fatty acids.

11 y hydrolyses glycerophospholipids to release free fatty acids.

12 as a control group with elevated circulating free fatty acids.

13 eling indicated that these cells accumulated free fatty acids.

14 ns and increased circulating levels of total free fatty acids.

15 , lipid oxidation products, antioxidants and free fatty acids.

16 oximately eightfold basal) while suppressing free fatty acids.

17 or of their actions is the G protein-coupled free fatty acid 2 (FFA2) receptor, and this has been sug

18 In pxa1 mutants, in which free fatty acids accumulated rapidly under extended dark

19 ssue (VAT), intrahepatic lipid (IHL), plasma free fatty acids, adipose cell diameter, and percentage

20 , but the ability of this solvent to extract free fatty acids also decreases.

21 This initial difference in free fatty acid amount was only partially buffered by th

22 Culturing renal proximal tubular cells with free fatty acid and FXR agonists showed that FXR activat

23 isted aqueous process had a lower content of free fatty acid and lower color imparting components tha

24 Spectroscopic profiles, fatty acid, free fatty acid and total phenol contents of pumpkin see

25 on, and growth hormone levels increased, and free fatty acids and 3-hydroxybutyrate concentrations an

26 nd their deaminated metabolites, and lowered free fatty acids and acylcarnitines.

27 onged to the classes of carbohydrates, while free fatty acids and amino acids, among which precursors

28 ted hepatic triglycerides, attenuated plasma free fatty acids and attenuated lipoprotein lipase activ

29 ations in glucose metabolism, divergences in free fatty acids and carnitine conjugated lipid levels,

30 is a critical lipid hydrolase that generates free fatty acids and cholesterol.

31 cium (Ca(2+)) depletion induced by saturated free fatty acids and cytokines causes beta-cell ER stres

32 Maternal HFD increased free fatty acids and decreased phospholipids (male > fem

33 howed slightly higher or similar hydrolysis (free fatty acids and diacylglycerols), similar primary (

34 gether with triacylglycerides, diglycerides, free fatty acids and ergosterol in salmon oil.

35 inflammation markers, as well as individual free fatty acids and free amino acids, were measured in

36 hondrial dysfunction; however, the impact of free fatty acids and glucose on mitochondrial traffickin

37 lipases are added to transform the fat into free fatty acids and glycerol, which elute at the end of

38 that hydrolyze triacylglycerols (TAGs) into free fatty acids and glycerol.

39 idylcholine (PC) results in the formation of free fatty acids and glycerophosphocholine (GPC) in the

40 ver disease (ALD) in alcoholics by releasing free fatty acids and inflammatory mediators.

41 levels of some lipid species but accumulate free fatty acids and lipid droplets.

42 to ambient UFP (diameter < 180 nm) increased free fatty acids and lipid metabolites in the mouse smal

43 Moreover, SAA sequestered free fatty acids and lysophospholipids to form stable pr

44 ugh generation of small neoantigens, such as free fatty acids and lysophospholipids, from common phos

45 rolyzes mammalian cell membranes, liberating free fatty acids and lysophospholipids.

46 Conversely, free fatty acids and monoacylglycerides significantly in

47 ols in rapeseed oil by the esterification of free fatty acids and monoacylglycerols.

48 easant taste and odor) due to the binding of free fatty acids and peroxide compounds.

49 Free fatty acids and peroxide value, as indicators of en

50 An adsorption mechanism of free fatty acids and peroxides is proposed.

51 ilic molecules are observed in obesity (e.g. free fatty acids and phosphatidic acid) and plastics exp

52 els of cytotoxic lipid intermediates such as free fatty acids and phosphatidic acid, suggesting a buf

53 However, treatment did reduce total free fatty acids and phospholipids and was dose dependen

54 ase and showed the highest concentrations of free fatty acids and reactive carbonyl compounds in the

55 mediates stemming from exposure to saturated free fatty acids and rescues hepatocytes from death.

56 The AAC-mediated H(+) current requires free fatty acids and resembles the H(+) leak via the the

57 ntermediate or high vapor pressures, such as free fatty acids and semi-volatile organic compounds (SV

58 Activation of endogenous free fatty acids and the subsequent reduction of fatty a

59 s was observed by analysing amount of formed free fatty acids and their antioxidant capacities.

60 actions: polar lipids (PL), diacylglycerols, free fatty acids and triacylglycerols (TAG) using thin l

61 oups and decreased the circulating levels of free fatty acids and triglycerides in the HFD-FBX4w anim

62 sphingolipids, amino acids and derivatives, free fatty acids, and bile acid.

63 Cardiolipin, free fatty acids, and branched lipids can access the poc

64 , pyrimidines, phospholipids, sphingolipids, free fatty acids, and glycerolipids) which were mapped t

65 ized triglyceride monomers, diglycerides and free fatty acids, and induction period based on Rancimat

66 Elevated levels of glucose, free fatty acids, and inflammatory cytokines due to diab

67 Lipid, leptin, free fatty acids, and inflammatory marker levels were al

68 lesterol, cholesterol esters, triglycerides, free fatty acids, and phosphatidylcholine, which is bloc

69 hepatocyte death upon exposure to saturated free fatty acids, and secretion of both IL-8 and TNFalph

70 increase in fasting plasma LDL cholesterol, free fatty acids, and total ketone bodies by 25, 49, and

71 d acid lipase expression and accumulation of free fatty acids are also present in a Pex19-deficient p

72 Obesity and elevation of circulating free fatty acids are associated with an accumulation and

73 the fact that both diacylglycerol (DAG) and free fatty acids are not interdependent after mild refin

74 f4)-induced lipotoxicity and accumulation of free fatty acids as the cause for mitochondrial damage i

75 of different lipids has been identified and free fatty acids as well as free cholesterol have been i

76 steatosis (i.e., high glucose, insulin, and free fatty acids), as well as drug-induced steatosis (i.

77 nalyses indicate that TAG lipolysis releases free fatty acids at a time that correlates well with mei

78 d is stimulated by exposure to nonesterified free fatty acids at concentrations observed in obese sub

79 nificantly different between groups included free fatty acids, bile acids, and amino acid metabolites

80 GPR84 is a medium chain free fatty acid-binding G-protein-coupled receptor assoc

81 -L-methionine (SAM) dependent methylation of free fatty acids, but FAME production by this route has

82 e tissue insulin sensitivity (suppression of free fatty acids by insulin) showed a continuous worseni

83 the organoids incubated with versus without free fatty acids by live imaging.

84 , there were marked elevations in myocardial free fatty acids, ceramides and diacylglycerols, consist

85 f female mice developed marked elevations in free fatty acids, ceramides and diacylglycerols.

86 Free fatty acids, ceramides, and triacylglycerol classes

87 To evaluate the impact of free fatty acids compared to hyperglycemia on mitochondr

88 with remarkably less oxidation products and free fatty acids compared with acid pH-shift process or

89 ctor, and the ability of the lipase to alter free fatty acid composition and sensory characteristics

90 Maternal EGP per unit insulin and plasma free fatty acid concentration during hyperinsulinemia mo

91 dams also had lower serum glucose and higher free fatty acid concentrations than controls on GD 21.

92 content of the seed reduced by 22% while its free fatty acid content increased by 4.3 folds after 10

93 The increases in phospholipid and free fatty acid content were observed with hydrolysis ti

94 Sensitivity analysis showed that free fatty acid content, peroxide value, L( *)Cab( *)hab

95 m ions in the intestinal fluid decreased the free fatty acids content and decreased the bioaccessibil

96 The free fatty acids content of sesame oil after processing

97 es in NAD(+), arginine, saturated long chain free fatty acids, diacylglycerides, triacylglycerides, a

98 Unexpectedly, high glucose and free fatty acids did not alter cellular ZnT8 levels, but

99 The use of molar excesses of the free fatty acids did not improve direct esterification r

100 Preventing the reduction of free fatty acids during the clamp with Intralipid infusi

101 to their coenzyme A derivatives, accumulates free fatty acids during the stationary phase of growth.

102 Acute free fatty acid elevation therefore induces a redistribu

103 Incubation of organoids with free fatty acid-enriched media resulted in structural ch

104 l that the coexistence of hypoxia along with free fatty acids exacerbates macrophage-mediated inflamm

105 h and inflammatory responses after saturated free fatty acid exposure by activating NF-kappaB through

106 esterification of vanillyl alcohol (VA) with free fatty acids (FA) and coconut oil (CO) as acyl donor

107 Free fatty acids (FA) are a vital component of cells and

108 bic glycolysis while reducing utilization of free fatty acid (FFA) and branched-chain amino acid (BCA

109 (TG) levels and HOMA-IR and positively with free fatty acid (FFA) and HDL after control for age and

110 release, triglyceride and glycogen contents, free fatty acid (FFA) content and release, and cholester

111 fatty acid and triacylglycerol composition, free fatty acid (FFA) content, peroxide index, thermal p

112 cycle were analyzed for 3-MCPD esters, GEs, free fatty acid (FFA) contents, specific extinction at 2

113 nistration significantly reduced insulin and free fatty acid (FFA) levels (P < 0.001) and ameliorated

114 increased adipose lipolysis, elevated plasma free fatty acid (FFA) levels, and impaired insulin signa

115 of CD36/fatty acid translocase and elevated free fatty acid (FFA) levels.

116 irectly or indirectly, determine the rate of free fatty acid (FFA) oxidation.

117 cell function and dysfunction in a model of free fatty acid (FFA) palmitate-induced oxidative stress

118 of biologic responses through members of the free fatty acid (FFA) receptor family, which includes FF

119 Leptin receptor and free fatty acid (FFA) receptor, GPR120, are upregulated

120 , finding several triacylglyceride (TAG) and free fatty acid (FFA) species to be significantly increa

121 Here, we present a novel near-infrared (NIR) free fatty acid (FFA) tracer suitable for in vivo imagin

122 Alterations in hepatic free fatty acid (FFA) uptake and metabolism contribute t

123 p4 and Fabp5 (Fabp4/Fabp5) impairs exogenous free fatty acid (FFA) uptake by CD8(+) TRM cells and gre

124 When applied to free fatty acid (FFA) uptake in 3T3-L1 adipocytes, this

125 at this defect would be exacerbated by acute free fatty acid (FFA)-induced insulin resistance.

126 hox activation of calpain-1 degrades Erk5 in free fatty acid (FFA)-stressed cardiomyocytes, whereas t

127 sis of ceramide to produce sphingosine and a free fatty acid (FFA).

128 ated lipid levels in the form of circulating free fatty acids (FFA) also have toxic effects, and that

129 ocytes, spleen and thymus weights, increased free fatty acids (FFA) and produced hyperglycemia and gl

130 centration of triacylglycerols, DAG, MAG and free fatty acids (FFA) and the concentration of saturate

131 s, combined with in vitro assays, identified free fatty acids (FFA) as circulating plasma factors tha

132 pression of SRA inhibits ATGL expression and free fatty acids (FFA) beta-oxidation.

133 ucted: (i) fish oil extraction, (ii) Omega-3 free fatty acids (FFA) concentration (low temperature wi

134 Free fatty acids (FFA) content of beer affects the abili

135 pothesis that an inherent increase in plasma free fatty acids (FFA) in the HFD together with an HFD-i

136 n insulin-mediated suppression of lipolysis, free fatty acids (FFA), and endogenous glucose productio

137 culating and tissue levels of triglycerides, free fatty acids (FFA), and leptin?

138 te degradation of lipids in the fillets were free fatty acids (FFA), lipid hydroperoxides (PV) and th

139 d, largely due to a reduction in circulating free fatty acids (FFA).

140 uated Adipo-IR (fasting and mean OGTT plasma free fatty acid [FFA] x insulin concentrations), periphe

141 y via circulating metabolic mediators (e.g., free fatty acids, FFA) associated with excess adiposity

142 epatic stellate (HSCs) cells were exposed to free fatty acids (FFAs) alone or in combination with OCA

143 mice showed a significant increase in serum free fatty acids (FFAs) and decrease in subcutaneous/per

144 crophages results in increased intracellular free fatty acids (FFAs) and elevated expression of uncou

145 with a higher abundance of saturated C16-C20 free fatty acids (FFAs) and long polyunsaturated complex

146 Free fatty acids (FFAs) are known to induce lipoapoptosi

147 In cultured podocytes, the presence of free fatty acids (FFAs) associated with serum albumin st

148 Nutrients such as free fatty acids (FFAs) contribute to precise regulation

149 nic acids (OAs), free amino acids (FAAs) and free fatty acids (FFAs) during ripening of raw sheep's m

150 ch in n-3 were produced by extraction of the free fatty acids (FFAs) from flaxseed oil, concentration

151 rprinting, although its power in analysis of free fatty acids (FFAs) is limited.

152 Faba bean LOX preferred free fatty acids (FFAs) over triacylglycerols as substra

153 Serum free fatty acids (FFAs) profile is highlighted in its as

154 We investigated the mechanism by which free fatty acids (FFAs) regulate MIR122 expression and t

155 Empagliflozin did not affect myocardial free fatty acids (FFAs) uptake but reduced myocardial gl

156 Circulating free fatty acids (FFAs) were less suppressed in IR than

157 We find that acyl-CoAs, rather than free fatty acids (FFAs), are the preferred substrate for

158 flecting chylomicrons of intestinal origin), free fatty acids (FFAs), insulin, glucose, glucagon, glu

159 aste frying oils (WFOs) with high content of free fatty acids (FFAs), otherwise unsuitable for biodie

160 The hydrolytic cleavage of TGs generates free fatty acids (FFAs), which can serve as energy subst

161 s is responsible for the slow suppression of free fatty acids (FFAs), which in turn is responsible fo

162 This led to elevated plasma free fatty acids (FFAs), which were transported to the a

163 nied by dyslipidemia with elevated levels of free fatty acids (FFAs).

164 gnize and are activated by nonesterified or "free" fatty acids (FFAs).

165 The chemical components (hydrocarbons - HCs, free fatty acids - FFAs, free fatty alcohols - FALs and

166 beta-carotene) and lipid digestion products (free fatty acids, FFAs, and monoacylglycerides, MAGs) du

167 dance of biomarkers such as HDL cholesterol, free fatty acids, FGF21, bilirubin, and lactate depend o

168 extracts, predominantly presented lipids as free fatty acids, followed by aminoacids, organic acids,

169 at autophagy is necessary for the release of free fatty acids from intracellular stores within neutro

170 ids and phospholipids can promote release of free fatty acids from lipid stores, detoxify antimicrobi

171 (2)H5]glycerol turnover rate and circulating free fatty acids, glycerol, and triglycerides), lipid ox

172 (TAG) and glucose uptake decreased, and the free fatty acid/glycerol ratio increased during the anta

173 Organoids incubated with free fatty acids had gene expression signatures similar

174 Hexadecanoic acid was the most abundant free fatty acid in the cheeses, which followed by cis-9-

175 nal processes also enabling the reduction of free fatty acids in crude oils.

176 y weight, and transiently alters circulating free fatty acids in lean mildly hypoleptinemic individua

177 ght; increased concentrations of glucose and free fatty acids in plasma; enhanced lipid accumulation

178 ell as SMc01003 contribute to the release of free fatty acids in S. meliloti, neither one can use pho

179 The defect in the mobilization of free fatty acids in the elderly is accompanied by increa

180 ted to the higher amount of monounstaturated free fatty acids in the micelle fraction.

181 ough transport phenomena not yet understood, free fatty acids in the oil binding medium migrate throu

182 Neutralization of free fatty acids in the vegetable oil before the deodori

183 yao, has been shown to bind leukotrienes and free fatty acids in vitro Therefore, here we assessed wh

184 phospholipase A(2) (sPLA(2)) enzymes release free fatty acids, including arachidonic acid, and genera

185 It is established that long-chain free fatty acids includingomega-3 fatty acids mediate an

186 Stimulation with albumin and free fatty acid increased mitochondrial reactive oxygen

187 oup, the EPA and DHA contributions to plasma free fatty acids increased (P = 0.0003 and P = 0.003, re

188 Additionally, BBR decreased the free fatty acid-induced lipid accumulation and tunicamyc

189 wed that FXR activation protected cells from free fatty acid-induced oxidative stress and endoplasmic

190 adipose tissue area (cm(2)), lipids, leptin, free fatty acids, inflammatory markers, and activity rat

191 to partial incorporation of diglycerides and free fatty acids into gum bilayers after PLC and 3G ED.

192 a radical increase in the esterification of free fatty acids into triacylglycerol.

193 encompass increased 1) circulating levels of free fatty acid, ketone bodies, and long-chain acylcarni

194 se increased responsiveness of host cells to free fatty acid, leading to a radical increase in the es

195 creatic beta cells to high concentrations of free fatty acids leads to lipotoxicity (LT)-mediated sup

196 ns, metabolic function, and triglyceride and free fatty acid levels and reverses weight gain in T2DM.

197 olytic effect of insulin, and reduces plasma free fatty acid levels during OGTT.

198 res during exercise and elevated circulating free fatty acid levels postexercise.

199 Consistently, free fatty acid levels were reduced in portal but not in

200 leaner with reduced plasma triglyceride and free fatty acid levels.

201 r binding domains tightly bind the essential free fatty acid linoleic acid (LA) in three composite bi

202 mplex lipids with relatively small amount of free fatty acids (<15%).

203 six triacylglycerides (m/z 726-860) and six free fatty acids (m/z 271-345).

204 hanced lipolysis, elevated concentrations of free fatty acids, maximal beta-oxidation, and mitochondr

205 eats bind directly to medium- and long-chain free fatty acids (MCFA and LCFA).

206 ssiveness were identified, including various free fatty acids, metabolites, and complex lipids such a

207 etabolism and contributes to fasting-induced free fatty acid mobilization.

208 to characterize the molecular speciation of free fatty acids, monoacylglycerol species, unmodified a

209 with the fastest release and lower amount of free fatty acids observed at 100 MPa.

210 eptors are those that respond to short-chain free fatty acids of carbon chain length 2-6.

211 ma BCAs in both insulin groups but increased free fatty acids only in the high insulin group, however

212 ression profiles of organoids incubated with free fatty acids or without.

213 nors, while acyl-ACP (acyl carrier protein), free fatty acids, or galactolipid-bound fatty acids were

214 patic de novo lipogenesis, decreased hepatic free fatty acid oxidation, or decreased very-low-density

215 In these subjects fasting levels of the free fatty acid palmitate are raised.

216 s in circulating levels of the most abundant free fatty acids (palmitic, linoleic and oleic acids).

217 The increase of free fatty acid, peroxide value, p-anisidine value, K(23

218 Free fatty acid, peroxide, p-anisidine, and total oxidat

219 The following analyses were carried out: free fatty acids, peroxide value, specific extinction at

220 rous cow milk fat was evaluated by measuring Free Fatty Acids, peroxide value, Thiobarbituric Acid va

221 ng lipids and signaling molecules, including free fatty acids, phospholipids, lyso-phospholipids, and

222 lyzes the hydrolysis of fatty acyl-CoAs into free fatty acids plus CoASH.

223 method was developed for elucidation of the free fatty acid profile in mussel samples, avoiding a pr

224 eted metabolomic measurement of serum BA and free fatty acid profiles was applied to sera of 381 indi

225 a, provided excellent predictive results for free fatty acid (R(2) = 0.97) and peroxide values (R(2)

226 Indeed, whereas the plasma free fatty acid Ra and Rd were twice those of YL in both

227 the absence of biliary CA leads to increased free fatty acids reaching the ileal L cells.

228 colon, the involvement of the SCFA receptor free fatty acid receptor (FFA)3, one of the free fatty a

229 The contribution of an SCFA receptor, free fatty acid receptor (FFA)3, to the enteric nervous

230 Free fatty acid receptor (FFAR) 1 (FFA1), which binds lo

231 The free fatty acid receptor 1 (FFA1 or GPR40) is establishe

232 The free fatty acid receptor 1 (FFA1/GPR40) is a potential t

233 in-coupled receptor 40 (GPR40) also known as free fatty acid receptor 1 (FFAR1) is highly expressed i

234 Moreover, we identify a lipid sensor, free fatty acid receptor 1 (Ffar1), that curbs glucose u

235 lin secretion from isolated human islets via free fatty acid receptor 1 (FFAR1/GPR40).

236 second long-chain free fatty acid receptor, free fatty acid receptor 1, the simple chemical structur

237 ot the other candidate transporters CD36 and free fatty acid receptor 1.

238 Free Fatty Acid Receptor 2 (FFA2) is a beta cell-express

239 Free fatty acid receptor 2 (FFA2) is expressed on entero

240 The free fatty acid receptor 2 (FFA2/GPR43) is considered a

241 We investigated the mechanisms by which free fatty acid receptor 2 (FFAR2), a receptor for short

242 phils and ILC3s through its cognate receptor free fatty acid receptor 2 (FFAR2).

243 idue produced a signalling-biased variant of Free Fatty Acid Receptor 2 in which Gi-mediated signalli

244 Free Fatty Acid Receptor 2 is a GPCR activated by short

245 s to homology models of both human and mouse Free Fatty Acid Receptor 2 suggested that a single lysin

246 d SCFAs, propionate, activates ileal mucosal free fatty acid receptor 2 to trigger a negative feedbac

247 Free fatty acid receptor 3 (FFA3, previously GPR41) is a

248 ssion of the short-chain fatty acid receptor free fatty acid receptor 3 and a population of regulator

249 The free fatty acid receptor 4 (FFA4 or GPR120) has appeared

250 there is great interest in the potential of free fatty acid receptor 4 (FFA4) as a novel therapeutic

251 Next, we hypothesize that free fatty acid receptor 4 (Ffar4), a functional recepto

252 free fatty acid receptor (FFA)3, one of the free fatty acid receptor family members, has not been cl

253 upled receptor 40 (GPR40), a Galphaq-coupled free fatty acid receptor linked to MAPK networks and glu

254 r lipid-based antigens presentation, and the free fatty acid receptor, CD36.

255 614 did not antagonize the second long-chain free fatty acid receptor, free fatty acid receptor 1, th

256 Free-fatty acid receptor-4 (FFA4) is a cell-surface G pr

257 Free-fatty acid receptor-4 (FFA4), previously termed GPR

258 Free fatty acid receptors (FFARs) for long-chain fatty a

259 Free fatty acid receptors 1 and 4 (FFAR1 and FFAR4) are

260 -derived short-chain fatty acids (SCFAs) and free fatty acid receptors including GPR43 are thought to

261 receptors 1 and 4 (FFAR1 and FFAR4) are two free fatty acid receptors under increasing investigation

262 ycerol, glycerol, and medium- and long-chain free fatty acids, reflective of lipid mobilization and c

263 Free fatty acids regulate numerous cellular processes, l

264 rom SGA newborns, whereas GPR120 (related to free fatty acid regulation) was hypomethylated in placen

265 The rate of free fatty acid release during lipid digestion decreased

266 ype cytokine signaling in adipocytes induces free fatty acid release from visceral adipocytes, thereb

267 Free fatty acid release profiles suggested that the stab

268 Free fatty acids released from the plastid need to be co

269 In the wild type, in which free fatty acids remained low and unchanged under dark t

270 The incorporation of excess saturated free fatty acids (SFAs) into membrane phospholipids with

271 Furthermore, the peroxide value and free fatty acids showed that the quality of the food pro

272 emulsion (MPE) droplets comprising particle-free fatty acid-stabilized apertures.

273 ary, we have described a novel nonesterified free fatty acid-stimulated pathway that selectively driv

274 FLD, causes more oxidative damage than other free fatty acids such as palmitic acid, and mediates sel

275 weight, serum cholesterol, triglycerides and free fatty acids, suggesting altered lipid metabolism in

276 lights the critical role of plasma saturated free fatty acids that are abundant in the WD with respec

277 ulated metabolic process called lipolysis to free fatty acids that serve as energy substrates for bet

278 ic islets that is activated by nonesterified free fatty acids, the major fuel used by beta-cells duri

279 In the presence of free fatty acids, the transfer of prooxidants between dr

280 se (FAAH) degrades NAE into ethanolamine and free fatty acid to terminate its signaling function.

281 ing a hybrid fatty acid synthase shifted the free fatty acids to a medium chain-length scale.

282 wn adipose tissue (BAT) utilizes glucose and free fatty acids to produce heat, thereby increasing ene

283 ated process that maintains plasma levels of free fatty acids to supply energy to cells.

284 w that autophagy-mediated lipolysis provides free fatty acids to support a mitochondrial respiration

285 A2 (PLA2) to mobilize lysophospholipids and free fatty acids to sustain fatty acid oxidation and oxi

286 of Sinorhizobium meliloti, unable to convert free fatty acids to their coenzyme A derivatives, accumu

287 Free fatty acids translocate through the interior over h

288 Unlike albumin, SAA effectively removed free fatty acids under acidic conditions, which characte

289 triglyceride accumulation pathways including free fatty acid uptake and de novo lipogenesis.

290 hat cathepsin B regulates VLDL secretion and free fatty acid uptake via cleavage of LFABP, which occu

291 sulted in successful quantification of their free fatty acid value, total phenol and major fatty acid

292 nhanced fat oxidation, circulating levels of free fatty acids were reduced in the HFD-fed AdSod2 KO m

293 ties responsible for the generation of these free fatty acids were unknown in rhizobia.

294 ow volatility, such as medium and long-chain free fatty acids, whereas compounds with high volatility

295 ative ion mode, MALDI favored sulfatides and free fatty acids, whereas NAPA spectra were dominated by

296 lysis take place leading to the formation of free fatty acids, which in turn interact with the differ

297 The effect of free fatty acids with different chain lengths or unsatur

298 C2C12 myotubes exposed to excess free fatty acids with or without UT were also evaluated

299 ow hypoxia modulates the response of ATMs to free fatty acids within obese adipose tissue is limited.

300 lasma insulin [FPI]), and adipocyte (fasting free fatty acid x FPI).