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1 ng the development of various regions of the frontal bone.
2 ly larger defect in the neural crest-derived frontal bone.
4 cell mixing between the neural crest-derived frontal bone and mesoderm-derived parietal bone accompan
5 higher maximum principal strains across the frontal bone and mid-face, a strain regime not observed
6 Cranial (mandible, maxilla, parietal, and frontal) bones and ulnae were analyzed for mineralizatio
8 axilla, the anterior-posterior length of the frontal bone, and the main body of the mandible, did not
10 ed at a neural crest-mesoderm interface: the frontal bones are neural crest-derived and the parietal
11 resulted in an increased bone volume in the frontal bone compared to the other groups at 4 weeks pos
14 mediated FGF signaling cascade in regulating frontal bone development, suggests that TGFbeta function
15 deletion of Fn1 or Wasl leads to diminished frontal bone expansion by altering cell shape and focal
16 ular Trauma Score, initial visual acuity and frontal bone fractures were predictive of NLP (P = .006
23 The frontal sinuses are cavities inside the frontal bone located at the junction between the face an
30 Both the quantity and proliferation of the frontal bone skeletogenic mesenchyme are reduced in Msx2
32 he FTD, measured from the inner table of the frontal bone to the posterior margin of the thalamus, wa
35 aniofacial bones were not much affected, the frontal bone was larger in width and volume, and the max
36 ed to the developing calvarial aspect of the frontal bone, whereas the orbital aspect forms despite t
37 2 signaling in osteogenic front cells of the frontal bones, which regulate WNT signaling in neighbori