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1  sample, where lamin B1 was increased in the frontal lobe.
2 es, temporal lobe, parietal lobe, and medial frontal lobe.
3 umor type arising with high frequency in the frontal lobe.
4  (k(f)) of the creatine kinase enzyme in the frontal lobe.
5 gradients, with particular enrichment in the frontal lobe.
6 of behavior mediated in part by the maturing frontal lobe.
7 rmation is conveyed from the parietal to the frontal lobe.
8 plexity specific to the human lineage in the frontal lobe.
9 ow grade gliomas (LGG) localized in the left frontal lobe.
10 rietal and occipital regions compared to the frontal lobe.
11  the temporal lobes, right basal ganglia and frontal lobe.
12 rguing for an initiation of selection in the frontal lobe.
13 , and culminating as causal inference in the frontal lobe.
14 twork that includes all 7 motor areas in the frontal lobe.
15 s that from any of the premotor areas in the frontal lobe.
16 s have implicated abnormal activities in the frontal lobe.
17 he corpus callosum connecting left and right frontal lobes.
18 redict the level of functional access to the frontal lobes.
19 atients showed increased connectivity in the frontal lobes.
20 he subarachnoid space, particularly over the frontal lobes.
21 that it involved relative enlargement of the frontal lobes.
22 regions predominantly influenced activity in frontal lobes.
23 ignificant hypometabolism in the temporal or frontal lobes.
24 d towards subtle abnormalities in the medial frontal lobes.
25 % of survivors, occurring more frequently in frontal lobes.
26 stro-caudal hierarchical organization of the frontal lobes.
27 the greatest increases in lateral and medial frontal lobes.
28 ng action selection signals generated in the frontal lobes.
29 associated with abnormal connectivity of the frontal lobes.
30 e encephalopathy predominantly affecting the frontal lobes, 1 hemorrhagic shock and encephalopathy, 1
31 elirium was associated with smaller superior frontal lobe (-2.11 cm(3) [-3.89, -0.32]; p = .03) and h
32 er, other atypical regions involved were the frontal lobes (30.4%), temporal lobes (8.69%), basal gan
33 8.89 [1.1] cm3; cortical lesion count in the frontal lobe, 5.0 [5.7]; r = -0.60; P < .01).
34 hite matter water diffusion metrics from the frontal lobe accounted for poor performance on both memo
35 esponse characterized by inferior and middle frontal lobe activation (right>left) which bears some si
36 0% and 13.1% of the total effect of inferior frontal lobe activation on angina severity, respectively
37                                     Inferior frontal lobe activation with mental stress is independen
38           For every doubling in the inferior frontal lobe activation, angina frequency was increased
39 animal's decision was reliably maintained in frontal lobe activity through a selective state sequence
40 unilateral and unilobar focal lesions in the frontal lobe affecting the cortical grey matter or the c
41 localised disease, frequently located in the frontal lobes, affecting the cortical grey matter and ad
42                   Damage to the ventromedial frontal lobe also impaired learning about the relevant d
43               Decision-making ability in the frontal lobe (among other brain structures) relies on th
44 th lesions to dissociated regions within the frontal lobe, amongst other regions.
45 olumetric/area measurements obtained for the frontal lobe, amygdala, and hippocampus and subregions o
46 ociated with structural abnormalities of the frontal lobe, amygdala, and insula.
47  genes differentially expressed within human frontal lobe and a striking increase in transcriptional
48 abnormalities extended rostrally towards the frontal lobe and dorsocaudally towards the superior temp
49 sconsin Card Sorting Test, a classic test of frontal lobe and executive function in humans, to assess
50 rformance on neuropsychological tests of the frontal lobe and executive functioning, the Trail Making
51 pect of the fissure moving anteriorly to the frontal lobe and laterally in the direction of the tempo
52 ludes all of the cortical motor areas in the frontal lobe and portions of somatosensory cortex.
53  of 10 regions-of-interest including insula, frontal lobe and putamen in AD compared with controls, b
54 ion for imagined words in the temporal lobe, frontal lobe and sensorimotor cortex, consistent with pr
55  Network disorganization primarily involving frontal lobe and subcortical regions in nonpsychotic rel
56  the patient group, most pronouncedly in the frontal lobe and temporal cortex.
57 injection or needle insertion into the right frontal lobe and were euthanized at different times to e
58               In humans, Broca's area in the frontal lobe and Wernicke's area in the temporal lobe ar
59 th premotor (PM) and motor (M1) areas of the frontal lobe and with other areas of parietal and occipi
60 volume difference of approximately 8% in the frontal lobes and 12% in the occipital lobes-these regio
61 erns of coherence are identified between the frontal lobes and between frontal and distant brain regi
62  puncture after enucleation and biopsied the frontal lobes and optic nerves of a macaque experimental
63 t there are no inhibitory modules within the frontal lobes and that behavioural inhibition is an emer
64 rtical structures including bilateral medial frontal lobes and the right inferior frontal and tempora
65  more likely in subnetworks that include the frontal lobe, and can be opposite in nature depending on
66 in the olfactory epithelium, olfactory bulb, frontal lobe, and lung tissues in cadavers from the city
67 er WMH load, specifically in the subcortical frontal lobe, and periventricular frontal and parietal c
68 articularly those located in the subcortical frontal lobe, and the periventricular frontal and pariet
69 netic methods reveals that the size of human frontal lobes, and of specific frontal regions, is as ex
70                  Since larger ECVs, expanded frontal lobes, and reduced olfactory bulbs are already p
71  callosum, occipital, temporal, parietal and frontal lobes, and right hippocampus (p < 0.025 after fa
72             We found that stimulation of the frontal lobes [anode electrode at the left dorsolateral
73 reases of grey matter were identified in the frontal lobes, anterior cingulate gyrus, left temporal l
74 and cerebral microdialysis monitoring (right frontal lobe, apparently normal tissue) combined with ce
75  and that event-related potentials above the frontal lobe are affected by both word reversal and the
76  glutamatergic and GABAergic deficits in the frontal lobe are potential targets for symptomatic drug
77 r cingulate cortex in the medial wall of the frontal lobe are the source of this increased theta powe
78                                          The frontal lobes are necessary for cognitive control at all
79  data have motivated the hypothesis that the frontal lobes are organized hierarchically, such that co
80 butyric acid (GABA), particularly within the frontal lobes, are some of the most reliable alterations
81 al white matter fiber tracts associated with frontal lobe areas critical in cognitive processing has
82  rather significantly in the hippocampus and frontal lobe as a function of learning and memory.
83 as correlated with cortical thickness in the frontal lobe as a whole.
84  one subtype showing grey matter loss in the frontal lobes as well as loss of the temporal lobes (fro
85 cluding the cingulate cortex, operculum, and frontal lobe, as well as in the temporal lobe.
86              It is typically associated with frontal lobe atrophy, although patterns of atrophy are h
87 superior temporal gyrus (BA21), dorsolateral frontal lobe (BA9), and dorsal parietal lobe (BA7) of ea
88 e anatomical and functional abnormalities in frontal lobe, basal ganglia and the limbic system.
89  or CTx-), with prominent alterations in the frontal lobes bilaterally.
90 obacterium necrophorum who developed a right frontal lobe brain abscess.
91 ignificant white matter tract changes in the frontal lobe, brainstem and hippocampal regions of the A
92 in four major brain structures (hippocampus, frontal lobe, brainstem, and cerebellum) using a techniq
93 uman primates indicate that the ventromedial frontal lobe carries information about both losses and g
94 orrelated with volumetric differences in the frontal lobes, caudate nucleus and cerebellum, and hyper
95 ical ROI and links were observed in superior frontal lobe, cingulate cortex, fusiform, putamen, and m
96 aboratory, there is also evidence that other frontal lobe circuits have evolved to make other types o
97 hip between striatal dopamine processing and frontal lobe cognitive function.
98                                Impairment of frontal lobe cognitive functions has been reported in pa
99 loid burden was significantly reduced in the frontal lobe compared to the placebo group.
100 ows that humans have a greater proportion of frontal lobe connections compared with monkeys, when nor
101  the disproportionally large volume of human frontal lobe connections is accompanied by a reduction i
102  and control groups, whilst the structure of frontal lobe connections showed relationships with execu
103                       However, uptake in the frontal lobe continued to increase with age but it decre
104                                  We explored frontal lobe contributions to cue-induced drug craving u
105           These results provide insight into frontal lobe contributions to procedural knowledge.
106                                          The frontal lobes control wide-ranging cognitive functions;
107 y to studies of HCS and the pH values in the frontal lobe correlated negatively with the YMRS values.
108 ded neurons from different somatosensory and frontal lobe cortices of monkeys performing a vibrotacti
109 y linked by a cascade of transient events in frontal lobe cortices.
110 ling Test clinical measures are sensitive to frontal lobe damage although only the suppression error
111 ds (impulsivity) characterizes subjects with frontal lobe damage and behavioral disorders including s
112                                Patients with frontal lobe damage are impaired on response sequencing
113               Participants with left lateral frontal lobe damage attributed rewards to irrelevant dim
114                          Patients with focal frontal lobe damage completed a demanding reward learnin
115 and education (N = 24) and with a group with frontal lobe damage sparing the VMF (N = 12).
116 est but it has been primarily used to detect frontal lobe damage.
117 e diseases such as Parkinson's disease (PD), frontal lobe dementia (FLD) and diffuse Lewy-body dement
118 esearch has demonstrated that performance on frontal lobe-dependent tasks is associated with dopamine
119 ive effects of socioeconomic disadvantage on frontal lobe development (with implications for function
120 ject via the occipitofrontal fascicle to the frontal lobe (dorsal area 6, and areas 8Ad, 8B, and 46);
121 s that migrate and integrate widely into the frontal lobe during infancy.
122 sistently, anatomical studies have indicated frontal lobe dysfunction in the disease.
123 is slow with frequent protracted symptoms of frontal lobe dysfunction.
124               Possible complications include frontal lobe edema and even the rare formation of delaye
125 n implicated in autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE) and epilepsy of infancy w
126                 Autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE) is linked with high penet
127 novel brain pathology in young patients with frontal lobe epilepsy (FLE) that is distinct from focal
128  reported in patients with schizophrenia and frontal lobe epilepsy and may contribute to their associ
129                 Autosomal dominant nocturnal frontal lobe epilepsy can be caused by the alpha4 mutati
130 associated with autosomal dominant nocturnal frontal lobe epilepsy have been found to show spontaneou
131  of seizures in autosomal dominant nocturnal frontal lobe epilepsy mice remain unknown.
132 nrapid eye movement parasomnia and nocturnal frontal lobe epilepsy remain.
133 (eg, epilepsia partialis continua, nocturnal frontal lobe epilepsy).
134 ions in nAChRs subunits have been related to frontal lobe epilepsy, neurodegenerative diseases, and o
135 e been noted in autosomal dominant nocturnal frontal lobe epilepsy.
136 ancer and directly inoculated into the mouse frontal lobe, exhibit striking differences in proliferat
137 enrichment of methylation QTLs (P<0.001) and frontal lobe expression quantitative trait loci (eQTLs)
138 mary sensorimotor cortices and with enriched frontal lobe expression.
139 l lobe, left posterior cingulate gyrus, left frontal lobe expressive language regions, and left basal
140  lateral, dorsal, and mesial surfaces of the frontal lobe, extending into primary sensory, superior p
141 fy differential diagnosis of temporal versus frontal lobe EZ localization.
142  and pointing HPs are strongly predictive of frontal lobe EZ; cup, politician's fist, and pincer are
143 omplex mixture extracted from a normal fetal frontal lobe (FL37).
144  lobes for the 100-130 ms timeframe, and the frontal lobe for the 130-160 ms timeframe.
145 ulate cortex and orbitofrontal cortex in the frontal lobe from a total of 90 elderly subjects (age ra
146 pe showing loss relatively restricted to the frontal lobes (frontal-dominant subtype).
147 s in 60 (45%) of 133 patients on the test of frontal lobe function (FAB), and in between 13 (10%) and
148 dence of a positive effect of simvastatin on frontal lobe function and a physical quality-of-life mea
149                   FA was not associated with frontal lobe function or visuospatial perception.
150 unger adults who performed worse on tests of frontal lobe function showed greater FMT signal in right
151 ze data from a classical task used to assess frontal lobe function, the Wisconsin Card Sorting Test.
152                         Critically, abnormal frontal-lobe function was correlated with the number of
153 ry modality and information domain influence frontal lobe functional organization, they also demonstr
154 ous research has associated abnormalities in frontal lobe functioning with alcohol relapse.
155 ntial underlying mechanism for impairment of frontal lobe functions in both patients and siblings, an
156 22q11.2DS subjects compared with controls in frontal lobe (g = -0.47; p < 0.001), temporal lobe (g =
157 an developmental in vivo evidence confirming frontal lobe GABA maturation, which was linked to impuls
158 ystem, also matures during adolescence, with frontal lobe GABA receptors reaching adult levels late i
159  diffusivity [RD, AxD, MD, respectively]) of frontal lobe, genu, and splenium of the corpus callosum
160                         Abnormalities within frontal lobe gray and white matter of bipolar disorder (
161 gray matter, total neocortical white matter, frontal lobe gray matter, frontal lobe white matter, and
162                                          The frontal lobes had high PiB retention with little grey ma
163 rain structures, such as the hippocampus and frontal lobe, has only been observed in humans.
164  how state desirability signals decoded from frontal lobe hemodynamics, as measured with near-infrare
165                                  Significant frontal lobe hyperactivation to support WM was found in
166 re parieto-temporal lobe, cerebellar cortex, frontal lobe, hypothalamus and striatum.
167               Although the role of the right frontal lobe in interference control is evident, the whi
168 ribed structural abnormalities in the medial frontal lobe in juvenile myoclonic epilepsy.
169  and enhanced connectivity within the dorsal frontal lobe in subjects with psychopathy.
170 of Interest) was defined in 7 locations: the frontal lobe in the right and left hemispheres, the basa
171                              The role of the frontal lobes in cognition and behavior has long been en
172 niqueness should therefore focus less on the frontal lobes in isolation and more on distributed neura
173 king circuits working in parallel within the frontal lobes in order to make different types of decisi
174 ion revealed a pathological mass in the left frontal lobe, in close topographical relationship to the
175 ciated with decreased GM density in the left frontal lobe, in the right frontoparietal cortex and in
176 ymmetric reduction in CTh/CSA in most of the frontal lobes, in addition to large temporoparietal area
177 isual and auditory attention networks in the frontal lobe, independent of sensory modality.
178 spontaneous long-term recovery from isolated frontal lobe injury and isolated frontoparietal injury.
179 the microcircuitry of agranular areas of the frontal lobe involved in cognitive control and responsib
180  studies using drug cue paradigms have shown frontal lobe involvement in this contextual influence on
181                                              Frontal lobe involvement is characteristic of CBD, and i
182 rcuits control executive function within the frontal lobe involving the dorsolateral prefrontal corte
183                                 The inferior frontal lobe is an important area of the brain involved
184                                          The frontal lobe is central to distinctive aspects of human
185                                    The human frontal lobe is important for social behavior and execut
186      A fundamental challenge in studying the frontal lobe is to parcellate this cortex into "natural"
187 bs of high connectivity (many located in the frontal lobe) is associated with amyloid accumulation.
188 CC), which lies on the medial surface of the frontal lobes, is important in regulating cognitive cont
189 disorder with dysfunction and atrophy of the frontal lobes leading to changes in personality, behavio
190 male Vietnam combat veterans consisting of a frontal lobe lesion group with focal penetrating head in
191 ity of lesion or correlation between lateral frontal lobe lesions and impairment on any task.
192 ation approaches developed for patients with frontal lobe lesions and pharmacotherapy approaches desi
193                                  Also, right frontal lobe lesions have been implicated, but typically
194                                       Larger frontal lobe lesions were associated with greater number
195 ue studies in an experiment with humans with frontal lobe lesions, asking whether behavioral impairme
196 ter numbers of terminal boutons than smaller frontal lobe lesions.
197  some but not all "executive" deficits after frontal lobe lesions.
198 cits similar to those found in patients with frontal lobe lesions.
199 hich suggests that structural changes in the frontal lobe may have an indirect influence on age-relat
200 l spatial codes are used in conjunction with frontal lobe mechanisms to plan routes during navigation
201  by considering the role of the anterior and frontal lobes, mesolimbic connections and the right hemi
202 t recent evidence suggests that parts of the frontal lobe might also play a key role.
203 subplate visualization [complete + partial]: frontal lobe, n = 243 vs n = 117; temporal lobe, n = 244
204 rily located in the posterior portion of the frontal lobe, near the controlling site, and non-linear
205 el tractography approach to demonstrate that frontal lobe networks, extending within and beyond the f
206 matter volume in nearly all networks, except frontal lobe networks, where differences in grey matter
207 otheses that (i) cerebellar efferents target frontal lobe neurons involved in forming action represen
208 g firing rates and correlated variability of frontal lobe neurons while monkeys perform a vibrotactil
209  the temporal lobe with predilection for the frontal lobes.New type III is one of the above dysplasia
210 brain has principal and arcuate sulci of the frontal lobe not seen in the stem catarrhine Aegyptopith
211 be networks, extending within and beyond the frontal lobes, occupy 66% of total brain white matter in
212  identical analyses of EEG recorded over the frontal lobe of macaque monkeys (one male, one female) p
213 is procedure to 17 injections, placed in the frontal lobe of nine marmosets as part of earlier studie
214 t parietal and occipital regions, the mesial frontal lobe of the right hemisphere, and the cuneus and
215 ng direct inoculation of M002 into the right frontal lobes of A. nancymae; (iii) there was no histopa
216 stone acetylation in the white matter of the frontal lobes of aged subjects and in patients with chro
217 cid (DHA) accumulates in the hippocampus and frontal lobes of the fetal brain during the last trimest
218  lower FA and significantly higher RD in the frontal lobes of the RHU group, relative to the TS group
219                                  The ventral frontal lobe, or inferior frontal gyrus, plays an import
220 of the basal ganglia, thalamus, hippocampus, frontal lobe, orbitofrontal cortex, and gyrus rectus.
221 han either olfactory epithelium (p = 0.071), frontal lobe (p < 0.001), or lungs (p = 0.037).
222 ly in the left internal capsule and the left frontal lobe (P = .02-.05).
223 magnetic resonance imaging with an executive frontal lobe paradigm, we investigated cortical activati
224 ory analysis moreover showed that the mesial frontal lobes, parahippocampal gyrus, and lateral tempor
225 ial diffusivity, and radial diffusivity from frontal lobes, parietal lobes, and in the frontostriatal
226 rom 12 patients with temporal, occipital, or frontal lobe partial onset seizures.
227 ing associated cognitive abilities, in other frontal lobe pathologies; however, in clinical trials, M
228 actional anisotropy in regions that included frontal lobe pathways.
229 rnally using brain stimulation, and that the frontal lobes play a causal role in mind-wandering behav
230 ng in 19% of the whole cortex and 24% of the frontal lobes, primarily in the right superior, medial o
231 neurons in several areas of the human medial frontal lobe prior to voluntary movement.
232  in spatial working memory while the ventral frontal lobe processes language and communication signal
233 nsisted of significantly increased CT within frontal lobe regions and reduced SA in the orbitofrontal
234 ormation about the complex interplay between frontal lobe regions in mediating these processes and ac
235 antly smaller gray matter volume was seen in frontal lobe regions in patients homozygous for the A al
236  focus on recent evidence that suggests four frontal lobe regions make distinct contributions to rewa
237  phase coding in four temporal lobe and four frontal lobe regions of the human brain using local fiel
238 receptive aphasia; (iii) widespread temporal/frontal lobe regions of the left hemisphere and expressi
239                              We propose that frontal lobe regions that contribute to complex decision
240       These functional changes correspond to frontal lobe regions where we previously reported struct
241  brain regional analysis demonstrated only 3 frontal lobe regions with an SUV estimation bias of 5% o
242 d morphological relationships primarily with frontal lobe regions, and their network-level alteration
243  activity was augmented in the left temporal/frontal lobe regions, as well as left inferior-parietal
244 g the middle and anterior temporal lobe with frontal lobe regions, including the pars triangularis.
245                      Significance statement: Frontal lobe-related function is vital for a variety of
246 s under dynamic conditions, with the lateral frontal lobe required for selecting the relevant dimensi
247  associate with reward, and the ventromedial frontal lobe required to learn the reward association it
248 ting PLA2/5-LOX/LTB4 cascade using a partial frontal lobe resection SBI rat model.
249             A biopsy specimen from the right frontal lobe revealed demyelination and perivascular lym
250 lated with oral naming speed in both lateral frontal lobes (rho = 0.48 and 0.47 for right and left fr
251 yzed ictal semiology of 489 temporal lobe or frontal lobe seizures recorded over a 6-year period at t
252 y- and deoxyhemoglobin concentrations in the frontal lobe show differences in response to both primar
253 s were recorded from temporal, parietal, and frontal lobe sites in 12 pediatric patients undergoing e
254  processes mediated by anatomically distinct frontal lobe subregions are both critical for adaptive c
255  we tested the critical involvement of human frontal lobe subregions in a probabilistic, multidimensi
256 , we examined the necessary contributions of frontal lobe subregions in attributing feedback to relev
257             Findings were principally in the frontal lobe, superior parietal lobes, and in the parame
258  results revealed regions of occipital lobe, frontal lobe, supplementary motor area, cingulate cortex
259 e, including gray matter of the total brain, frontal lobe, temporal lobe, and hippocampus.
260                               Regions in the frontal lobe, temporal lobe, and parietal lobe exhibited
261 and may occur in other locations such as the frontal lobes, thalami, basal ganglia and brainstem.
262                             Smaller superior frontal lobes, thalamus, and cerebellar volumes at 3 mon
263 tex or less cortical folding in temporal and frontal lobes than control subjects.
264  theta band, involving mainly regions in the frontal lobe that were sending information to a more dis
265 gions, there is extensive involvement of the frontal lobes that has been missed by past ROI analyses.
266 e occurrence of 5hmC in DNA from human brain frontal lobe tissue.
267 le contributions of different regions of the frontal lobes to probabilistic learning.
268 tion of the right hemisphere, especially the frontal lobe, to reading comprehension performance on th
269                    Lastly, rats induced with frontal lobe tumors and treated with a single intratumor
270  speech information from the temporal to the frontal lobe utilizes two different streams and conducti
271 riety of executive functions governed by the frontal lobes via actions on D1 and D2 receptors.
272 ome evidence from patients with ventromedial frontal lobe (VMF) damage argues against a very general
273                             The ventromedial frontal lobe (VMF) has been implicated in learning in dy
274 conomic models propose that the ventromedial frontal lobe (VMF) supports multiattribute decisions by
275 ypes were characterized by a large amount of frontal lobe volume loss, with one subtype showing grey
276 ion, and smaller medial, lateral and orbital frontal lobe volumes showed faster rates of decline.
277 of altered variability of caudate nucleus or frontal lobe volumes.
278                   Blood flow to the inferior frontal lobe was evaluated as a ratio compared with whol
279                                          The frontal lobe was involved in 14 patients (77.8%) and the
280 feedback attribution, while the ventromedial frontal lobe was necessary for learning the value of fea
281                             The left lateral frontal lobe was required for filtering option dimension
282 comprising the precuneus, cingulate & middle frontal lobe was significantly higher in psALS and affec
283 Society of Paris and announced that the left frontal lobe was the seat of speech.
284  thinning in children primarily in the right frontal lobes was associated with exposure to prenatal m
285 ger resting-state absolute EEG powers in the frontal lobe were associated with wiser advising from th
286 ration change and the beta band power in the frontal lobe were found to differ the most between the t
287                     Volumetric reductions of frontal lobes were largest in the ADHD+ODD group, possib
288 nificantly fewer correlated areas within the frontal lobes when compared to term controls.
289 ated by anatomically distinct regions of the frontal lobes when reinforcement contingencies are assur
290 entified a small cluster in the left lateral frontal lobe where children with greater upper-body musc
291         Abnormal ERPs were recorded over the frontal lobes, where FXTAS patients showed both P300 amp
292 wed that microstate 3 encompassed the medial frontal lobe, whereas microstate 4 involved the occipita
293 sions with a hypoechogenic halo in the right frontal lobe, which could correspond to brain abscesses.
294 er volume changes in both hemispheres of the frontal lobe, which suggests that structural changes in
295  a predilection for iron accumulation in the frontal lobes, which when combined with the subcortical
296 ical white matter, frontal lobe gray matter, frontal lobe white matter, and the hippocampus in a cros
297 eneration were limited to focal areas in the frontal lobe white matter.
298 and document increases in activity in medial frontal lobe with decreased activity in the dorsolateral
299 l as in distinct regions of the temporal and frontal lobes with increasing age.
300 , various cortical areas in the parietal and frontal lobes work together effortlessly to analyze obje

 
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