ライフサイエンスコーパス: fume

1 ), and nontraditional emissions (e.g., paint fumes).

2 nts present in simulated road paving asphalt fumes.

3 g simulated occupational exposure to asphalt fumes.

4 with increasing lifetime exposure to welding fumes.

5 s without exposing the laboratory to reagent fumes.

6 ix to calculate lifetime exposure to welding fumes.

7 jor component of cigarette smoke and cooking fumes.

8 ught to be mediated by manganese (Mn) in the fumes.

9 MMA-HS) or gas metal arc-mild steel (GMA-MS) fumes.

10 o green upon grinding and green to blue upon fuming.

11 2.05; median, 1.8 mg/m3 x years) for welding fumes, 1.85 (95% CI: 1.35, 2.54; median, 1.4 mug/m3 x ye

12 l exposure to particulate matter, gases, and fumes (15.6%).

13 Lifetime exposure to welding fumes above the median of exposed controls was associate

14 ogical materials, such as fly ash and silica fume, according to the residue amounts on sieves.

15 population, occupational exposure to welding fumes accounted for approximately 4% of lung cancer case

16 ned high porosity and ultrastability even in fuming acids.

17 Specific WTC exposures, including smelling fumes (AHR, 1.05 [95% CI, 1.01-1.09]; P = .007) or sewag

18 ulus and show diminished avoidance of acidic fumes, ammonia, and carbon dioxide.

19 aline and pozzolanic methods, such as silica fume and kaolin surface coating.

20 nual job/industry-specific estimates of lead fume and lead dust exposure, derived from a statistical

21 379) to estimate cumulative exposure to lead fume and lead dust.

22 ence of bentonite in combination with silica fume and polypropylene fibers (PPFs) on the workability,

23 at the use of bentonite together with silica fume and PPF significantly reduced chloride migration, i

24 with addition of bentonite along with silica fume and PPF.

25 al-fold, as do other exposures such as metal-fume and wood-dust exposure.

26 , cells at higher voltages exhibited sparks, fumes and fire.

27 tional agents, such as epoxy resins, welding fumes and hand-arm vibration, have been investigated, bu

28 e an effective method for scrubbing spent SF fumes and preventing SF from reaching the atmosphere.

29 the association between exposure to welding fumes and the risk of biliary tract, male breast, bone,

30 posure of Homo to these elements, via fires, fumes and their ashes, which could have played certain r

31 organic matter, is also released in exhaust fumes and widely used in chemical manufacturing.

32 th increasing cumulative exposure to welding fumes and with increasing exposure duration for Cr(VI) a

33 .e., the mixture of 5% bentonite, 10% silica fume, and 0.75% PPF resulted in the lowest chloride migr

34 mixtures containing 5% bentonite, 10% silica fume, and 0.75% PPF resulted in the lowest mass loss, th

35 The mixtures with 5% bentonite, 10% silica fume, and 1% PPF presented the best compressive strength

36 levels of 5% and 15%, a constant 10% silica fume, and different PPF dosages of 0.25%, 0.50%, 0.75%,

37 time occupational history, exposure to metal fume, and potential confounding factors.

38 Magnesite, silica fumes, and aluminum slag were used to create foam cordie

39 coryza, and exposure to cigarettes, cooking fumes, and other children in the home were each signific

40 xposure to diesel exhaust, solvents, welding fumes, and other respiratory irritants.

41 oking of meats at high temperatures produces fumes, and these fumes can be suspended as aerosols via

42 es to biological dusts, mineral dusts, gases/fumes, and vapors, gases, dusts, or fumes (VGDF) (high,

43 es to biological dusts, mineral dusts, gases/fumes, and VGDF were associated with incidence of COPD o

44 odels indicate that condensates from asphalt fumes are genotoxic and can promote skin tumorigenesis.

45 LCINS, including exposure to radon, cooking fumes, asbestos, heavy metals, and environmental tobacco

46 dicate that this technique could treat CH3Br fumes at approximately $5/kg, roughly one-third of the c

47 Repeated instillations of the two fumes at doses that mimic approximately 1 to 5 yr of wor

48 Exposure to the OF or ether fumes both produced increases in plasma corticosterone (

49 de high temperature environments and exhaust fumes, but electromagnetic fields have not been implicat

50 Behavioral avoidance of acidic fumes, but no increased labeling in the trigeminal pain

51 ation of total organic matter of the asphalt fume by electron impact ionization of isotope dilution g

52 clic aromatic hydrocarbons (PAHs) in asphalt fume by selected ion monitoring.

53 In addition, topical application of asphalt fumes by painting the tail skin of mice increased AP-1 a

54 irst report showing that exposure to asphalt fumes can activate AP-1 and intracellular signaling that

55 high temperatures produces fumes, and these fumes can be suspended as aerosols via the vapor-to-part

56 These results demonstrate that asphalt fume composition could be characterized and specific pri

57 Generated at 150 degrees C, the asphalt fume concentration in the animal exposure chamber ranged

58 With the developed method, asphalt fumes could be characterized into three fractions: (1) f

59 Welding fumes could contribute to an increased risk of some rare

60 sult of their exposures, and therefore these fumes could not be a hazard to the general public's heal

61 Our results showed that welding fumes, Cr(VI), and nickel might contribute independently

62 profiles, whereas asthma related to welding fumes differed.

63 nfertility and exposure to shift work, metal fumes, electromagnetic fields, solvents, lead, paint, pe

64 Flight crews complain of illness following a fume event in aircraft.

65 ed (p < 0.001) in the lung tissue of asphalt-fume-exposed mice relative to tissue from control animal

66 elevated (p < 0.001) in the urine of asphalt fume-exposed rats relative to controls.

67 In the urine of the asphalt fume-exposed rats, benzo[a]pyrene and its metabolites of

68 hydroxy metabolites in the urine of asphalt fume-exposed rats.

69 is study investigated the effects of welding fume exposure on correlates of oxidative stress in the s

70 applications: a study on the effect of metal fume exposure on immune response and a study of gene exp

71 ma related to flour, isocyanate, and welding fume exposure to be tested and clinically validated in f

72 step to study the health effects of asphalt fume exposure, an analytical method was developed to cha

73 g, the odds ratios for persons with dust and fume exposures for chronic cough, chronic phlegm, persis

74 laces are constructed such that they have no fume extraction system, and so all of the gases from com

75 umented in a variety of exposures, including fumes from flavoring plants, smoke from burn pits, and e

76 Capture of CH3Br fumes from fumigation chambers onto GAC, and electrolyti

77 ing individual particulate PhIP as simulated fumes from meat cooking, were constantly produced via co

78 Behaviorally, naked mole-rats did not avoid fumes from moderately high concentrations of acetic acid

79 nition of Gulf War illness, with exposure to fumes from munitions having the highest odds ratio (odds

80 He stated that the fumes from such establishments were not hazardous.

81 dried for reuse to capture and destroy CH3Br fumes from the next fumigation.

82 by other members of their households and to fumes from the use of gas as a cooking fuel.

83 satile method for the removal of bone cement fumes from the vicinity of health care workers in a simu

84 Occupational exposure to gas, dust, and fumes (GDF) increases the risk of asthma and eczema.

85 nd validated for characterization of asphalt fume generated under simulated road paving conditions.

86 A dynamic asphalt fume generation system was modified to provide consisten

87 In the process of fume generation, asphalt was initially preheated in an o

88 that welders exposed to manganese-containing fumes had plasma exosomes that contained more ASC than d

89 reused for multiple runs, and avoids use of fuming HCl.

90 isk of lung cancer after exposure to welding fumes, hexavalent chromium (Cr(VI)), and nickel, we anal

91 th active feedback to alert lab users when a fume hood is left open and unused.

92 In this work, we developed a low-cost fume hood monitoring device with active feedback to aler

93 ire process is safe and operated in standard fume hood settings, ensuring practicality and convenienc

94 bly, the reaction is performed in a standard fume hood setup, ensuring ease of handling and enhanced

95 Energy-conscious fume hood usage-for example, closing the sash when a hoo

96 The procedures are performed in a typical fume hood using ordinary laboratory glassware.

97 ions of a typical American vehicle, for each fume hood.

98 nt of half the size of a standard laboratory fume hood.

99 d at room temperature (20-25 degrees C) in a fume hood.

100 Ventilation, including fume hoods, consumes 40-70% of the total energy used by

101 rs, crystallizers, and filters in laboratory fume hoods.

102 mice were exposed daily (4h/day) to asphalt fume in a whole-body inhalation chamber for 10 days; 16

103 with reported occupational exposure to metal fume in the previous year (adjusted odds ratio (OR) = 1.

104 with 8 as controls and 16 exposed to asphalt fumes in a whole-body inhalation chamber for 10 days (4

105 Occupational exposure to dust and fumes in men and women is similarly associated with airf

106 ing was greater in those exposed to dust and fumes in men and women.

107 biological dust, mineral dust, and gases and fumes in relation to FEV1 levels.

108 potential involvement of exposure to asphalt fumes in skin carcinogenesis.

109 n of an occupational exposure (e.g., welding fume inhalation) in combination with diet, age, and stra

110 Although occupational exposure to dust and fumes is considered a risk factor for chronic obstructiv

111 coarse aggregates, superplasticizers, silica fume, marble dust, and water, with compressive strength

112 Occupational exposure to asphalt fumes may pose a health risk.

113 t to assess the effect of mild steel welding fumes (MS-WF) on PAFR-dependent pneumococcal adhesion an

114 ic acid derivatives has been developed using fuming nitric acid as the nitrating agent.

115 s also observed in dye 5, where grinding and fuming of a solid sample gave blue- and red-shifted emis

116 nonporous surfaces consists of cyanoacrylate fuming of the fingerprint material, followed by impregna

117 that these macroscale dynamics are 'exhaust fumes' of more relevant processes.

118 vestigated the effect of exposure to asphalt fumes on AP-1 activation in mouse JB6 P+ epidermal cells

119 risk of chronic cough seen with occupational fumes or smoke exposure disappeared after adjusting for

120 without prior development with cyanoacrylate fuming or Vacuum Metal Deposition, was also examined.

121 bar pneumonia and recent exposure to ferrous fume (OR = 2.3, 95% CI: 1.2, 4.3).

122 bability of exposures to dusts, gas, vapors, fumes, or sensitizers also contributed significantly to

123 Exposure to dusts, gas, vapors, fumes, or sensitizers was associated with a significantl

124 with both previous exposure (PE) to dust and fumes (P = 0.006) and airflow limitation (AFL) (P = 0.03

125 nfirmed occupational exposure to dust and/or fumes, physical inactivity, maternal or secondhand tobac

126 8 (95% CI: 1.01, 1.38) was found for welding fumes (prevalence controls: 22.8%), increasing to 1.38 f

127 Exposure to asphalt fumes promoted basal as well as epidermal growth factor-

128 n task for many workers, exposure to welding fumes represents an important risk factor for lung cance

129 test the hypothesis that inhalation of metal fume reversibly increases susceptibility to pneumonia, t

130 thesis that ferrous and possibly other metal fumes reversibly predispose to infectious pneumonia.

131 terials such as cement, fly ash (FA), silica fume (SF), ground granulated blast furnace slag (GGBFS),

132 ral wheat straw fiber, bentonite, and silica fume (SF).

133 oal gangue ash (CGA) replacement with silica fume (SF: 0-20%) were varied in the alkaline solution by

134 Exposure to asphalt fumes significantly increased AP-1 activity in JB6 P+ ce

135 ere less than those measured on a nonporous, fumed silica (Cabosil) and were also found to decrease a

136 es (MMMs) by incorporating a surface-treated fumed silica (FS) into a PDMS polymer matrix to enhance

137 of Pt nanoparticles on titania-incorporated fumed silica (Pt/Ti-FS) supports was examined using X-ra

138 Specifically, we find for fumed silica a positive correlation of toxicity with hyd

139 atic interactions of the silanol surfaces of fumed silica aggregates with the extracellular plasma me

140 lial and macrophage cells, we discovered for fumed silica an important toxicity relationship to posts

141 ensive routes to high-purity precipitated or fumed silica and compounds with single Si-C bonds.

142 Lewis acid-decorated PS when reinforced with fumed silica as a filler.

143 ica zeolite ITQ-12 has been synthesized with fumed silica as the silica source in the presence of 1,3

144 gold electrodes modified with carbon ink or fumed silica can compete with the oxidation of mediators

145 eated equal and that the unusual toxicity of fumed silica compared to that of colloidal silica derive

146 The stability of the fumed silica derivative, however, is greatly compromised

147 al silica into aggregates mimicking those of fumed silica had no effect on cell viability or hemolysi

148 Easy to prepare solid materials based on fumed silica impregnated with polyethylenimine (PEI) wer

149 As a potential remedy, here we use fumed silica nanoparticles (FSN) have shown excellent po

150 to generate hydroxyl radicals for Stober and fumed silica NPs with comparable primary particle sizes

151 ation of various nitric oxide (NO)-releasing fumed silica particles (0.2-0.3 microm) are reported.

152 urther shown that the resulting NO-releasing fumed silica particles can be embedded into polymer film

153 physical dispersion of nonporous, nanoscale, fumed silica particles in glassy amorphous poly(4-methyl

154 bstantially more stable derivative made from fumed silica possess equivalent local framework wall str

155 e sodium - free mesostructure assembled from fumed silica retains an open framework under the same hy

156 We propose fumed silica toxicity stems from its intrinsic populatio

157 silica) or high-temperature pyrolysis (e.g., fumed silica) routes.

158 xtural properties and chemical reactivities (fumed silica, amorphous silica and MCM-41) was evaluated

159 ic acid hydrazide (INAH) chemically modified fumed silica, as a novel adsorbent, was designed for the

160 l radicals generated by the strained 3MRs in fumed silica, but largely absent in colloidal silicas, m

161 lystyrene), various silica precursors (TEOS, fumed silica, or zeolite seed), and many oils (decane, p

162 conventional filled polymer systems, reflect fumed silica-induced disruption of polymer chain packing

163 A highly active and durable fumed silica-supported heterogeneous molybdenum(VI) cata

164 on of hydrophobic silicon dioxide particles (fumed silicon dioxide), as model air pollutants, and Lan

165 For fumed SiO(2) NP in a highly complex food matrix, a metho

166 n nanotubes, graphene, carbon black, Ag, and fumed SiO2 nanoparticles.

167 estimates of occupational exposure to dust, fumes, smoke, and gas.

168 sitol 3-kinase activation eliminated asphalt fume-stimulated AP-1 activation and formation of anchora

169 Fuming sulfuric acid charges SWNTs and promotes their or

170 arbon nanotube fibers were swollen in oleum (fuming sulfuric acid), and organic spacer groups were co

171 lear Pd(III) intermediate in concentrated or fuming sulfuric acid.

172 to benchmark the accuracy and sensitivity of FUME-TCRseq against existing methods demonstrated excell

173 Spatially resolved FUME-TCRseq analysis of colorectal cancers using macrodi

174 Furthermore, FUME-TCRseq detected more clonotypes than a commercial R

175 FUME-TCRseq incorporates unique molecular identifiers in

176 FUME-TCRseq is a TCR sequencing methodology that support

177 In summary, FUME-TCRseq represents an accurate, sensitive, and low-c

178 Here, we developed and validated FUME-TCRseq, a robust and sensitive RNA-based TCR sequen

179 Characterization of the asphalt fume test atmospheres included the following: (1) determ

180 Welding generates and releases fumes that are hazardous to human health.

181 llergens, (ii) isocyanates and (iii) welding fumes the day after relevant exposure.

182 in part mediated by the capacity of welding fumes to increase PAFR-dependent pneumococcal adhesion a

183 Asphalt fumes transiently activated c-Jun NH2-terminal kinases w

184 iously, we described a system in which CH3Br fumes vented from fumigation chambers could be captured

185 s, gases/fumes, and vapors, gases, dusts, or fumes (VGDF) (high, low, or unexposed as reference).

186 rted job exposure to vapors, gases, dust, or fumes (VGDF) (PR 4.3; 95% CI 2.2 to 8.6).

187 tional exposure to vapors, gases, dusts, and fumes (VGDF) and pesticides is associated with a lower l

188 pational exposures to vapors, gas, dust, and fumes (VGDF) are associated with high-attenuation areas

189 The fume was conducted from the generator to an exposure cha

190 The asphalt fume was generated at 180 degrees C and the concentratio

191 Total organic matter of the asphalt fume was quantified over the 5 exposure days.

192 Occupational exposure to both dust and fumes was reported by 47.9% of men and 20.1% of women.

193 , isocyanate (isocyanate asthma), or welding fumes (welding asthma) and identify potential biomarkers

194 s that superplasticizers, cement, and silica fume were the most weighted factors that affected CS.

195 reatened the closure of many factories whose fumes were considered hazardous to the public's health.

196 Asphalt fumes were generated from a dynamic generation system th

197 An important health concern of welding fume (WF) exposure is neurological dysfunction akin to P

198 Welding fumes (WFs) are a complex mix of metallic oxides, fluori

199 nhalational exposures (eg, vapor, dust, gas, fumes), which are known correlates of reduced lung funct

200 at associated with another irritant, ammonia fumes, which elicited an increase in trigeminal but not

201 responses of this species to airborne acidic fumes, which would be expected to affect the trigeminal

202 tate, which vanished upon grinding, and upon fuming with acetone, the fluorescence turned yellowish o

203 1G can be restored by grinding 1Y or 1G* or fuming with dichloromethane (DCM) vapor.