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1 y), which have been implicated as plant anti-fungal toxins.
2 ve sequence similarity with other viral anti-fungal toxins.
3                                The plant and fungal toxin 3-nitropropionic acid, an irreversible inac
4 cystin (ST), a precursor of the better known fungal toxin aflatoxin (AF).
5  in some of the cytotoxic effects of the ETP fungal toxins and their synthetic analogues.
6                We previously showed that the fungal toxin brefeldin A (BFA) induces the ADP-ribosylat
7 yphae escape this mechanism by releasing the fungal toxin candidalysin, which causes eosinophil membr
8                                          The fungal toxin cytochalasin D (CD) interferes with the nor
9 ce of a programmed cell death (PCD)-inducing fungal toxin FB1.
10 ine by ceramide synthase is inhibited by the fungal toxin fumonisin B(1).
11 ltration of leaf tissues with a PCD-inducing fungal toxin fumonisin B1 (FB1) and increased sensitivit
12                                          The fungal toxin fumonisin B1 (FB1) induces apoptosis-like p
13 cal inhibition of ceramide production by the fungal toxin, fumonisin B1, impaired TCR-induced interle
14 e type of mammalian channel affected by this fungal toxin further support our contention that KP4 inh
15                                              Fungal toxin fusicoccin (FC) promotes stomatal opening b
16 cess of nuclear export can be blocked by the fungal toxin leptomycin B (LMB).
17 roscopy (cryo-EM) structure of the two-part, fungal toxin Pleurotolysin (Ply), together with crystal
18 erium Pseudomonas syringae) or fusicoccin (a fungal toxin produced by the fungus Fusicoccum amygdali)
19                     KP4 is a virally encoded fungal toxin secreted by the P4 killer strain of Ustilag
20 fully depolarized membranes by fusicoccin, a fungal toxin that activates the H(+)-ATPase, indicates t
21                         Fusicoccin (FC) is a fungal toxin that activates the plant plasma membrane H+
22 very of pH upon treatment with fusicoccin (a fungal toxin that activates the plasma membrane proton p
23 inds and severs filaments, and phalloidin, a fungal toxin that binds and stabilizes F-actin.
24 n addition, treatment with cytochalasin D, a fungal toxin that depolymerizes actin filaments, reduced
25 in is an epidithiopiperazine-2,5-dione (ETP) fungal toxin that disrupts cellular functions likely via
26                                Phalloidin, a fungal toxin which stabilizes F-actin and increases its
27  a family of structurally related polyketide fungal toxins with nearly universal toxicity.
28 ketopiperazines, leading to the formation of fungal toxins with prolific biological activities.