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1 GAA repeats in M9/pMGA gene expression in M. gallisepticum.
2 in the avian respiratory pathogen Mycoplasma gallisepticum.
3 tiserum recognizes a 150-kDa protein from M. gallisepticum.
4 ved interaction between house finches and M. gallisepticum.
5 ding of molecular pathogenesis in Mycoplasma gallisepticum.
6 plasmal conjunctivitis, caused by Mycoplasma gallisepticum.
7  days after experimental inoculation with M. gallisepticum.
8 n direct contact of HD-11 cells with live M. gallisepticum.
9  the first genome-scale reconstruction of M. gallisepticum.
10 transmission potential, among isolates of M. gallisepticum.
11 of naive juveniles or the introduction on M. gallisepticum.
12 increased rapidly after reintroduction of M. gallisepticum.
13 date genomic variability among strains of M. gallisepticum.
14 ntical homolog in four strains of Mycoplasma gallisepticum.
15 understanding of the relationship between M. gallisepticum and LPAIV during copathogenesis in the nat
16 ses to monoinfection and coinfection with M. gallisepticum and LPAIV highlighted the involvement of d
17 es suggest that coinfection with virulent M. gallisepticum and LPAIV induces decreases in the express
18 exposed to the bacterial pathogen Mycoplasma gallisepticum and showed signatures of balancing selecti
19 (Leptospira, Salmonella enterica, Mycoplasma gallisepticum) and their functionality showed a direct c
20 omoterless lacZ gene and transformed into M. gallisepticum by using transposon Tn4001 as a vector.
21 te catabolism and all genes examined from M. gallisepticum, C. perfringens, and S. pneumoniae were un
22                  The initial emergence of M. gallisepticum caused a rapid halving of house finch dens
23                                           M. gallisepticum cgMLST represents a standardized, accurate
24 ountries on 4 continents were typed using M. gallisepticum cgMLST.
25                      Southern analysis of M. gallisepticum chromosomal DNA confirmed that the phase-v
26 rchived isolates of the bacterium Mycoplasma gallisepticum collected during sequential emergence even
27      We inoculated 55 isolates of Mycoplasma gallisepticum, collected over 20 y from outbreak, into h
28                                   Mycoplasma gallisepticum colonizes the chicken respiratory mucosa a
29                            A total of 425 M. gallisepticum conserved genes (49.85% of M. gallisepticu
30                  For the transformants of M. gallisepticum containing the reporter with deletion of n
31 t, vaccination, and surveillance, Mycoplasma gallisepticum continues to cause significant morbidity,
32 rotein (heat shock protein 60) of Mycoplasma gallisepticum could induce apoptosis in peripheral blood
33                 By injecting live Mycoplasma gallisepticum cultures into the ankle joint a typical my
34  that both GapA and CrmA are required for M. gallisepticum cytadherence and pathogenesis.
35  CrmA might play an essential role in the M. gallisepticum cytadherence process.
36 house finch epizootic outbreak strains of M. gallisepticum displayed a greater ability to adhere to,
37 nt was used to probe a genomic library of M. gallisepticum DNA.
38 of the avian respiratory pathogen Mycoplasma gallisepticum encode a family of hemagglutinins that are
39 ic and include the lipoprotein of Mycoplasma gallisepticum, encoded by the MGA0674 gene.
40  isolates of the pathogen collected after M. gallisepticum established itself in western North Americ
41  proteins important for cytadherence, few M. gallisepticum factors or pathways contributing to host c
42            To effectively analyze Mycoplasma gallisepticum for virulence-associated determinants, the
43 y emerged pathogen, the bacterium Mycoplasma gallisepticum, form a system in which evidence suggests
44 ermits the comprehensive screening of the M. gallisepticum genome for the identification of novel vir
45  gallisepticum conserved genes (49.85% of M. gallisepticum genome) were selected as core genome targe
46  obtained in this manner to the annotated M. gallisepticum genome, the precise locations of transposo
47                             A total of 81 M. gallisepticum genomes from 5 countries on 4 continents w
48 f this scheme, a diverse collection of 37 M. gallisepticum genomes was used to identify cgMLST target
49 ovel insights into inter- and intrastrain M. gallisepticum genomic variability and the genetic basis
50 -kDa cytadhesin-like protein from Mycoplasma gallisepticum has been identified.
51                    House finch-associated M. gallisepticum (HFMG) spread rapidly and increased in vir
52                             MGA_1199, the M. gallisepticum homologue of the cytadherence-associated p
53  Mycoplasma pneumoniae, and MGA_0928, the M. gallisepticum homologue of the M. pneumoniae cytoskeleta
54 thought to be important for mechanisms of M. gallisepticum-host interaction, pathogenesis, and immune
55 d with virulent, immunopathologic Mycoplasma gallisepticum; however, mechanisms delineating these fro
56 e that glycerol metabolism is required by M. gallisepticum in a niche that we have yet to study.
57 lisepticum vaccines, and the detection of M. gallisepticum in game and free-flying song birds has str
58  disease manifestations characteristic of M. gallisepticum infection.
59  second hypothesis that reintroduction of M. gallisepticum into a multiage group of previously expose
60                                   Mycoplasma gallisepticum is a significant respiratory and reproduct
61                                   Mycoplasma gallisepticum is among the most economically significant
62                                   Mycoplasma gallisepticum is an avian respiratory and reproductive t
63                                   Mycoplasma gallisepticum is an etiologic agent of chronic respirato
64                                   Mycoplasma gallisepticum is an important pathogen of chickens and t
65 low) strain of the avian pathogen Mycoplasma gallisepticum is capable of producing H2O2 when grown in
66 se finches and that the reintroduction of M. gallisepticum is sufficient to cause a new outbreak, eve
67                                   Mycoplasma gallisepticum is the most virulent and economically impo
68                                   Mycoplasma gallisepticum is the primary etiologic agent of chronic
69 oducible method for differentiation among M. gallisepticum isolates.
70                                   Mycoplasma gallisepticum, known primarily as a respiratory pathogen
71 omparative transcriptomic analysis of the M. gallisepticum live attenuated vaccine strain F and the v
72 elated to bacterial infections, including M. gallisepticum, M. synoviae, E. coli, and other gram-nega
73 pulations, the bacterial pathogen Mycoplasma gallisepticum (MG) has been increasing in virulence.
74 characteristics drive patterns of Mycoplasma gallisepticum (MG) infections in the house finch (Carpod
75 e conjunctival bacterial pathogen Mycoplasma gallisepticum (MG), to experimentally examine the impact
76                                    Global M. gallisepticum mutagenesis is currently limited to the us
77                An annual pulse of Mycoplasma gallisepticum-naive juveniles increasing the number of s
78  experimentally determined growth rate of M. gallisepticum of 0.244+/-0.03[Formula: see text].
79  M. synoviae, but not on the sialidase of M. gallisepticum or the sialidases or other enzymes essenti
80 2, a novel virulence mechanism in Mycoplasma gallisepticum Our findings lead to a better understandin
81                        The reemergence of M. gallisepticum outbreaks among poultry, the increased use
82       Moreover, molecular epidemiology of M. gallisepticum outbreaks can be performed using RFLP and/
83 with > 50 isolates of their novel Mycoplasma gallisepticum pathogen collected over a 20-year period d
84 -kDa cell surface antigen (M9) of Mycoplasma gallisepticum PG31 that mediates antibody-induced agglut
85       This construct was transformed into M. gallisepticum PG31.
86 ptomic vlhA gene expression directly from M. gallisepticum populations present on tracheal mucosae du
87                  Chickens coinfected with M. gallisepticum R(low) followed by LPAIV H3N8 exhibited si
88 ted first with H3N8 and subsequently with M. gallisepticum R(low) Recovery of live M. gallisepticum w
89 ickens given a mock infection followed by M. gallisepticum R(low) The transcriptional responses to mo
90 ected first with LPAIV H3N8 and then with M. gallisepticum R(low), compared to chickens given a mock
91  in the natural host compared to virulent M. gallisepticum R(low).
92               These results indicate that M. gallisepticum regulates gene expression upon exposure to
93  the avian respiratory tract with Mycoplasma gallisepticum results in a profound inflammatory respons
94                                   Mycoplasma gallisepticum Rhigh, the attenuated, high-passage deriva
95 rom tracheas of 20 chickens infected with M. gallisepticum Rlow and 20 mock-infected animals at days
96    Fibronectin was found to be present in M. gallisepticum Rlow protein extracts by Western blotting
97 and the sequenced genomes compared to the M. gallisepticum Rlow reference genome.
98 leotide identity with the pMGA1.1 gene of M. gallisepticum S6.
99 rst description of a functional gene from M. gallisepticum showing homology to cytadhesin genes from
100  to determine the feasibility of using an M. gallisepticum-specific gene encoding a phase-variable pu
101 ions, and significantly higher numbers of M. gallisepticum-specific IgG- and IgA-secreting plasma/B c
102                                Currently, M. gallisepticum strain differentiation based on sequence a
103 mic sequence of the virulent, low-passage M. gallisepticum strain R (R(low)) has been reported, genom
104 n of the phenotypic expression of Mycoplasma gallisepticum strain R low (passage 15) to that of strai
105 ously demonstrated that avirulent Mycoplasma gallisepticum strain R(high) (passage 164) is lacking th
106 fected or coinfected with either virulent M. gallisepticum strain R(low) or LPAIV H3N8 (A/duck/Ukrain
107 to investigate transcriptional changes in M. gallisepticum strain R(low) upon exposure to eukaryotic
108 n following challenge with the pathogenic M. gallisepticum strain R(low).
109  We have determined that virulent Mycoplasma gallisepticum strain Rlow is capable of binding the extr
110 locus sequence typing (cgMLST) scheme for M. gallisepticum strains and field isolates.
111 of cgMLST allowed differentiation between M. gallisepticum strains of the same outbreak.
112 AMA assays varied between 10(1) and 10(4) M. gallisepticum template copies/reaction, while that of th
113                                   Mycoplasma gallisepticum, the cause of chronic respiratory infectio
114                The avian pathogen Mycoplasma gallisepticum, the etiological agent of chronic respirat
115                                   Mycoplasma gallisepticum, the primary etiologic agent of chronic re
116 of a widespread poultry pathogen, Mycoplasma gallisepticum, through a previously unknown host, the ho
117 l microsatellite of the bacterium Mycoplasma gallisepticum, through the collection and analysis of in
118 lts can initiate an epidemic and transmit M. gallisepticum to naive house finches and that the reintr
119 igned to target 5 sequences unique to the M. gallisepticum ts-11 strain: vlhA3.04a, vlhA3.04b, vlhA3.
120 5, and mg0359 was able to distinguish the M. gallisepticum ts-11 vaccine strain from field isolates.
121 othetical protein gene, mg0359, unique to M. gallisepticum ts-11 vaccine strain.
122                   A total of 239 samples (M. gallisepticum vaccine and type strains, pure cultures, a
123             Analyses of the genome of the M. gallisepticum vaccine strain F revealed numerous differe
124  isolates, whole-genome sequencing of the M. gallisepticum vaccine strain ts-11 and several "ts-11-li
125                         A live attenuated M. gallisepticum vaccine strain, GT5, was previously shown
126 d in the present study to distinguish the M. gallisepticum vaccine strains and field isolates based o
127 current approaches to quickly distinguish M. gallisepticum vaccine strains from field isolates.
128 nscriptomic responses to two experimental M. gallisepticum vaccines were assessed during the first 2
129  among poultry, the increased use of live M. gallisepticum vaccines, and the detection of M. gallisep
130  an attenuated strain and plays a role in M. gallisepticum virulence.
131 omic variability and the genetic basis of M. gallisepticum virulence.
132               The GroEL gene from Mycoplasma gallisepticum was cloned and expressed in Escherichia co
133 metabolic model for the bacterium Mycoplasma gallisepticum was created.
134 , at epizootic outbreak in house finches, M. gallisepticum was particularly adept at using the intra-
135  M. gallisepticum R(low) Recovery of live M. gallisepticum was significantly higher in chickens infec
136 h have not evolved protective immunity to M. gallisepticum We show using 3 different metrics of virul
137 trains of the bacterial pathogen, Mycoplasma gallisepticum, which jumped from poultry into North Amer
138 eproducible methods for comparisons among M. gallisepticum whole genomes.
139 recently reported that the interaction of M. gallisepticum with chicken tracheal epithelial cells (TE
140 he notion that the initial interaction of M. gallisepticum with host respiratory epithelial cells con

 
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