ライフサイエンスコーパス: gamma-aminobutyric acid

1 enrichment of glutamate, glutamine and GABA (gamma-aminobutyric acid).

2 the major inhibitory neurotransmitter GABA (gamma-aminobutyric acid).

3 H variant on the inhibitory neurotransmitter gamma-aminobutyric acid.

4 t coexpress galanin and the neurotransmitter gamma-aminobutyric acid.

5 gamma-Aminobutyric acid, 4-hydroxyproline, glycine, leuc

6 id and fatty acid compositions and levels of gamma-aminobutyric acid (54.9 mg/100 g), free phenolics

7 hat bilateral infusions into the RMTg of the gamma-aminobutyric acid A (GABAA) agonist, muscimol, ind

8 n the Nlrp3 inflammasome were independent of gamma-aminobutyric acid A receptor activation or N-methy

9 elective action on the omega1 subtype of the gamma-aminobutyric acid A receptor, zolpidem tartrate pr

10 Antagonism of gamma-aminobutyric acid A receptors (GABA(A)Rs) by bilob

11 in two neurotransmitter receptor genes, the gamma-Aminobutyric acid-A receptor delta and gamma-amino

12 iated protein-like 2), glycine receptor, and gamma-aminobutyric acid-A receptor.

13 thyl-4-isoxazolepropionic acid, glycine, and gamma-aminobutyric acid-A receptors), were prevalent in

14 gamma-Aminobutyric acid aminotransferase (GABA-AT) is a

15 od-brain barrier and inhibit the activity of gamma-aminobutyric acid aminotransferase (GABA-AT), the

16 al activator that regulates transcription of gamma-aminobutyric acid aminotransferase (GABA-AT; GabT)

17 GAD65 synthesizes gamma-aminobutyric acid, an important autocrine and para

18 idative stress, and is associated with GABA (gamma-aminobutyric acid, an inhibitory neurotransmitter)

19 In particular, bicyclic gamma-amino acids-gamma-aminobutyric acid analogues-were synthesized.

20 with furosine (2-FM-lysine), 2-furoylmethyl-gamma-aminobutyric acid and 2-FM-arginine were detected.

21 es in some neurotransmitter systems, such as gamma-aminobutyric acid and glutamate, mainly in the cor

22 the method to monitor glutamate, glutamine, gamma-aminobutyric acid and lactate in the brains of una

23 elease the inhibitory neurotransmitter GABA (gamma-aminobutyric acid) and are inhibited by iSPNs and

24 ve antagonist bicuculline, the agonist GABA (gamma-aminobutyric acid), and the classical benzodiazepi

25 as well as regulating release of glutamate, gamma-aminobutyric acid, and acetylcholine from presynap

26 tivated by l-glutamate (l-Glu), l-aspartate, gamma-aminobutyric acid, and acetylcholine, with l-Glu e

27 mediated by the neurotransmitters glutamine, gamma-aminobutyric acid, and dopamine.

28 Interactions between the endocannabinoid, gamma-aminobutyric acid, and glutamate systems and their

29 biogenesis including synthesis of glutamate/gamma-aminobutyric acid as a potential transcriptional t

30 that the neurons also release glutamate and gamma-aminobutyric acid as cotransmitters, with striking

31 etabotropic glutamate receptors (mGluRs) and gamma-aminobutyric acid B (GABA(B)) receptors (GABA(B)Rs

32 ons (MSNs) triggered astrocyte signaling via gamma-aminobutyric acid B (GABA(B)) receptors.

33 gamma-Aminobutyric acid-A receptor delta and gamma-aminobutyric acid B receptor subunit 1; their diff

34 s, including two pathways, phenylacetate and gamma-aminobutyric acid catabolism, which were found to

35 BrdU(+) /GABA(+) (gamma-aminobutyric acid) cells were also found but no ne

36 s and the specific impairment of perisomatic gamma-aminobutyric acid circuits are hallmarks of the sc

37 per and lower limb was associated with lower gamma-aminobutyric acid concentration in the sensorimoto

38 Additionally, the reduced gamma-aminobutyric acid concentration may contribute to

39 n older adults to a likely decrease in GABA (Gamma Aminobutyric Acid) concentration in visual cortex,

40 n models were used to compare differences in gamma-aminobutyric acid concentrations between patients

41 microinjection to the NAcSh decreased local gamma-aminobutyric acid concentrations.

42 Fast glutamate and gamma-aminobutyric acid cotransmission convey discrete p

43 iologically relevant concentrations, whereas gamma-aminobutyric acid does not.

44 imaged the distribution of neurotransmitters-gamma-aminobutyric acid, dopamine and serotonin-with hig

45 during real-time-corrected three-dimensional gamma-aminobutyric acid-edited magnetic resonance (MR) s

46 As the inhibitory gamma-aminobutyric acid-ergic (GABAergic) transmission h

47 a variety of neurotransmitters, such as the gamma-aminobutyric acid-ergic system, the study of prefr

48 large part by direction-selective release of gamma-aminobutyric acid from starburst amacrine cells on

49 The gamma aminobutyric acid (GABA) neurotransmission system

50 lly, increased mitochondrial activity causes gamma aminobutyric acid (GABA) sequestration in the mito

51 major excitatory (glutamate) and inhibitory (gamma aminobutyric acid (GABA)) neurotransmitter circuit

52 Gamma aminobutyric acid (GABA), a neurotransmitter of th

53 ants changes the number of dopamine (DA)- or gamma aminobutyric acid (GABA)-expressing neurons, with

54 napse (93%) on dendrites that do not contain gamma aminobutyric acid (GABA).

55 Abnormalities in prefrontal gamma aminobutyric acid (GABA)ergic transmission, partic

56 Type A gamma-aminobutyric acid (GABA(A)) receptors are pentamer

57 The effects of gamma-aminobutyric acid (GABA) A receptor activation on

58 determine whether antecedent stimulation of gamma-aminobutyric acid (GABA) A receptors with the benz

59 Gamma-Aminobutyric acid (GABA) accumulates in plants fol

60 gamma-Aminobutyric acid (GABA) administration has been s

61 The differentiation of gamma-aminobutyric acid (GABA) and dopamine neurons, but

62 Gamma-aminobutyric acid (GABA) and glutamate are the mos

63 s containing dopamine (DA), serotonin (5HT), gamma-aminobutyric acid (GABA) and glutamate neurons.

64 lated PVN -> NAc neurons and quantified both gamma-aminobutyric acid (GABA) and glutamate release and

65 atory amino acid neurotransmitter systems of gamma-aminobutyric acid (GABA) and glutamate, respective

66 Levels of gamma-aminobutyric acid (GABA) and glutamic acid decarbo

67 and the inhibitory transmitters include both gamma-aminobutyric acid (GABA) and glycine (GLY).

68 ion of health-promoting metabolites, such as gamma-aminobutyric acid (GABA) and phenolic compounds.

69 Glutamate (GLU) and gamma-aminobutyric acid (GABA) are the major excitatory

70 that release the inhibitory neurotransmitter gamma-aminobutyric acid (GABA) at non-image-forming brai

71 antihypertensive activity due to their large gamma-aminobutyric acid (GABA) content (6.8-10.6 mg/g) a

72 his enhanced vulnerability may be related to gamma-aminobutyric acid (GABA) deficits observed in schi

73 valuable structures, including amido-diols, gamma-aminobutyric acid (GABA) derivatives, and heterocy

74 lobe of the cockroach Periplaneta americana, gamma-aminobutyric acid (GABA) has been identified as th

75 Altered neurotransmission of gamma-aminobutyric acid (GABA) has been implicated in th

76 scopy to map histamine (HA), FMRF-amide, and gamma-aminobutyric acid (GABA) immunoreactivity in the c

77 Abnormally elevated levels of gamma-aminobutyric acid (GABA) in the medial prefrontal

78 -fat milk and regular-fat cheese enriched in gamma-aminobutyric acid (GABA) influences daytime ambula

79 Despite its importance for gamma-aminobutyric acid (GABA) inhibition and involvemen

80 nked to reductions of parvalbumin containing gamma-aminobutyric acid (GABA) interneurons and volumes

81 d-aspartate receptor (NMDAR) hypofunction on gamma-aminobutyric acid (GABA) interneurons disinhibitin

82 Gamma-aminobutyric acid (GABA) is a widely conserved sig

83 ted, the role of VGLUT3 in neurons releasing gamma-aminobutyric acid (GABA) is not settled.

84 gamma-Aminobutyric acid (GABA) is one of the major inhib

85 gamma-Aminobutyric acid (GABA) is the major inhibitory n

86 gamma-Aminobutyric acid (GABA) is the major inhibitory t

87 gamma-Aminobutyric acid (GABA) is the most abundant inhi

88 gamma-Aminobutyric acid (GABA) is the primary inhibitory

89 In contrast, KATPHI islets had low gamma-aminobutyric acid (GABA) levels and lacked (13)C i

90 Low-pressure plasma also increased gamma-aminobutyric acid (GABA) levels from approximately

91 es brain excitatory glutamate and inhibitory gamma-aminobutyric acid (GABA) levels, including in brai

92 (inhibitory), schizophrenia (excitatory and gamma-aminobutyric acid (GABA) mediated) and bipolar dis

93 nia, the density of cartridges detectable by gamma-aminobutyric acid (GABA) membrane transporter 1 im

94 xylase enzyme GAD67, a critical actor of the gamma-aminobutyric acid (GABA) metabolism as it catalyse

95 Early-born gamma-aminobutyric acid (GABA) neurons (EBGNs) are major

96 netic stimulation of mouse zona incerta (ZI) gamma-aminobutyric acid (GABA) neurons or their axonal p

97 Stimulation of dorsal raphe gamma-aminobutyric acid (GABA) neurons promoted movement

98 at to show that optogenetic inhibition of LH gamma-aminobutyric acid (GABA) neurons restricted to cue

99 d c-fos expression in VTA 5-HT2CR expressing gamma-aminobutyric acid (GABA) neurons, but not 5-HT2CR

100 Aberrant glutamate and gamma-aminobutyric acid (GABA) neurotransmission contrib

101 sure (ACE) resulted in an enhancement of the gamma-aminobutyric acid (GABA) neurotransmitter system i

102 lease either the amino acid (AA) transmitter gamma-aminobutyric acid (GABA) or glutamate.

103 proteins, scaffold and adaptor proteins, and gamma-aminobutyric acid (GABA) or glycine receptors.

104 riments did not confirm colocalizations with gamma-aminobutyric acid (GABA) or the circadian coupling

105 The non-protein amino acid, gamma-aminobutyric acid (GABA) rapidly accumulates in pl

106 Given the heterogeneity within the gamma-aminobutyric acid (GABA) receptor and transporter

107 ering neurites are also immunopositive for a gamma-aminobutyric acid (GABA) receptor subunit (GABAA R

108 Nonselectively reducing intrastriatal gamma-aminobutyric acid (GABA) receptor-A inhibition syn

109 rprisingly, severe antagonists of ionotropic gamma-aminobutyric acid (GABA) receptors.

110 dent drinking, and basal and alcohol-induced gamma-aminobutyric acid (GABA) release in the central nu

111 Our results demonstrate that the gamma-aminobutyric acid (GABA) release is lower and opio

112 We found that local gamma-aminobutyric acid (GABA) release on dendrites of m

113 studies revealed a significant reduction in gamma-aminobutyric acid (GABA) release probability witho

114 tion of the metabotropic GABA(B) receptor by gamma-aminobutyric acid (GABA) results in prolonged inhi

115 se and nitrate stimulated PGC1alpha-mediated gamma-aminobutyric acid (GABA) secretion from muscle.

116 Gamma-aminobutyric acid (GABA) serves diverse biological

117 search, Gao and colleagues identify that the gamma-aminobutyric acid (GABA) shunt is upregulated with

118 and exendin-4, a GLP-1 receptor agonist, on gamma-aminobutyric acid (GABA) signaling in hippocampal

119 Altered gamma-aminobutyric acid (GABA) signaling in the prefront

120 rtical inflammation, synaptic plasticity and gamma-aminobutyric acid (GABA) signaling, and liver infl

121 f one target, NKCC1, initiates the switch in gamma-aminobutyric acid (GABA) signaling, limits early s

122 tal landmark is the excitatory to inhibitory gamma-aminobutyric acid (GABA) switch caused by reciproc

123 is associated with reduced concentrations of gamma-aminobutyric acid (GABA) that are normalized by an

124 additionally stained with antibodies against gamma-aminobutyric acid (GABA) to identify GABAergic int

125 ors (HA) exhibited a significant increase in gamma-aminobutyric acid (GABA) transmission compared wit

126 lutamate signaling (P-value=7.22 x 10(-15)), gamma-aminobutyric acid (GABA) transport (P-value=1.36 x

127 f NL3 leads to a large increase in vesicular gamma-aminobutyric acid (GABA) transporter (vGAT) and gl

128 We saw that stimulation of vesicular gamma-aminobutyric acid (GABA) transporter (VGAT)-expres

129 AT-1, encoded by SLC6A1, is one of the major gamma-aminobutyric acid (GABA) transporters in the brain

130 PFC) levels of glutamate+glutamine (Glx) and gamma-aminobutyric acid (GABA) were measured before, dur

131 d caffeic acids, as well as higher amount of gamma-aminobutyric acid (GABA) were observed in biodynam

132 trol of endogenous alpha5-subunit-containing gamma-aminobutyric acid (GABA)(A) receptors (alpha5-GABA

133 important ion channels, gated by glutamate, gamma-aminobutyric acid (GABA), and acetylcholine, also

134 or inhibitory neurotransmitter of the brain, gamma-aminobutyric acid (GABA), can be released through

135 o find the relative quantity of glutamate to gamma-aminobutyric acid (GABA), DA, and glutamate neuron

136 ent enzyme synthesizing the neurotransmitter gamma-aminobutyric acid (GABA), define how artemisinins

137 differentially methylated probes implicated gamma-aminobutyric acid (GABA), dopamine and serotonin n

138 the synthesis of neurotransmitters including gamma-aminobutyric acid (GABA), dopamine, and serotonin.

139 ors for various neurotransmitters-glutamate, gamma-aminobutyric acid (GABA), dopamine, serotonin, ace

140 re predicted to form anion channels gated by gamma-aminobutyric acid (GABA), glutamate, histamine, or

141 the major sleep-promoting neurotransmitter, gamma-aminobutyric acid (GABA), in the GABA shunt genera

142 pathway of the inhibitory neurotransmitter, gamma-aminobutyric acid (GABA), in this putative imbalan

143 The effects of postharvest treatments with gamma-aminobutyric acid (GABA), methyl jasmonate (MeJA)

144 Low levels of gamma-aminobutyric acid (GABA), one of two major neurotr

145 markers for the inhibitory neurotransmitter gamma-aminobutyric acid (GABA), particularly within the

146 Recently, it has been demonstrated that gamma-aminobutyric acid (GABA), the chief inhibitory neu

147 gamma-Aminobutyric acid (GABA), the major inhibitory neu

148 A considerable increase in gamma-aminobutyric acid (GABA), together with some other

149 fast-acting neurotransmitters glutamate and gamma-aminobutyric acid (GABA), two major neurotransmitt

150 sor of a potential lipophilic bioisostere of gamma-aminobutyric acid (GABA), various late-stage diver

151 The inhibitory neurotransmitter, gamma-aminobutyric acid (GABA), was then actively delive

152 brospinal fluid (CSF)-induced enhancement of gamma-aminobutyric acid (GABA)-A receptor activity was f

153 yl-d-aspartate (NMDA) glutamate receptor and gamma-aminobutyric acid (GABA)-A receptor during progres

154 ated with a positive allosteric modulator of gamma-aminobutyric acid (GABA)-A receptors in cerebrospi

155 how a unique role of GABARAPs, in particular gamma-aminobutyric acid (GABA)-A-receptor-associated pro

156 mission is likely due to a reduced number of gamma-aminobutyric acid (GABA)-ergic boutons, which may

157 ic interactions between parvalbumin-positive gamma-aminobutyric acid (GABA)-ergic interneurons and py

158 hat houses stress-sensitive, PVH-projecting, gamma-aminobutyric acid (GABA)-ergic neurons as represen

159 Dysfunction related to gamma-aminobutyric acid (GABA)-ergic neurotransmission i

160 This study describes the gamma-aminobutyric acid (GABA)-immunoreactive (GABA-ir)

161 Calretinin- and gamma-aminobutyric acid (GABA)-immunoreactive (IR) cells

162 yl-D-aspartate (NMDA) excitation balanced by gamma-Aminobutyric acid (GABA)-mediated inhibition and l

163 lation, salt reabsorption in the kidney, and gamma-aminobutyric acid (GABA)-mediated modulation in ne

164 lase 2 but not the parvalbumin subset of SNr gamma-aminobutyric acid (GABA)-releasing (GABAergic) neu

165 Here, we found that gamma-aminobutyric acid (GABA)-releasing neurons of the

166 At the circuit level, these gamma-aminobutyric acid (GABA)-releasing projections tar

167 and release the inhibitory neurotransmitter, gamma-aminobutyric acid (GABA).

168 roduction of the inhibitory neurotransmitter gamma-aminobutyric acid (GABA).

169 alterations in regulation of the HPA axis by gamma-aminobutyric acid (GABA).

170 ions due to significant, tonic inhibition by gamma-aminobutyric acid (GABA).

171 a rate-limiting enzyme in the production of gamma-aminobutyric acid (GABA).

172 t, with slightly different sensitivities, on gamma-aminobutyric acid (GABA)A , alpha-amino-3-hydroxy-

173 nized to involve functional deficits in both gamma-aminobutyric acid (GABA)ergic and glutamatergic sy

174 ide exchange factor selectively localized to gamma-aminobutyric acid (GABA)ergic and glycinergic post

175 Convergent findings indicate that cortical gamma-aminobutyric acid (GABA)ergic circuitry is altered

176 d orexinergic systems and is extended to the gamma-aminobutyric acid (GABA)ergic elements involved wi

177 d orexinergic systems and is extended to the gamma-aminobutyric acid (GABA)ergic elements of these nu

178 Finally, we revealed a role for the gamma-aminobutyric acid (GABA)ergic functions of LTSIs i

179 ontains glutamatergic excitatory neurons and gamma-aminobutyric acid (GABA)ergic inhibitory interneur

180 the superior colliculus receives inhibitory gamma-aminobutyric acid (GABA)ergic input from the basal

181 gamma-Aminobutyric acid (GABA)ergic inputs are strategic

182 dial tegmental nucleus (RMTg), which contain gamma-aminobutyric acid (GABA)ergic neurons that mediate

183 cell types (cholinergic, glutamatergic, and gamma-aminobutyric acid (GABA)ergic neurons) across its

184 for cognitive alterations and damage to the gamma-aminobutyric acid (GABA)ergic system in the hippoc

185 hout the retina, the ratio of glycinergic to gamma-aminobutyric acid (GABA)ergic to amacrine cells re

186 synaptic connectivity and function of a few gamma-aminobutyric acid (GABA)ergic wide-field amacrine

187 s present solely in neurons, specifically in gamma-aminobutyric acid (GABA)ergic, glutamatergic, and

188 ompared mPFC glutamate + glutamine (Glx) and gamma-aminobutyric acid (GABA+) concentrations in 64 wom

189 The gamma-aminobutyric acid (GABAA) receptor gamma2 subunit

190 associated with mutations in the inhibitory gamma-aminobutyric acid (GABAA) receptor gamma2 subunit

191 nanolone, a positive allosteric modulator of gamma-aminobutyric acid (GABAA) receptors, for the treat

192 izing synaptic inhibition mediated by type A gamma-aminobutyric acid (GABAA) receptors, which are Cl(

193 Ionotropic receptors of gamma-aminobutyric acid (GABAAR) regulate neuronal inhib

194 ntributing to the risk of each first IA, and gamma-aminobutyric acid (GABAs) associated with the firs

195 n with altered brain levels of glutamate and gamma-aminobutyric acid have been identified in both ani

196 The neuropil organization and the pattern of gamma-aminobutyric acid immunostaining of the medulla an

197 d somatostatin (SST), a marker of inhibitory gamma-aminobutyric acid interneurons that target pyramid

198 Gamma-aminobutyric acid is the principle inhibitory neur

199 ficant differences in prefrontal or striatal gamma-aminobutyric acid level.

200 he aims of this study were to investigate if gamma-aminobutyric acid levels (i) are abnormal in patie

201 Specifically for each unit decrease in gamma-aminobutyric acid levels (in mM), there was a pred

202 Patients had significantly lower gamma-aminobutyric acid levels in the hippocampus (adjus

203 glutamate + glutamine, N-acetylaspartate, or gamma-aminobutyric acid levels in the left DLPFC.

204 c resonance spectroscopy at 3 T, to quantify gamma-aminobutyric acid levels in the prefrontal cortex,

205 This study suggests that reduced gamma-aminobutyric acid levels reflect pathological abno

206 Abnormal glutamate and GABA (gamma-aminobutyric acid) levels have been found in the e

207 r abnormalities (e.g., serotonin, glutamate, gamma-aminobutyric acid) likely interact to facilitate i

208 king interneurons (FSIs), a strong source of gamma-aminobutyric acid-mediated synaptic inhibition in

209 train impulsive actions, most likely through gamma-aminobutyric acid-mediated synaptic inhibition of

210 ched pathways related to ethanol production, gamma-aminobutyric acid metabolism, and endotoxin biosyn

211 The gamma-aminobutyric acid modulator propofol induces neuro

212 Mice lacking GIRK2 in gamma-aminobutyric acid neurons (GAD-Cre:Girk2(flox/flox

213 rons and a specific population of inhibitory gamma-aminobutyric acid neurons (i.e., parvalbumin-conta

214 RK-dependent signaling in dorsal hippocampal gamma-aminobutyric acid neurons but no evident behaviora

215 It is principally composed of gamma-aminobutyric acid neurons.

216 y exciting and inhibiting neighbouring GABA (gamma-aminobutyric acid) neurons in the ventral tegmenta

217 s in the pre- and postsynaptic components of gamma-aminobutyric acid neurotransmission and in the den

218 s study supports the idea that modulation of gamma-aminobutyric acid neurotransmission may be an impo

219 lation is in part mediated by specific GABA (gamma-aminobutyric acid)-producing neurons in the bed nu

220 Here, we asked whether mesoaccumbal GABA (gamma-aminobutyric acid) projections enhance adaptive re

221 oxication causes changes in the rodent brain gamma-aminobutyric acid receptor (GABAAR) subunit compos

222 Mechanisms controlling the metabotropic gamma-aminobutyric acid receptor (GABAB) cell surface st

223 ange the type of excitability: a depolarized gamma-Aminobutyric acid receptor (GABAR) reversal potent

224 ethyl-d-aspartate receptor in 4 patients and gamma-aminobutyric acid receptor A in 1 patient of 111 p

225 peptide (AgRP) neurons because inhibition of gamma-aminobutyric acid receptor in the ARC did not prev

226 s, and behavioral results suggest that local gamma-aminobutyric acid receptor signaling mediates the

227 g amylin's effects on energy balance through gamma-aminobutyric acid receptor signaling.

228 of reduced lysosomal degradation of RhoB in Gamma-aminobutyric acid receptor-associated protein (GAB

229 le-associated protein 1 light chain 3 (LC3), gamma-aminobutyric acid receptor-associated protein (GAB

230 The gamma-aminobutyric acid receptor-associated protein (GAB

231 onal cell-surface antibodies, mainly against gamma-aminobutyric acid receptors (53% vs 11%; P < .001)

232 Metabotropic gamma-aminobutyric acid receptors (GABA(B)) are involved

233 cently become appreciated that activation of gamma-aminobutyric acid receptors (GABA-Rs) on ss-cells

234 The accumulation of gamma-aminobutyric acid receptors (GABAARs) at the appro

235 also includes the metabotropic glutamate and gamma-aminobutyric acid receptors.

236 cient synaptic inhibition upon activation of gamma-aminobutyric acid receptors.

237 tor (CB1R); 3) binds to CB1R, which inhibits gamma-aminobutyric acid release from the cholecystokinin

238 in mouse brain, prevented ethanol-stimulated gamma-aminobutyric acid release in the central amygdala,

239 Elevated gamma-aminobutyric acid release under chronic stress is

240 arachidonoylglycerol-dependent inhibition of gamma-aminobutyric acid release without altering postsyn

241 ts that chronic stress increases presynaptic gamma-aminobutyric acid release.

242 ting cell types include hypocretin and GABA (gamma-aminobutyric-acid)-releasing neurons of the latera

243 inly result from the synchronous activity of gamma-aminobutyric acid-releasing cells.

244 events including the migration of GABAergic (gamma-aminobutyric-acid-releasing) neurons from ventral

245 GABAergic (gamma-aminobutyric-acid-releasing) oviposition inhibitor

246 Here we show that a GABAergic (gamma-aminobutyric-acid-releasing) pathway originating f

247 Moreover, up-regulation of the gamma-aminobutyric acid shunt and alanine metabolism exp

248 succinate-semialdehyde dehydrogenase in the gamma-aminobutyric acid shunt pathway and an aconitase f

249 e, fermentation, alanine metabolism, and the gamma-aminobutyric acid shunt, while [(13)C]glutamate an

250 ce can be temporarily reverted by inhibiting gamma-aminobutyric acid signaling but not by a Ras-ERK b

251 ature links impulsive behavior in rodents to gamma-aminobutyric acid signaling in the NAc.

252 This results in delayed gamma-aminobutyric acid switch and higher susceptibility

253 mental process, the excitatory-to-inhibitory gamma-aminobutyric acid switch; defects in this switch h

254 Immunohistochemistry for the gamma aminobutyric acid synthesizing enzyme glutamic aci

255 Current techniques for monitoring GABA (gamma-aminobutyric acid), the primary inhibitory neurotr

256 o have strong antagonism toward receptors of gamma-aminobutyric acid, the major inhibitory transmitte

257 The binding of GABA (gamma-aminobutyric acid) to extrasynaptic GABA(A) recept

258 uits treated with melatonin exhibited higher gamma-aminobutyric acid transaminase (GABA-T) enzyme act

259 elevates the CG7433 protein, a mitochondrial gamma-aminobutyric acid transaminase (GABAT), and reduce

260 ansmission or pharmacological enhancement of gamma-aminobutyric acid transmission.

261 : inhibitory synaptic currents and vesicular gamma-aminobutyric acid transporter (vGAT) staining inte

262 ntal disorder genes, SLC6A1, which encodes a gamma-aminobutyric acid transporter, was associated with

263 olarising synaptic inhibition through GABAA (gamma aminobutyric acid type A) and glycine receptors de

264 variants in the gene GABRB2, coding for the gamma-aminobutyric acid type A (GABA(A) ) receptor subun

265 Gabazine, a gamma-aminobutyric acid type A (GABA(A)) receptor antago

266 ze and predict peak current responses of the gamma-aminobutyric acid type A (GABA(A)) receptor.

267 1, beta3, and gamma2 subunits comprising the gamma-aminobutyric acid type A (GABA(A)) receptor.

268 ine drugs act through positive modulation of gamma-aminobutyric acid type A (GABA(A)) receptors to da

269 potential firing by local application of the gamma-aminobutyric acid type A (GABA-A) agonist muscimol

270 tly identified four de novo mutations in the gamma-aminobutyric acid type A (GABAA ) receptor beta3 s

271 Proteostasis maintenance of gamma-aminobutyric acid type A (GABAA) receptors dictate

272 OEt) displayed micromolar affinity for brain gamma-aminobutyric acid type A (GABAA) receptors.

273 The extrasynaptic alpha-5 gamma-aminobutyric acid type A receptor (alpha5-GABAAR)

274 ctive steroids are efficacious modulators of gamma-aminobutyric acid type A receptor (GABA(A)) recept

275 hat is highly selective for the slow type of gamma-aminobutyric acid type A receptor (GABA(A)R) could

276 The gamma-aminobutyric acid type A receptor (GABAA-R) is a m

277 -RICS-deficient neurons show reduced surface gamma-aminobutyric acid type A receptor (GABAAR) levels

278 drugs that inhibit alpha5 subunit-containing gamma-aminobutyric acid type A receptor function improve

279 cotransporter expression and impaired spinal gamma-aminobutyric acid type A receptor function, indica

280 However, DCS-LTD persists despite either gamma-aminobutyric acid type A receptor or N-methyl-D-as

281 , including GABRB3, GABRA5, GABRG3, encoding gamma-aminobutyric acid type A receptor subunits (beta3,

282 Alpha-5 gamma-aminobutyric acid type A receptors (alpha5-GABAARs

283 Subunit-selective modulation of gamma-aminobutyric acid type A receptors (GABA(A)R) is c

284 The function and pharmacology of gamma-aminobutyric acid type A receptors (GABA(A)Rs) are

285 eurosteroids (NS) are the main modulators of gamma-aminobutyric acid type A receptors (GABA(A)Rs), wh

286 cotinic acetylcholine receptors (nAChRs) and gamma-aminobutyric acid type A receptors (GABAARs) are m

287 ed that act as convulsants and inhibitors of gamma-aminobutyric acid type A receptors (GABAARs) rathe

288 Extrasynaptic gamma-aminobutyric acid type A receptors (GABAARs),which

289 sults suggest that alpha5 subunit-containing gamma-aminobutyric acid type A receptors are novel targe

290 g of the scaffolding protein Gephyrin and of gamma-aminobutyric acid type A receptors at inhibitory n

291 ogic inhibition of alpha5 subunit-containing gamma-aminobutyric acid type A receptors attenuated cogn

292 ther inhibition of alpha5 subunit-containing gamma-aminobutyric acid type A receptors attenuated defi

293 ed that inhibiting alpha5 subunit-containing gamma-aminobutyric acid type A receptors would improve c

294 t is selective for alpha5 subunit-containing gamma-aminobutyric acid type A receptors, 30 minutes bef

295 excess function of alpha5 subunit-containing gamma-aminobutyric acid type A receptors.

296 pileptic animals, supporting the notion that gamma-aminobutyric acid type A signaling can also initia

297 sustained neuronal inhibition is mediated by gamma-aminobutyric acid type B (GABAB) receptors, which

298 Ethanol, like rapid antidepressants, alters gamma-aminobutyric acid type B receptor (GABABR) express

299 tly bound the sushi 1 domain specific to the gamma-aminobutyric acid type B receptor subunit 1a (GABA

300 The GABA(B) (gamma-aminobutyric acid type B) receptor is one of the p