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1 ate activated AKT as a tumorigenic driver in ganglioneuroma.
2 al nerve sheath tumor (PNST) that arose in a ganglioneuroma.
3 ude neuroblastoma, ganglioneuroblastoma, and ganglioneuroma.
4 tomas, musculoskeletal anomalies and mucosal ganglioneuromas.
5 ing from immature malignant tumors to benign ganglioneuromas.
6 distal colonic aganglionosis and intestinal ganglioneuromas.
7 he disease course and clinical management of ganglioneuromas.
9 onal cell vitronectin was detected in 7 of 9 ganglioneuromas, 5 of 8 peripheral ganglia, and 14 of 21
11 ies that could reduce the size and extent of ganglioneuroma and therefore limit surgical morbidity.
17 f mTOR inhibitors as a means to shrink large ganglioneuromas before resection in order to reduce surg
18 retroperitoneal, intra-abdominal, and pelvic ganglioneuromas between January 1, 2000, and January 1,
20 Homozygotes did not develop gastrointestinal ganglioneuromas, but displayed ganglioneuromas of the ad
21 patients with malignant PNST that arose in a ganglioneuroma, contrast-enhanced CT showed a large, mar
22 wnstream AKT target, mTOR, in zebrafish with ganglioneuroma effectively reduced the tumor burden.
23 ected identical band shifts in the leukemia, ganglioneuroma, ERMS, and normal tissues, consistent wit
26 al tumors, histologically identical to human ganglioneuromas, in their sympathetic nervous systems an
28 trointestinal ganglioneuromas, but displayed ganglioneuromas of the adrenal medulla, enlargement of t
29 and adrenal chromaffin cells, together with ganglioneuromas of the gastrointestinal tract and other
33 nine patients with ganglioneuroma, two with ganglioneuroma-pheochromocytoma, and two with malignant
40 ed of imaging findings in nine patients with ganglioneuroma, two with ganglioneuroma-pheochromocytoma
41 Surgery is the only effective therapy for ganglioneuroma, which may be challenging due to the loca
42 athetic nervous system were found to develop ganglioneuroma without progression to neuroblastoma.