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1 patient presenting with aggressive pyoderma gangrenosum.
2 confirmed the clinical suspicion of pyoderma gangrenosum.
3 included pathergy, cystic acne, and pyoderma gangrenosum.
4 e discussed regarding psoriasis and pyoderma gangrenosum.
5 cers with a clinical resemblance to pyoderma gangrenosum.
6 and pretibial lesions diagnosed as pyoderma gangrenosum.
7 itides such as Sweet's syndrome and pyoderma gangrenosum.
9 ing of a disease locus for familial pyoderma gangrenosum-acne-arthritis to the long arm of chromosome
13 mmatory disease; pyogenic arthritis pyoderma gangrenosum and acne; Muckle-Wells syndrome; familial co
14 ents with Sweet's syndrome (SW) and pyoderma gangrenosum and found numerous novel splice variants in
16 s of PSTPIP1 in the pathogenesis of pyoderma gangrenosum and suggest that the cytoskeleton is a ratio
17 case of CB, initially diagnosed as pyoderma gangrenosum and treated with steroids, leading to dissem
20 the syndrome of pyogenic arthritis, pyoderma gangrenosum, and acne (PAPA), a dominantly inherited aut
24 ndrome (pyogenic sterile arthritis, pyoderma gangrenosum, and acne, OMIM #604416) and familial recurr
27 aneous spread, clinically mimicking pyoderma gangrenosum, and subsequently progressed to disseminated
29 associated disease and suggest that pyoderma gangrenosum can be classified as a dynamical disease at
30 exclusion, and the misdiagnosis of pyoderma gangrenosum can result in substantial complications in p
32 er entities, CB may be mistaken for pyoderma gangrenosum due to overlap of findings on histopathologi
33 ts studied, 64 had been treated for pyoderma gangrenosum for a median of 10 months (range, 3 to 180).
35 enosum (PPG), an unusual variant of pyoderma gangrenosum, has been reported almost exclusively in pat
36 Here we report the first case of pyoderma gangrenosum in a patient with refractory celiac disease.
37 in all patients suspected of having pyoderma gangrenosum in order to rule out alternative diagnoses.
42 cers in the 64 patients treated for pyoderma gangrenosum, it was clear that those in 23 patients (36
44 d in all other patients with either pyoderma gangrenosum or SW, it was always associated with splice
55 ot respond to treatment directed at pyoderma gangrenosum, those in 8 (12 percent) were exacerbated by
57 of 240 patients with a diagnosis of pyoderma gangrenosum who were evaluated at our institution from 1
58 y occurring, extra-hepatic onset of pyoderma gangrenosum, with the AIH in remission, strengthening th