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1 udimentary pattern of inspiratory movements (gasping).
2 as changes in sensory feedback, sighing, and gasping.
3 d that this burster discharge could underlie gasping.
4 44 of 113 (39%) of all arrested patients had gasping.
5 t 5-HT2A receptor activation is critical for gasping.
6 eas only one burst mechanism is critical for gasping.
7 markedly from eupnoea but are identical with gasping.
8 nerve evoked by airway closure that leads to gasping.
9 t-care reports to determine the incidence of gasping after arrest in relation to the various EMS arri
10            The incidence and significance of gasping after cardiac arrest in humans are controversial
11                      The association between gasping and 1-year survival with favorable neurological
12  strong cardiorespiratory response including gasping and a pronounced bradycardia; however, the mecha
13            Here, we demonstrate that fictive gasping and CI-pacemaker bursting were selectively elimi
14 ude that PGE2 differentially modulates sigh, gasping and eupnoeic activity by differentially increasi
15 eased breathing frequency, often followed by gasping and fatal respiratory failure.
16 ) of 1-year survival with CPC </=2 versus no gasping and no shockable rhythm.
17                   The frequency of sighs and gasping are stimulated at low concentrations, while the
18                The stroke volume produced by gasping averaged 23 +/- 6 mL, which represented approxim
19                                              Gasping combined with a shockable initial recorded rhyth
20 demonstrated that the presence or absence of gasping correlated with EMS arrival time.
21     During hypoxia, bradycardia, and hypoxic gasping develop after a brief period of hyperactivity.
22 95% confidence interval [CI]: 0.43 to 0.76), gasping during CPR (OR: 3.94; 95% CI: 2.09 to 7.44), sho
23                                              Gasping during CPR was independently associated with inc
24 dy sought to assess the relationship between gasping during out-of-hospital cardiac arrest and 1-year
25         Our studies confirm that preterminal gasping during ventricular fibrillation increases both v
26                  This progresses to gagging, gasping, eye bulging, bradycardia, cyanosis, and vomitin
27                              Remarkably, the gasping face (traditionally considered a display of fear
28 to others on the basis of facial behavior: A gasping face can be seen as showing "fear" and intent to
29 s threatening; Trobrianders chose the "fear" gasping face whereas Spaniards chose an "angry" scowling
30 challenge the Western assumption that "fear" gasping faces uniformly express fear or signal submissio
31 lood" of the US administrative state, is now gasping for air.
32 inely recognize, monitor, and record data on gasping in all future cardiac arrest trials and registri
33 ating NEBs or vagal PVALB neurons eliminated gasping in response to airway closure.
34 imulating NEBs or vagal PVALB neurons evoked gasping in the absence of airway threats, whereas ablati
35 rize the ventilatory patterns of eupnoea and gasping in the neonatal rat.
36                                              Gasping is a natural reflex that enhances oxygenation an
37                                              Gasping is associated with increased survival.
38 ate repression, which correlated to enhanced gasping latency and impaired autoresuscitation during an
39 authors prospectively collected incidence of gasping on all evaluable subjects in a multicenter, rand
40                                              Gasping or abnormal breathing is common after cardiac ar
41 for individuals with and without spontaneous gasping or agonal respiration during CPR, respectively.
42                                    Nocturnal gasping or choking is the most reliable indicator of obs
43 esuscitation, after phrenic nerve-stimulated gasping, or during spontaneous ventilation, the impedanc
44 ed pups was associated with delayed onset of gasping (P < 0.001), delayed recovery of HR from hypoxic
45 that eventually died took longer to initiate gasping (p = 0.0001), recover heart rate (p = 0.0001), a
46 not terminating resuscitation prematurely in gasping patients and the need to routinely recognize, mo
47  Treatment with 5,7-DHT affected neither the gasping pattern once initiated, nor HR, V(E)/V(O(2)) or
48 in both groups, with no effect of DNE on the gasping pattern.
49 ivity then ceased, but later returned with a gasping pattern.
50 e day of birth, rats can exhibit eupnoea and gasping patterns which are very similar to those of adul
51 , AR occurs if oxygen is restored during the gasping period where an initial heart rate increase is r
52 the central respiratory oscillator, in which gasping relies on persistent sodium current, whereas eup
53 pathways in regions of the brain involved in gasping, response to hypoxia, and arousal.
54 ggest that the recognition and importance of gasping should be taught to bystanders and emergency med
55 nter text files to determine the presence of gasping soon after collapse.
56 ructive sleep apnea was nocturnal choking or gasping (summary likelihood ratio [LR], 3.3; 95% CI, 2.1
57 equires minimal control effort to complete a gasping task.
58                During severe hypoxia-induced gasping, the low-dimensional dynamics of the VRC reconfi
59                                              Gasping was characterized by a sudden onset of phrenic a
60                    As heat stress continued, gasping was evoked in both groups, with no effect of DNE
61                     Upon exposure to anoxia, gasping was observed, even in animals in which phrenic a
62                                              Gasping was present in 39 of 119 patients (33%) who arre
63  agonal respiratory movements in the form of gasping were observed in 6 of 8 pigs in the nonclamp gro
64 ity is supplanted by the neural correlate of gasping, which is proposed to depend only on CI-pacemake
65 , and eupneic activity was reconfigured into gasping, which like eupnea was insensitive to 4 microM c