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1 d in lymphoepithelial lesions, a hallmark of gastric MALT lymphoma.
2 [p-ERK]) is associated with HP dependence of gastric MALT lymphoma.
3 ith the clinical management of patients with gastric MALT lymphoma.
4 n Helicobacter pylori-positive gastritis and gastric MALT lymphoma.
5 he splenic marginal zone in a single case of gastric MALT lymphoma.
7 ocation t(11;18)(q21,q21) is found in 30% of gastric MALT lymphomas and is associated with a failure
9 of the depth of infiltration of the wall by gastric MALT lymphoma as measured by endoscopic ultrasou
10 8Ga]Pentixafor-PET for detection of residual gastric MALT lymphoma at follow-up were 97.0%, 95.0%, 10
11 ential involvement of the marginal zone when gastric MALT lymphomas disseminate to the spleen, which
15 confirm a higher than expected incidence of gastric MALT lymphoma in immunosuppressed transplant rec
19 ications have furthered the understanding of gastric MALT lymphoma pathogenesis, clinical behavior, a
20 c resonance imaging (MRI) examinations of 26 gastric MALT lymphoma patients, and 20 [68Ga]Pentixafor-
22 consensus for the treatment of patients with gastric MALT lymphoma requiring further treatment beyond
25 es, and medical records of six patients with gastric MALT lymphoma were retrospectively reviewed.
27 our consecutive patients with stage I to IIE gastric MALT lymphoma who obtained a pathologic remissio
28 opsied at endoscopy demonstrated early-stage gastric MALT lymphoma with associated Helicobacter pylor