ライフサイエンスコーパス: gastric acid secretion

1 t (CWR) and also inhibits gastrin-stimulated gastric acid secretion.

2 astric parietal cell that is responsible for gastric acid secretion.

3 tric environment may be its ability to alter gastric acid secretion.

4 d following a rise in gastric pH, leading to gastric acid secretion.

5 limiting food-stimulated gastrin release and gastric acid secretion.

6 axis, a neurotransmitter, and a regulator of gastric acid secretion.

7 e, hormonal, and intracellular regulation of gastric acid secretion.

8 e, hormonal, and intracellular regulation of gastric acid secretion.

9 ameters and mechanism of P-CAB inhibition of gastric acid secretion.

10 estigate the requirement for this protein in gastric acid secretion.

11 e2 channel facilitates its essential role in gastric acid secretion.

12 mulation-associated membrane recruitment and gastric acid secretion.

13 Gastrin is a key regulator of gastric acid secretion.

14 e regarding the regulation and assessment of gastric acid secretion.

15 een performed on colonic fluid transport and gastric acid secretion.

16 gulatory role for the CFTR protein in normal gastric acid secretion.

17 onal, and paracrine pathways finely regulate gastric acid secretion.

18 g a gene-dose effect and a primary defect in gastric acid secretion.

19 n made in understanding of the regulation of gastric acid secretion.

20 sight into the complex regulation of in vivo gastric acid secretion.

21 ponses, inflammation, neurotransmission, and gastric acid secretion.

22 ation of proliferation and downregulation of gastric acid secretion.

23 ward an understanding of the cell biology of gastric acid secretion.

24 lead to new insights into the regulation of gastric acid secretion.

25 ased numbers of parietal cells and decreased gastric acid secretion.

26 n kinase Akt, we explored the role of Akt in gastric acid secretion.

27 100-23 contribute to tolerance towards host gastric acid secretion.

28 lary VLRF area in the sympathetic control of gastric acid secretion.

29 mmatory cytokine and a powerful inhibitor of gastric acid secretion.

30 r to play a role in inhibition of stimulated gastric acid secretion.

31 Slc26a9 deletion resulted in the loss of gastric acid secretion and a moderate reduction in the n

32 ers, paracrine agents, and hormones regulate gastric acid secretion and are themselves regulated.

33 Morphine treatment also increases gastric acid secretion and causes delays in gastric empt

34 tion of acid in the stomach due to increased gastric acid secretion and delayed gastric emptying.

35 esults demonstrate that AE2 is essential for gastric acid secretion and for normal development of sec

36 Epidermal growth factor (EGF) stimulates gastric acid secretion and H(+)/K(+)-ATPase alpha-subuni

37 In vivo, gastric acid secretion and in vitro histamine release fr

38 w emphasizes the importance and relevance of gastric acid secretion and its regulation in health and

39 mpairments in basal and histamine-stimulated gastric acid secretion and markedly reduced levels of th

40 disease.Proton pump inhibitors (PPIs) reduce gastric acid secretion and modulate gut microbiota compo

41 the vagus (DMV) has been shown to stimulate gastric acid secretion and motility, respectively, via v

42 ons into the DMV may mediate the increase in gastric acid secretion and motor activity associated wit

43 rin receptor is one of several regulators of gastric acid secretion and mucosal growth.

44 t analysis of its role during organogenesis, gastric acid secretion and neoplastic transformation.

45 biological processes including inflammation, gastric acid secretion and neuromodulation.

46 limited to allergic reactions, wakefulness, gastric acid secretion and neurotransmission.

47 potassium channel beta subunit gene ablates gastric acid secretion and predisposes to gastric neopla

48 ine alone showed partial suppression of both gastric acid secretion and progression to neoplasia.

49 g cellular restitution as well as inhibiting gastric acid secretion and reactive oxygen species (ROS)

50 ent strategies including improved control of gastric acid secretion and role for surgery, and new app

51 lly linked to epithelial processes including gastric acid secretion and thyroid hormone biosynthesis.

52 Recent milestones in the understanding of gastric acid secretion and treatment of acid-peptic diso

53 ximal tubular and intestinal Na+ absorption, gastric acid secretion, and cAMP-induced jejunal Cl- sec

54 siological vagal control of gastrin release, gastric acid secretion, and gastric motility.

55 wed mild hypergastrinemia, increased maximal gastric acid secretion, and increased parietal cell numb

56 prove our understanding of the regulation of gastric acid secretion at the central, peripheral, and i

57 buting to understanding of the regulation of gastric acid secretion at the central, peripheral, and i

58 ocin, which acts within the DMV to stimulate gastric acid secretion, but inhibits gastric motor funct

59 ovides direct evidence for the regulation of gastric acid secretion by a TRP channel; TRPML1 is an im

60 Endotoxemia from LPS inhibits gastric acid secretion by an unknown mechanism.

61 e hypothesized that LPS causes inhibition of gastric acid secretion by down-regulating the H/K-ATPase

62 These data suggest that inhibition of gastric acid secretion by LPS is due to inhibition of H/

63 contributes to galanin-induced inhibition of gastric acid secretion by means of the suppression of en

64 Gastric acid secretion by parietal cells is precisely re

65 Gastric acid secretion by parietal cells requires traffi

66 Inhibition of gastric acid secretion by proton pump inhibitors like om

67 ass of protein may account for inhibition of gastric acid secretion by PYY released from the small in

68 heir ability of immune defense responses and gastric acid secretion, consistent with their ability to

69 Our understanding of the regulation of gastric acid secretion continues to advance.

70 es central neurotensin-induced inhibition of gastric acid secretion does not appear to be the high-af

71 Stimulation of gastric acid secretion during P. yoelii infection restor

72 RECENT FINDINGS: Gastric acid secretion facilitates the digestion of prot

73 Caffeine, generally known as a stimulant of gastric acid secretion (GAS), is a bitter-tasting compou

74 exchanger, is important for normal levels of gastric acid secretion, gastric epithelial cell differen

75 Different aspects of regulation of gastric acid secretion have been focused either in terms

76 ed vascular permeability, and stimulation of gastric acid secretion, histamine plays important roles

77 detectable tissue histamine levels, impaired gastric acid secretion, impaired passive cutaneous anaph

78 nd 2c (L-736,380) dose-dependently inhibited gastric acid secretion in anesthetized rats (ID(50), 0.0

79 edge, the understanding of the regulation of gastric acid secretion in health and disease is far from

80 es in our understanding of the regulation of gastric acid secretion in health and disease, as well as

81 ss in our understanding of the regulation of gastric acid secretion in health and disease.

82 d distal renal tubular acidosis or decreased gastric acid secretion in humans.

83 ociated with any significant change in basal gastric acid secretion in monkeys and occurred despite a

84 Helicobacter pylori infection increases gastric acid secretion in patients with duodenal ulcers

85 esis that inhibition of gastric emptying and gastric acid secretion in response to dietary lipid is d

86 Secretagogues that stimulate gastric acid secretion induce Shh gene expression throug

87 The regulation of gastric acid secretion is achieved in the periphery by i

88 Gastric acid secretion is mediated by the H/K-ATPase of

89 fluence of central and peripheral stimuli on gastric acid secretion is mediated via activation of his

90 Gastric acid secretion is regulated by biologic agents p

91 Gastric acid secretion is tightly regulated by overlappi

92 histamine H3 receptors in the regulation of gastric acid secretion is unclear.

93 ar target for peptide YY (PYY) inhibition of gastric acid secretion is unknown.

94 h advancing age has no independent effect on gastric acid secretion, it is associated with reduced pe

95 ng of the pathways and mechanisms regulating gastric acid secretion may lead to the development of ne

96 Gastric acid secretion must be precisely controlled at a

97 ical processes including allergic reactions, gastric acid secretion, neurotransmitter release, and in

98 onse to counteract infection-related damage, gastric acid secretion or gastrointestinal motility for

99 Gastric acid secretion plays a pivotal role in the physi

100 more potent and longer-lasting inhibition of gastric acid secretion provided by proton pump inhibitor

101 ished the stimulatory effect of histamine on gastric acid secretion, providing evidence for the regul

102 Global Kcne2 deletion, which inhibits gastric acid secretion, reduced the relative abundance o

103 ists for the treatment of allergy and excess gastric acid secretion, respectively.

104 After excision of the tumor, WDS ceased and gastric acid secretion returned.

105 oltage-dependent K(+) channel that regulates gastric acid secretion, salt and glucose homeostasis, an

106 ng of the pathways and mechanisms regulating gastric acid secretion should lead to improved managemen

107 ng of the pathways and mechanisms regulating gastric acid secretion should lead to the development of

108 ed into the cisterna magna potently inhibits gastric acid secretion stimulated by intravenous infusio

109 licobacter pylori interaction with the human gastric acid secretion system.

110 minant gastritis and profound suppression of gastric acid secretion that is partially reversible with

111 data demonstrate that KCNE2 is essential for gastric acid secretion, the first genetic evidence that

112 agents have been implicated as regulators of gastric acid secretion, their site and mechanism of acti

113 Reduction of gastric acid secretion therefore appears to promote over

114 nal role of Akt appears to be stimulation of gastric acid secretion through induction of H(+)/K(+)-AT

115 e whereby endogenous somatostatin suppresses gastric acid secretion through inhibition of gastrin act

116 the VLRF to inhibit pentagastrin-stimulated gastric acid secretion through spinal pathways, suggesti

117 re, we identified a novel pathway modulating gastric acid secretion through the stomach calcium-sensi

118 actor (EGF) inhibits secretagogue-stimulated gastric acid secretion via an EGF receptor located on pa

119 pose that Slc26a9 plays an essential role in gastric acid secretion via effects on the viability of t

120 ction of this amidated peptide in regulating gastric acid secretion via the CCK2 receptor is now well

121 naesthetized A-IV(+/+) mice, meal-stimulated gastric acid secretion was 59% inhibited by intestinal l

122 Decreased gastric acid secretion was associated with cholera but n

123 Gastric acid secretion was blocked and stimulated by ML1

124 A rat model to study gastric acid secretion was created.

125 enervated rats with a gastric fistula, basal gastric acid secretion was depressed 3-fold, and plasma

126 When gastric acid secretion was measured after maximal stimul

127 art of an intravenous pentagastrin infusion; gastric acid secretion was monitored every 10 min for 20

128 la oblongata where bombesin acts to suppress gastric acid secretion were investigated in urethane-ane

129 mmunoreactivity, and pentagastrin-stimulated gastric acid secretion, were similar in both infected an

130 n in the somatostatin pathway have increased gastric acid secretion, which confirms an important nega

131 rticularly the gastrin (G) cell, co-ordinate gastric acid secretion with the arrival of food in the s