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1 c states that have an increased incidence of gastric carcinoids.
2  was to determine whether SRS could localize gastric carcinoids.
3  the upper abdomen on SRS may be caused by a gastric carcinoid and not a pancreatic endocrine tumor.
4 ciencies (including B12 and hypomagnesemia), gastric carcinoid and rebound hyperacidity are biologica
5                       Sixteen patients had a gastric carcinoid, and 12 of these patients had SRS loca
6  the connection between hypergastrinemia and gastric carcinoids, and most importantly, improving upon
7                             The incidence of gastric carcinoid appears to be increasing without a cor
8                                     Multiple gastric carcinoids are a well-recognized complication of
9                                     However, gastric carcinoids are difficult to diagnose.
10                                              Gastric carcinoids are of increasing clinical concern be
11                                              Gastric carcinoids evolved from enterochromaffin-like (E
12 mys species exhibits a genetic propensity to gastric carcinoid formation that can be accelerated by a
13 three patients who underwent splenectomy for gastric carcinoid, gastric adenocarcinoma and cancer of
14                      Individuals with type I gastric carcinoids >2 cm should undergo cross-sectional
15              BEST PRACTICE ADVICE 13: Type I gastric carcinoids in individuals with atrophic gastriti
16          Endoscopists may consider resecting gastric carcinoids <1 cm and should endoscopically resec
17 neoplasia type 1 (MEN-1) gene was studied in gastric carcinoids of patients with MEN-1 and chronic at
18                      Individuals with type I gastric carcinoids should undergo surveillance, but the
19 ypersecretion, acute interstitial nephritis, gastric carcinoid tumor, cardiovascular risk with clopid
20                                              Gastric carcinoid tumors are rare lesions but have been
21 s the understanding of the biologic basis of gastric carcinoid tumors increases, the treatment will l
22 nd clinical arenas, the optimal treatment of gastric carcinoid tumors is still a matter of debate.
23                                              Gastric carcinoid tumors occurred as a consequence of ch
24 ed with an increased risk of gastric polyps, gastric carcinoid tumors, and possibly adenocarcinomas.
25 cusses the current biologic understanding of gastric carcinoid tumors, including the role of hypergas
26 omaffin-like cells and to the development of gastric carcinoid tumors.
27 ne expression is useful in the assessment of gastric carcinoid tumors.
28 hronic atrophic gastritis and multiple large gastric carcinoid tumors.
29                                              Gastric carcinoids (types I and II) involve the transfor
30       The sensitivity of SRS in localizing a gastric carcinoid was 75%, with a specificity of 95%.
31 ing a different increased risk of developing gastric carcinoids, were studied.
32 asive method that can identify patients with gastric carcinoids with a reasonable sensitivity and a h