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1 ation, noncardia gastric adenocarcinoma, and gastric lymphoma.
2 cinoma of the distal esophagus and 1 case of gastric lymphoma.
3 s peptic ulcers, gastric adenocarcinoma, and gastric lymphoma.
4 treated before the development of high-grade gastric lymphoma.
5 peptic ulcer disease, gastric carcinoma, and gastric lymphoma.
6 pattern of Ig light chain restriction as the gastric lymphoma.
7 ation, noncardia gastric adenocarcinoma, and gastric lymphoma.
8 remains the best frontline therapy for early gastric lymphoma.
9 peptic ulcer disease, gastric carcinoma, and gastric lymphoma.
10  for pathologically staged IE or IIE-1 (pN1) gastric lymphoma.
11 gastrointestinal stromal tumors, and primary gastric lymphoma.
12 ceration, distal gastric adenocarcinoma, and gastric lymphoma.
13 ous group of patients with stage IE or IIE-1 gastric lymphoma after complete surgical resection.
14 mpt to establish clonal identity between the gastric lymphoma and cells in the splenic marginal zone.
15 ed risk of noncardia gastric adenocarcinoma, gastric lymphoma, and peptic ulcer.
16                                Nearly 60% of gastric lymphomas are high-grade lesions with or without
17                                 About 40% of gastric lymphomas are low-grade, and nearly all these lo
18                                      Primary gastric lymphomas are non-Hodgkin lymphomas that origina
19       The proportions of PUD, dyspepsia, and gastric lymphoma attributable to H pylori (95% confidenc
20                             The treatment of gastric lymphoma continues to evolve, and surgical resec
21                                              Gastric lymphomas demonstrated characteristic lymphoepit
22 lative risk of developing PUD, dyspepsia, or gastric lymphomas due to H pylori to calculate the popul
23 ecords of all patients with the diagnosis of gastric lymphoma from 1980 to 1991 were reviewed retrosp
24                                          All gastric lymphomas had an abnormal EUS: eight with discre
25  Enthusiasm for multimodality approaches for gastric lymphoma has lead to the current trend of using
26 he splenic marginal zone infiltrates and the gastric lymphoma in the histologically involved cases.
27                                  Surgery for gastric lymphoma is now often reserved for patients with
28 s peptic ulcer disease (PUD), dyspepsia, and gastric lymphomas, is often overlooked in guidelines and
29 tes varied with lowest PAFs (PUD, dyspepsia, gastric lymphomas) observed in the United States (35% [2
30                                              Gastric lymphoma of mucosa-associated lymphoid tissue (M
31 early illustrated by developments concerning gastric lymphomas of mucosa-associated lymphoid tissue (
32 ession in approximately 50% of patients with gastric lymphomas of mucosa-associated lymphoid tissue,
33 be the most common site on lymphoma, primary gastric lymphoma (PGL).
34                            The management of gastric lymphoma remains controversial.
35                                              Gastric lymphoma resembling gastric mucosa-associated ly
36                                  Non-Hodgkin gastric lymphoma should be recognized as another cause o
37          All nine patients (six with primary gastric lymphoma, three with generalized lymphoma with s
38 with ante- or postmortem evidence of primary gastric lymphoma were described.
39 osis, and treatment of patients with primary gastric lymphoma, with special attention to the changing