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1 e released only at pH 4 or 5 (typical infant gastric pH).
2 he others because the caseins clotted at the gastric pH.
3  potentially useful therapeutics to regulate gastric pH.
4      PEP 1:2 ratio inversely correlates with gastric pH.
5 onstrate equivalency and ability to increase gastric pH.
6 estion of a meal and maintain homeostasis of gastric pH.
7 pH paper are not interchangeable measures of gastric pH.
8 -secreting parietal cells and an increase in gastric pH.
9                 Neither rHSP nor ITF altered gastric pH.
10         P. yoelii-infected mice had elevated gastric pH.
11 fs by an in vitro digestion model at varying gastric pHs.
12  vitamin B(6) in baby foods is lower in both gastric pHs.
13 foodstuffs (declared vitamin C fortified) at gastric pH 1.5 and 4, respectively.
14  from 1.3 to 53.8%, and from 0.3 to 26.3% at gastric pH 1.5 and 4, respectively.
15 accessibility of the PL, PN, and PM forms in gastric pH 1.5 were 53%, 76%, and 50%, respectively.
16 ils, H. pylori-infected gerbils had a higher gastric pH, a higher incidence of gastric ulcers, and a
17    24-h median [IQ] percentages of time with gastric pH above 3 and 4 were higher with rabeprazole th
18  a basic center imparting high solubility at gastric pH, addressing the dissolution limitation observ
19                                              Gastric pH affected the composition of the aspirate micr
20 fter omeprazole was first administered, mean gastric pH after omeprazole was started, and the lowest
21 omeprazole mean gastric pH was 7.1, the mean gastric pH after starting omeprazole was 6.8, and the me
22 aling was sufficient to prevent elevation of gastric pH and enhance S. Typhimurium colonization durin
23 n terms of histologic healing and increasing gastric pH and had a larger overall treatment effect.
24 is of the mice included measurement of basal gastric pH and plasma gastrin levels.
25 er endoscopic hemostasis, and in controlling gastric pH and protecting against upper gastrointestinal
26 onization, the bacterium neutralizes the low gastric pH and recruits immune cells to the stomach.
27             The impact of enzymes, different gastric pHs, and food-covering liquids on the bioaccessi
28 e; in the remaining 16 subjects, low vs high gastric pH changed between tests.
29 ly low bioaccessibility [i.e., solubility at gastric pH conditions (pH 1.5 to 3)].
30 min C in baby foodstuffs is very low in both gastric pH conditions.
31 received <3 mg/kg/day of ranitidine had poor gastric pH control as compared with 19% who received a m
32 ric drug references resulted in unsuccessful gastric pH control in a high percentage of pediatric int
33                                              Gastric pH control with ranitidine was considered unsucc
34 after omeprazole was started, and the lowest gastric pH during omeprazole therapy.
35     The effects of different factors such as gastric pH, enzymes, and food matrix on the solubility a
36  alkaline pH, and excystation in response to gastric pH followed by alkaline pH and protease.
37                             The median basal gastric pH for the cohort after stabilization on therapy
38 nt mice exhibited a marked increase in basal gastric pH (from 3.2 to 5.2) and an approximately 10-fol
39 y (0.05 mg x kg(-1) x hr(-1)) until reaching gastric pH &gt; or =4 for > or =75% of a 24-hr period, afte
40 ate ranitidine concentration associated with gastric pH &gt; or =4 was 287 +/- 133 ng/mL.
41 ial ranitidine dosage regimen that targets a gastric pH &gt; or =4.
42 termittent secretion, and 11% for consistent gastric pH higher than 3.5.
43  corpus is excised, presents a challenge for gastric pH homeostasis.
44 nzymes and bile salts, as well as the higher gastric pH in the infant model.
45 sibility of BPS in the intestinal phase with gastric pH increase.
46 ngle intravenous dose (1.52 +/- 0.47 mg/kg), gastric pH increased from 1.6 +/- 1.0 to 5.1 +/- 1.1 (p
47 had more severe gastric inflammation, higher gastric pH, increased parietal cell loss, increased gast
48      Gastrin is secreted following a rise in gastric pH, leading to gastric acid secretion.
49             Secondary efficacy measures were gastric pH measured 4 hrs after omeprazole was first adm
50 tic determinants, especially those affecting gastric pH, might contribute to eradication therapy fail
51 per gastrointestinal bleeding and maintained gastric pH of > 5.5 in mechanically ventilated critical
52 itidine and the dose should be titrated to a gastric pH of > or =4.
53 e than intravenous cimetidine in maintaining gastric pH of >4 in critically ill patients.
54  - 8.4 with a median pentagastrin stimulated gastric pH of 1.60 (range 1.0 - 8.2).
55 study, we detailed the consequences on intra-gastric pH, pepsin concentration and proteolysis by samp
56        For Salmonella, transient exposure to gastric pH prepares invading bacteria for the stresses o
57           Before its inactivation by the low gastric pH, salivary alpha-amylase released about 80% of
58                                  Thereafter, gastric pH should be monitored and the dose of ranitidin
59 onal status but it inversely correlated with gastric pH (Spearman's rho = -0.34; P = 0.0001).
60 nts in pigs key digestive parameters such as gastric pH, stomach emptying kinetics, and intestinal tr
61 stion (i.e. specific dynamic action or SDA), gastric pH, the postprandial blood alkalosis (the "alkal
62 location of the pH step-up point (shift from gastric pH to a pH >4) was also measured before and afte
63                                              Gastric pH values were lower in sst2 (-/-) mice (3.8 +/-
64                                       Median gastric pH was > or =6 on all trial days with omeprazole
65   In the omeprazole suspension group, median gastric pH was >4 on each trial day in 95% of patients.
66                                     When the gastric pH was 4, the average bioaccessibility of PL, PN
67                 The 4-hr postomeprazole mean gastric pH was 7.1, the mean gastric pH after starting o
68                                              Gastric pH was determined at the end of the ranitidine d
69                                              Gastric pH was monitored for 24 hours on three separate
70                                              Gastric pH was monitored hourly via nasogastric pH probe
71                              Poor control of gastric pH was not associated with feeding, intubation s
72                                              Gastric pH was poorly controlled in 36% of patients.
73 ritional status, smoking, alcohol intake and gastric pH were also analysed.