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1 e released only at pH 4 or 5 (typical infant gastric pH).
2 he others because the caseins clotted at the gastric pH.
3 potentially useful therapeutics to regulate gastric pH.
4 PEP 1:2 ratio inversely correlates with gastric pH.
5 onstrate equivalency and ability to increase gastric pH.
6 estion of a meal and maintain homeostasis of gastric pH.
7 pH paper are not interchangeable measures of gastric pH.
8 -secreting parietal cells and an increase in gastric pH.
9 Neither rHSP nor ITF altered gastric pH.
10 P. yoelii-infected mice had elevated gastric pH.
11 fs by an in vitro digestion model at varying gastric pHs.
12 vitamin B(6) in baby foods is lower in both gastric pHs.
15 accessibility of the PL, PN, and PM forms in gastric pH 1.5 were 53%, 76%, and 50%, respectively.
16 ils, H. pylori-infected gerbils had a higher gastric pH, a higher incidence of gastric ulcers, and a
17 24-h median [IQ] percentages of time with gastric pH above 3 and 4 were higher with rabeprazole th
18 a basic center imparting high solubility at gastric pH, addressing the dissolution limitation observ
20 fter omeprazole was first administered, mean gastric pH after omeprazole was started, and the lowest
21 omeprazole mean gastric pH was 7.1, the mean gastric pH after starting omeprazole was 6.8, and the me
22 aling was sufficient to prevent elevation of gastric pH and enhance S. Typhimurium colonization durin
23 n terms of histologic healing and increasing gastric pH and had a larger overall treatment effect.
25 er endoscopic hemostasis, and in controlling gastric pH and protecting against upper gastrointestinal
26 onization, the bacterium neutralizes the low gastric pH and recruits immune cells to the stomach.
31 received <3 mg/kg/day of ranitidine had poor gastric pH control as compared with 19% who received a m
32 ric drug references resulted in unsuccessful gastric pH control in a high percentage of pediatric int
35 The effects of different factors such as gastric pH, enzymes, and food matrix on the solubility a
38 nt mice exhibited a marked increase in basal gastric pH (from 3.2 to 5.2) and an approximately 10-fol
39 y (0.05 mg x kg(-1) x hr(-1)) until reaching gastric pH > or =4 for > or =75% of a 24-hr period, afte
46 ngle intravenous dose (1.52 +/- 0.47 mg/kg), gastric pH increased from 1.6 +/- 1.0 to 5.1 +/- 1.1 (p
47 had more severe gastric inflammation, higher gastric pH, increased parietal cell loss, increased gast
50 tic determinants, especially those affecting gastric pH, might contribute to eradication therapy fail
51 per gastrointestinal bleeding and maintained gastric pH of > 5.5 in mechanically ventilated critical
55 study, we detailed the consequences on intra-gastric pH, pepsin concentration and proteolysis by samp
60 nts in pigs key digestive parameters such as gastric pH, stomach emptying kinetics, and intestinal tr
61 stion (i.e. specific dynamic action or SDA), gastric pH, the postprandial blood alkalosis (the "alkal
62 location of the pH step-up point (shift from gastric pH to a pH >4) was also measured before and afte
65 In the omeprazole suspension group, median gastric pH was >4 on each trial day in 95% of patients.