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4 Chemoradiation After Resection of Gastric or Gastroesophageal Adenocarcinoma) assessed whether a post
6 rognostic factor in patients with resectable gastroesophageal adenocarcinomas with and without neoadj
7 nset in 6 of 16 patients (38%): prostate and gastroesophageal adenocarcinomas, myeloma, melanoma, col
10 Amplification of the MET proto-oncogene in gastroesophageal cancer (GEC) may constitute a molecular
12 ative chemotherapy plus surgery for operable gastroesophageal cancer from July 1, 1994, through April
13 ) expression may help to stratify breast and gastroesophageal cancer patients for HER2-targeting ther
14 SI, and survival in patients with resectable gastroesophageal cancer randomized to surgery alone or p
20 ater understanding of the roles of miRNAs in gastroesophageal carcinogenesis could provide insights i
24 s study aimed to determine the prevalence of gastroesophageal disease (GERD) and extraesophageal mani
25 PIs) are used for the long-term treatment of gastroesophageal disorders and the non-prescription medi
29 sease-specific survival (DSS) for resectable gastroesophageal (GE) junction and gastric adenocarcinom
30 chemotherapy regimen for advanced gastric or gastroesophageal junction (GEJ) adenocarcinoma and is as
31 iate esophageal versus gastric epithelium in gastroesophageal junction (GEJ) biopsies, the histology
34 ction of stage IB through IV (M0) gastric or gastroesophageal junction adenocarcinoma were randomly a
35 gnosis in patients with distal esophageal or gastroesophageal junction adenocarcinoma who have receiv
36 rpose After curative resection of gastric or gastroesophageal junction adenocarcinoma, Intergroup Tri
37 ion After a curative resection of gastric or gastroesophageal junction adenocarcinoma, postoperative
39 radiochemotherapy for high risk gastric and gastroesophageal junction adenocarcinoma: Demonstrated s
40 veness in reducing mortality from esophageal/gastroesophageal junction adenocarcinomas has not been e
41 ed by gastroesophageal reflux disease in the gastroesophageal junction and associated with tumorigene
42 ined as eradication of IM (in esophageal and gastroesophageal junction biopsy specimens), documented
43 reviously untreated metastatic esophageal or gastroesophageal junction cancer were randomly assigned
44 otherapy for locally advanced esophageal and gastroesophageal junction cancers is based on a few posi
46 ith adenocarcinoma of the gastric cardia and gastroesophageal junction have no history of reflux.
48 the lower frequency of tumors arising at the gastroesophageal junction in comparison with distal gast
49 carcinomas arising in the stomach or in the gastroesophageal junction in patients with HER2-positive
51 tients with resectable adenocarcinoma of the gastroesophageal junction treated with or without neoadj
52 thoracic esophageal carcinoma, including the gastroesophageal junction tumors (Siewert I), were rando
53 cancer localized in the distal esophagus or gastroesophageal junction undergoing McKeown TMIE or Ivo
57 py of local and locally advanced esophageal, gastroesophageal junction, and gastric adenocarcinomas,
58 lumnar junction and 3) 5-10 mm distal to the gastroesophageal junction, as demarcated by the top of t
62 ogy, and recurrence patterns associated with gastroesophageal malignancies suggest the need to split
63 with advanced lung, colorectal, pancreatic, gastroesophageal, or breast cancer between 1998 and 2005
64 doscopic capture from patients with gastric, gastroesophageal, or esophageal cancer who are administe
65 moderate to severe abdominal pain, a severe gastroesophageal reflex, and moderate to severe depressi
66 point), obstructive sleep apnea (+1 point), gastroesophageal reflux (+1 point), and depression (+1 p
67 omorbidities were allergic rhinitis (62.4%), gastroesophageal reflux (42.1%), sinusitis (37.9%), nasa
73 f a disease engendered more controversy than gastroesophageal reflux (GER), a highly prevalent condit
74 sts with erosive tooth wear have significant gastroesophageal reflux (GERD), despite minor reflux sym
77 tissue remodeling is associated with chronic gastroesophageal reflux and constitutes a premalignant l
80 also significant improvements in symptoms of gastroesophageal reflux and rhinitis, bronchial reversib
81 index greater than 4 on pH monitoring and/or gastroesophageal reflux and/or herniated wrap on upper g
87 gus (n = 1059) with those from subjects with gastroesophageal reflux disease (gastroesophageal reflux
88 .1; 95% confidence interval [CI], 2.9-12.9), gastroesophageal reflux disease (GERD) (RR, 1.9; 95% CI,
89 lin, leptin, and ghrelin are associated with gastroesophageal reflux disease (GERD) and Barrett's eso
90 and evaluate esophageal disorders, including gastroesophageal reflux disease (GERD) and eosinophilic
92 might represent an alternative treatment of gastroesophageal reflux disease (GERD) and may provide d
93 agement of extraesophageal manifestations of gastroesophageal reflux disease (GERD) and to compare th
96 .4)] were included; 70% had been treated for gastroesophageal reflux disease (GERD) during infancy.
97 surgery, highly variable rates of recurrent gastroesophageal reflux disease (GERD) have been reporte
98 The histologic changes associated with acute gastroesophageal reflux disease (GERD) have not been stu
101 RAs) are frequently used in the treatment of gastroesophageal reflux disease (GERD) in children; howe
102 n (TF) can decrease or eliminate features of gastroesophageal reflux disease (GERD) in some patients
103 There are few data on the prevalence of gastroesophageal reflux disease (GERD) in the United Sta
113 illustrated by the Montreal classification, gastroesophageal reflux disease (GERD) is much more than
115 ump inhibitor (PPI) therapy in patients with gastroesophageal reflux disease (GERD) is reported in up
120 f an underlying acid peptic disorder such as gastroesophageal reflux disease (GERD) nor should it pre
123 has been developed for use in patients with gastroesophageal reflux disease (GERD) symptoms despite
124 tailed critique of objective measurements of gastroesophageal reflux disease (GERD) would improve man
126 cluding Crohn disease (CrD), celiac disease, gastroesophageal reflux disease (GERD), and eosinophilic
127 frequency of irritable bowel syndrome (IBS), gastroesophageal reflux disease (GERD), and overactive b
128 rm use of PPIs for three common indications: gastroesophageal reflux disease (GERD), Barrett's esopha
129 this study was to appraise the prevalence of gastroesophageal reflux disease (GERD), esophagitis, and
130 n several pathophysiological states, such as gastroesophageal reflux disease (GERD), functional dyspe
133 ed the accuracy of frequency and duration of gastroesophageal reflux disease (GERD), using data from
136 can individuals have a similar prevalence of gastroesophageal reflux disease (GERD), yet esophageal a
137 defined as a decrease of 50% or more in the Gastroesophageal Reflux Disease (GERD)-Health Related Qu
153 nths after fundoplication for PPI-refractory gastroesophageal reflux disease (n = 14 270 degrees LPF
154 ad EoE (n = 17), indeterminate EoE (n = 15), gastroesophageal reflux disease (n = 7), or normal esoph
155 onitoring on therapy in patients with proven gastroesophageal reflux disease [GERD]), to document phy
156 ether patient-identified domains (dysphagia, gastroesophageal reflux disease [GERD], nausea/vomiting,
157 er improvement in quality of life related to gastroesophageal reflux disease and a 50% or greater red
159 ulosis is associated with conditions such as gastroesophageal reflux disease and diabetes mellitus, a
160 allenges to diagnosis, including the role of gastroesophageal reflux disease and proton pump inhibito
161 75-year-old woman with a medical history of gastroesophageal reflux disease and type II diabetes pre
163 pump inhibitors and has focused attention on gastroesophageal reflux disease as a causative factor in
164 We prospectively assessed 100 patients with gastroesophageal reflux disease before and after sphinct
165 iteria consisted of a confirmed diagnosis of gastroesophageal reflux disease by an abnormal esophagea
166 , screening 50-year-old men with symptoms of gastroesophageal reflux disease by Cytosponge is cost ef
167 emerging disease that is distinguished from gastroesophageal reflux disease by the expression of a u
169 95% confidence interval [CI]: 1.04-2.67) or gastroesophageal reflux disease controls (OR = 1.61; 95%
170 bjects with gastroesophageal reflux disease (gastroesophageal reflux disease controls, n = 1332), and
172 es for detailed drugs (lipid-lowering drugs, gastroesophageal reflux disease drugs, antihypertensive
173 ugs in 8 drug classes (lipid-lowering drugs, gastroesophageal reflux disease drugs, diabetes drugs, a
175 rm outcome of ARS in pediatric patients with gastroesophageal reflux disease have shown good to excel
176 months improved in patients with normal GE (Gastroesophageal Reflux Disease Health-Related Quality o
177 imary laparoscopic antireflux surgery due to gastroesophageal reflux disease in adults (>18 years).
178 ed strong genetic correlations of BE/EA with gastroesophageal reflux disease in male individuals and
179 related to the acidic environment caused by gastroesophageal reflux disease in the gastroesophageal
181 is (EoE) was historically distinguished from gastroesophageal reflux disease on the basis of histolog
182 ated with EoE (or indeterminate EoE) but not gastroesophageal reflux disease or normal esophagus and
184 ted with a relatively high rate of recurrent gastroesophageal reflux disease requiring treatment, dim
186 men in the United Kingdom with histories of gastroesophageal reflux disease symptoms, assuming the p
190 screening ages for patients with symptomatic gastroesophageal reflux disease were older (58 for men a
191 as chronic sinusitis, allergic rhinitis, and gastroesophageal reflux disease were only associated wit
193 structive sleep apnea may be associated with gastroesophageal reflux disease, a strong risk factor fo
194 allergic rhinitis, chronic rhinitis, asthma, gastroesophageal reflux disease, adenotonsillitis, sleep
196 ibly due to increased abdominal pressure and gastroesophageal reflux disease, although this pathogeni
197 lization for asthma, mechanical ventilation, gastroesophageal reflux disease, and aspiration or other
198 lation), and prior diagnoses (eg, pneumonia, gastroesophageal reflux disease, and other comorbidities
199 the individual to irritable bowel syndrome, gastroesophageal reflux disease, and peptic ulcer diseas
200 factors for EAC have been identified-mainly gastroesophageal reflux disease, Barrett's esophagus, ob
201 shown to be a safe and effective therapy for gastroesophageal reflux disease, but its effect on the L
203 terms: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
204 lowing: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
205 l gastrointestinal complications of obesity: gastroesophageal reflux disease, erosive esophagitis, Ba
206 This issue provides a clinical overview of gastroesophageal reflux disease, focusing on diagnosis,
207 on airway disease, including rhinosinusitis, gastroesophageal reflux disease, obesity and dysfunction
208 , including history of peptic ulcer disease, gastroesophageal reflux disease, or gastrointestinal ble
209 premorbid prevalence of anxiety, headaches, gastroesophageal reflux disease, sleep apnea, and infect
210 44 patients (52%) had objective findings of gastroesophageal reflux disease, such as esophagitis.
212 ic conditions (hypertension, hyperlipidemia, gastroesophageal reflux disease, thyroid disease, diabet
214 ministered a modified version of a validated gastroesophageal reflux disease-specific QOL tool to pat
228 is a laryngeal symptom that can be caused by gastroesophageal reflux disease; however, treatment outc
229 cough may have abnormal proximal exposure to gastroesophageal reflux documented by HMII that would ha
230 sphincter (UES) to simulated or spontaneous gastroesophageal reflux have shown conflicting results.
232 he diagnosis of cough, LPR, or asthma due to gastroesophageal reflux is difficult, as no criterion st
234 allograft injury from donor-directed Abs or gastroesophageal reflux led to new ColV and KAT Abs post
237 rative complications, sleep difficulties and gastroesophageal reflux progressively worsened during fo
238 ireflux surgery, 17.7% experienced recurrent gastroesophageal reflux requiring long-term medication u
239 poorly described, and the risk of worsening gastroesophageal reflux requiring revision may be higher
242 based on the modified Rome III criteria) and gastroesophageal reflux symptoms (GERS) in a population-
244 eep apnea and to determine whether nocturnal gastroesophageal reflux symptoms affect the relationship
245 estioned regarding severity of their typical gastroesophageal reflux symptoms and presence of nocturn
246 nal GERD in 1999 (>/=3 episodes of nocturnal gastroesophageal reflux symptoms per week) had an OR of
247 Barrett's esophagus patients with nocturnal gastroesophageal reflux symptoms should be further evalu
248 's esophagus was associated with more severe gastroesophageal reflux symptoms, and nocturnal reflux s
260 s have failed to demonstrate that preventing gastroesophageal reflux with antireflux surgery halts th
261 f unproven efficacy and focus on controlling gastroesophageal reflux with reflux-reducing medication
264 is used variably to prevent complications of gastroesophageal reflux, but its effectiveness is unprov
265 trophy (SMA) notes that patients suffer from gastroesophageal reflux, constipation and delayed gastri
266 discussed: rhinitis, chronic rhinosinusitis, gastroesophageal reflux, obstructive sleep apnoea, vocal
267 in the postoperative period from SSc-related gastroesophageal reflux, renal impairment, and skin fibr
268 , sex, percent predicted FEV1, self-reported gastroesophageal reflux, St. George's Respiratory Questi
269 pump inhibitors (PPIs) are popular drugs for gastroesophageal reflux, which are now available for lon
270 BACKGROUND & AIMS: Central obesity promotes gastroesophageal reflux, which may be related to increas
283 nclude younger age at presentation, previous gastroesophageal surgery or ulcers as an etiology, and a
286 y is common in cirrhotic patients with acute gastroesophageal variceal bleeding and is an independent
287 a lower rate of abdominal and chest varices, gastroesophageal variceal bleeding and refractory ascite
288 cotropin stimulation test in 157 episodes of gastroesophageal variceal bleeding in 143 patients with
290 We present a case of a patient with massive gastroesophageal variceal bleeding refractory to numerou
291 utcomes of patients with cirrhosis and acute gastroesophageal variceal bleeding remains unknown.
293 ich may induce lethal complications, such as gastroesophageal variceal hemorrhage and hepatic encepha
297 eeded to eradicate varices; no bleeding from gastroesophageal varices was observed after eradication.
298 tension (PH) has been exclusively devoted to gastroesophageal varices-related events at different fra