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1 ion, visceral hypersensitivity, and abnormal gastrointestinal motility.
2                                Both regulate gastrointestinal motility.
3  in a similar progression to embryonic mouse gastrointestinal motility.
4 optimizing therapeutic design and evaluating gastrointestinal motility.
5  increased fecal water content and increased gastrointestinal motility.
6 y contribute to these mechanisms by altering gastrointestinal motility.
7 lucuronidation, phospholipid metabolism, and gastrointestinal motility.
8 cts of antibiotics on the gut microbiome and gastrointestinal motility.
9  be influenced by drug dosage via changes in gastrointestinal motility.
10 iological effects and involves in regulating gastrointestinal motility.
11 lly explore the effect of GPER deficiency on gastrointestinal motility.
12 egulation of appetite, immune responses, and gastrointestinal motility.
13  It is an important physiologic regulator of gastrointestinal motility.
14 urmeric-interacts with gut bacteria to alter gastrointestinal motility.
15 acrophages and enteric neurons that controls gastrointestinal motility.
16  to increase survival of enteric neurons and gastrointestinal motility.
17  this effect of H2S modulates CNS control of gastrointestinal motility.
18  system (ENS), but profound abnormalities in gastrointestinal motility.
19 ntribute to enteric inhibitory regulation of gastrointestinal motility.
20 aling in neurons might be developed to alter gastrointestinal motility.
21 uired for body weight homeostasis and normal gastrointestinal motility.
22 s nerve as a key component underlying normal gastrointestinal motility.
23 signaling in survival of enteric neurons and gastrointestinal motility.
24 ptide initially discovered as a regulator of gastrointestinal motility.
25 ide-expressing neurons and are essential for gastrointestinal motility.
26  cells compromises epithelial maintenance or gastrointestinal motility.
27 t of increased beta-cell response or reduced gastrointestinal motility.
28 cells of Cajal (ICC) are required for normal gastrointestinal motility.
29 e Y (NPY) is an important peptide regulating gastrointestinal motility.
30 al physiological functions of these cells in gastrointestinal motility.
31 sting that female steroid hormones influence gastrointestinal motility.
32 utic agents for disorders involving impaired gastrointestinal motility.
33 r interstitial cells of Cajal in controlling gastrointestinal motility.
34 dentified two novel endogenous regulators of gastrointestinal motility.
35 ticipate in the Ca2+-dependent regulation of gastrointestinal motility.
36 teric plexus innervation density and reduced gastrointestinal motility.
37 y be of fundamental importance in regulating gastrointestinal motility.
38  to treat gastrointestinal bleeding, affects gastrointestinal motility.
39 the neuroendocrine factors controlling upper gastrointestinal motility, (2) noninvasive techniques to
40 that leukocyte-derived inducible NO inhibits gastrointestinal motility after manipulation and plays a
41  showing that CO is an integral regulator of gastrointestinal motility and an important factor in the
42 ptamine receptor (5-HT(4)R) agonists promote gastrointestinal motility and attenuate visceral pain, b
43 el with high strength to withstand long-term gastrointestinal motility and can be triggered to dissol
44  Although other methods exist for monitoring gastrointestinal motility and contractility, this study
45 ve that methane challenge is associated with gastrointestinal motility and fat metabolism.
46        Other acute effects include decreased gastrointestinal motility and food intake and increased
47 on between hunger, satiation, and integrated gastrointestinal motility and hormonal responses in morb
48                Intraduodenal lipid modulates gastrointestinal motility and hormone release and suppre
49 agastrically to assess its effect on fasting gastrointestinal motility and hunger ratings, motilin an
50     Tachykinins contribute to the control of gastrointestinal motility and modulation of somatic and
51  findings identify C1q as a key regulator of gastrointestinal motility and provide enhanced insight i
52             There was a significant delay in gastrointestinal motility and reduced numbers of nitrerg
53 esis that GAL-R1 mediates galanin actions on gastrointestinal motility and secretion by modulating th
54 to play important roles in the regulation of gastrointestinal motility and secretion, appetite, and f
55 e multiple physiological responses including gastrointestinal motility and secretion, glucose homeost
56 fects of food, meal intake and microbiota on gastrointestinal motility and sensation are discussed.
57                                      Altered gastrointestinal motility and sensation, changed activit
58 c ileus characterized by total inhibition of gastrointestinal motility and were excluded from further
59 sensitization, contributing to vasodilation, gastrointestinal motility, and airway resistance.
60 ceral perception, cardioautonomic responses, gastrointestinal motility, and brain activation patterns
61 ecreased epithelial cell turnover, increased gastrointestinal motility, and decreased lipid absorptio
62 se and xylose appearance after oral loading, gastrointestinal motility, and glucose transepithelial t
63 , airway mucus secretion, dysphagia, altered gastrointestinal motility, and itchy swollen skin.
64 les that act on enteric neurons to influence gastrointestinal motility, and metabolites that stimulat
65 late diverse biological processes, including gastrointestinal motility, angiogenesis, and circadian r
66 t have been shown to play important roles in gastrointestinal motility, angiogenesis, circadian rhyth
67 ation of enteric neurons, but its effects on gastrointestinal motility are not well characterized.
68 een used in the study of normal and abnormal gastrointestinal motility, as well as for the characteri
69 a, using immunofluorescence, immunoblot, and gastrointestinal motility assays.
70 rity to withstand the forces associated with gastrointestinal motility, be triggerable to address any
71 ve acquisition of functional and coordinated gastrointestinal motility before birth.
72                                              Gastrointestinal motility causes movement of food during
73                  Patients from a US academic gastrointestinal motility clinic, who failed medical the
74 1(flox/flox); Phox2b-Cre mice have increased gastrointestinal motility compared with controls.
75             They also suggest that increased gastrointestinal motility contributes to elimination of
76       Observations suggest that constitutive gastrointestinal motility depends more on neuronal than
77 : Cell therapy offers the potential to treat gastrointestinal motility disorders caused by diseased o
78      Cell therapy has the potential to treat gastrointestinal motility disorders caused by diseases o
79 s of muscles from patients with a variety of gastrointestinal motility disorders have raised the exci
80                                              Gastrointestinal motility disorders involve alterations
81 ay be a potential therapeutic target for the gastrointestinal motility disorders related to diabetes.
82 ptions currently available for patients with gastrointestinal motility disorders, especially patients
83 stinal pacemaker cells, underlie a number of gastrointestinal motility disorders, including diabetic
84 tients with Hirschsprung disease suffer from gastrointestinal motility disorders, whereas Waardenburg
85 he peripheral autonomic nerves can result in gastrointestinal motility disorders.
86 ve potential for therapeutic applications in gastrointestinal motility disorders.
87                                              Gastrointestinal motility disturbances are a hallmark of
88  of the forebrain/midbrain and impairment of gastrointestinal motility due to a reduction in postmito
89 s allows visualization and quantification of gastrointestinal motility during development and highlig
90 ation between subjective hunger feelings and gastrointestinal motility during the MMC is largely unkn
91 on-related damage, gastric acid secretion or gastrointestinal motility for these genes.
92 ld enhance effects of intraduodenal lipid on gastrointestinal motility, gut hormones, appetite, and e
93 eart rate, blood pressure, respiratory rate, gastrointestinal motility, hormone release, and body tem
94                         The effects of CT on gastrointestinal motility, however, have not been adequa
95 staltic reflex has been a central concept in gastrointestinal motility; however, evidence was publish
96                                     Immature gastrointestinal motility impedes preterm infant surviva
97                           Recovery of fasted gastrointestinal motility in dogs is equally rapid after
98 val of neurons in the intestine, but altered gastrointestinal motility in female, but not male, mice.
99    We provided the first recordings of fetal gastrointestinal motility in living embryos without anes
100 Blocking LPAR1 in vivo with AM966 attenuated gastrointestinal motility in mice and produced marked en
101 s and microbes increases neuron survival and gastrointestinal motility in mice.
102 testing of pharmacological agents to enhance gastrointestinal motility in prematurity.
103      The authors investigate the recovery of gastrointestinal motility in the fed and fasted state af
104               We implicate genes influencing gastrointestinal motility in the pathogenesis of gallsto
105  We measured epithelial barrier function and gastrointestinal motility in these mice and littermate c
106         Finally, Giardia infection increased gastrointestinal motility in wild-type mice, but not in
107                                              Gastrointestinal motility is altered in PWH.
108                                      Altered gastrointestinal motility is associated with significant
109                     A requirement for normal gastrointestinal motility is the tight regulation of ion
110                                              Gastrointestinal motility is the underlying program that
111 nt of a fully functional ENS is required for gastrointestinal motility, little is known about the ont
112 human fetal tissue and find that human fetal gastrointestinal motility matures in a similar progressi
113 , significance, pitfalls, and guidelines for gastrointestinal motility measurements by scintigraphy,
114 The A-ZIP/F-1 mice also did not show reduced gastrointestinal motility or 24-h food intake during bet
115 ant developments in our approach to studying gastrointestinal motility over the past few years has be
116  actual effect of a laparoscopic approach on gastrointestinal motility, particularly fed-state motili
117 We investigated the effect of propranolol in gastrointestinal motility, permeability and bacterial ov
118  and microcephaly, neurodevelopmental delay, gastrointestinal motility problems, and respiratory comp
119 tion of cannabinoid CB(1) receptors inhibits gastrointestinal motility, propulsion, and transit, wher
120 ce the expression of nNOS, a key molecule in gastrointestinal motility regulation.
121 HT(4) agonist for the potential treatment of gastrointestinal motility-related disorders.
122 s to be designed to therapeutically modulate gastrointestinal motility, secretion, and sensation.
123 by years, and include anosmia, problems with gastrointestinal motility, sleep disturbances, sympathet
124 in tandem, leading to disturbances in infant gastrointestinal motility that manifests clinically as c
125  advent of antidiabetes therapies that alter gastrointestinal motility, the effect of bariatric surge
126 gic links between HIV-associated AN, altered gastrointestinal motility, the gastrointestinal microbio
127 e effects that genetic knockouts may have on gastrointestinal motility; therefore, an understanding o
128 e enteric nervous system (ENS), glia control gastrointestinal motility through bidirectional communic
129 that enteric glial LPAR1 signaling regulates gastrointestinal motility through enteric glia and could
130    To determine the effect of CD8 T cells on gastrointestinal motility, transgenic mice expressing ov
131  indicates antipsychotic agents might affect gastrointestinal motility via varying microbiome-related
132                                              Gastrointestinal motility was assessed by measuring gast
133 the vagus nerve on metabolic homeostasis and gastrointestinal motility, we generated and characterize
134 n of pancreatic enzymes and secretin-induced gastrointestinal motility, which are mediated by vago-va
135 P-1 has effects on glucose metabolism and on gastrointestinal motility years after RYGB.
136          The enteric nervous system controls gastrointestinal motility, yet it is unknown when the hu
137 ages and the enteric nervous system regulate gastrointestinal motility, yet we have an incomplete und

 
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