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1 e yegM promoter was demonstrated in vitro by gel retardation assay.
2          This result is further supported by gel retardation assay.
3  and DNA binding ability of Cbfa1 based on a gel retardation assay.
4 igrating specific DNA-protein complexes in a gel retardation assay.
5 ance the DNA binding activity of EmBP-1 in a gel retardation assay.
6  sensitive and specific liquid hybridization gel retardation assay.
7 d abasic site was supershifted by XRCC1 in a gel retardation assay.
8 ed by the operator variants, and in vitro by gel-retardation assay.
9 ity DNA binding sequences were identified by gel retardation assays.
10 rms that include these exons fail to bind in gel retardation assays.
11  inability of each protein to bind to DNA in gel retardation assays.
12 anscription, using cotransfection assays and gel retardation assays.
13 electron microscopy and competitive assembly gel retardation assays.
14 rom each of these cell lines are examined by gel retardation assays.
15  site binding studies to be undertaken using gel retardation assays.
16                                 Moreover, in gel retardation assays, a 38-bp oligomer in the shortest
17                   However, data derived from gel retardation assays, a modified ligation-mediated pol
18                                              Gel retardation assays also demonstrated that staurospor
19                                           In gel retardation assays, an RCS cell-specific protein and
20  the effects of mutation on competition in a gel retardation assay and on promoter activity were stud
21 ts with single and multiple PRH sites, using gel retardation assays and DNase I and chemical footprin
22 s on Fur binding was examined using in vitro gel retardation assays and DNase I footprinting experime
23                                              Gel retardation assays and DNase I footprinting studies
24 rate and product analogs was evaluated using gel retardation assays and footprinting experiments.
25 d by chromatin immunoprecipitation, in vitro gel retardation assays and gene expression analyses.
26                                              Gel retardation assays and mutational analysis demonstra
27 nd to cellular SSDB factors by use of common gel-retardation assays and PCR.
28  (Zetasizer), encapsulation or complexation (gel retardation assay) and transfection (Fluorescence mi
29 ational analyses using reporter gene assays, gel retardation assays, and immunostaining experiments,
30 d E7 genes by in vitro DNase I footprinting, gel retardation assays, and transfection studies, we als
31                            As estimated by a gel retardation assay, binding saturates at a stoichiome
32                                         In a gel retardation assay both the phosphatidic acid-depende
33                                 Quantitative gel retardation assays confirm this observation and show
34 ounds of random oligonucleotide selection in gel retardation assays, coupled with PCR amplification o
35                                              Gel retardation assay demonstrated that H2O2 (hydrogen p
36                                              Gel retardation assays demonstrated that an increase in
37                                              Gel retardation assays demonstrated that in vitro transl
38                                              Gel retardation assays demonstrated the binding of homog
39                                              Gel-retardation assays demonstrated binding of IHF to th
40                                            A gel retardation assay detected two sites within the pali
41                                              Gel retardation assays employing T.brucei mRNAs demonstr
42                                              Gel retardation assay experiments showed that DNA fragme
43 pearance of an NF-kappaB complex as shown in gel retardation assays; however, induction of the HSR by
44                                       An RNA gel retardation assay identified a 37-nucleotide cognate
45 irected mutagenesis experiments and in vitro gel retardation assays identified Ets and GATA elements
46                                              Gel retardation assays identified Sp1, Sp3 and DeltaSp3
47 lex RT/DNA/dNTP could be readily detected by gel retardation assays if the DNA had a template overhan
48                                              Gel retardation assays indicated that the Ap1/NF-E2 moti
49                                              Gel retardation assays indicated that the master negativ
50 esults from P. aeruginosa mutant studies and gel retardation assays indicated that this regulation wa
51                                              Gel retardation assays involving the purified QscR have
52  and monitoring changes to RepB binding in a gel-retardation assay, it was shown that the central, GC
53                                           In gel retardation assays, Mot21 and MotA bind DNA containi
54                    Using DNA competition and gel retardation assays, NaeI-L43K showed reduced affinit
55                   Here, circular permutation gel retardation assays provide evidence that the polymer
56         Consistent with this observation, in gel retardation assays, purified TraM abolished the DNA
57                                              Gel retardation assays reveal ribonucleoprotein complexe
58                                              Gel retardation assays revealed that another islet-enric
59                                              Gel retardation assays revealed that full-length GTF3, a
60                                              Gel retardation assays revealed that the reason for this
61 acting factors, in vitro transcriptional and gel retardation assays revealed that the RpoN-recognized
62                                              Gel retardation assays revealed that the three proteins
63                                              Gel retardation assays revealed time-dependent, hypoxia-
64                               As assessed by gel retardation assays RpoAAt, but not RpoAEc, formed a
65 rease the binding of E2 to non-specific DNA, gel retardation assays show that these ions have no effe
66                                          The gel retardation assay showed that the N/P > 1 exhibited
67 ia-mediated activation of C/EBPbeta-NF-IL-6, gel retardation assays showed no change in the intensity
68                                              Gel retardation assays showed PPARgamma bound hexamers 3
69                                              Gel retardation assays showed that dephosphorylation of
70                                              Gel retardation assays showed that IL-4 induced the form
71                                              Gel retardation assays showed that nuclear proteins from
72                                              Gel retardation assays showed that the F-F-x-R architect
73                                              Gel retardation assays showed that while the mutant MITF
74 n gene transfection and proteolytic clipping gel retardation assays suggest that the AP-1 flanking se
75     In UV cross-linking, filter binding, and gel retardation assays, the MSH2-msh6-F337A complex disp
76                                           In gel retardation assays, the specific binding seen with e
77 ogen receptor-specific antisera and shown by gel retardation assay to bind oligonucleotides containin
78 erized tomato homeobox protein, was shown by gel retardation assay to interact with the promoter of L
79                              Lastly, we used gel retardation assays to show that alterations in trans
80                We identified this complex by gel retardation assay using short, linear fragments and
81                                              Gel retardation assays using a probe containing the repe
82                                              Gel retardation assays using extracts from both Jurkat T
83                                              Gel retardation assays using the Hol75 DNA confirm these
84      To understand this phenomenon better, a gel retardation assay was used to measure the equilibriu
85                                            A gel retardation assay was used to purify a potential reg
86 y the mechanistic details of this process, a gel-retardation assay was used to measure the dissociati
87 oupling novel immunoblotting techniques to a gel retardation assay, we observed that nucleosome cores
88                                      Using a gel retardation assay, we show here that nuclear extract
89                           Furthermore, using gel retardation assay, we show that 12-O-tetradecanoylph
90                                        Using gel retardation assays, we mapped an IE2-binding site wi
91                                        Using gel retardation assays, we now demonstrate that the bind
92                                        Using gel retardation assays, we show that the hierarchy of E2
93 nce, circular dichroism, and electrophoretic gel retardation assays were carried out on recombinant 8
94 wo-dimensional (2D) and one-dimensional (1D) gel retardation assays were used to analyze occludin pho
95                                              Gel retardation assays were used to investigate the pair
96 or the AC-rich region was also shown using a gel retardation assay with an ACBF recombinant protein e
97 extracts from these strains were inactive in gel retardation assays with a 158-bp fragment of the DNA
98 ter with increased methylation level, and in gel retardation assays with an oligonucleotide containin
99                                              Gel retardation assays with crude cell extracts confirme
100 essing the recombinant proteins were used in gel retardation assays with double stranded oligonucleot
101                                              Gel retardation assays with ER+ and ER- cell lines ident
102                                              Gel retardation assays with extracts from the cells grow
103                                              Gel retardation assays with extracts from the complement
104                                              Gel retardation assays with mutant MotA boxes are consis
105                              Electrophoretic gel retardation assays with purified M.RSR:I were perfor
106 rmined by RNAII and RNAIII transcription and gel retardation assays with the P2 and P3 promoters of a

 
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