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1 ge-specific to an AP-1-driven injury-induced gene expression program.
2 nalyses revealed that CBX6 governs a complex gene expression program.
3 uronal activity and turn on a detoxification gene expression program.
4 ent epidermal cells, initiating the follicle gene expression program.
5 effect gene through regulation of the oocyte gene expression program.
6 a switch from an adaptive to an inflammatory gene expression program.
7 and triggering a vascular stability-related gene expression program.
8 export, which are central to the eukaryotic gene expression program.
9 ts as a major regulator of the TAL1 leukemic gene expression program.
10 26(N1ICD/+)) actively promotes a SC-specific gene expression program.
11 ption factors responsible for the basal-like gene expression program.
12 ription factors that represses the erythroid gene expression program.
13 ates Smad2/3 signaling and induces a complex gene expression program.
14 ctures intimately controls every step of the gene expression program.
15 estrate sequential deployment of the cardiac gene expression program.
16 positive transcriptional regulator of the PC gene expression program.
17 cess that requires the MRTF-dependent motile gene expression program.
18 as well as suppression of an injury-related gene expression program.
19 on of MRTFs and the activation of the motile gene expression program.
20 s to a block of the KLF1-dependent erythroid gene expression program.
21 at controls the Lgr5(+) intestinal stem cell gene expression program.
22 r36, which is required for the proadipogenic gene expression program.
23 ile Pseudomonas aeruginosa induce a specific gene expression program.
24 ing genome, leading to a senescence-specific gene expression program.
25 s an essential regulator of the pro-fibrotic gene expression program.
26 required for the activation of the myogenic gene expression program.
27 in vitro and express the expected B-lineage gene expression program.
28 l key transcription factors in the Th17 cell gene-expression program.
29 with HOXA9 and modulates a HOXA9-controlled gene-expression program.
30 onization, and had a different developmental gene-expression program.
31 eractions have profound effects on embryonic gene expression programs.
32 nd transduce them to the nucleus to regulate gene expression programs.
33 Type I or II IFN activate highly overlapping gene expression programs.
34 rograms, while H3F3A adapted for a subset of gene expression programs.
35 itive to Notch signals that rapidly regulate gene expression programs.
36 it chromatin machinery to activate oncogenic gene expression programs.
37 encing of transposable elements rewires host gene expression programs.
38 tors are required to control tissue-specific gene expression programs.
39 or the chromatin landscape for cell-specific gene expression programs.
40 nes, leading to the suppression of oncogenic gene expression programs.
41 rinciples for stochastic, mutually exclusive gene expression programs.
42 omplexes to establish and maintain oncogenic gene expression programs.
43 nd RNA-binding proteins (RBPs) can influence gene expression programs.
44 lator that antagonizes T and B cell-specific gene expression programs.
45 re transcription factors that drive specific gene expression programs.
46 lance between proapoptotic and antiapoptotic gene expression programs.
47 kappaB and MAPK pathways allow dose-specific gene expression programs.
48 sible to preferentially induce four distinct gene expression programs.
49 hancer activity is important for controlling gene expression programs.
50 adaptive changes in cellular physiology and gene expression programs.
51 hromatin remodeling and regulate tumorigenic gene expression programs.
52 e been attributed to its ability to regulate gene expression programs.
53 organs and tissues generally have conserved gene expression programs.
54 for specialized cell-type and developmental gene expression programs.
55 sms by which cis-regulatory elements control gene expression programs.
56 ion process involving major modifications in gene expression programs.
57 TFs can recognize their motifs and regulate gene expression programs.
58 opmentally appropriate expression and switch gene expression programs.
59 bryo development and regulating cellular and gene expression programs.
60 nt binding patterns and driving differential gene expression programs.
61 balance developmental and stress-responsive gene expression programs.
62 sses are modulated by regulation of specific gene expression programs.
63 oliferating cells enriched for developmental gene expression programs.
64 ites are functionally linked to latency type gene expression programs.
65 n microRNAs, which may regulate bat-specific gene-expression programs.
66 to the nucleus by GLI proteins to influence gene-expression programs.
67 ific milieus for the maintenance of defining gene-expression programs.
68 ecific genomic sequences to regulate complex gene-expression programs.
69 eristics, which are a reflection of distinct gene-expression programs.
70 es residing in different organs have diverse gene-expression programs.
71 ions of IRF4 and IRF8 in regulating distinct gene-expression programs.
72 ganization of DNA to establish cell-specific gene-expression programs.
73 somes, are required for proper physiological gene-expression programs.
74 y needs to be inhibited to make the PrE-like gene expression program accessible for activation by GAT
75 e a resource for the analysis of EC-specific gene expression programs across heterogeneous vascular b
76 We found that both repression and activation gene expression programs, affecting hundreds of genes, a
77 clusion, IL-3 produces a distinct eosinophil gene expression program among the beta-chain receptor cy
78 otypic outcomes and divergent alterations in gene expression programs among different tumors, despite
80 tions as a modulator of a neurodevelopmental gene expression program and binds to important regulator
81 1 (Gfi1) orchestrates the fidelity of the DP gene expression program and developmental maturation int
82 te that IKZF2 regulates a HOXA9 self-renewal gene expression program and inhibits a C/EBP-driven diff
83 nscription of a key set of genes within this gene expression program and might therefore be exception
84 ate to regulate the basal-like breast cancer gene expression program and provides the basis for impro
86 lar processes ensure timely execution of the gene expression program and survival under conditions of
87 of neurogenesis to fully repress the neural gene expression program and to promote glial gene expres
88 hy tumor-specific MYC levels induce specific gene expression programs and alter defined biological pr
90 nd recruited macrophages to acquire distinct gene expression programs and corresponding functions.
91 element evolution that shape tissue-specific gene expression programs and defines regulatory elements
94 rplay of transcription factors that initiate gene expression programs and epigenetic mechanisms that
96 cessive activation and silencing of specific gene expression programs and is driven by tissue-specifi
97 ng protein, thereby mediating stage-specific gene expression programs and post-meiotic chromatin reor
98 cardiac precursors (CPs) to initiate cardiac gene expression programs and repress non-cardiac express
99 eceptor family member signaling in postnatal gene expression programs and select ontogeny-specific ph
100 e the participation of HvVP1 in antagonistic gene expression programs and support its central role as
101 logical signals link acetylation to specific gene expression programs and whether such responses are
102 omatin-remodeling SWI/SNF complex determines gene expression programs and, consequently, contributes
103 hat controlled an iNKT-cell lineage-specific gene-expression program and epigenetic landscape in a de
104 activation, turning on its tumor-suppressive gene-expression program and turning off STAT3's oncogeni
105 n, regulation of an E2F-dependent cell-cycle gene expression program, and estrogen-dependent mitogeni
106 m of a regulatory switch controlling crucial gene expression programs, and provide a framework for un
107 protein associated with fetal hematopoietic gene expression programs, and these cells acquired a fet
109 regulatory evolution and cell-type-dependent gene-expression programs, and provides a resource for fu
111 owever, the mechanisms driving such atypical gene expression programs are incompletely understood.
112 cultures using nitrogen-limited chemostats, gene expression programs are strikingly similar regardle
114 d/or PMA on Jurkat T cells, we show that the gene expression program associated with activation of TC
115 is and is linked to a previously unexplained gene expression program associated with anti-PD-1 immuno
116 We also show that SHP099 normalizes the gene expression program associated with increased cell p
117 lysaccharide (LPS) revealed the induction of gene expression programs associated with a native defens
118 n brown adipose tissue controls antagonistic gene expression programs associated with energy balance
119 therapeutic targets for modulating aberrant gene expression programs associated with MLL-fusion leuk
120 d to more robust induction of IL-10-mediated gene expression programs at low ligand concentrations in
121 as cells enter G0, their survival and global gene expression programs become increasingly dependent o
122 these mRNA populations revealed the diverged gene expression programs between filial and maternal com
123 data provide molecular support for distinct gene expression programs between the AC-type SAMs of Sel
124 tazoan cells requires execution of different gene expression programs but recent single-cell transcri
125 activation of species- and context-specific gene expression programs, but such regulation must be ba
126 ated process that requires tightly regulated gene expression programmed by transcription factors and
128 the soluble nucleoplasmic pool over time as gene expression programs change during development or di
130 e in the establishment of cell-type specific gene expression programs characteristic of different cel
131 events is the acquisition of a mature T-cell gene expression program characterized by the induction o
132 s known to be important in the regulation of gene expression programs conferring cellular identities.
133 t bind the serum-response factor to activate gene expression programs critical in smooth muscle (SM)
134 r calcium flux with activation of an anergic gene-expression program dependent on the transcription f
135 uggesting that Scriptaid maintains stem cell gene expression programs despite expansion in HSC number
136 factors ETS1 and ETS2 mediate activation of gene expression programs downstream of RAS/MAPK signalin
137 s distinct signaling complexes that initiate gene expression programs downstream of the transcription
138 rate that, rather than controlling essential gene expression programs, Drosophila JmjC proteins gener
139 n cancers, but how they influence the cancer gene expression program during cancer initiation and pro
140 We show that RUNX1 represses the erythroid gene expression program during megakaryocytic differenti
142 essential for establishing lineage-specific gene expression programs during cell differentiation.
143 chromatin remodeling establishes coordinated gene expression programs during development, yet importa
145 on of chromatin states is thought to control gene expression programs during lineage specification.
146 chanisms play a major role in the control of gene expression programs during normal development and a
147 eedforward loops to control lineage-specific gene expression programs during progressive differentiat
148 n by different splicing factors to fine tune gene expression programs during these physiological and
150 portion of the noncoding genome and control gene expression programs either in cis or in trans Putat
152 or post-translational modification to induce gene expression programs essential in tumorigenicity.
154 rotein level but provide novel insights into gene expression programs expected to define different T
156 rovide a powerful way to construct synthetic gene expression programs for a wide range of application
159 nthetic systems, and the faculty to engineer gene expression programs from a minimal set of first pri
161 nfection both induce a similar IRF-dependent gene expression program, gene expression driven by the N
162 ors and histone-modifying enzymes to control gene expression programs governing lineage allocation in
163 occurs pervasively during stress to activate gene expression programs; however, the convenience of RN
165 n arrest, suggesting that the injury-related gene expression program in Dgcr8 cKOs cannot be attribut
166 be due to differences between species in the gene expression program in each cell type, but may also
167 ption factors (TFs) dominates control of the gene expression program in embryonic stem cells and othe
168 o-regulated genes, thereby altering the host gene expression program in favor of viral persistence.
169 ETS1 and ETS2 can regulate a cell migration gene expression program in opposite directions, and prov
170 emia that is strictly dependent on a defined gene expression program in the cell of origin, which inc
171 dent mechanism to restrict the smooth muscle gene expression program in the developing mesothelium an
174 th in vivo and shifted the immunosuppressive gene expression program in tumor-associated macrophages
175 -coding genes to maintain cell-type-specific gene expression programs in all human cells is a fundame
177 ihydroergotamine (DHE), regulate overlapping gene expression programs in AML and repress transcriptio
180 spond to Notch signaling, broadly regulating gene expression programs in cell development and functio
181 mediated in part by epigenetic remodeling of gene expression programs in discrete brain regions.
184 -binding proteins (RBPs) critically regulate gene expression programs in mammalian cells by modulatin
188 d as key regulatory molecules of immune cell gene expression programs in response to microbial and ti
189 of key transcriptional regulators mediating gene expression programs in response to pathogen-specifi
190 omputational normalization to compare global gene expression programs in steady-state and dynamic con
192 cription factors coordinate subtype-specific gene expression programs in subtypes in which their expr
193 PRDM16 regulates convergent developmental gene expression programs in the cortex and MGE, which ut
194 stinct Polycomb components in the control of gene expression programs in the disorders of epidermal d
195 elements, transcription factor binding, and gene expression programs in three regions of the human v
196 incident with activation and differentiation gene-expression programs in a cell-division-dependent ma
197 g cells abrogated the cell of origin-derived gene expression program, including the expression of Hox
198 ion by partially restoring the EGFR-promoted gene expression program, including the sustained express
199 7 signaling in AML cells activates stem cell gene expression programs, including the Wnt pathway, and
200 ed upon inhibition, resulting in an abnormal gene expression program influencing the glial lineage.
201 al that RAC1(P29S) activates PAK, AKT, and a gene expression program initiated by the SRF/MRTF transc
202 ing, we performed a deep characterization of gene expression programs involved in the early and late
207 epression of pre-existing ES cell-associated gene expression program is followed by activation of TS
210 together, induction of liver STAT3-dependent gene expression programs is essential to countering the
212 An outstanding issue in understanding T cell gene expression programs is whether RUNX1 and ETS1 have
213 els in adulthood, with particular focus on a gene expression program known as the conserved transcrip
216 l nervous system development, spatiotemporal gene expression programs mediate specific lineage decisi
217 able to implement extensive changes to their gene-expression programs, metabolism, and cellular struc
218 cription factor (TF) network establishes the gene expression program necessary for pluripotency.
219 anisms by which SOX10 guides the appropriate gene expression programs necessary to promote the melano
220 n to repress the ability of HSF1 to activate gene-expression programs necessary for cancer survival.
222 ntly reported that oncogenic KRAS promotes a gene expression program of de novo lipogenesis in non-sm
224 melanoma cells become pigmented and enact a gene expression program of melanocyte differentiation.
226 t regulates the inducible cell type-specific gene expression program of the human TNF/LT locus and pr
228 ypes, we were able to derive and compare the gene expression programs of ASC subsets that were respon
230 vide the structural framework for the global gene expression programs of the individual chromosomes.
231 ormation and lineage differentiation involve gene expression programs orchestrated by transcription f
232 tion, both the mTOR and extracellular matrix gene expression programs paralleled the activation of pr
233 neage priming, activation, and repression of gene expression programs) provides insight into fundamen
234 apoptosis, depending on the cell type, with gene expression programs, rather than extent of mitochon
236 Thus, the ETS/AP-1 sequence defines a unique gene expression program regulated by the relative levels
240 Transcription factor networks shape the gene expression programs responsible for normal cell ide
241 entiation, keratinocytes sequentially switch gene expression programs, resulting in terminal differen
242 s and transcriptional drivers to single-cell gene expression programs, significantly extending our un
244 on to mediate a compensatory neuroprotective gene expression program that desensitizes neurons to glu
245 that ES7c is cleaved at early stages of the gene expression program that enables cells to successful
246 y for the cholesterol biosynthesis metabolic gene expression program that generates RORgammat agonist
247 strates, thereby acting to restrain a neural gene expression program that is aberrantly expressed in
248 -modified germline transcripts to maintain a gene expression program that is conducive for progressio
249 iving a transcriptionally encoded adrenergic gene expression program that was selectively reversed by
250 s beyond the chromosome, instilling adaptive gene expression programs that are heritable over long bi
251 ylation (DNAm) is an epigenetic regulator of gene expression programs that can be altered by environm
254 cular regulators, which control the aberrant gene expression programs that drive and maintain the can
255 suggest that LIN28 may regulate splicing and gene expression programs that drive breast cancer subtyp
256 ernative splicing (AS) is a key component of gene expression programs that drive cellular differentia
257 matopoietic stem cells (HSCs) possess unique gene expression programs that enforce their identity and
258 n of LTP at PV-IN output synapses, involving gene expression programs that need to be addressed in fu
259 , activated CD4(+) T cells initiate distinct gene expression programs that produce multiple functiona
260 struction).Conclusions: Two distinct stromal gene expression programs that promote cancer initiation
261 changes, thereby allowing the activation of gene expression programs that promote cardiomyocyte dedi
262 se distinct cell fates are driven by massive gene expression programs that promote the necessary chan
263 DNA methylation and histone modifications on gene expression programs that promote this malignancy.
264 specific properties are encoded by selective gene expression programs that shape molecular repertoire
265 hat lung stromal cells activate pathological gene expression programs that support oncogenesis.Object
266 Chromatin modifiers affect spatiotemporal gene expression programs that underlie organismal develo
267 allel, activation initiates context-specific gene-expression programs that drive effector functions a
268 tablish and reinforce the cell-type-specific gene-expression program; the ensemble of core TFs and th
270 ption factors are essential for implementing gene expression programs, they do not function in isolat
271 squamous cells activates a context-specific gene expression program through lineage-specific regulat
272 that m(6)A MTase activity promotes erythroid gene expression programs through selective translation o
273 knowledge of how transcription factors drive gene expression programs through their interactions with
274 nvironmental signals with activation-induced gene-expression programs through modulation of the epige
275 distinct and specific roles in regulation of gene-expression programs throughout postnatal developmen
276 required to fully execute the rpoS-dependent gene expression program to allow E. coli to adapt to sus
278 BRN2 also suppresses an apoptosis-associated gene expression program to protect against UVB-, chemoth
279 the MYC regulatory network that orchestrates gene expression programs to control self-renewal for the
280 ory sensory epithelium, but how it activates gene expression programs to generate distinct cell-types
281 pied by various key TFs, regulating specific gene expression programs triggered by divergent macropha
282 signalling axis that fine-tunes inflammatory gene expression programs under both physiological and pa
286 chromatin remodeling activities to regulate gene expression programs underlying cIN development.
288 amily of transcription factors that regulate gene expression programs underlying key immunological pr
289 that Dgcr8 is responsible for modulation of gene expression programs underlying myelin formation and
290 ion, nor do they specifically activate early gene expression programs upon short exposure to ER stres
291 is required in ESCs to initiate appropriate gene expression programs upon somatic multi-lineage diff
292 f identifying new regulators of this massive gene expression program, we have used a GFP-based protei
293 NF complex target sites to tumor-suppressive gene expression programs, we clarify the transcriptional
294 cells activate a previously dormant tip cell gene expression program, which likely underlies the adap
295 , IL-10 did not induce key components of its gene expression program, which may explain its lack of e
296 hanges in the H3K27ac enhancer landscape and gene expression program, which was also seen with direct
297 determination requires faithful execution of gene expression programs, which are increasingly recogni
298 mitotic ON SACs and promotes the ON fate and gene expression program while repressing the OFF fate an
299 hanistically distinct, senescence-associated gene expression programs, with altered expression of dis
300 a recently discovered feature of eukaryotic gene expression programs, yet its function remains large