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1 s, and during the development of dietary and genetic obesity.
2 s, and during the development of dietary and genetic obesity.
3 and metabolic abnormalities associated with genetic obesity.
4 e 2 in vitro and in vivo in diet-induced and genetic obesity.
5 evidence for a role of AT LPL in one type of genetic obesity.
6 as setmelanotide used to treat rare forms of genetic obesity.
7 abnormally activated in mice with dietary or genetic obesity.
8 ents and rhesus monkeys with diet-induced or genetic obesity and diabetes exerts strong antihyperglyc
9 lowing partial hepatectomy (PH) in mice with genetic obesity and hepatic steatosis and also in wild-t
10 nditure, protection against diet-induced and genetic obesity and insulin resistance, thus suggesting
11 or the ob protein leptin in rodent models of genetic obesity, and insulin has been reported to direct
14 onal status in a context of diet-induced and genetic obesity; food deprivation increased Deptor mRNA
16 ents, or reverses, high-fat diet-induced and genetic obesity, insulin resistance and hepatic steatosi
17 We now demonstrate that either dietary or genetic obesity is a potent bona fide liver tumor promot
20 dy size and body composition associated with genetic obesity susceptibility inform the mechanisms tha
23 uscle of the ob/ob mouse, an animal model of genetic obesity with extreme insulin resistance, exhibit