1 A
genetic screen for A. tumefaciens mutants deficient for
2 Genetic screening for a germline mutation at the RET gen
3 arcoded mutants unlocks the power of reverse
genetic screening for a malaria parasite and will enable
4 In a
genetic screen for aberrant synaptic growth at the neuro
5 In a
genetic screen for adaptive defective mutants, a nonsens
6 are available to the fly community, forward
genetic screens for adult heart phenotypes have been rar
7 A
genetic screen for allele-specific suppressors of the G(
8 of the SIR complex, we performed a targeted
genetic screen for alleles of SIR3 that dominantly disru
9 In a reverse
genetic screen for altered drought tolerance, we identif
10 A forward
genetic screen for altered PD transport identified incre
11 e lhr1 mutant was isolated through a forward
genetic screening for altered expression of the lucifera
12 nents that have been identified by extensive
genetic screens for altered sensitivities to osmotic str
13 ng regulator, was uncovered during a forward-
genetic screen for angiogenesis-deficient mutants in the
14 .1 mutant, which was identified in a forward
genetic screen for Arabidopsis (Arabidopsis thaliana) mu
15 ation-sensitive PCR (CHOP-PCR)-based forward
genetic screen for Arabidopsis DNA hyper-methylation mut
16 A
genetic screen for Arabidopsis mutants affecting tempera
17 for this feedback regulation, we performed a
genetic screen for Arabidopsis mutants that exhibit enha
18 A
genetic screen for Arabidopsis thaliana mutants compromi
19 t protein fluorescence imaging-based forward
genetic screen for Arabidopsis thaliana mutants that sho
20 test this hypothesis, we employed a forward
genetic screen for Arabidopsis thaliana mutants tolerati
21 function in auxin signaling, we performed a
genetic screen for Arabidopsis thaliana mutants with an
22 Through forward
genetic screens for Arabidopsis mutants showing DNA hype
23 and bio2 auxotrophs identified using forward
genetic screens for arrested embryos rescued on enriched
24 ic causes of atopy, we carried out a forward
genetic screen for atopy in mice.
25 Forward
genetic screens for auxin-related mutants have led to th
26 By deploying an RNAi-based
genetic screen for bak1/serk4 cell death suppressors, we
27 s have been extended by the development of a
genetic screen for BLIP function in Escherichia coli.
28 The
genetic screen for BLIP function was used to sort the li
29 addition, ENA21 was identified in a forward
genetic screen for C. albicans genomic sequences that in
30 From a forward
genetic screen for C. elegans genes required for RNAi, w
31 In a
genetic screen for C. elegans mutants exhibiting partial
32 To seek such genes, we performed a
genetic screen for C. elegans mutants that age premature
33 In a
genetic screen for C. elegans mutants with defects in do
34 A
genetic screen for C. elegans mutants with PKD-2 ciliary
35 A
genetic screen for C. elegans mutations that cause the s
36 In a previous
genetic screen for Caenorhabditis elegans mutants that s
37 A
genetic screen for Caenorhabditis elegans mutants with b
38 We describe here the results of
genetic screens for Caenorhabditis elegans mutants in wh
39 g Beauty is a new tool for unbiased, forward
genetic screens for cancer genes in vivo.
40 A conditional
genetic screen for CckA mislocalization mutants, using a
41 tors, are likely to alter fundamentally both
genetic screening for celiac disease and its therapy.
42 A
genetic screen for cell division cycle mutants of Caulob
43 In a new
genetic screen for cell shape mutants, we identified Rod
44 ion motif-containing protein, from a forward
genetic screen for cellular antisilencing factors in Ara
45 In a
genetic screen for cellular factors involved in preventi
46 s a binding partner for TopBP1 and also in a
genetic screen for checkpoint regulators in zebrafish.
47 d in zinc metabolism, we conducted a forward
genetic screen for chemically induced mutations that cau
48 In a forward
genetic screen for chemotaxis mutants in Dictyostelium d
49 A
genetic screen for Chlamydomonas reinhardtii mutants wit
50 Based on the success of
genetic screens for circadian mutants in Drosophila mela
51 ensing receptors, we carried out an unbiased
genetic screen for cold-sensing mutants in C. elegans an
52 will enable large-scale forward and reverse
genetic screens for complex behaviors.
53 In a
genetic screen for components involved in the early immu
54 Here, using a yeast
genetic screen for components that control nucleoid dist
55 In a Drosophila
genetic screen for components that promote Wingless sign
56 1p-induced DNA lesions, we described a yeast
genetic screen for conditional tah (top1T722A-hypersensi
57 ation (GAIT) of ceruloplasmin (Cp) mRNA by a
genetic screen for Cp 3'-UTR binding proteins.
58 Using a
genetic screen for cVA-insensitive mutants, we have iden
59 network in Arabidopsis, we used a sensitized
genetic screen for deetiolation-defective seedlings.
60 lsy-6 mutants that we retrieved from a
genetic screen for defects in neuronal left/right asymme
61 d trypanosome VSG exclusion-1 (VEX1) using a
genetic screen for defects in telomere-exclusive express
62 A
genetic screen for defects in the organization of interm
63 es cerevisiae SPT4, previously identified in
genetic screens for defects in chromosome transmission f
64 gevity control genes, we performed a forward
genetic screen for delayed reproductive aging in C. eleg
65 A zebrafish
genetic screen for determinants of susceptibility to Myc
66 s and demonstrates the promise of functional
genetic screens for dissecting therapeutically relevant
67 A
genetic screen for dominant enhancers of the mod(mdg4) p
68 gpp is an essential gene identified in a
genetic screen for dominant suppressors of pairing-depen
69 A
genetic screen for dominant suppressors of wing blisters
70 morphogenesis, we performed two large-scale
genetic screens for dominant enhancers of the malformed
71 From a
genetic screen for Drosophila melanogaster mutants with
72 We identified moody in a
genetic screen for Drosophila mutants with altered cocai
73 ga Chlamydomonas reinhardtii, we developed a
genetic screen for early components of singlet oxygen si
74 We undertook a forward
genetic screen for effectors of EpiSC reprogramming, emp
75 el activity, as shown by (i) TrIP and (ii) a
genetic screen for effects of the oncogenic suppressors
76 s bearing N-end rule sequences isolated in a
genetic screen for efficient degradation tags.
77 We identified mutations in Rab8 in a
genetic screen for enhancement of an FTD phenotype assoc
78 In a
genetic screen for enhancers of sic-1, we isolated a los
79 mb pathway in plants, we performed a forward
genetic screen for enhancers of the like heterochromatin
80 on of ARK1 (Armadillo-Repeat Kinesin1) via a
genetic screen for enhancers of the rhd3 mutant phenotyp
81 In a
genetic screen for enhancers of the tir1-1 auxin respons
82 (EMC) was identified over a decade ago in a
genetic screen for ER protein homeostasis.
83 A
genetic screen for ethanol sedation mutants in Drosophil
84 Using a
genetic screen for exported proteins in GBS, we identifi
85 We identified HTP-3 in a
genetic screen for factors necessary to maintain SCC unt
86 bly pathways, we have performed a systematic
genetic screen for factors required for centromeric hete
87 induction of ER stress in yeast to enable a
genetic screen for factors that augment stress resistanc
88 cherichia coli, CsrC, was discovered using a
genetic screen for factors that regulate glycogen biosyn
89 s for Pro-16, Asp-18, and Asn-19 followed by
genetic screening for functional proteins.
90 We identified WHSC1 in a
genetic screen for genes involved in responding to repli
91 In a forward
genetic screen for genes regulating endodermal organ dev
92 , and we exploit this phenotype to conduct a
genetic screen for genes required for the homeostatic re
93 We conducted a forward
genetic screen for genes that are required for salivary
94 negative regulators of MEN, we carried out a
genetic screen for genes that are toxic to cdc5-1 mutant
95 We identified Abl in a
genetic screen for genes that contribute to Netrin-depen
96 We identified IRF2 in a CRISPR-based forward
genetic screen for genes that controlled MHC-I Ag presen
97 In a gain-of-function
genetic screen for genes that influence fruit developmen
98 ) gene, which were identified in an unbiased
genetic screen for genes that modify parkin phenotypes.
99 From a
genetic screen for genes that modulate biofilm formation
100 Here we report an RNAi-based
genetic screen for genes that suppress transformation of
101 s initially discovered in budding yeast in a
genetic screen for genes whose deletion confers defects
102 In a
genetic screen for germ-line proliferation-defective mut
103 proteostasis, we performed a tissue targeted
genetic screen for germline modifiers of polyglutamine a
104 Research addressing
genetic screening for hereditary hemochromatosis remains
105 te the benefit from, widespread or high-risk
genetic screening for hereditary hemochromatosis.
106 Limitations: This review considered
genetic screening for HFE-related hereditary hemochromat
107 In a forward
genetic screen for homeostatic plasticity genes, we iden
108 roup (PcG) proteins were first identified in
genetic screens for homeotic transformations in Drosophi
109 Direct comparison to
genetic screens for homologous recombination or Fanconi
110 In a forward
genetic screen for host ERAD components hijacked by US11
111 We identified POS5 in a S.cerevisiae
genetic screen for hyperoxia-sensitive mutants, or cells
112 ation sites in murine tumors to be used as a
genetic screen for identification of large numbers of ca
113 A
genetic screen for identifying additional genes that, wh
114 pathways in Arabidopsis, we describe here a
genetic screen for identifying mutants that display a ch
115 orm and report a set of quantitative forward
genetic screens for identifying regulatory mechanisms of
116 ese data demonstrate high-throughput in vivo
genetic screens for immunotherapy target discovery and e
117 To investigate this, we used a
genetic screen for impaired development to isolate four
118 We performed a
genetic screen for impaired walking in Drosophila and is
119 We performed a forward
genetic screen for imperial pils (imp) mutants that enha
120 ditis elegans homolog of rictor in a forward
genetic screen for increased body fat.
121 mutation, repro5, was isolated in a forward
genetic screen for infertility mutations induced by ENU
122 In a forward
genetic screen for interaction with mitochondrial iron c
123 A yeast
genetic screen for IpaJ substrates identified ADP-ribosy
124 In a
genetic screen for Kinesin heavy chain (Khc)-interacting
125 In addition, in a
genetic screen for klarsicht-interacting genes, we ident
126 We also carried out a large-scale
genetic screen for lethal mutations in the region.
127 During a forward
genetic screen for liver and pancreas mutants, we identi
128 cidate LON2 functions, we executed a forward-
genetic screen for lon2 suppressors, which revealed mult
129 A forward
genetic screen for lysozyme-sensitive mutants led to the
130 se of hematopoietic stem cells to facilitate
genetic screening for malaria host factors.
131 A
genetic screen for markedly enhanced fat storage in tub-
132 regulators of Dpp signaling, we conducted a
genetic screen for maternal-effect suppressors of dpp ha
133 udy, we identified the NIMA kinase Nek4 in a
genetic screen for mediators of the response to Taxol, a
134 A forward
genetic screen for mice with defects in thalamocortical
135 We performed a
genetic screen for micro-RNAs that are differentially ex
136 utant, cassiopeia (csp), was identified by a
genetic screen for mitotic mutant.
137 Using a
genetic screen for modifiers of Drosophila Myc (dMyc)-in
138 In a
genetic screen for modifiers of wingless signaling in th
139 dfxr function in the testes paves the way to
genetic screens for modifiers of dfxr-induced male steri
140 Genetic screening for molecules involved in Shiga toxin
141 Caenorhabditis elegans, which emerged from a
genetic screen for molting-defective mutants sensitized
142 CDC55 was identified in a
genetic screen for monopolins performed by isolating sup
143 In a
genetic screen for mutants affecting endosomal trafficki
144 A
genetic screen for mutants defective for GAL gene memory
145 In a
genetic screen for mutants defective in this noxious hea
146 In an unbiased
genetic screen for mutants exhibiting neurodegeneration
147 idopsis mutant Atcand1-1 that emerged from a
genetic screen for mutants insensitive to sirtinol.
148 In a
genetic screen for mutants lacking this regulation, we i
149 solated a novel allele of hid-1 in a forward
genetic screen for mutants mislocalizing RBF-1 rabphilin
150 an inhibitor of cardioblast development in a
genetic screen for mutants that affect heart development
151 Using a forward
genetic screen for mutants that can sustain hyphal elong
152 mitochondrial protein, was identified with a
genetic screen for mutants that diminish RDD.
153 involved in oxygen toxicity, we conducted a
genetic screen for mutants that display altered survival
154 By performing an unbiased
genetic screen for mutants that impair the somnogenic ef
155 In a
genetic screen for mutants that lack AvrB-dependent chlo
156 In a
genetic screen for mutants that mislocalize the dendriti
157 Using a chemical-
genetic screen for mutants that specifically require the
158 To address this issue, we conducted a
genetic screen for mutants that suppress a partial loss
159 A
genetic screen for mutants that suppress acd6-1-conferre
160 CTN-1 was identified in a
genetic screen for mutants that suppressed a lethargic p
161 In this study, we conducted a
genetic screen for mutants with altered leaf transpirati
162 In a
genetic screen for mutants with defective neuromuscular
163 A Drosophila forward
genetic screen for mutants with defective synaptic devel
164 C. elegans eri-1 was identified in a
genetic screen for mutants with enhanced sensitivity to
165 was previously identified in four different
genetic screens for mutants affecting chromosome transmi
166 In
genetic screens for mutants with disruptions in myelinat
167 From a
genetic screen for mutations able to suppress the bloate
168 From a
genetic screen for mutations affecting early-endosome di
169 We isolated mutations in Liprin-alpha in a
genetic screen for mutations affecting the pattern of sy
170 tion in neural crest, we performed a forward
genetic screen for mutations causing DRG deficiencies in
171 in the optic tectum, we undertook a forward
genetic screen for mutations disrupting visual responses
172 We conducted a
genetic screen for mutations in myospheroid, the gene en
173 We performed a
genetic screen for mutations in the catalytic subunit th
174 A
genetic screen for mutations in thioredoxin that render
175 eterochromatin formation system to perform a
genetic screen for mutations that abolish heterochromati
176 ts of the cell death pathway, we performed a
genetic screen for mutations that abolish the death of t
177 In a
genetic screen for mutations that affect Drosophila eye
178 the mutant motionless (mot), identified in a
genetic screen for mutations that affect neuronal develo
179 In a
genetic screen for mutations that affect the biosynthesi
180 During a
genetic screen for mutations that affect trichome shape,
181 In a forward
genetic screen for mutations that alter intracellular No
182 in tau neurotoxicity, we conducted a forward
genetic screen for mutations that ameliorate tau-induced
183 In a forward
genetic screen for mutations that block PHP we identifie
184 e chromatin or transcription, we performed a
genetic screen for mutations that cause lethality in the
185 Here, we performed a forward
genetic screen for mutations that de-repress Pho1 acid p
186 In a forward
genetic screen for mutations that destabilize the neurom
187 To address this question, we conducted a
genetic screen for mutations that differentially affecte
188 RNA pathway, Pasha and Dicer-1, in a forward
genetic screen for mutations that disrupt wiring specifi
189 A
genetic screen for mutations that dominantly suppress or
190 migration, we conducted a nonbiased forward
genetic screen for mutations that enhanced the nuclear m
191 ing and tissue repair, we have carried out a
genetic screen for mutations that impair regeneration in
192 ed DAM1-765, a dominant allele of DAM1, in a
genetic screen for mutations that increase stress on the
193 We performed an unbiased
genetic screen for mutations that permit the survival of
194 In a
genetic screen for mutations that restrict cell growth a
195 In a forward
genetic screen for mutations that result in loss of habe
196 nsduction pathway(s), we conducted a forward
genetic screen for mutations that suppressed edr1-mediat
197 Our
genetic screening for mutations that resist CLE peptide
198 a valuable resource for forward and reverse
genetic screens for mutations affecting a wide array of
199 To identify these proteins, we carried out
genetic screens for mutations affecting Drosophila melan
200 Unbiased forward
genetic screens for mutations causing increased gross ch
201 ndant functions of LIN-35 were identified in
genetic screens for mutations that display synthetic phe
202 Forward
genetic screens for mutations that rescue the paralysis
203 Genetic screens for mutations that result in increased p
204 From a forward
genetic screen for myelination defects in zebrafish, we
205 In an ongoing forward
genetic screen for N-ethyl-N-nitrosourea (ENU)-induced m
206 A
genetic screen for negative regulators of Mis4 yielded a
207 A
genetic screen for negative regulators of olfaction unco
208 proteins, FitA and FitB, was identified in a
genetic screen for Neisseria gonorrhoeae determinants th
209 o understand this process, here we conduct a
genetic screen for nematodes defective in transmitting R
210 approaches have hindered systematic forward
genetic screening for NMD factors in human cells.
211 Fireworks) that enables CRISPR-based forward
genetic screening for NMD pathway defects in human cells
212 ri1 allele, bri1-5, in an activation-tagging
genetic screen for novel brassinosteroid (BR) signal tra
213 Using a murine forward
genetic screen for novel determinants of axon guidance,
214 We further performed an unbiased
genetic screen for novel modifiers of instability.
215 FAM83B was recently discovered in a forward
genetic screen for novel oncogenes that drive human mamm
216 In a
genetic screen for novel TOR interactors in Drosophila m
217 8-1) mutant was identified in an independent
genetic screen for NPC assembly (npa) mutants.
218 Patients underwent
genetic screening for NPM1, FLT3-ITD, FLT3-D835, and CEB
219 Furthermore, a
genetic screen for NTZ-resistant bacterial mutants isola
220 In this study, in a genome-wide
genetic screen for other ion channel subunits required f
221 tumour suppressor gene by using a mammalian
genetic screen for p53-dependent genes involved in tumor
222 2delta-2), were also identified in a forward
genetic screen for pain genes (alpha2delta-3).
223 In a
genetic screen for pans1 suppressors, we identified SEPA
224 r during muscle degeneration and performed a
genetic screen for parkin modifiers.
225 Using a
genetic screen for PCL defect, we identified a mutation
226 From a forward
genetic screen for phagocytosis mutants in Drosophila me
227 In a
genetic screen for picloram resistance, we identified th
228 atopoietic stem cells to carry out a forward
genetic screen for Plasmodium falciparum host determinan
229 This observation instigated further
genetic screening for prostacyclin receptor variants on
230 brain 4.1 proteins, in yeast two-hybrid and
genetic screens for proteins that interact with and loca
231 is thaliana mutant smd1b was identified in a
genetic screen for PTGS deficiency, revealing the involv
232 i named PEANUT1-5 (PNT) were identified in a
genetic screen for radially swollen embryo mutants.
233 In a
genetic screen for rapamycin-sensitive mutations, we iso
234 d in axon navigation, we conducted a forward
genetic screen for recessive alleles affecting motor neu
235 In the course of a forward
genetic screen for recessive mouse mutants, we identifie
236 Genetic screens for recessive mutations continue to prov
237 In a forward
genetic screen for regulators of C-REPEAT BINDING FACTOR
238 In a forward
genetic screen for regulators of C. elegans PKD-2 ciliar
239 n minus-end directed microtubule motor, in a
genetic screen for regulators of EGFR signaling.
240 A previous
genetic screen for regulators of inv identified RovA, wh
241 In a
genetic screen for regulators of muscle development in D
242 We performed a
genetic screen for regulators of RAN translation and ide
243 ore this facet of myogenesis, we performed a
genetic screen for regulators of somatic muscle morpholo
244 In a
genetic screen for regulators of synaptic morphology, we
245 uction have already been defined, continuous
genetic screening for regulators of innate immunity may
246 The par genes were discovered in
genetic screens for regulators of cytoplasmic partitioni
247 We carried out two forward
genetic screens for regulators of endodermal organ devel
248 A
genetic screen for relevant mutations in Drosophila gene
249 A
genetic screen for resistance to ethanol intoxication in
250 entified EDM2 (enhanced downy mildew 2) in a
genetic screen for RPP7 suppressors.
251 Here, we describe two independent
genetic screens for rsc suppressors that yielded mutatio
252 In a
genetic screen for Saccharomyces cerevisiae mutants hype
253 challenges faced by families as a result of
genetic screening for SADS to enable equitable access to
254 To identify salt tolerance determinants, a
genetic screen for salt overly sensitive (sos) mutants w
255 of the MVA pathway in plants, we performed a
genetic screen for second-site suppressor mutations of t
256 From a
genetic screen for second-site suppressors of the DNA de
257 m therapeutic entry points, we carried out a
genetic screen for secondary mutations that improved phe
258 mechanisms we performed a GFP-based forward
genetic screen for seedling-lethal biosynthetic membrane
259 In independent
genetic screens, for shade-avoidance response and cytoki
260 required for silencing in S. bayanus using a
genetic screen for silencing-defective mutants.
261 A
genetic screen for similar lumbar maintenance mutants re
262 ated gene disruption procedure and performed
genetic screening for single P-element insertion mutatio
263 In a chemical
genetic screen for small molecules that dampened the inh
264 A chemical
genetic screen for small molecules that suppress growth
265 al genes involved in body size regulation, a
genetic screen for small mutants was previously performe
266 We performed a
genetic screen for spoIIE mutants that were impaired in
267 These elements were identified in a
genetic screen for spontaneous mutations that caused col
268 In a
genetic screen for Staphylococcus aureus secreted virule
269 lated paf1 and leo1 mutations in an unbiased
genetic screen for suppressors of a cold-sensitive spt5
270 In a
genetic screen for suppressors of a lethargic phenotype
271 Here we describe a
genetic screen for suppressors of a postrecruitment-defe
272 on (lf) mutants of hrpu-2 were isolated in a
genetic screen for suppressors of a sluggish phenotype c
273 A
genetic screen for suppressors of BS A-to-G mutants, whi
274 By deploying a
genetic screen for suppressors of cell death triggered b
275 rt machinery component Tic40, we performed a
genetic screen for suppressors of chlorotic tic40 knocko
276 From a forward
genetic screen for suppressors of heat-shock-induced gen
277 In a
genetic screen for suppressors of reduced neurotransmitt
278 Here, we report the results of a forward
genetic screen for suppressors of ref4-3.
279 In a
genetic screen for suppressors of sni1, we discovered th
280 A
genetic screen for suppressors of the Arabidopsis bri1-9
281 In a
genetic screen for suppressors of the ibm2 mutation, we
282 y and chromosome segregation, we performed a
genetic screen for suppressors of the increase-in-ploidy
283 In a
genetic screen for suppressors of the SIN mutant sid2-25
284 Genetic screens for suppressors of SWI6 mutants have bee
285 ) component vha100-1 in flies in an unbiased
genetic screen for synaptic malfunction.
286 In a Drosophila
genetic screen for synaptogenesis mutants, we identified
287 Genetic screens for synaptogenesis mutants have been per
288 A
genetic screen for synergistic increase in fat storage o
289 nd-P metabolism is defective, we developed a
genetic screen for synthetic interactions which, in comb
290 Here, via a forward
genetic screen for TAG homeostasis, we isolated a Chlamy
291 Here, we report a
genetic screen for the enhancement of maize inflorescenc
292 In this study, we developed a forward
genetic screen for the identification of host factors re
293 Using a confocal microscopy forward
genetics screen for the identification of Arabidopsis th
294 Currently, there is no effective therapy or
genetic screens for these diseases; however, nuclear gen
295 is important for basic and medical research;
genetic screening for those genes in Caenorhabditis eleg
296 )) has also been identified in C. elegans in
genetic screens for touch insensitivity (MEC-2(P134S)).
297 In a
genetic screen for transcription factors regulating sene
298 This work provides a sensitive
genetic screen for uncovering auxin-resistant mutants in
299 e results of a large-scale, microscopy-based
genetic screen for Vibrio cholerae mutants that are defe
300 t influence liver lipid mass, we performed a
genetic screen for zebrafish mutants with hepatic steato