ライフサイエンスコーパス: genital ulcer disease

1 Haemophilus ducreyi causes chancroid, a genital ulcer disease.

2 uction contributing to the pathogenesis of a genital ulcer disease.

3 creyi is the etiologic agent of chancroid, a genital ulcer disease.

4 HSV-2) infection is the most common cause of genital ulcer disease.

5 c agent of chancroid, a sexually transmitted genital ulcer disease.

6 asma HIV-1 RNA levels among, and presence of genital ulcer disease among HIV-1-infected partners and

7 irus type 2, human papillomavirus (HPV), and genital ulcer disease among men, and it reduces HPV, gen

8 HSV-2 infection is a common cause of genital ulcer disease and a significant public health co

9 uld have additional benefits beyond reducing genital ulcer disease and HSV-associated HIV transmissio

10 herpes simplex virus (HSV) type 2-associated genital ulcer disease and lesional HSV shedding.

11 Among 40 consecutive patients with genital ulcer disease and with sufficient sample for bot

12 mples from patients with chancroid and other genital ulcer diseases and from normal subjects containe

13 on, and only at the STI clinic were marital, genital ulcer disease, and HIV-infection status associat

14 Sexually transmitted diseases, genital ulcer disease, and progesterone therapy increase

15 h and without sexually transmitted diseases, genital ulcer disease, and progesterone-predominant cond

16 d with increased risk of HIV-1 infection and genital ulcer disease, and these effects remained after

17 s who had an AIDS-defining illness or active genital ulcer disease, and those that were taking antire

18 - and late-stage infection, higher HIV load, genital ulcer disease, and younger age of the index part

19 vir had a smaller effect on the frequency of genital ulcer disease as well as a smaller effect on the

20 chancroid, a sexually transmitted cutaneous genital ulcer disease associated with increased heterose

21 ulcer disease among men, and it reduces HPV, genital ulcer disease, bacterial vaginosis, and trichomo

22 eople by reducing the prevalence of herpetic genital ulcer disease, but could also have an additional

23 Measuring incidence density in the genital ulcer disease cases directly gave the highest es

24 reening), and a cohort study of seronegative genital ulcer disease cases were compared.

25 us lesions of the human sexually transmitted genital ulcer disease chancroid are characterized by the

26 i, a Gram-negative bacterium that causes the genital ulcer disease chancroid, activates inflammasomes

27 philus ducreyi, the etiological agent of the genital ulcer disease chancroid, binds extracellular mat

28 etiologic agent of the sexually transmitted genital ulcer disease chancroid, has been shown to assoc

29 Haemophilus ducreyi, which causes the genital ulcer disease chancroid, requires high basal lev

30 etiologic agent of the sexually transmitted genital ulcer disease chancroid.

31 tive obligate human pathogen that causes the genital ulcer disease chancroid.

32 i is a hemin-requiring bacterium causing the genital ulcer disease chancroid.

33 to assess the possible benefits of treating genital ulcer disease, chorioamnionitis, mastitis, and m

34 ubjects and patients with chancroid or other genital ulcer diseases contained antibodies to purified

35 We evaluated genital ulcer disease (GUD) and HSV-2-associated GUD at

36 higher virus set point, and the presence of genital ulcer disease (GUD) during the early phase of HI

37 es simplex virus type 2 (HSV-2) shedding and genital ulcer disease (GUD) has not been evaluated.

38 dard laboratory methods for the diagnosis of genital ulcer disease (GUD) in 105 patients; 36% were hu

39 d with cervicovaginal HSV-2 DNA shedding and genital ulcer disease (GUD) in a cohort of women living

40 Viremia was increased with genital ulcer disease (GUD) in both subjects with incide

41 Monocytes recruited to genital ulcer disease (GUD) sites express increased leve

42 ltaneously detects the three major causes of genital ulcer disease (GUD), Haemophilus ducreyi, Trepon

43 uman immunodeficiency virus (HIV) infection, genital ulcer disease (GUD), penile epithelial trauma, m

44 tive men with urethritis and with or without genital ulcer disease (GUD).

45 roid has dramatically declined as a cause of genital ulcer disease (GUD).

46 pecimens obtained from 163 patients (96 with genital ulcer disease [GUD]).

47 ates for genital-tract infections (syphilis, genital ulcer disease [GUD], Neisseria gonorrhoeae infec

48 Genital ulcer disease has been epidemiologically linked

49 ; 95% confidence interval [CI], 1.9-8.3) and genital ulcer disease (HRR=2.5; 95% CI, 1.1-5.3).

50 eyi causes chancroid, a sexually transmitted genital ulcer disease implicated in increased heterosexu

51 on an increasingly important role in causing genital ulcer disease in addition to being the primary n

52 ed diseases and are the most common cause of genital ulcer disease in the United States.

53 emophilus ducreyi, is one of the most common genital ulcer diseases in developing countries.

54 ducreyi, the etiologic agent of chancroid, a genital ulcer disease, produces a cell-associated hemoly

55 Transmission of HIV was associated with genital ulcer disease, syphilis, and vaginal or penile d

56 c agent of chancroid, a sexually transmitted genital ulcer disease that facilitates the transmission

57 Haemophilus ducreyi causes chancroid, a genital ulcer disease that facilitates the transmission

58 In multivariate models, vulvitis, genital ulcer disease, vaginal discharge, and Candida va

59 es simplex virus is the most common cause of genital ulcer disease worldwide.