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1 roper mitotic progression and maintenance of genomic stability.
2 anscriptional regulation, and maintenance of genomic stability.
3 by resolving replicative stress to maintain genomic stability.
4 e 1 (FEN1) removes the 5'-flap and maintains genomic stability.
5 formation is critical for the maintenance of genomic stability.
6 A and PUMILIO proteins in the maintenance of genomic stability.
7 ericentric H3K9 trimethylation (H3K9me3) and genomic stability.
8 stress and contribute to the maintenance of genomic stability.
9 in a dynamic yet regulated manner to ensure genomic stability.
10 y both result from mutations that compromise genomic stability.
11 double-strand breaks (DSBs) is essential for genomic stability.
12 ir protein, which plays an important role in genomic stability.
13 critical role of MMR in maintaining general genomic stability.
14 NBN, plays an important role in maintaining genomic stability.
15 feration, meiosis, and DNA repair to control genomic stability.
16 ell cycle progression and the maintenance of genomic stability.
17 the genome, with important implications for genomic stability.
18 mosomal segregation during mitosis to ensure genomic stability.
19 sor p53 plays a critical role in maintaining genomic stability.
20 bed as playing a prominent role in promoting genomic stability.
21 ch forks is essential for the maintenance of genomic stability.
22 criptional regulation and the maintenance of genomic stability.
23 activity of retrotransposons that may alter genomic stability.
24 ome regulates the replication checkpoint and genomic stability.
25 king on MMR signaling processes critical for genomic stability.
26 tudies have implicated Apc in the control of genomic stability.
27 mitigating replication stress and promoting genomic stability.
28 isite for replication fork establishment and genomic stability.
29 etic and epigenetic features and to preserve genomic stability.
30 iR-155 levels improved telomere function and genomic stability.
31 cycle and ultimately for the maintenance of genomic stability.
32 nts between alleles is important to maintain genomic stability.
33 chromosome segregation and thereby maintain genomic stability.
34 RecQ[Bs]'s possible functions in preserving genomic stability.
35 ouble strand break repair and maintenance of genomic stability.
36 itical for homologous recombination (HR) and genomic stability.
37 which MK2 contributes to the maintenance of genomic stability.
38 e function is critical for cell division and genomic stability.
39 ctions in metabolism, stress resistance, and genomic stability.
40 nucleus, which is important for maintaining genomic stability.
41 e that regulates transcription and maintains genomic stability.
42 airing DNA replication forks and for overall genomic stability.
43 of genes involved in cell growth control and genomic stability.
44 nd is essential for proper cell division and genomic stability.
45 c recombination intermediates that undermine genomic stability.
46 logous recombination, a process that ensures genomic stability.
47 A helicase which is critical for maintaining genomic stability.
48 orchestrate DNA repair, thereby maintaining genomic stability.
49 ipolar mitotic spindle and are essential for genomic stability.
50 aks (DSBs) are dangerous lesions threatening genomic stability.
51 ught to encompass a defect in maintenance of genomic stability.
52 int and, thus, contributes to maintenance of genomic stability.
53 ogous recombination (HR) and is critical for genomic stability.
54 r day and is critical for the maintenance of genomic stability.
55 ccurs in human cancers, may adversely affect genomic stability.
56 f metabolism, longevity, gene expression and genomic stability.
57 in T-cell development and the maintenance of genomic stability.
58 egrity is critical for telomere function and genomic stability.
59 ously unknown property of the MRN complex in genomic stability.
60 n of Fanconi anaemia proteins is to maintain genomic stability.
61 ting the DNA damage response and maintaining genomic stability.
62 us recombination (HR) and the maintenance of genomic stability.
63 hanced transcription activities and impaired genomic stability.
64 of gene expression, chromatin structure, and genomic stability.
65 olecular target by which autophagy maintains genomic stability.
66 a non-canonical function of ATRX in guarding genomic stability.
67 esponses to DNA damage that help to maintain genomic stability.
68 n, WRN, are involved in maintaining cellular genomic stability.
69 dulated the DNA damage response and affected genomic stability.
70 chromosome segregation and thereby maintain genomic stability.
71 erochromatin and euchromatin is essential to genomic stability.
72 NA damage checkpoints and the maintenance of genomic stability.
73 NA repair that are essential for maintaining genomic stability.
74 r suppression by Bre1 through maintenance of genomic stability.
75 anaphase inhibitors to efficiently preserve genomic stability.
76 roups for DNA methylation, both critical for genomic stability.
77 onstrating a link between WRN, telomeres and genomic stability.
78 localizes to the nucleus and contributes to genomic stability.
79 on and thus contribute to the maintenance of genomic stability.
80 ential for cell viability and maintenance of genomic stability.
81 ucidate an important pathway for maintaining genomic stability.
82 the alcohol effect on DNA damage/repair and genomic stability.
83 th pro- and anti-inflammatory processes, and genomic stability.
84 oss-link (ICL) repair and the maintenance of genomic stability.
85 air, and it is important for preservation of genomic stability.
86 damage, and it is necessary for maintaining genomic stability.
87 efore nuclear envelope breakdown to maintain genomic stability.
88 n shaping chromatin landscape and preserving genomic stability.
89 undly impairs the global DNA methylation and genomic stability.
90 lation donor for NHEJ repair and maintaining genomic stability.
91 gY, demonstrating that DFMO directly affects genomic stability.
92 role for Ca(2+) influx in the maintenance of genomic stability.
93 a role for this modification in safeguarding genomic stability.
94 R/Chk1 pathway activation and helps maintain genomic stability.
95 licase important in telomere maintenance and genomic stability.
96 les template-directed DNA repair to maintain genomic stability.
97 hromosome segregation and the maintenance of genomic stability.
98 kinase pathway is important for maintaining genomic stability.
99 ion is of crucial importance for maintaining genomic stability.
100 gregation is required for the maintenance of genomic stability.
101 on from repetitive DNA sequences and ensures genomic stability.
102 be coordinated with cytokinesis to preserve genomic stability.
103 be coordinated with cytokinesis to preserve genomic stability.
104 hat POT1, like SSB1, is required to maintain genomic stability.
105 that PI3K-C2alpha expression is required for genomic stability.
106 thylation that influence gene expression and genomic stability.
107 tion that is critical for the maintenance of genomic stability.
108 ansformation in the pancreas via maintaining genomic stability.
109 apoptosis; vascular tone; host defenses; and genomic stability.
110 n of replication initiation events to insure genomic stability.
111 pair of DNA double-strand breaks to maintain genomic stability.
112 roliferation-quiescence decision to maintain genomic stability.
113 te cAMP signaling as a critical regulator of genomic stability against platinum-induced mutagenesis.
115 ortant role for RECQL5 in the maintenance of genomic stability and a new insight into the decatenatio
117 V contribute to lymphomagenesis by affecting genomic stability and by subverting the cellular molecul
123 ng cell division; however, its importance to genomic stability and cellular fitness is not understood
124 the possible influence of such structures on genomic stability and cellular processes, such as transc
125 liferated vigorously while maintaining their genomic stability and could be redifferentiated in vitro
126 osome segregation is crucial for maintaining genomic stability and dependent on separase, a conserved
133 w a RAD51 paralog is involved in maintaining genomic stability and how its deficiency may predispose
138 air of DNA alkylation damage is critical for genomic stability and involves multiple conserved enzyma
140 ing yeast F-box protein Dia2 is required for genomic stability and is targeted for ubiquitin-dependen
141 ether with its known function in maintaining genomic stability and its mislocalization in cancers, su
143 olved in regulatory pathways for maintaining genomic stability and play important roles in regulating
146 s the telomerase RNA component TERC, confers genomic stability and promotes DNA repair, which have co
147 somatic homologous recombination, providing genomic stability and promoting resistance to DNA damage
149 howcase the influence of repeat sequences on genomic stability and structural variant complexity and
150 DNA replication is vital for maintenance of genomic stability and suppression of cancer development
151 re each critical for maintenance of cellular genomic stability and suppression of lymphomas harboring
152 linked to its function in the maintenance of genomic stability and suppression of oxidative damage.
154 ns of cells in a few weeks while maintaining genomic stability and the ability to undergo multipotent
156 equirement for methylation in maintenance of genomic stability and the integrity of both the tubulin
157 a key pathway involved in the maintenance of genomic stability and the prevention of oncogenic transf
158 e examine the effect of the loss of RAP80 on genomic stability and the susceptibility to cancer devel
160 Biological self-defense systems to ensure genomic stability and to eliminate tetraploid cells exis
163 Telomere length homeostasis is essential for genomic stability and unlimited self-renewal of embryoni
165 serve as surveillance mechanisms to maintain genomic stability, and are regulated by ATM/ATR-mediated
167 (RE) suppresses their mobility and maintains genomic stability, and decreases in it are frequently ob
169 ower proportion of UV mutagenesis, increased genomic stability, and harbor fewer functionally resista
170 of DNA double-strand breaks, maintenance of genomic stability, and prevention of developmental disor
172 ssed in MM cells, as an adaptive response to genomic stability, and that high SIRT6 levels are associ
173 into the relationship between mitochondria, genomic stability, and tumor suppressive control, with i
174 (1) blocking differentiation; (2) impairing genomic stability; and (3) increasing self-renewal in he
175 mediates and cellular contexts that endanger genomic stability are discussed as well as the emerging
176 pluripotency, differentiation potential, and genomic stability are typically maintained during the cl
177 d with control of cell-cycle progression and genomic stability as well as production of Th2 cytokines
178 iption factor involved in the maintenance of genomic stability, as a functional and physical interact
180 pport its role in the overall maintenance of genomic stability at sites of alternatively structured D
181 We therefore propose that ZRANB3 maintains genomic stability at stalled or collapsed replication fo
182 findings suggest that nuclear FMRP regulates genomic stability at the chromatin interface and may imp
185 ect temporal regulation of mitosis underpins genomic stability because it ensures the alignment of ch
186 53 tumor suppressor is a pivotal guardian of genomic stability, but its contributions to epigenetic r
187 suppression function that normally maintains genomic stability by controlling the epigenetic landscap
188 spindle assembly checkpoint (SAC) maintains genomic stability by delaying chromosome segregation unt
189 Thus, the REV1/Polzeta complex maintains genomic stability by directly participating in DSB repai
190 s are important editing enzymes that protect genomic stability by excising chemically modified nucleo
191 ymerization by Pol alpha might contribute to genomic stability by limiting the amount of inaccurate D
192 t PUM binding is required for maintenance of genomic stability by NORAD whereas binding of RBMX is di
193 te the mechanism by which TRIP/NOPO promotes genomic stability by performing a yeast two-hybrid scree
194 ween guanines in G-rich sequences, threatens genomic stability by perturbing normal DNA transactions
195 he Spindle Assembly Checkpoint (SAC) ensures genomic stability by preventing sister chromatid separat
200 have important implications for DNA repair, genomic stability, carcinogenesis and aging in human cel
201 itical in the regulation of gene expression, genomic stability, cell cycle and nuclear architecture.
202 56 acetylation is important for replication, genomic stability, chromatin assembly, and the response
203 gulation of both the DNA damage response and genomic stability, culminating in increased susceptibili
204 Proper chromatin function and maintenance of genomic stability depends on spatiotemporal coordination
205 chromatin must distort and somehow maintain genomic stability despite ever-present double-strand bre
207 a novel role for USP9X in the maintenance of genomic stability during DNA replication and provide pot
210 e an essential role for Dnmt1 in maintaining genomic stability during intestinal development and the
212 the existence of an error threshold limiting genomic stability during such transitions, but does not
213 ed clonal lines displayed robust morphology, genomic stability, expression and localization of the ta
214 e that lamin A/C is required for maintaining genomic stability following replication fork stalling, i
215 t aspects of bacterial physiology, including genomic stability, formation of persister cells under an
216 r vascular functionality, maintained greater genomic stability, harbored decreased lineage-primed gen
217 ipate in the DNA damage response to maintain genomic stability has expanded significantly to include
218 s of genes that are required for maintaining genomic stability have traditionally relied on reporter
219 erhouses, and telomeres, which help maintain genomic stability, have been implicated in cancer and ag
221 oints, Atm activation, or the maintenance of genomic stability in B lymphocytes and primary fibroblas
224 of developing thymocytes, and maintenance of genomic stability in cycling alphabeta T-lineage cells.
229 fication of Filia as a specific regulator of genomic stability in mouse embryonic stem cells (ESCs).
230 cs in normal mouse development, promotion of genomic stability in mouse fibroblasts, and in IgH class
231 eptional and prenatal toxicant exposures for genomic stability in offspring is difficult to analyze i
233 r understanding of the factors that regulate genomic stability in PSCs could help address this issue.
234 acetylase SirT1 regulates gene silencing and genomic stability in response to nutrient deprivation an
235 Dnmt1 maintains DNA methylation patterns and genomic stability in several in vitro cell systems.
236 bination (HR) is a central process to ensure genomic stability in somatic cells and during meiosis.
237 However, the role of p21 in maintaining genomic stability in the absence of exogenous DNA-damagi
238 long term ethanol exposure on DNA repair and genomic stability in the brain results from OCM dysfunct
241 exposures during gestation and conception on genomic stability in the offspring, stability was assess
244 tes multiple cellular pathways that maintain genomic stability including cell cycle checkpoints, DNA
245 S) cluster family of helicases important for genomic stability including XPD (nucleotide excision rep
246 homeoprotein involved in early development, genomic stability, insulin sensitivity, and hematopoiesi
247 molecule targeting of pathways that maintain genomic stability is an attractive chemotherapeutic appr
250 Maintenance of proper chromatin states and genomic stability is vital for normal development and he
252 gase BRCA1 is a key player in maintenance of genomic stability, its role in growth signaling remains
255 emain as important mechanisms for preserving genomic stability, metabolic functions of p53 show incre
258 have established that ATM and MDC1 maintain genomic stability not only by controlling the DNA damage
259 tes DNA double-strand break (DSB) repair and genomic stability not only in c-NHEJ-proficient but also
260 function of nucleostemin in maintaining the genomic stability of actively dividing stem/progenitor c
263 he passaged viruses, demonstrating long-term genomic stability of complex VSV recombinants carrying l
265 understanding of the mechanisms maintaining genomic stability of hESC and our ability to modulate th
267 st that MYB potentially regulates growth and genomic stability of pancreatic cancer cells via targeti
272 int and, thus, contributes to maintenance of genomic stability.Oncogene advance online publication, 1
273 detectably affect resection, maintenance of genomic stability or viability, whereas T847 is essentia
274 ablish that the cellular Fanconi anemia (FA) genomic stability pathway is exploited by herpes simplex
275 modification involved in the maintenance of genomic stability, preservation of cellular identity, an
276 Although RAD51AP1 is involved in maintaining genomic stability, recent studies have shown that RAD51A
279 DNA damage response is essential to maintain genomic stability, suppress replication stress, and prot
280 nces inflammatory tumorigenesis by affecting genomic stability, the inflammatory microenvironment, an
281 , which are essential for the maintenance of genomic stability, these proteins may synergistically fu
282 amentally significant role in maintenance of genomic stability through a DDR-independent pathway.
283 uppressor protein that functions to maintain genomic stability through critical roles in DNA repair,
285 a partner of BRCA1 and BRCA2 in maintaining genomic stability through homologous recombination (HRR)
286 icate that Tpx2 is essential for maintaining genomic stability through its role in spindle regulation
287 an important player not only in maintaining genomic stability through NHEJ-dependent functions, but
288 er, the malignant clone often re-establishes genomic stability through overexpression of telomerase.
289 nserved antimutagenic pathway that maintains genomic stability through rectification of DNA replicati
291 gation, the Arg/N-end rule pathway regulates genomic stability through the degradation-mediated contr
292 echanism to maintain centromere identity and genomic stability through the FACT-mediated degradation
295 fine balance between genetic variability and genomic stability tunes plasticity of these chromosomal
296 d homeostasis and is involved in maintaining genomic stability under conditions of oxidative DNA dama
298 multi-subunit polymerase that contributes to genomic stability via its roles in leading strand replic
299 ge-induced cell death for the maintenance of genomic stability, we examined whether Nfkb1 acts as a t
300 racting RNAs (piRNAs) have a central role in genomic stability, which is inextricably linked to germ-