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1 transcriptional programme similar to that of germ cell tumours.
2 chemotherapy-induced apoptosis in testicular germ cell tumours.
3 elatively frequent loss of heterozygosity in germ cell tumours.
4 ng response assessment for patients with CNS germ cell tumours.
5 better international clinical trials for CNS germ cell tumours.
6 miology, genetics, and biology of testicular germ cell tumours.
7 s in the clinical management of intracranial germ-cell tumours.
8 cal management of patients with intracranial germ-cell tumours.
9 progression-free survival for patients with germ-cell tumours.
10 maeric embryos, and is associated with human germ-cell tumours.
11 cryptorchidism, risk factors for testicular germ-cell tumours.
12 34 patients with cancer, 53% had testicular germ-cell tumours.
13 eview the assessment and management of early germ-cell tumours.
14 e of approaches offer high survival in early germ-cell tumours.
15 tility and increases risk for development of germ-cell tumours.
17 ere defined for the most common sites of CNS germ cell tumour and for the definition of locoregional
18 ise the consensus on how to treat testicular germ cell tumours and focus on a few controversies and i
20 ntly overexpressed in human non-seminomatous germ cell tumours and transitional carcinoma of the blad
21 ion or death in patients with poor prognosis germ-cell tumours and an unfavourable tumour marker decl
31 the results obtained in treating metastatic germ-cell tumours are superior to those with other solid
32 operitoneal, or mediastinal non-seminomatous germ cell tumours based on histological findings or clin
36 a indicating fewer second primary testicular germ-cell tumours favour carboplatin use, these findings
41 rasellar masses, such as craniopharyngiomas, germ cell tumours, gliomas, cysts of Rathke's pouch and
42 stratification, and treatment approaches for germ-cell tumours have evolved disparately along several
46 of in certain pathological contexts such as germ-cell tumours, melanoma or human immunodeficiency vi
47 Fewer than 70 new cases of malignant ovarian germ cell tumours (MOGCTs) are seen each year in the UK.
48 rs, the malignant cells in 73% of testicular germ-cell tumours (seminomas and teratomas), expressed h
49 ovarian cancers, including malignant ovarian germ cell tumours, sex cord-stromal tumours, and small c
50 Tumours within this group are testicular germ-cell tumours (such as benign teratoma, epidermoid c
51 icipants of the 2013 Third International CNS Germ Cell Tumour Symposium (Cambridge, UK) agreed to und
54 identified multiple risk loci for testicular germ cell tumour (TGCT), revealing a polygenic model of
64 es that control susceptibility to testicular germ-cell tumours (TGCTs), the most common cancer affect
67 of response to therapy in patients with CNS germ cell tumours to be more consistent, and facilitate
69 tements encompassing aspects of intracranial germ-cell tumour work-up, staging, treatment, and follow