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1 were more susceptible to infection than were glandular epithelial cells.
2 mmunoglobulin receptor (pIgR) on mucosal and glandular epithelial cells.
3 to the androgen-responsiveness of prostatic glandular epithelial cells.
4 DNA was present in the nuclei of >90% of the glandular epithelial cells.
5 seen predominantly within the carcinomatous glandular epithelial cells.
6 ar stroma in normal prostates but not in the glandular epithelial cells.
7 ymphocytes, autoantibodies, interferons, and glandular epithelial cells.
8 monstrated previously that the protein GEC1 (glandular epithelial cell 1) bound to the human kappa op
9 aran sulfate in the apical region of uterine glandular epithelial cells and can be released by hepari
11 was present primarily in uterine luminal and glandular epithelial cells and in the epithelial cells o
12 oblasts, indicating that alterations in both glandular epithelial cells and their surrounding microen
13 icrobes interact with host mucins lining the glandular epithelial cells and trigger inflammation.
14 responses, was upregulated in the uterus, on glandular epithelial cells, and adjacent to glands in re
17 ction of HESCs, HEECs, and human endometrial glandular epithelial cells followed by real time, quanti
22 ates, cytokine production by lymphocytes and glandular epithelial cells, neuroendocrine and hormonal
23 PA1 expression was predominately detected in glandular epithelial cells of prostate by in situ hybrid
24 opin receptor, PROK1, PROKR1, and LIF to the glandular epithelial cells of the first trimester decidu
27 Chlamydial infection was detected in the glandular epithelial cells, potentially leading to infla
28 The intermediate filament network in simple glandular epithelial cells predominantly consists of het
29 nderstand how HCMV is replicating within the glandular epithelial cells so that it might be possible
30 further revealed the cytoplasm of the tumor glandular epithelial cells to be strongly positive for c