ライフサイエンスコーパス: glia

1 is known about integrin's regulators in the glia.

2 that functions to prevent the overgrowth of glia.

3 gment epithelium (RPE) and/or retinal Muller glia.

4 ell adhesion molecule that binds to ClC-2 in glia.

5 eneration via distinct molecular pathways in glia.

6 nd deubiquitinase SAGA complex in peripheral glia.

7 l accumulation of tau protein in neurons and glia.

8 for genes expressed in interneurons, but not glia.

9 s and cytosol of their surviving neurons and glia.

10 lls but little to no infection of neurons or glia.

11 nervous system, CLC-2 appears in neurons and glia.

12 es in the IFN-alpha/beta pathway compared to glia.

13 zone, and decreased and disorganized Muller glia.

14 BP1 promotes Cd40 mRNA translation in Muller glia.

15 eural crest-derived lineages and myelinating glia.

16 expression of interferon-inducible genes in glia.

17 where it localizes to the axons and midline glia.

18 rinsically and also extrinsically by retinal glia.

19 m (RPE) and limited expression in the Muller glia.

20 sualize the response of microglia and Muller glia.

21 translocator protein (TSPO), a biomarker of glia.

22 se isogenic comparisons of human neurons and glia.

23 d layers including amacrine cells and Muller glia.

24 Infection was also increased in glia.

25 m most types of cells, including neurons and glia.

26 been implicated as cells of origin, as have glia.

27 ty of cells to differentiate into neurons or glia.

28 endritic shafts (47.9 +/- 1.2%), followed by glia (37.7 +/- 2.5%), and dendritic spines (14.3 +/- 2.6

29 ith a conditionally inactivated Tsc1 gene in glia, 8 significantly reduced the loss of Tsc1-induced m

30 Changes in other brain cells (glia) across the sleep-wake cycle and their role in slee

31 glial cytoplasm, indicating that phagocytic glia act as obligatory intermediates in aggregate spread

32 Myelin destruction is followed by resident glia activation and mobilization of endogenous progenito

33 revented 5-FU-induced neuronal loss, enteric glia activation, intestinal inflammation, oxidative stre

34 n SCI, via beneficial effects on neurons and glia after injury to facilitate axonal outgrowth.SIGNIFI

35 pathway are dynamically regulated by Muller glia after neuronal damage or treatment with growth fact

36 HLA-DQ2.5-restricted gluten epitopes, DQ2.5-glia-alpha1a (PFPQPELPY) and DQ2.5-glia-omega1 (PFPQPEQP

37 e the most abundant cells that present DQ2.5-glia-alpha1a in the inflamed mucosa.

38 ting the immunodominant gluten peptide DQ2.5-glia-alpha1a in the tissues from these patients.

39 the 33-mer and peptides containing the DQ2.5-glia-alpha1a/DQ2.5-glia-alpha2 (P1), DQ2.5-glia-alpha3 (

40 ides containing the DQ2.5-glia-alpha1a/DQ2.5-glia-alpha2 (P1), DQ2.5-glia-alpha3 (P2) and DQ2.5-glia-

41 5-glia-alpha1a/DQ2.5-glia-alpha2 (P1), DQ2.5-glia-alpha3 (P2) and DQ2.5-glia-gamma1 (P3) epitopes, in

42 oencephalopathy with spheroids and pigmented glia (ALSP) is an autosomal dominant leukoencephalopathy

43 veal remarkable similarities between enteric glia and brain neural stem cells in teleosts and open ne

44 ese requirements are partially met by Muller glia and cells of the retinal pigment epithelium (RPE),

45 a, we found Jedi-1 expression in perineurial glia and endothelial cells, but not in sensory neurons.

46 rized its cellular targets including neuron, glia and endothelial cells.

47 f the ventral nerve cord, compression of the glia and extracellular matrix in the peripheral nerves,

48 through regulation of the relative timing of GliA and GliR production, which is important in understa

49 synapses when cultured with mouse cerebellar glia and granule cells and fired large calcium currents,

50 Despite their different origins, Drosophila glia and hemocytes are related cell populations that pro

51 ge in the process that discriminates between glia and hemocytes.

52 ivity in non-neuronal cells (such as enteric glia and innate immune cells).

53 orting an l-serine shuttle mechanism between glia and neurons in generating the NMDAR coagonist d-ser

54 na leads to age-related degeneration of both glia and neurons, preceded by an abnormal accumulation o

55 ittle is known about epigenetic signaling in glia and nothing is known about the intersection of glia

56 responses consistent with hypoxia in Muller glia and retinal neurons, and we find a metabolic shift

57 as become a powerful model in which to study glia and their neuronal interactions.

58 It has not been described, however, if glia and various components of the neuroretina form befo

59 ion of neural cells and a lack of functional glia and vasculature.

60 stingly, the Drosophila immune cells within (glia) and outside (hemocytes) the nervous system require

61 nscription factors that play both activator (GliA) and repressor (GliR) roles.

62 layer of outer glial cells, the perineurial glia, and a specialized extracellular matrix, the neural

63 al neurons from intrinsic stem cells, Muller glia, and are a powerful model to understand how stem ce

64 fferentiated cell types, such as neurons and glia, and brain organoids from cells obtained from patie

65 ze the ovary, differentiate into neurons and glia, and form a dense neural network within the ovarian

66 m healthy volunteers was incubated with TIMP-GLIA, and hemolysis, platelet activation and aggregation

67 d) ACOX1 is mostly expressed and required in glia, and loss of ACOX1 leads to developmental delay, pu

68 he fact that signaling between immune cells, glia, and neurons is now recognized as indispensable for

69 t growth of neural stem cells (neuroblasts), glia, and trachea is coordinated and whether coordinated

70 ts sbp-1 activation to promote the growth of glia, and when animals reach the adult stage, this inhib

71 lls, were cultured in medium containing TIMP-GLIA, anti-CD3 antibody, or lipopolysaccharide (controls

72 We have identified neuron-glia antigen 2 (NG2) as a unique molecular marker of S10

73 Neuron/glia antigen 2 (NG2) is a type I transmembrane proteogly

74 endogenous GDNF, and new enteric neurons and glia appeared to arise from Schwann cells within extrins

75 Glia are active in a complementary pattern.

76 odies that bind to the surface of neurons or glia are associated with a wide range of rare but treata

77 literature of nuanced roles, it follows that glia are consequential to behavior in adult animals, wit

78 tion and neuronal differentiation of enteric glia are regulated by Notch signalling.

79 Recent work has revealed that Muller glia are the source of regenerated neurons in zebrafish.

80 Caenorhabditis elegans amphid sheath (AMsh) glia as a model and show that a conserved cis-Golgi memb

81 w possibilities for use of mammalian enteric glia as a potential source of neurons to restore the act

82 dings expand our understanding of phagocytic glia as double-edged players in neurodegeneration-by cle

83 tokine interactions in brain and spinal cord glia as well as dorsal root ganglia satellite glia have

84 shared molecular mechanisms that make Muller glia attractive targets for cellular reprogramming and h

85 In myelinating glia, autophagy has expanded roles, particularly in phag

86 possesses glial types similar to vertebrate glia, based on molecular, morphological, and functional

87 Top results in neurons, glia, bulk brain, T cells, monocytes, and whole blood we

88 gical characteristics with mammalian enteric glia but cannot be identified by the expression of canon

89 expression in excitatory neurons and radial glias but depleted expression in inhibitory neurons.

90 tle information is available on autophagy in glia, but it is paramount for glia to perform their crit

91 by TDP-43 proteinopathy in motor neurons or glia, but not muscle, suggesting that metabolic dysregul

92 ate morphologies in concert with surrounding glia, but the molecules that coordinate dendrite and gli

93 ar identity and the regulation of the neuron-glia cell fate switch.

94 mouse Muller glial cells and a human Muller glia cell line (M10-M1 cells).

95 Herein, we show that three distinct glia cell types differentially wrap around the axonal an

96 is robustly enhanced in mutant outer radial glia cells (oRGs), a subtype of NPCs barely detectable i

97 n reconstructions and train CMNs to identify glia cells in a supervised classification paradigm, whic

98 r show late-emerging shh-GFP positive radial glia cells in the medial zone of the dorsal telencephalo

99 al nerves contain axons and their enwrapping glia cells named Schwann cells (SCs) that are either mye

100 duced a specific transcriptional response in glia cells.

101 lly conserved autoimmunity module induced in glia cells.

102 irectional communication between neurons and glia cells.

103 y accumulation of tau protein in neurons and glia cells.

104 ace glia (SG), wrapping glia (WG) and carpet glia (CG).

105 n cell numbers and myelination in PNS neuron-glia co-cultures.

106 Together, these results reveal that neuron-glia communication is ubiquitously altered at the NMJ in

107 al cell at the nose, reminiscent of dendrite-glia contacts in the mammalian brain.

108 Glia continuously survey neuronal health during developm

109 ic lateral sclerosis (ALS), immune cells and glia contribute to motor neuron (MN) degeneration.

110 Interactions between motor neurons and glia contribute to motor neuron loss, but the spatiotemp

111 ese results suggest that fornix white matter glia damage may cause hippocampal gray matter damage dur

112 ques possess significantly higher neuron and glia densities relative to the other three species, howe

113 d activation of this pathway in human radial glia, dependent on two receptors upregulated specificall

114 n Drosophila, such brain sparing relies on a glia-derived growth factor to sustain proliferation of n

115 Thus, glia-derived miR-274 coordinates growth of synaptic bout

116 e known to influence the formation of Muller glia-derived progenitor cells (MGPCs), but the mechanism

117 d to enhance adaptive immunity in TNBC while glia development, along with a few other neural function

118 Bergmann glia, Lphn3 deletion from Bergmann glia did not detectably impair excitatory or inhibitory

119 TIMP-GLIA did not increase markers of maturation on cultured

120 monstrated that zebrafish spinal cord radial glia differentiate into cells that are similar to mammal

121 Our findings suggest that this novel glia-to-glia differentiation is both dependent on early lineage

122 adjacent to dorsal midline Nestin(+) radial glia (dmNes+RG) down-regulate apical polarity proteins,

123 ar fragment of Robo upon cleavage to attract glia during migration in Caenorhabditis elegans.

124 Nearly 50% of Muller glia engaged in phagocytosis.

125 nd increased expression of glial markers and glia-enriched genes.

126 ivate from quiescence and ventral nerve cord glia expand their membranes.

127 Peripheral immune cells, neurons and glia express CB(2); however, the involvement of CB(2) fr

128 cal adhesions to uphold the structure of the glia-extracellular matrix sheath.

129 factor/cytokine that is expressed by Muller glia following neuronal death, is required for Muller gl

130 proper trafficking of lipids to neighboring glia for lipid exchange and disposal of potentially lipo

131 We discuss here the relevance of glia for maintaining circadian rhythms and also for serv

132 ion studies within subpopulations of neurons/glia for the brain data and granulocytes/T cells/B cells

133 Sorted neurons/glia from a fourth postmortem brain collection (n = 58)

134 ession and chromatin accessibility in Muller glia from zebrafish, chick, and mice in response to diff

135 lpha2 (P1), DQ2.5-glia-alpha3 (P2) and DQ2.5-glia-gamma1 (P3) epitopes, in a set of 60 German hexaplo

136 in offspring, including ones in white matter/glia, glucocorticoid receptors, neuroimmune outcomes, ce

137 vitro) from primary neurons co-cultured with glia grown on ECM coatings from decellularized brain tis

138 Once reactivated, neuroblasts promote cortex glia growth to ultimately form a selective membrane barr

139 guidance mechanisms, with evidence for both glia-guided growth and fasciculation along a neuronal sc

140 uster of differentiation 40 (CD40) in Muller glia has been implicated in the initiation of diabetes-i

141 lia as well as dorsal root ganglia satellite glia have been identified important- in pain modulation.

142 Glia have been implicated in Alzheimer's disease (AD) pa

143 In the retina of zebrafish, Muller glia have the ability to reprogram into stem cells capab

144 Loss of Fbxo7 in myelinating glia, however, led to axonal degeneration in the CNS and

145 roteins (C1q, C3), TLR4, and colabeling with glia (IBA1, GFAP) were examined using gene expression, i

146 the diversity of signals used by neurons and glia in a closed-loop fashion is necessary to establish

147 may serve a protective role for neurons and glia in adult Drosophila melanogaster brains.

148 teral sclerosis (ALS) emphasizes the role of glia in disease development.

149 Mice given GDNF developed neurons and glia in distal bowel tissues that were aganglionic in co

150 Resident GFAP(+) glia in dorsal root ganglia (DRG) known as satellite gli

151 levated levels of reactive oxygen species in glia in flies and murine Schwann cells.

152 efforts directed toward reprogramming Muller glia in mammals.

153 ypes and further implicate the importance of glia in neurodegeneration.

154 did not detect PI16 expression in neurons or glia in spinal cord, DRG, and nerve.

155 ed an explosion of research into the role of glia in supporting and modulating neuronal activity, pro

156 Our work identifies a new role for dSarm and glia in suppression of bystander neuron function after i

157 growth coordination between neuroblasts and glia in the brain.

158 l progenitor cells that generate neurons and glia in the developing mammalian cerebral cortex(1-4).

159 a phagocytic receptor expressed by satellite glia in the DRG involved in clearing apoptotic neurons d

160 The absence of classical glia in the ENS further suggests that neural crest-deriv

161 orphology of genetically defined neurons and glia in the mammalian brain.

162 etinal neurogenesis and lamination of Muller glia in the mouse retina.

163 egarding spontaneous reprogramming of Muller glia in zebrafish and compares this knowledge to researc

164 ntrolling the stem cell properties of Muller glia in zebrafish may provide cues to unlock the regener

165 articles encapsulating gliadin protein (TIMP-GLIA) in 3 mouse models of celiac disease.

166 en the peripheral and central nervous system glia, indicating common immunological features across di

167 function required Draper/MEGF10 signaling in glia, indicating glial cells spread injury signals and a

168 ptic and cell cycle genes to disrupted adult glia, inhibitory synapses, and behavior suggests a mecha

169 ss-of-function mutants of both sexes, Muller glia initiate the appropriate reprogramming response to

170 ligodendrocyte entities was termed as neuron-glia integrity recently.

171 Here we monitored the status of neuron-glia integrity via non-invasive neuroimaging methods and

172 aling at synapses is widely studied, but how glia interact with neuronal somas to regulate their acti

173 Axon-glia interactions converge on ankyrin and spectrin cytos

174 on necessary for studies to elucidate neuron-glia interactions in this neuropil.

175 dentified: they include two independent axon-glia interactions that converge on distinct axonal cytos

176 re implicated in synaptic plasticity, neuron-glia interface, neuroprotection, neuroregeneration, and

177 romatin peaks, and transcriptomes for radial glia, intermediate progenitor cells, excitatory neurons,

178 ntibodies that bind to molecules in neurons, glia, interstitial cells of Cajal, and muscularis macrop

179 (submucosal > myenteric) and is not seen in glia, interstitial cells of Cajal, or smooth muscle.

180 ms that suppress the reprogramming of Muller glia into neurogenic progenitors is key to harnessing th

181 ne regulatory networks that reprogram Muller glia into progenitor cells, we profiled changes in gene

182 that suppresses the reprogramming of Muller glia into proliferating MGPCs and this 'hub' coordinates

183 ammation, during the reprogramming of Muller glia into proliferating progenitors.

184 While the function of axonal ensheathment by glia is well studied, less is known about the functional

185 we have found that mouse hippocampal radial glia-like (RGL) neural stem cells express the synaptic c

186 Each part's neurogenic potential, radial glia-like neural stem cells (NSCs) proliferation and dif

187 Radial glia-like neural stem cells (RGLs) in the dentate gyrus

188 RHO GTPase-activating protein, in the radial glia-like neural stem cells within the ventricular zone

189 Specifically, radial glia-like type 1 NSCs were shifted from a proliferative

190 uire protection from the reactive astrocytic Glia Limitans not only during neuroinflammation but also

191 tration of a low dose of maraviroc protected glia limitans partially, maintained the integrity of end

192 ent to stimulate a protective barrier at the Glia Limitans that limits the severity of subsequent neu

193 ing conditions of a secondary barrier at the Glia Limitans with protective effects against subsequent

194 n barrier between reactive astrocytes at the Glia Limitans.

195 othelial Dhh on the integrity of the BBB and Glia Limitans.

196 in to study the cellular constituents of the glia limitans.

197 nd induces a tight junctional barrier at the Glia Limitans.

198 ellar Lphn3 protein is expressed in Bergmann glia, Lphn3 deletion from Bergmann glia did not detectab

199 The glia-lymphatic (glymphatic) system facilitates brain flu

200 carring of intraventricular and parenchymal (glia-lymphatic) CSF pathways.

201 resident neurogenic precursors or classical glia marked by sox10, plp1a, gfap or s100 Rather, lineag

202 iated neural progenitor had little effect on glia marker expression, suggesting that RAF1 is required

203 This underlies the importance of glia-matrix communication for structural and functional

204 nsitive to debranching by fission yeast GMF (glia maturation factor) than branches with ADP-P (i) -Ar

205 ATPalpha (adenosine triphosphatase alpha) in glia may be modulated by serotonin/dopamine signaling, c

206 though cell-intrinsic defects in neurons and glia may partially explain this decline, cell-extrinsic

207 lts suggest that aberrant translation within glia may suffice to cause severe neurological symptoms a

208 ycolytic response, paired with enhanced axon-glia metabolic coupling, supports the survival of axons.

209 on in the zebrafish retina stimulates Muller glia (MG) to proliferate and generate multipotent progen

210 s, fish have robust regeneration from Muller glia (MG).

211 r suggests that neural crest-derived enteric glia might have evolved after the teleost lineage.This a

212 ring embryonic development, when neurons and glia migrate to their destinations and axons project to

213 +/- 5763 neuron/mm(3) and 129,321 +/- 25,356 glia/mm(3).

214 brillary acidic protein expressing (GFAP(+)) glia modulate nociceptive neuronal activity in both the

215 orescent reporters Slits distribution and FP glia morphology.

216 nally differentiated postmitotic neurons and glia must cope with the accumulation of damage over the

217 with gliadin sensitivity, injection of TIMP-GLIA nanoparticles induced unresponsiveness to gliadin a

218 ytes from HLA-DQ8 transgenic mice given TIMP-GLIA nanoparticles, but not control nanoparticles, had i

219 We found that GOF disrupts mitosis of radial-glia neural progenitors (RGCs), inside-out radial migrat

220 onal regulator PRDM16 is expressed by radial glia, neural progenitors present in both regions; howeve

221 , we review recent progress in understanding glia-neuron interactions in C. elegans.

222 ingly, single-cell RNA-Sequencing implicated glia, not neurons, in this effect; there are relatively

223 ng accumulation of sTDP43 within neurons and glia of ALS patients.

224 Injury induces retinal Muller glia of certain cold-blooded vertebrates, but not those

225 D40 protein expression is elevated in Muller glia of diabetic mice; however, the mechanisms responsib

226 ) are specialized, non-myelinating, synaptic glia of the neuromuscular junction (NMJ), that participa

227 Here we show that Schwann cells (SCs), the glia of the peripheral nervous system, protect injured a

228 es, DQ2.5-glia-alpha1a (PFPQPELPY) and DQ2.5-glia-omega1 (PFPQPEQPF).

229 resulting clones are derived from two radial glia on average, span cortical layers 2-6, and are compo

230 re given 1 or 2 tail-vein injections of TIMP-GLIA or control nanoparticles.

231 sive cell population similar to outer radial glia (oRG), a fetal cell type that expands the stem cell

232 Most adult neurons and glia originate from radial glial progenitors (RGs), a ty

233 Prion-induced alterations in glia overlapped with those identified in other neurodege

234 th more GNs (p = .0003) and a trend for more glia (p = .0160) in whole DG in MDDSui and control subje

235 This is also evidence that glia participate in presynaptic homeostatic plasticity,

236 The mechanisms by which glia, particularly astrocytes and microglia, may contrib

237 ts show paradoxical responses to neurons and glia, patient-derived lymphoblasts appear to carry poten

238 ver, the roles of another important class of glia present in the retina, microglia, during this regen

239 but also impairs RGC neurogenesis and radial glia processes.

240 tation leads to eventual reduction in radial glia progenitors (RGPs).

241 RNA binding protein (RBP), Elavl4, in radial glia progenitors and early neurons depends on its altern

242 atively high proportion of zebrafish enteric glia proliferate under physiological conditions giving r

243 on during retinogenesis and regeneration and glia proliferation in proliferative retinopathies.

244 The retinal basal glia (RBG) is a group of glia that migrates from the opt

245 show that the activin ligand Myoglianin from glia regulates the temporal factor Imp in mushroom body

246 using 7T (1)H-MRS and findings indicate that glia-related metabolites could be valuable in cognitive

247 We found higher levels of glia-related metabolites in all brain regions in older i

248 , we assessed levels of mIns, tCr, and tCho (glia-related metabolites) and tNAA and Glu (neuron-relat

249 orrelated negatively with regional levels of glia-related metabolites.

250 gnitive ageing and higher regional levels of glia-related metabolites.SIGNIFICANCE STATEMENT Neuroche

251 oglia influence how NF-kappaB impacts Muller glia reprogramming.

252 -term effects of colitis on neurogenesis and glia responses in the hippocampus.

253 Loss of Basigin in the glia results in an overall compression of the nervous sy

254 CNTF responsive cell types, including Muller glia, rod and cone photoreceptors, and bipolar cells.

255 tal organization of oRG cells and the radial glia scaffold.

256 r and morphological characteristics: surface glia (SG), wrapping glia (WG) and carpet glia (CG).

257 liac disease, intravenous injections of TIMP-GLIA significantly decreased gliadin-specific T-cell pro

258 (Flox) knockdown mice-to determine whether a glia-specific, upstream complement cascade contributes t

259 This raises the issue of how do glia specifically differentiate in the nervous system, a

260 ermine the lineage relationships among these glia subtypes and the temporal profile of the lineage de

261 pressed at lower levels and more abundant in glia, suggesting their roles are at least partially cell

262 nt such as the transcriptome of outer-radial glia, suggesting use of a human-derived model remains im

263 clearance and waste removal during sleep via glia-supported perivascular channels.

264 ential negative regulator of integrin in the glia, supporting proper glial and extracellular matrix e

265 MPRIN is highly expressed in the perineurial glia surrounding the Drosophila larval nervous system.

266 rations of mIns, tCr, and tCho are higher in glia than neurons, the findings of this study suggest a

267 ganglia exist as a collection of neurons and glia that are arranged in a series of plexuses throughou

268 Multiple types of neurons and glia that are diploid at eclosion, become polyploid in t

269 s work in this system examined dendrites and glia that develop within epithelia, similar to mammalian

270 The numerous neurons and glia that form the brain originate from tightly controll

271 The retinal basal glia (RBG) is a group of glia that migrates from the optic stalk into the third i

272 dies identified multiple findings in neurons/glia that were detected across brain collections and wer

273 e findings demonstrate that independently of glia, the immune receptor TLR4 directly regulates post-t

274 he precursor form of miR-274 is expressed in glia, the mature form of miR-274 distributes broadly, in

275 critical to investigate the contribution of glia to alpha-synuclein aggregation and spread.

276 ved in the glutamate/GABA/glutamine cycle in glia to control neurotransmitter levels in neurons and t

277 Dilp-2 induces brain cortex glia to initiate membrane growth and make first contact

278 ts in neurons and glia, while GRDN-1 acts in glia to non-autonomously promote dendrite extension.

279 n autophagy in glia, but it is paramount for glia to perform their critical responses to nervous syst

280 owing neuronal death, is required for Muller glia to progress through the cell cycle.

281 ntain and restore quiescence, induces Muller glia to proliferate and generate neurons in adult mice a

282 EVs may mediate signaling between cilia and glia to shape sensory organ compartments.

283 Mature miR-274 is secreted from glia to the circulating hemolymph as an exosomal cargo,

284 Our findings suggest that this novel glia-to-glia differentiation is both dependent on early

285 nt-more specifically, the ventricular radial glia-to-intermediate progenitor cell transition at gesta

286 This glia-to-neuron cell fate switch occurs during male sexua

287 other three species, however we observed no glia-to-neuron ratio differences among species.

288 Here, we show that both neurons and glia together give rise to the pathology that is mitigat

289 g to progress through the cell cycle, Muller glia undergo reactive gliosis, a pathological hallmark i

290 With cell cycle progression stalled, Muller glia undergo reactive gliosis, a pathological hallmark o

291 neural progenitors, differentiated neurons, glia, undifferentiated neurons and non-neural cells.

292 Knockdown of Basigin in perineurial glia using RNAi results in significant shortening of the

293 In addition to satellite glia, we found Jedi-1 expression in perineurial glia and

294 ury and histological features of neurons and glia were also investigated using immunofluorescent labe

295 characteristics: surface glia (SG), wrapping glia (WG) and carpet glia (CG).

296 art with vimentin- and nestin-immunopositive glia whereas GFAP and the water-channel aquaporin 4 were

297 muscles interact with surrounding satellite glia, which enhances the sensitivity of the inflamed neu

298 actor, XBP-1s, in a subset of astrocyte-like glia, which extended the life span in Caenorhabditis ele

299 SAX-7 acts in neurons and glia, while GRDN-1 acts in glia to non-autonomously prom

300 manipulation in different tissues including glia, wing and eye resulted in multiple phenotype modifi