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1 y accumulation of tau protein in neurons and glia cells.
2 tested in cultures of rat hippocampal neuron glia cells.
3 e surface of adult fly brains in a subset of glia cells.
4 ession and enhanced ATP release from enteric glia cells.
5 olonged cell cycle and maintenance of radial glia cells.
6  cells generate non-dopaminergic neurons and glia cells.
7 duced a specific transcriptional response in glia cells.
8  enhanced Hippo pathway-driven overgrowth in glia cells.
9 lly conserved autoimmunity module induced in glia cells.
10 teractions between photoreceptors and Muller glia cells.
11 irectional communication between neurons and glia cells.
12  control excitability from either neurons or glia cells.
13 ation development of all retinal neurons and glia cells.
14 n the nervous system as a potent mitogen for glia cells.
15 fish retina, serpine3 is expressed in Muller glia cells, a cell type essential for survival and maint
16                                      Enteric glia cell abundancy correlates with worse disease outcom
17 eurons, disinhibition in the dorsal horn and glia cell activation.
18 ptic contacts between CFs and NG2-expressing glia cells, adding to the intrigue of neuro-glial intera
19 in-associated glycoprotein (MAG), and neuron-glia cell adhesion molecule (L1).
20 cell adhesion molecules (L1-CAMs) [L1/neuron-glia cell adhesion molecule (L1/NgCAM) and neurofascin (
21 tion of the cell adhesion molecule L1/neuron-glia cell adhesion molecule (NgCAM) depends on endocytos
22              The adhesion molecule L1/neuron-glia cell adhesion molecule (NgCAM) travels to axons via
23                      In contrast, the neuron-glia cell adhesion molecule (NgCAM), which binds heterop
24  the axonal cell adhesion molecule L1/neuron-glia cell adhesion molecule (NgCAM).
25 lysine and either laminin (LN) or the neuron-glia cell adhesion molecule (NgCAM).
26 he axonal adhesion molecule L1/NgCAM (neuron-glia cell adhesion molecule) and, if so, in what compart
27  containing the axonal protein NgCAM (neuron-glia cell adhesion molecule) were transported into both
28 , tenascin-C, thrombospondin, and the neuron-glia cell adhesion molecule, Ng-CAM.
29 m low-density lipoprotein receptor or neuron-glia cell-adhesion molecule to polarize expression to ei
30 hondroitin sulfate proteoglycans produced by glia cells and containing Alzheimer amyloid precursor pr
31 use brains prolongs the cell cycle of radial glia cells and extends cortical neurogenesis into postna
32 onstrated to promote the formation of radial glia cells and Notch signaling is one such signal.
33 Both GLYT1 and HMGN3 are highly expressed in glia cells and the eye, and we show that both proteins a
34 LRP1) in the metabolism of Abeta in neurons, glia cells, and along the brain vasculatures.
35   In the retina, sEH was localized in Muller glia cells, and Muller cell-specific sEH deletion reprod
36 plexiform vascular endothelial cells, Muller glia cells, and the basolateral side of the retinal pigm
37 IRDs, the LNPs are limited to RPE and Muller glia cells, and their application has been limited to tr
38 ors expressed by Purkinje cells and Bergmann glia cells are activated predominantly by synaptic and e
39 lts demonstrate that astrocytes and Bergmann glia cells are the first cells of the brain parenchyma i
40 ts cell proliferation, whereas untransformed glia cells are unaffected by similar IMPDH2 perturbation
41                                              Glia cells are uniquely positioned at synapses, contacti
42 icate in utero binding of AQP4-IgG to radial glia cells as a mechanism for alterations of the develop
43 r than being composed of specialized midline glia cells as in many other species, the embryonic midli
44 rogenitors, resulting in an excess of radial glia cells at the expense of intermediate progenitors an
45 B1, which interacts with ephrin-B2 on radial glia cells at the optic chiasm to repulse VT axons away
46 ate, suggesting that the appican-transfected glia cells attach more avidly to their substrate than do
47                  In the cerebellum, Bergmann glia cells (BGs) are intimately associated with PF synap
48                                       Radial glia cells both generate neurons and physically guide na
49 646329 is a lncRNA enriched in single radial glia cells but is detected at low abundance in tissues.
50 H1 hinders viral control in B7-H1 expressing glia cells, but does not mediate resistance to CD8 T cel
51 ically active Fic on capitate projections of glia cells, but not neurons, supporting a role in the re
52 s seen in both WT (CD-1) and S-100B KO (-/-) glia cells, but S-100B KO (-/-) GFAP-IR cells appeared l
53 Reducing GAD1 in metastatic cells by primary glia cell coculture abolished the capacity of metastatic
54 nsheath the neuroepithelium, suggesting that glia cells communicate with the neuroepithelium.
55 and of nitric oxide (NO) production in these glia cells contributes to neuroinflammation and neurodeg
56             In embryonic rat midbrain neuron/glia cell cultures exposed to LPS, even delayed administ
57  and rod markers; but coinciding with Muller glia cell cycle withdrawal.
58 o exhibited a severity-dependent increase in glia cell density, white matter thinning and reduced neu
59 t to alterations in immune system signaling, glia cell differentiation, and synaptic signaling in the
60    Ependymal cells are generated from radial glia cells during embryonic brain development and acquir
61 t expression of copper transporter (CTR1) on glia cells during neuroinflammation.
62                                      Enteric glia cells (EGC) play an important role in the maintenan
63                                      Enteric glia cells (EGCs) form a dense network around myenteric
64  cerebellar cortex the processes of Bergmann glia cells encase synapses between presynaptic climbing
65 e cells express neuregulin (NRG), and radial glia cells express erbB4 in the developing cerebellum an
66 c plexus layer, and cultured primary enteric glia cells expressed IL-6 and the chemokine monocyte che
67  they control bipolar interneuron and Muller glia cell fate specification and promote proliferative q
68 ue of Neuron, Li et al. show that the neuron/glia cell fate switch of cortical progenitors is regulat
69 ar identity and the regulation of the neuron-glia cell fate switch.
70  precursors of genes critical for the Muller glia cell fate, which are rarely generated from terminal
71 ify Dmrt5 as a novel regulator of the neuron-glia cell-fate switch in the developing hippocampus.
72                     Regulation of the neuron-glia cell-fate switch is a critical step in the developm
73 y regulated network that controls the neuron-glia cell-fate switch.SIGNIFICANCE STATEMENT We identify
74 te transporter-mediated currents in Bergmann glia cells follow the rules of synaptic release more clo
75 s protective effects by direct modulation of glia cell function (i.e., either astrocytes, microglia,
76                                       Radial glia cells function as guide cells for neuronal migratio
77 of PrP in neurons (NSE-HPrP/mPrP(0/0)) or in glia cells (GFAP-HPrP/mPrP(0/0)).
78        Specific assays show that in Bergmann glia cells Gpr37l1 is associated with primary cilium mem
79  contrast, gap junctions between neurons and glia cells have rarely been reported and are poorly unde
80      Here, we demonstrated that mouse Muller glia cells have the capacity to be reprogrammed into the
81 sed within the apical domain of human radial glia cells (hRGCs).
82      Conditionally immortalized mouse Muller glia cells (ImM10) were cultured under nonimmortalizing
83 n reconstructions and train CMNs to identify glia cells in a supervised classification paradigm, whic
84 how that the survival of a subset of midline glia cells in Drosophila depends upon direct suppression
85               We found that embryonic radial glia cells in neocortex express AQP4.
86  receptor HTR2A selectively activates radial glia cells in the developing human, but not mouse, neoco
87 r show late-emerging shh-GFP positive radial glia cells in the medial zone of the dorsal telencephalo
88                                 In addition, glia cells in the optic lamina that contact photorecepto
89                   These results suggest that glia cells in the prefrontal cortex are particularly sen
90                                       Radial glia cells in the VZ primarily divide with a vertical or
91              Studies in cultured neurons and glia cells indicate that these changes in MAO A and B ar
92 iased identification of mechanisms of neuron-glia cell interactions.
93 f autophagy and immune activity within brain glia cells is poorly understood.
94 synaptic Schwann cells (PSCs), which are the glia cells juxtaposed to the motor nerve terminal, activ
95 ably, a distinct human-specific outer radial glia cell layer.
96  mouse Muller glial cells and a human Muller glia cell line (M10-M1 cells).
97 uch as brain-derived neurotrophic factor and glia cell line-derived neurotrophic factor also reduced
98 h factor, brain-derived neurotrophic factor, glia cell line-derived neurotrophic factor, or ciliary n
99 ived cone photoreceptors and an adult Muller glia cell line.
100 otent and give rise only to cells within the glia cell lineage, although they are capable of forming
101 ncentration glutamate transients at Bergmann glia cell membranes that are necessary to activate low-a
102 provide a geographical cue to guide Bergmann glia cell membranes to surround active synapses and ensu
103 ed abundant expression of harmonin in Muller glia cells (MGCs) and retinal neurons.
104 by the processes of one type of glia, Muller glia cells (MGs).
105 al nerves contain axons and their enwrapping glia cells named Schwann cells (SCs) that are either mye
106 euroepithelium (NE) give rise to neurons and glia cells of the medulla cortex.
107  is robustly enhanced in mutant outer radial glia cells (oRGs), a subtype of NPCs barely detectable i
108                                       Radial glia cells play an important role in guiding migrating n
109 ole of FASN-dependent lipogenesis for radial glia cell polarity in human brain organoids.
110              Panx1 deletion in GFAP-positive glia cells prevented hypersensitivity completely, wherea
111       LncND expression is enriched in radial glia cells (RGCs) in the ventricular and subventricular
112 ulin G (IgG) antibodies binding to satellite glia cells (SGC) in the dorsal root ganglia.
113 ehavior via the LPA(1) receptor on satellite glia cells (SGCs), which is expressed by both human and
114 pulations of rare bipolar cells and a Muller glia cell subpopulation.
115  intrinsic ganglia, comprised of neurons and glia cells that innervate airway smooth muscle, is a rec
116 roglia and astrocytes are subgroups of brain glia cells that support and protect neurons within the c
117 e (VZ) of the nervous system contains radial glia cells that were originally considered relatively ho
118 ina with changes in the morphology of radial glia cells, the aberrant migration of Cajal-Retzius cell
119 reased numbers or mitotic activity of radial glia cells, the principal neural stem cells.
120 hown that morphine modulates the function of glia cells through both opioid receptor dependent and in
121  neurotoxicity and their ability to activate glia cells, two biological activities of amyloid peptide
122      These data are the first to demonstrate glia cell type-dependent B7-H1 regulation in vivo, resul
123          Herein, we show that three distinct glia cell types differentially wrap around the axonal an
124 fidelity to neuroepithelial and early radial glia cell types, but improve as neurogenesis progresses.
125 ons form overlapping subsets of neuronal and glia cell types.
126 d Cry2 in the hemispherical layer of laminal glia cells underneath the retina.
127 ransgenic mouse that expresses Cre in Muller glia cells was generated.
128 real-time imaging of cohorts of mouse radial glia cells, we show that the radial glial scaffold, upon
129                                              Glia cells were largely negative.
130 s with a disrupted basement membrane, radial glia cells were retracted from the pial surface, and rad
131  and expressed constitutively in the Bergman glia cells within the molecular layer of the cerebellum.

 
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