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1 nd induces a tight junctional barrier at the Glia Limitans.
2 n barrier between reactive astrocytes at the Glia Limitans.
3 monocytes facilitate penetration through the glia limitans.
4 sappearance of the basement membrane and the glia limitans.
5 had direct contacts with astrocytes near the glia limitans.
6  of glial fibrillary acidic protein-positive glia limitans.
7 ciated with small blood vessels and with the glia limitans.
8 es that terminate on blood vessels or on the glia limitans.
9 d with the ependyma, choroid plexus, and the glia limitans.
10 othelial Dhh on the integrity of the BBB and Glia Limitans.
11  to the endothelial cells and by forming the glia limitans.
12 in to study the cellular constituents of the glia limitans.
13     AQP4, found in astrocyte foot processes, glia limitans and ependyma, facilitates water movement i
14 action composed primarily of the superficial glia limitans and pial vessels, but trended toward a dec
15 ches in basement membrane and the underlying glia limitans are the key initial events of the cellular
16 ches in basement membrane and the underlying glia limitans are the key initial events of the cellular
17       Some cortical NeuN(+) neurons, GFAP(+) glia limitans astrocytes, Iba-1(+) microglia and S100bet
18 sappearance of the basement membrane and the glia limitans at the cerebral cortical surface during de
19                    Barrier properties of the glia limitans during health and neuroinflammation are in
20                                          The glia limitans ensheathes the entire central nervous syst
21 ascular glia limitans, we here show that the glia limitans forms a barrier for soluble mediators, bea
22 barrier (BBB) and then across the astrocytic glia limitans (GL).
23         Radial glial endfeet, which comprise glia limitans, grew out of the neural boundary.
24 -feet that surround capillaries and form the glia limitans; however, the structural organization of A
25  our understanding of the active role of the glia limitans in CNS immune privilege.
26 olleagues demonstrate that astrocytes of the glia limitans induce tight junction formation in respons
27 sms regulating their penetration through the glia limitans into the parenchyma are less well studied,
28                            We found that the glia limitans layer of the olfactory nervous system is m
29 uire protection from the reactive astrocytic Glia Limitans not only during neuroinflammation but also
30  layer of the olfactory bulb constitutes the glia limitans of the olfactory nervous system and that O
31                             In contrast, the glia limitans of the rest of the nervous system consists
32 tration of a low dose of maraviroc protected glia limitans partially, maintained the integrity of end
33 ation in the arcuate nucleus/median eminence/glia limitans region.
34        The blood-brain barrier (BBB) and the glia limitans serve to prevent the migration of cells an
35 y indicate that the inducible barrier of the glia limitans should be further explored as a therapeuti
36 face of PPC with features in common with the glia limitans that is formed by endfeet in other cortica
37 ent to stimulate a protective barrier at the Glia Limitans that limits the severity of subsequent neu
38 ntinuities in the pial surface basal lamina (glia limitans) that probably underlie the neuronal migra
39    Furthermore, the olfactory nervous system glia limitans (the glial layer defining the PNS-CNS bord
40 ycan stabilizes the basement membrane of the glia limitans, thereby supporting the cortical infrastru
41    Sensory axons must traverse a spinal cord glia limitans to connect the brain with the periphery.
42  imaging of the superficial and perivascular glia limitans, we here show that the glia limitans forms
43 n astrocyte endfeet, at synapses, and at the glia limitans, where it mediates water exchange across t
44 ing conditions of a secondary barrier at the Glia Limitans with protective effects against subsequent