ライフサイエンスコーパス: globus

1 Only globus and VP insufficiency had a significant incidence

2 older, choking or pill-induced dysphagia or globus caused 37.6% (95% CI, 29.1 to 46.2) of all emerge

3 entiated somatoform disorder, panic attacks, globus hystericus, vocal cord dysfunction, scombroid poi

4 nificantly higher GM volumes in the putamen, globus pailldus, and inferior frontal gyrus after donepe

5 .04 ppm +/- 0.05 and 24.96 sec(-1) +/- 5.3), globus pallidi (case participants: 0.14 ppm +/- 0.05 and

6 07 ppm +/- 0.06 and 25.65 sec(-1) +/- 6.37), globus pallidi (first MRI: 0.13 ppm +/- 0.07 and 27.53 s

7 the thalamus, medial prefrontal cortex, and globus pallidum compared to euglycemia for both PASL-MRI

8 the thalamus, medial prefrontal cortex, and globus pallidum in response to hypoglycemia.

9 orticospinal tract, and reduced thalamic and globus pallidum volumes relate to deficits in cognitive

10 ions (thalamus, medial prefrontal cortex and globus pallidum) were obtained using PASL-MRI and [(15)O

11 The splenium of the corpus callosum, left globus pallidum, thalamus and hippocampus (P < 0.01) wer

12 , resulting in a heterogeneous distribution (globus pallidus > cortical regions > cerebellum) consist

13 bstantia nigra (SN; +46%; p<0.02) and in the globus pallidus (+9%; p=0.06) and ventral pallidum (+11%

14 intensity increase per injection, P < .001), globus pallidus (0.23% increase, P = .009), posterior th

15 he nucleus accumbens (10 +/- 7%), and in the globus pallidus (13 +/- 15%) measured with [(11)C]PHNO,

16 telangiectasia also had higher metabolism in globus pallidus (16%, P = 0.05), which correlated negati

17 ording single-unit spiking activity from the globus pallidus (232 neurons) while the animals complete

18 spite loss of kinematic tuning, cells in the globus pallidus (63%, 10/16 cells) and VLo (84%, 46/55 c

19 joint position, velocity, or acceleration in globus pallidus (75%, 12/16 cells) and in the pallidal r

20 In the globus pallidus (GP in rodents, homolog of the primate G

21 -related increases in iron concentrations in globus pallidus (GP) and substantia nigra (SN), although

22 hat volumetric hemispheric lateralization of globus pallidus (GP) and thalamus (Th) explains individu

23 The firing patterns of neurons in the globus pallidus (GP) are affected by two major sources o

24 halamus before, during, and after DBS in the globus pallidus (GP) GP-DBS resulted in: (1) stimulus-en

25 SI) increase in the dentate nucleus (DN) and globus pallidus (GP) in relation to the middle cerebella

26 leus (STN) neurons and two types of external globus pallidus (GP) neuron inappropriately synchronise

27 Synchronization of globus pallidus (GP) neurons and cortically entrained os

28 Globus pallidus (GP) neurons fire rhythmically in the ab

29 MSNs and FSIs together with motor cortex and globus pallidus (GP) neurons, in rats performing a simpl

30 onsidered similar, if not homologous, to the globus pallidus (GP) of mammals.

31 levodopa responses was seen in the striatum/globus pallidus (GP) of the lesioned hemisphere.

32 The globus pallidus (GP) predominantly contains GABAergic pr

33 In comparison, the globus pallidus (GP), a main striatal output nucleus, ha

34 triatum and their subsequent growth into the globus pallidus (GP), an important (intermediate) target

35 s were measured in the dentate nucleus (DN), globus pallidus (GP), crus anterior of capsula interna (

36 The signal intensities (SIs) in the globus pallidus (GP), thalamus (T), dentate nucleus (DN)

37 substantia nigra (SN), pulvinar thalami, and globus pallidus (GP).

38 he reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

39 he reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

40 Reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

41 Reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

42 n-expressing (PV(+)) neurons in the external globus pallidus (GPe) are critically involved in the mot

43 ine-depleted rats indicate that the external globus pallidus (GPe) contains two main types of GABAerg

44 Neurons in the external globus pallidus (GPe) generate pacemaker activity that c

45 whether the manipulation of the DMS-external globus pallidus (GPe) iMSNs circuit alters the ethanol-c

46 tal dopamine, the striatum, and the external globus pallidus (GPe) in regulating RLS-like movements,

47 cal excitation and indirect pathway external globus pallidus (GPe) inhibition of the STN are critical

48 The external globus pallidus (GPe) is a critical node within the basa

49 The external globus pallidus (GPe) is a key contributor to motor supp

50 The external globus pallidus (GPe) is a key nucleus within basal gang

51 thin the basal ganglia circuit, the external globus pallidus (GPe) is critically involved in motor co

52 The external segment of the globus pallidus (GPe) is one of the core nuclei of the b

53 We show how external globus pallidus (GPe) neuron is sensitive to the phase o

54 analogous division of labor in the external globus pallidus (GPe) of Parkinsonian rats, showing that

55 of the neuronal composition in the external globus pallidus (GPe) undermines our ability to interrog

56 the constituent neurons within the external globus pallidus (GPe) was not fully appreciated.

57 ated activity of the external segment of the globus pallidus (GPe), a downstream structure whose comp

58 s that pathological activity of the external globus pallidus (GPe), a nucleus in the basal ganglia, c

59 In the external segment of the globus pallidus (GPe), neuronal populations have been de

60 ison to the adjacent external segment of the globus pallidus (GPe), remain unexplored.

61 Focusing on the mouse external globus pallidus (GPe), we demonstrate that the potential

62 ic nucleus (STN) and the external segment of globus pallidus (GPe).

63 ural circuits in the external segment of the globus pallidus (GPe).

64 highest levels in the striatum and external globus pallidus (GPe).

65 ng in neurons of the external segment of the globus pallidus (GPe).

66 , and neurons in the external segment of the globus pallidus (GPe)], and one neuromodulator group [st

67 nputs from the striatum (direct pathway) and globus pallidus (GPe, indirect pathway).

68 show that neurons in the habenula-projecting globus pallidus (GPh) in mice are essential for evaluati

69 uit, which regulates the habenula-projecting globus pallidus (GPh) neurons, exists within the basal g

70 eive GABAergic projections from the internal globus pallidus (GPi) and glutamatergic inputs from moto

71 of the subthalamic nucleus (STN) or internal globus pallidus (GPi) reduces dyskinesias remain largely

72 ive visualization scores of STN and internal globus pallidus (GPi) were recorded by two neuroradiolog

73 drove network activity through the internal globus pallidus (GPi), external globus pallidus, motor c

74 deep brain stimulation (DBS) at the internal globus pallidus (GPi).

75 for post-GBCA group [P < .001]) but not the globus pallidus (mean SI ratio for two-group comparison:

76 shortening in the dentate nucleus (n = 13), globus pallidus (n = 13), substantia nigra (n = 13), pos

77 ation of the subthalamic nucleus (n = 60) or globus pallidus (n = 61).

78 e gray matter of the whole brain (P < .001), globus pallidus (P = .002), dentate nucleus (P = .046),

79 ight caudate nucleus and ventral putamen and globus pallidus (P = .003) compared with that in control

80 years in the substantia nigra (P < .001) and globus pallidus (P = .035), which are both predictors of

81 caudate (P < 0.001), putamen (P < 0.001) and globus pallidus (P = 0.025) in patients with Parkinson's

82 0.005), putamen (r = -0.51; P = 0.025), and globus pallidus (r = -0.47; P = 0.030).

83 0.011), putamen (r = -0.48; P = 0.022), and globus pallidus (r = -0.70; P < 0.001).

84 reduce central thalamus activity and release globus pallidus activity in DOCs.

85 e alignments between subthalamic nucleus and globus pallidus amplified local neural synchrony in the

86 The reversal of globus pallidus and central thalamus profiles across BIs

87 ing single-unit activity in the sensorimotor globus pallidus and motor thalamus before, during, and a

88 y specific brain areas such as the thalamus, globus pallidus and orbitofrontal regions of the right h

89 ssociated with a lower T1R (P = 0.01) in the globus pallidus and putamen but were not associated with

90 nd most prolonged increases occurring in the globus pallidus and putamen.

91 y between external and internal parts of the globus pallidus and saliency of the sensory input, a low

92 t of the parvalbumin-positive neurons in the globus pallidus and striatum of the Ldb1 mutants.

93 e recapitulated in STN, but also in external globus pallidus and striatum.

94 put from the caudal BF, including the caudal globus pallidus and substantia innominata and moderate i

95 s, and projected their axons to the targeted globus pallidus and substantia nigra in a time-dependent

96 ar whether abnormal activity measured in the globus pallidus and substantia nigra pars reticulata is

97 Activity in the internal segment of the globus pallidus and subthalamic nucleus correlated stron

98 but also bilateral caudate and left putamen/globus pallidus and subthalamic nucleus.

99 tion of subcortical structures (specifically globus pallidus and thalamus) explains individual biases

100 ined the surface morphology of the striatum, globus pallidus and thalamus, and thickness of the cereb

101 the parieto-occipital regions, left putamen/globus pallidus and thalamus; and in white matter of the

102 al neurons in rodents), which project to the globus pallidus and the substantia nigra, and the locall

103 in the striatum and their projections to the globus pallidus and the substantia nigra.

104 eceptor-mediated inhibition arising from the globus pallidus and thus promote single-spike activity r

105 striatal D1-MSNs collateralized to both the globus pallidus and ventral mesencephalon.

106 ve been reported about hyperintensity of the globus pallidus and/or dentate nucleus on unenhanced T1-

107 indicating spontaneous beta oscillations in globus pallidus are not pathognomonic.

108 Globus pallidus binding best distinguished PSP patients

109 ared with NT mice, significantly increase in globus pallidus but decrease in entopeduncular nucleus/s

110 in the dorsal caudate nucleus, putamen, and globus pallidus but the observed variation did not assoc

111 nd caudate nucleus, red nucleus, putamen and globus pallidus by T2* MRI at baseline and after 3 and 6

112 al neurons, and magnocellular neurons in the globus pallidus did not demonstrate a similar extent of

113 uantification of single-unit activity in the globus pallidus externa (GPe) and substantia nigra retic

114 a from the globus pallidus interna (GPi) and globus pallidus externa (GPe) in children undergoing dee

115 strates that functional connections from the globus pallidus externa (GPe) to striatum are substantia

116 es in firing rates of single neurons in STN, globus pallidus externa (GPe), and substantia nigra pars

117 cortex, the globus pallidus interna, and the globus pallidus externa compared with patients with trem

118 o the homolog of the globus pallidus interna/globus pallidus externa express D1 or D2 receptors.

119 cortex, the globus pallidus interna, and the globus pallidus externa.

120 l neurons comprising subthalamic nucleus and globus pallidus external and suggest this approach for d

121 we report that a previously neglected CeA-to-globus pallidus external segment (GPe) circuit plays an

122 eptide somatostatin, send projections to the globus pallidus external segment (GPe), and this CeA-GPe

123 ent striatal (Str) neurons projecting to the globus pallidus external segment (GPe).

124 ectly through the caudal-ventral part of the globus pallidus externus (cvGPe).

125 ere we describe a direct projection from the globus pallidus externus (GP), a central nucleus of the

126 al perikarya, ENK-immunostained terminals in globus pallidus externus (GPe) were more abundant at 6 m

127 ne increased rabies-labelled inputs from the globus pallidus externus (GPe), a basal ganglia nucleus

128 Gamma oscillations arise in the subthalamic-globus pallidus feedback loop, and occur during movement

129 , and survival of forebrain interneurons and globus pallidus GABAergic neurons, thereby leading to th

130 the basal ganglia, the caudate, putamen and globus pallidus in brown capuchin monkeys (Cebus apella)

131 n metabolic rates including hyperactivity in globus pallidus indicative of basal ganglia involvement.

132 ance imaging of the brain revealed bilateral globus pallidus injury in the setting of dyskinetic CP.

133 We report microelectrode data from the globus pallidus interna (GPi) and globus pallidus extern

134 ndomised to subthalamic nucleus (STN) versus globus pallidus interna (GPi) DBS surgery.

135 ood effects of subthalamic nucleus (STN) vs. globus pallidus interna (GPi) deep brain stimulation (DB

136 ut a previous case suggested that DBS of the globus pallidus interna (GPi) might.

137 ither the subthalamic nucleus (STN, n=84) or globus pallidus interna (GPi, n=80), using standardised

138 o had undergone bilateral stimulation of the globus pallidus interna (pallidal stimulation) or subtha

139 The basal ganglia, including the striatum, globus pallidus interna and externa (GPe), subthalamic n

140 The globus pallidus interna and the subthalamic nucleus are

141 n of Medtronic quadripolar electrodes in the globus pallidus interna bilaterally.

142 Long-term data on the outcomes of globus pallidus interna DBS are limited and mostly confi

143 The results suggest anteromedial globus pallidus interna DBS for Tourette's syndrome is a

144 the effectiveness of DBS of the anteromedial globus pallidus interna on tic severity and common comor

145 ating that stimulation of an area called the globus pallidus interna partially reverses deficits in v

146 viously ignored structure in data describing globus pallidus interna responses to cortical stimulatio

147 ilateral dorsolateral prefrontal cortex, the globus pallidus interna, and the globus pallidus externa

148 ilateral dorsolateral prefrontal cortex, the globus pallidus interna, and the globus pallidus externa

149 targeting the subthalamic nucleus (STN) and globus pallidus interna, is a surgical therapy with clas

150 from the dorsal pallidum, the homolog of the globus pallidus interna.

151 tal neurons projecting to the homolog of the globus pallidus interna/globus pallidus externa express

152 paring the role of inputs originating in the globus pallidus internal segment (GPi), and lateral hypo

153 ion in the basal ganglia output nucleus, the globus pallidus internal segment.

154 trating that deep brain stimulation (DBS) of globus pallidus internus (GPi DBS) and subthalamic nucle

155 ed in the centromedial (CM) thalamus (n=51), globus pallidus internus (GPi) (n=47), nucleus accumbens

156 assess the safety and efficacy of bilateral globus pallidus internus (GPi) DBS in patient's with sev

157 tes during subthalamic nucleus (STN) DBS and globus pallidus internus (GPi) DBS.

158 ients to either subthalamic nucleus (STN) or globus pallidus internus (GPi) deep brain stimulation (D

159 implicates the subthalamic nucleus (STN) and globus pallidus internus (GPi) in reward and punishment

160 n (DBS) of the subthalamic nucleus (STN) and globus pallidus internus (GPi) is an effective treatment

161 studies have characterized beta power in the globus pallidus internus (GPi), an equally effective DBS

162 dies have characterized beta activity in the globus pallidus internus (GPi), another effective target

163 corded the activity of single neurons in the globus pallidus internus (GPi), the primary BG output nu

164 in skeletomotor output region of the BG, the globus pallidus internus (GPi).

165 f patients (n = 66) implanted bilaterally in globus pallidus internus (n = 34) or centromedial thalam

166 Deep brain stimulation of the globus pallidus internus alleviates involuntary movement

167 a small volume covering the ventroposterior globus pallidus internus and adjacent subpallidal white

168 direct pathway neurons were more abundant in globus pallidus internus and substantia nigra at 6 month

169 ed tonic inhibition of the thalamus from the globus pallidus internus could lead to an under-suppress

170 l studies reported the beneficial effects of globus pallidus internus deep brain stimulation (GPi DBS

171 amma synchronization (ERS) recorded from the globus pallidus internus in patients undergoing deep bra

172 ed increased interaction between putamen and globus pallidus internus, and decreased interaction betw

173 The results showed that for globus pallidus internus, connectivity to limbic network

174 l connectivity from the sensorimotor cortex, globus pallidus internus, ventral intermediate thalamic

175 DBS targets (STN, subthalamic nucleus; GPi, globus pallidus internus; NAc, nucleus accumbens) evoked

176 Deep brain stimulation of the internal globus pallidus is a highly effective and established th

177 ucleus, and movement-related activity of the globus pallidus leads to transient beta oscillations dur

178 mean uptake after BI; a relative increase in globus pallidus metabolism was evident in BI subjects wh

179 The subthalamic nucleus-globus pallidus network is a potential source of oscilla

180 We show that most globus pallidus neurons, but very few neocortical intern

181 om the Shh-expressing domain eliminated most globus pallidus neurons, whereas most cortical and stria

182 cortical interneurons; and pattern C, mainly globus pallidus neurons.

183 Striatal lesion core and globus pallidus of Abeta + ET1 rats showed extensive deg

184 recordings from the subthalamic nucleus and globus pallidus of five patients with Parkinson's diseas

185 activity occurs in the prefrontal cortex and globus pallidus of patients with nontremor-dominant PD c

186 onounced differences in substantia nigra and globus pallidus of PSP compared with control brains.

187 spectively, into the superior colliculus and globus pallidus of Sprague-Dawley rats.

188 Within the basal ganglia, the globus pallidus pars externa (GPe) has been hypothesized

189 ferentially to deep brain stimulation of the globus pallidus pars interna (GPi DBS).

190 e treated with deep brain stimulation of the globus pallidus pars interna during the period 1999-2010

191 rm efficacy of deep brain stimulation of the globus pallidus pars interna in the treatment of general

192 r predicting a better or poorer outcome from globus pallidus pars interna stimulation in this series

193 Projections to the globus pallidus showed solely D2-mediated presynaptic in

194 tside our Holmes tremor circuit, whereas the globus pallidus target was close, consistent with publis

195 are distributed more caudally in the caudal globus pallidus than noncholinergic projection neurons.

196 density of direct pathway collaterals in the globus pallidus that bridge the direct pathway with the

197 ing potential, which ranged from 2.42 in the globus pallidus to 8.48 in the nucleus accumbens.

198 ential values (from approximately 0.5 in the globus pallidus to approximately 3.5 in the insula) for

199 ly distinct projections through striatum and globus pallidus to EP targets within epithalamus and tha

200 discovered feedback projection from external globus pallidus to striatum is crucial for inhibitory co

201 n, TBF was solely negatively associated with globus pallidus volume.

202 However, shorter T1 relaxation times in the globus pallidus were found in group 1 compared with grou

203 alamus and thalamic subnuclei, striatum, and globus pallidus were segmented using a fully automated m

204 Protein components of the globus pallidus were studied including glial fibrillary

205 ith diabetes and MDD showed lower MTR in the globus pallidus when compared with the group with MDD.

206 Moreover, low signal intensity in globus pallidus with hyperintensive streaking and low si

207 basal ganglia nuclei (caudate, putamen, and globus pallidus) in children with ADHD.

208 " pathways that regulate the pallidal (e.g., globus pallidus) output nuclei involved in the control o

209 l striatum with the dorsal striatum (putamen/globus pallidus), and lower connectivity between these r

210 ance in the striatum and axon entry into the globus pallidus).

211 n: (1) stimulus-entrained firing patterns in globus pallidus, (2) a monophasic stimulus-entrained fir

212 esponses to joint articulation (3/4 cells in globus pallidus, 11/33 cells in VLo).

213 hod, we were able to map a homunculus in the globus pallidus, a key target area for deep brain stimul

214 tosensory cortex, contralateral amygdala and globus pallidus, and bilateral periaqueductal gray.

215 l gyrus, ventral posterior cingulate cortex, globus pallidus, and calcarine cortex using the threshol

216 ume reductions in the hippocampus, thalamus, globus pallidus, and caudate nucleus compared with 26 co

217 uit with nodes in the red nucleus, thalamus, globus pallidus, and cerebellum.

218 in contralateral amygdala, ventral pallidum, globus pallidus, and hippocampus, as well as decreases i

219 -A5 selectively binds areas of the striatum, globus pallidus, and substantia nigra containing EphA7+

220 itization was found in hypothalamus, cortex, globus pallidus, and substantia nigra of betaarr2-KO com

221 alamus, caudate, putamen, nucleus accumbens, globus pallidus, and substantia nigra).

222 nd distinct firing patterns in the striatum, globus pallidus, and subthalamic nucleus related to sens

223 n between cortex, corpus callosum, striatum, globus pallidus, and thalamus after cerebral injury.

224 ronal tissues from the dentate nuclei, pons, globus pallidus, and thalamus of these 23 deceased patie

225 ronal tissues from the dentate nuclei, pons, globus pallidus, and thalamus were harvested and analyze

226 ively express NuIP such as striatum, septum, globus pallidus, and the reticular thalamic nucleus.

227 D3 selective brain regions (limbic striatum, globus pallidus, and ventral pallidum (9-14%; p < 0.04)

228 ygdala) and sub-cortical (putamen, thalamus, globus pallidus, cerebellum) regions.

229 0.59, 0.51, and 0.50, respectively, for the globus pallidus, cingulate cortex, insula, caudate, puta

230 imaging demonstrated T2 hypointensity in the globus pallidus, confluent T2 white matter hyperintensit

231 on, regions of interest were measured in the globus pallidus, dentate nucleus, thalamus, and pons.

232 Within the globus pallidus, EphA7+ axons terminate primarily within

233 volumes (thalamus, caudate nucleus, putamen, globus pallidus, hippocampus, and nucleus accumbens) oth

234 ucleus accumbens, amygdala, caudate nucleus, globus pallidus, hippocampus, putamen, thalamus, and bra

235 re reward circuit regions including putamen, globus pallidus, insula and thalamus.

236 , 1.46, 0.80, and 0.77 for cingulate cortex, globus pallidus, insula, striatum, and frontal cortex, r

237 av1.8 expression in the amygdala, brainstem, globus pallidus, lateral and paraventricular hypothalamu

238 In the globus pallidus, medium spiny neurons connected strongly

239 the internal globus pallidus (GPi), external globus pallidus, motor cortex, thalamus, or cerebellum.

240 : white matter changes, hypointensity of the globus pallidus, ponto-cerebellar atrophy, and thin corp

241 0A is upregulated in striatal projections to globus pallidus, preferentially expressing D2 receptors

242 e group differences in the substantia nigra, globus pallidus, pulvinar thalamus, thalamus, and caudat

243 mbens, amygdala, brainstem, caudate nucleus, globus pallidus, putamen and thalamus, using genome-wide

244 structures examined (the amygdala, caudate, globus pallidus, putamen, and thalamus).

245 and magnetic field correlation (MFC) in the globus pallidus, putamen, caudate nucleus, and thalamus

246 ional hedonic (orbitofrontal cortex, insula, globus pallidus, putamen, hippocampus, and amygdala) and

247 nucleus compared to all other groups, and in globus pallidus, putamen, substantia nigra and the denta

248 rally elevated (18) F-flortaucipir uptake in globus pallidus, putamen, subthalamic nucleus, midbrain,

249 ith low FA values including caudate nucleus, globus pallidus, putamen, superior temporal gyrus, and s

250 s accumbens, amygdala, caudate, hippocampus, globus pallidus, putamen, thalamus, lateral ventricles.

251 f GPi from the internal capsule and external globus pallidus, respectively.

252 ei, including the subthalamic nucleus (STN), globus pallidus, striatum, and substantia nigra.

253 ojecting back to the same cortical areas via globus pallidus, substantia nigra, and thalamus.

254 severe degeneration in the cardinal nuclei - globus pallidus, subthalamic nucleus and substantia nigr

255 -Fos(+) neurons in the external and internal globus pallidus, subthalamic nucleus, and ventral motor

256 m and instead receiving more inputs from the globus pallidus, subthalamic nucleus, and zona incerta.

257 olinergic projection neurons in the external globus pallidus, suggesting a potential source of motor-

258 ed in the sensorimotor cortex, striatum, and globus pallidus, support the hypothesis of a circuitwide

259 capsule, anterior commissure), gray matter (globus pallidus, thalamus), and cortices (cingulate, mot

260 atients bilaterally in the ventral striatum, globus pallidus, thalamus, amygdala and right insula.

261 creased susceptibility of the neurons of the globus pallidus, thalamus, and substantia nigra pars com

262 The SI in the globus pallidus, thalamus, dentate nucleus, and pons was

263 ead of the caudate nucleus (hCaud), putamen, globus pallidus, thalamus-and four cortical regions-rost

264 in demonstrate the highest Mn content in the globus pallidus, the thalamus, and the substantia nigra

265 al model of subthalamic nucleus and external globus pallidus, we extend the concept of adaptive stimu

266 ation, including the dorsal striatum and the globus pallidus, were also activated.

267 l, particularly in the nucleus accumbens and globus pallidus, where the change in [(11)C]raclopride n

268 ers decreased in the thalamus, striatum, and globus pallidus, while iron-sensitive markers decreased

269 ging showed iron in the substantia nigra and globus pallidus, with a 'halo' of T1 hyperintense signal

270 iated by postsynaptic receptors, and that of globus pallidus-evoked inputs is mediated by presynaptic

271 ed basal ganglia output and thalamic nuclei (globus pallidus-internus [GPi] and ventrolateral anterio

272 Globus pallidus-thalamus and dentate nucleus-pons SI rat

273 intensity changes, signal intensity ratios (globus pallidus-to-thalamus and dentate nucleus-to-pons

274 the caudate, putamen, nucleus accumbens, and globus pallidus.

275 ortex shifts slightly caudally in the caudal globus pallidus.

276 d as disinhibited downstream activity in the globus pallidus.

277 pulate the cortex, hippocampus striatum, and globus pallidus.

278 ls were recorded throughout the sensorimotor globus pallidus.

279 tabolic activity in the central thalamus and globus pallidus.

280 ange projections to the substantia nigra and globus pallidus.

281 d prefrontal areas, and between amygdala and globus pallidus.

282 t brain pineal gland, nucleus accumbens, and globus pallidus.

283 mice after local infusion into the external globus pallidus.

284 isorder and prominent iron deposition in the globus pallidus.

285 llular component (Nkx2-1(-);Npas1(+)) of the globus pallidus.

286 s might indicate gadolinium retention in the globus pallidus.

287 ons at the junction between the striatum and globus pallidus.

288 ssue, most notably in the dentate nuclei and globus pallidus.

289 no changes being detected in the putamen or globus pallidus.

290 ar peduncle, colliculi, dentate nucleus, and globus pallidus.

291 lar nucleus/substantia nigra and to external globus pallidus.

292 ulate cortex and increased metabolism in the globus pallidus.

293 lly significant dose-dependent relationship (globus pallidus: rho = 0.90, P = .04).

294 modulation in firing patterns (2/12 cells in globus pallidus; 13/23 cells in VLo), and regularized fi

295 ral, associative, and sensorimotor striatum; globus pallidus; central thalamus and noncentral thalamu

296 e stria terminalis; the caudate-putamen; the globus pallidus; the lateral septum; and the islands of

297 Although the majority of FGIDs, including globus, rumination syndrome, IBS, bloating, constipation

298 , dysphagia (32.3% vs. 13.1%; p = 0.001) and globus sensation (27.7% vs. 13.8%; p = 0.021) were found

299 s (7.2%), dysgeusia (0.3%), dysosmia (0.2%), globus sensation (8.2%), surgical site infection (1.3%),

300 lly significant rate of VP insufficiency and globus sensation; however, studies lack details of surgi