ライフサイエンスコーパス: globus pallidus

1 ance in the striatum and axon entry into the globus pallidus).

2 ortex shifts slightly caudally in the caudal globus pallidus.

3 the caudate, putamen, nucleus accumbens, and globus pallidus.

4 d as disinhibited downstream activity in the globus pallidus.

5 pulate the cortex, hippocampus striatum, and globus pallidus.

6 ls were recorded throughout the sensorimotor globus pallidus.

7 tabolic activity in the central thalamus and globus pallidus.

8 ange projections to the substantia nigra and globus pallidus.

9 d prefrontal areas, and between amygdala and globus pallidus.

10 t brain pineal gland, nucleus accumbens, and globus pallidus.

11 mice after local infusion into the external globus pallidus.

12 isorder and prominent iron deposition in the globus pallidus.

13 llular component (Nkx2-1(-);Npas1(+)) of the globus pallidus.

14 s might indicate gadolinium retention in the globus pallidus.

15 ssue, most notably in the dentate nuclei and globus pallidus.

16 no changes being detected in the putamen or globus pallidus.

17 ar peduncle, colliculi, dentate nucleus, and globus pallidus.

18 ons at the junction between the striatum and globus pallidus.

19 lar nucleus/substantia nigra and to external globus pallidus.

20 ulate cortex and increased metabolism in the globus pallidus.

21 intensity increase per injection, P < .001), globus pallidus (0.23% increase, P = .009), posterior th

22 esponses to joint articulation (3/4 cells in globus pallidus, 11/33 cells in VLo).

23 he nucleus accumbens (10 +/- 7%), and in the globus pallidus (13 +/- 15%) measured with [(11)C]PHNO,

24 modulation in firing patterns (2/12 cells in globus pallidus; 13/23 cells in VLo), and regularized fi

25 telangiectasia also had higher metabolism in globus pallidus (16%, P = 0.05), which correlated negati

26 n: (1) stimulus-entrained firing patterns in globus pallidus, (2) a monophasic stimulus-entrained fir

27 ording single-unit spiking activity from the globus pallidus (232 neurons) while the animals complete

28 spite loss of kinematic tuning, cells in the globus pallidus (63%, 10/16 cells) and VLo (84%, 46/55 c

29 joint position, velocity, or acceleration in globus pallidus (75%, 12/16 cells) and in the pallidal r

30 bstantia nigra (SN; +46%; p<0.02) and in the globus pallidus (+9%; p=0.06) and ventral pallidum (+11%

31 hod, we were able to map a homunculus in the globus pallidus, a key target area for deep brain stimul

32 reduce central thalamus activity and release globus pallidus activity in DOCs.

33 e alignments between subthalamic nucleus and globus pallidus amplified local neural synchrony in the

34 The reversal of globus pallidus and central thalamus profiles across BIs

35 ing single-unit activity in the sensorimotor globus pallidus and motor thalamus before, during, and a

36 y specific brain areas such as the thalamus, globus pallidus and orbitofrontal regions of the right h

37 ssociated with a lower T1R (P = 0.01) in the globus pallidus and putamen but were not associated with

38 nd most prolonged increases occurring in the globus pallidus and putamen.

39 y between external and internal parts of the globus pallidus and saliency of the sensory input, a low

40 t of the parvalbumin-positive neurons in the globus pallidus and striatum of the Ldb1 mutants.

41 e recapitulated in STN, but also in external globus pallidus and striatum.

42 put from the caudal BF, including the caudal globus pallidus and substantia innominata and moderate i

43 s, and projected their axons to the targeted globus pallidus and substantia nigra in a time-dependent

44 ar whether abnormal activity measured in the globus pallidus and substantia nigra pars reticulata is

45 Activity in the internal segment of the globus pallidus and subthalamic nucleus correlated stron

46 but also bilateral caudate and left putamen/globus pallidus and subthalamic nucleus.

47 tion of subcortical structures (specifically globus pallidus and thalamus) explains individual biases

48 ined the surface morphology of the striatum, globus pallidus and thalamus, and thickness of the cereb

49 the parieto-occipital regions, left putamen/globus pallidus and thalamus; and in white matter of the

50 al neurons in rodents), which project to the globus pallidus and the substantia nigra, and the locall

51 in the striatum and their projections to the globus pallidus and the substantia nigra.

52 eceptor-mediated inhibition arising from the globus pallidus and thus promote single-spike activity r

53 striatal D1-MSNs collateralized to both the globus pallidus and ventral mesencephalon.

54 ve been reported about hyperintensity of the globus pallidus and/or dentate nucleus on unenhanced T1-

55 l striatum with the dorsal striatum (putamen/globus pallidus), and lower connectivity between these r

56 tosensory cortex, contralateral amygdala and globus pallidus, and bilateral periaqueductal gray.

57 l gyrus, ventral posterior cingulate cortex, globus pallidus, and calcarine cortex using the threshol

58 ume reductions in the hippocampus, thalamus, globus pallidus, and caudate nucleus compared with 26 co

59 uit with nodes in the red nucleus, thalamus, globus pallidus, and cerebellum.

60 in contralateral amygdala, ventral pallidum, globus pallidus, and hippocampus, as well as decreases i

61 -A5 selectively binds areas of the striatum, globus pallidus, and substantia nigra containing EphA7+

62 itization was found in hypothalamus, cortex, globus pallidus, and substantia nigra of betaarr2-KO com

63 alamus, caudate, putamen, nucleus accumbens, globus pallidus, and substantia nigra).

64 nd distinct firing patterns in the striatum, globus pallidus, and subthalamic nucleus related to sens

65 n between cortex, corpus callosum, striatum, globus pallidus, and thalamus after cerebral injury.

66 ronal tissues from the dentate nuclei, pons, globus pallidus, and thalamus of these 23 deceased patie

67 ronal tissues from the dentate nuclei, pons, globus pallidus, and thalamus were harvested and analyze

68 ively express NuIP such as striatum, septum, globus pallidus, and the reticular thalamic nucleus.

69 D3 selective brain regions (limbic striatum, globus pallidus, and ventral pallidum (9-14%; p < 0.04)

70 thalamic nucleus (STN), rostromedial lateral globus pallidus, and ventral pallidum.

71 indicating spontaneous beta oscillations in globus pallidus are not pathognomonic.

72 Globus pallidus binding best distinguished PSP patients

73 ared with NT mice, significantly increase in globus pallidus but decrease in entopeduncular nucleus/s

74 in the dorsal caudate nucleus, putamen, and globus pallidus but the observed variation did not assoc

75 nd caudate nucleus, red nucleus, putamen and globus pallidus by T2* MRI at baseline and after 3 and 6

76 ral, associative, and sensorimotor striatum; globus pallidus; central thalamus and noncentral thalamu

77 ygdala) and sub-cortical (putamen, thalamus, globus pallidus, cerebellum) regions.

78 0.59, 0.51, and 0.50, respectively, for the globus pallidus, cingulate cortex, insula, caudate, puta

79 imaging demonstrated T2 hypointensity in the globus pallidus, confluent T2 white matter hyperintensit

80 on, regions of interest were measured in the globus pallidus, dentate nucleus, thalamus, and pons.

81 al neurons, and magnocellular neurons in the globus pallidus did not demonstrate a similar extent of

82 s in CMR and increases in CBF in the putamen/globus pallidus, dorsal midbrain/pons, STN, and ventral

83 Within the globus pallidus, EphA7+ axons terminate primarily within

84 iated by postsynaptic receptors, and that of globus pallidus-evoked inputs is mediated by presynaptic

85 uantification of single-unit activity in the globus pallidus externa (GPe) and substantia nigra retic

86 a from the globus pallidus interna (GPi) and globus pallidus externa (GPe) in children undergoing dee

87 strates that functional connections from the globus pallidus externa (GPe) to striatum are substantia

88 es in firing rates of single neurons in STN, globus pallidus externa (GPe), and substantia nigra pars

89 cortex, the globus pallidus interna, and the globus pallidus externa compared with patients with trem

90 o the homolog of the globus pallidus interna/globus pallidus externa express D1 or D2 receptors.

91 cortex, the globus pallidus interna, and the globus pallidus externa.

92 s dopamine D2 receptors (D2+) project to the globus pallidus external and are thought to inhibit move

93 l neurons comprising subthalamic nucleus and globus pallidus external and suggest this approach for d

94 we report that a previously neglected CeA-to-globus pallidus external segment (GPe) circuit plays an

95 eptide somatostatin, send projections to the globus pallidus external segment (GPe), and this CeA-GPe

96 ent striatal (Str) neurons projecting to the globus pallidus external segment (GPe).

97 ectly through the caudal-ventral part of the globus pallidus externus (cvGPe).

98 ere we describe a direct projection from the globus pallidus externus (GP), a central nucleus of the

99 al perikarya, ENK-immunostained terminals in globus pallidus externus (GPe) were more abundant at 6 m

100 ne increased rabies-labelled inputs from the globus pallidus externus (GPe), a basal ganglia nucleus

101 Gamma oscillations arise in the subthalamic-globus pallidus feedback loop, and occur during movement

102 , and survival of forebrain interneurons and globus pallidus GABAergic neurons, thereby leading to th

103 In the globus pallidus (GP in rodents, homolog of the primate G

104 -related increases in iron concentrations in globus pallidus (GP) and substantia nigra (SN), although

105 hat volumetric hemispheric lateralization of globus pallidus (GP) and thalamus (Th) explains individu

106 The firing patterns of neurons in the globus pallidus (GP) are affected by two major sources o

107 halamus before, during, and after DBS in the globus pallidus (GP) GP-DBS resulted in: (1) stimulus-en

108 SI) increase in the dentate nucleus (DN) and globus pallidus (GP) in relation to the middle cerebella

109 leus (STN) neurons and two types of external globus pallidus (GP) neuron inappropriately synchronise

110 Synchronization of globus pallidus (GP) neurons and cortically entrained os

111 Globus pallidus (GP) neurons fire rhythmically in the ab

112 halamic nucleus (STN) and GABAergic external globus pallidus (GP) neurons normally exhibit weakly cor

113 Globus pallidus (GP) neurons recorded in brain slices sh

114 MSNs and FSIs together with motor cortex and globus pallidus (GP) neurons, in rats performing a simpl

115 onsidered similar, if not homologous, to the globus pallidus (GP) of mammals.

116 levodopa responses was seen in the striatum/globus pallidus (GP) of the lesioned hemisphere.

117 The globus pallidus (GP) predominantly contains GABAergic pr

118 In comparison, the globus pallidus (GP), a main striatal output nucleus, ha

119 triatum and their subsequent growth into the globus pallidus (GP), an important (intermediate) target

120 s were measured in the dentate nucleus (DN), globus pallidus (GP), crus anterior of capsula interna (

121 The signal intensities (SIs) in the globus pallidus (GP), thalamus (T), dentate nucleus (DN)

122 with those of the lateral striatum (LSt) and globus pallidus (GP).

123 substantia nigra (SN), pulvinar thalami, and globus pallidus (GP).

124 he reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

125 he reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

126 Reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

127 Reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

128 n-expressing (PV(+)) neurons in the external globus pallidus (GPe) are critically involved in the mot

129 ine-depleted rats indicate that the external globus pallidus (GPe) contains two main types of GABAerg

130 Neurons in the external globus pallidus (GPe) generate pacemaker activity that c

131 whether the manipulation of the DMS-external globus pallidus (GPe) iMSNs circuit alters the ethanol-c

132 tal dopamine, the striatum, and the external globus pallidus (GPe) in regulating RLS-like movements,

133 cal excitation and indirect pathway external globus pallidus (GPe) inhibition of the STN are critical

134 The external globus pallidus (GPe) is a critical node within the basa

135 The external globus pallidus (GPe) is a key contributor to motor supp

136 The external globus pallidus (GPe) is a key nucleus within basal gang

137 thin the basal ganglia circuit, the external globus pallidus (GPe) is critically involved in motor co

138 The external segment of the globus pallidus (GPe) is one of the core nuclei of the b

139 We show how external globus pallidus (GPe) neuron is sensitive to the phase o

140 analogous division of labor in the external globus pallidus (GPe) of Parkinsonian rats, showing that

141 of the neuronal composition in the external globus pallidus (GPe) undermines our ability to interrog

142 the constituent neurons within the external globus pallidus (GPe) was not fully appreciated.

143 ated activity of the external segment of the globus pallidus (GPe), a downstream structure whose comp

144 s that pathological activity of the external globus pallidus (GPe), a nucleus in the basal ganglia, c

145 In the external segment of the globus pallidus (GPe), neuronal populations have been de

146 ison to the adjacent external segment of the globus pallidus (GPe), remain unexplored.

147 Focusing on the mouse external globus pallidus (GPe), we demonstrate that the potential

148 ic nucleus (STN) and the external segment of globus pallidus (GPe).

149 ural circuits in the external segment of the globus pallidus (GPe).

150 highest levels in the striatum and external globus pallidus (GPe).

151 ng in neurons of the external segment of the globus pallidus (GPe).

152 , and neurons in the external segment of the globus pallidus (GPe)], and one neuromodulator group [st

153 nputs from the striatum (direct pathway) and globus pallidus (GPe, indirect pathway).

154 show that neurons in the habenula-projecting globus pallidus (GPh) in mice are essential for evaluati

155 uit, which regulates the habenula-projecting globus pallidus (GPh) neurons, exists within the basal g

156 eive GABAergic projections from the internal globus pallidus (GPi) and glutamatergic inputs from moto

157 of the subthalamic nucleus (STN) or internal globus pallidus (GPi) reduces dyskinesias remain largely

158 ive visualization scores of STN and internal globus pallidus (GPi) were recorded by two neuroradiolog

159 drove network activity through the internal globus pallidus (GPi), external globus pallidus, motor c

160 deep brain stimulation (DBS) at the internal globus pallidus (GPi).

161 onal activity in the internal segment of the globus pallidus (GPi).

162 , resulting in a heterogeneous distribution (globus pallidus > cortical regions > cerebellum) consist

163 volumes (thalamus, caudate nucleus, putamen, globus pallidus, hippocampus, and nucleus accumbens) oth

164 ucleus accumbens, amygdala, caudate nucleus, globus pallidus, hippocampus, putamen, thalamus, and bra

165 the basal ganglia, the caudate, putamen and globus pallidus in brown capuchin monkeys (Cebus apella)

166 basal ganglia nuclei (caudate, putamen, and globus pallidus) in children with ADHD.

167 n metabolic rates including hyperactivity in globus pallidus indicative of basal ganglia involvement.

168 ance imaging of the brain revealed bilateral globus pallidus injury in the setting of dyskinetic CP.

169 re reward circuit regions including putamen, globus pallidus, insula and thalamus.

170 , 1.46, 0.80, and 0.77 for cingulate cortex, globus pallidus, insula, striatum, and frontal cortex, r

171 We report microelectrode data from the globus pallidus interna (GPi) and globus pallidus extern

172 ndomised to subthalamic nucleus (STN) versus globus pallidus interna (GPi) DBS surgery.

173 ood effects of subthalamic nucleus (STN) vs. globus pallidus interna (GPi) deep brain stimulation (DB

174 ut a previous case suggested that DBS of the globus pallidus interna (GPi) might.

175 ither the subthalamic nucleus (STN, n=84) or globus pallidus interna (GPi, n=80), using standardised

176 o had undergone bilateral stimulation of the globus pallidus interna (pallidal stimulation) or subtha

177 The basal ganglia, including the striatum, globus pallidus interna and externa (GPe), subthalamic n

178 The globus pallidus interna and the subthalamic nucleus are

179 n of Medtronic quadripolar electrodes in the globus pallidus interna bilaterally.

180 Long-term data on the outcomes of globus pallidus interna DBS are limited and mostly confi

181 The results suggest anteromedial globus pallidus interna DBS for Tourette's syndrome is a

182 the effectiveness of DBS of the anteromedial globus pallidus interna on tic severity and common comor

183 ating that stimulation of an area called the globus pallidus interna partially reverses deficits in v

184 viously ignored structure in data describing globus pallidus interna responses to cortical stimulatio

185 ilateral dorsolateral prefrontal cortex, the globus pallidus interna, and the globus pallidus externa

186 ilateral dorsolateral prefrontal cortex, the globus pallidus interna, and the globus pallidus externa

187 targeting the subthalamic nucleus (STN) and globus pallidus interna, is a surgical therapy with clas

188 from the dorsal pallidum, the homolog of the globus pallidus interna.

189 tal neurons projecting to the homolog of the globus pallidus interna/globus pallidus externa express

190 jor output station of the basal ganglia, the globus pallidus internal segment (GPi) projects to the t

191 paring the role of inputs originating in the globus pallidus internal segment (GPi), and lateral hypo

192 ion in the basal ganglia output nucleus, the globus pallidus internal segment.

193 trating that deep brain stimulation (DBS) of globus pallidus internus (GPi DBS) and subthalamic nucle

194 ed in the centromedial (CM) thalamus (n=51), globus pallidus internus (GPi) (n=47), nucleus accumbens

195 assess the safety and efficacy of bilateral globus pallidus internus (GPi) DBS in patient's with sev

196 tes during subthalamic nucleus (STN) DBS and globus pallidus internus (GPi) DBS.

197 ients to either subthalamic nucleus (STN) or globus pallidus internus (GPi) deep brain stimulation (D

198 implicates the subthalamic nucleus (STN) and globus pallidus internus (GPi) in reward and punishment

199 n (DBS) of the subthalamic nucleus (STN) and globus pallidus internus (GPi) is an effective treatment

200 studies have characterized beta power in the globus pallidus internus (GPi), an equally effective DBS

201 dies have characterized beta activity in the globus pallidus internus (GPi), another effective target

202 corded the activity of single neurons in the globus pallidus internus (GPi), the primary BG output nu

203 in skeletomotor output region of the BG, the globus pallidus internus (GPi).

204 f patients (n = 66) implanted bilaterally in globus pallidus internus (n = 34) or centromedial thalam

205 Deep brain stimulation of the globus pallidus internus alleviates involuntary movement

206 a small volume covering the ventroposterior globus pallidus internus and adjacent subpallidal white

207 direct pathway neurons were more abundant in globus pallidus internus and substantia nigra at 6 month

208 ed tonic inhibition of the thalamus from the globus pallidus internus could lead to an under-suppress

209 l studies reported the beneficial effects of globus pallidus internus deep brain stimulation (GPi DBS

210 amma synchronization (ERS) recorded from the globus pallidus internus in patients undergoing deep bra

211 ed increased interaction between putamen and globus pallidus internus, and decreased interaction betw

212 The results showed that for globus pallidus internus, connectivity to limbic network

213 l connectivity from the sensorimotor cortex, globus pallidus internus, ventral intermediate thalamic

214 DBS targets (STN, subthalamic nucleus; GPi, globus pallidus internus; NAc, nucleus accumbens) evoked

215 ed basal ganglia output and thalamic nuclei (globus pallidus-internus [GPi] and ventrolateral anterio

216 Deep brain stimulation of the internal globus pallidus is a highly effective and established th

217 av1.8 expression in the amygdala, brainstem, globus pallidus, lateral and paraventricular hypothalamu

218 a unilateral excitotoxic injection into the globus pallidus leads to a gradual loss of dopaminergic

219 ucleus, and movement-related activity of the globus pallidus leads to transient beta oscillations dur

220 s dopamine cell death at 3 weeks following a globus pallidus lesion, but not thereafter.

221 for post-GBCA group [P < .001]) but not the globus pallidus (mean SI ratio for two-group comparison:

222 In the globus pallidus, medium spiny neurons connected strongly

223 mean uptake after BI; a relative increase in globus pallidus metabolism was evident in BI subjects wh

224 the internal globus pallidus (GPi), external globus pallidus, motor cortex, thalamus, or cerebellum.

225 shortening in the dentate nucleus (n = 13), globus pallidus (n = 13), substantia nigra (n = 13), pos

226 ation of the subthalamic nucleus (n = 60) or globus pallidus (n = 61).

227 The subthalamic nucleus-globus pallidus network is a potential source of oscilla

228 We show that most globus pallidus neurons, but very few neocortical intern

229 om the Shh-expressing domain eliminated most globus pallidus neurons, whereas most cortical and stria

230 cortical interneurons; and pattern C, mainly globus pallidus neurons.

231 Striatal lesion core and globus pallidus of Abeta + ET1 rats showed extensive deg

232 recordings from the subthalamic nucleus and globus pallidus of five patients with Parkinson's diseas

233 sed local field in the caudate, putamen, and globus pallidus of patients relative to control subjects

234 activity occurs in the prefrontal cortex and globus pallidus of patients with nontremor-dominant PD c

235 onounced differences in substantia nigra and globus pallidus of PSP compared with control brains.

236 spectively, into the superior colliculus and globus pallidus of Sprague-Dawley rats.

237 tial component compared with inhibition from globus pallidus or substantia nigra pars reticulata.

238 s by stimulation of afferents from striatum, globus pallidus, or pars reticulata have been shown to b

239 " pathways that regulate the pallidal (e.g., globus pallidus) output nuclei involved in the control o

240 e gray matter of the whole brain (P < .001), globus pallidus (P = .002), dentate nucleus (P = .046),

241 ight caudate nucleus and ventral putamen and globus pallidus (P = .003) compared with that in control

242 years in the substantia nigra (P < .001) and globus pallidus (P = .035), which are both predictors of

243 caudate (P < 0.001), putamen (P < 0.001) and globus pallidus (P = 0.025) in patients with Parkinson's

244 Within the basal ganglia, the globus pallidus pars externa (GPe) has been hypothesized

245 ferentially to deep brain stimulation of the globus pallidus pars interna (GPi DBS).

246 e treated with deep brain stimulation of the globus pallidus pars interna during the period 1999-2010

247 rm efficacy of deep brain stimulation of the globus pallidus pars interna in the treatment of general

248 r predicting a better or poorer outcome from globus pallidus pars interna stimulation in this series

249 : white matter changes, hypointensity of the globus pallidus, ponto-cerebellar atrophy, and thin corp

250 0A is upregulated in striatal projections to globus pallidus, preferentially expressing D2 receptors

251 e group differences in the substantia nigra, globus pallidus, pulvinar thalamus, thalamus, and caudat

252 mbens, amygdala, brainstem, caudate nucleus, globus pallidus, putamen and thalamus, using genome-wide

253 structures examined (the amygdala, caudate, globus pallidus, putamen, and thalamus).

254 and magnetic field correlation (MFC) in the globus pallidus, putamen, caudate nucleus, and thalamus

255 ional hedonic (orbitofrontal cortex, insula, globus pallidus, putamen, hippocampus, and amygdala) and

256 nucleus compared to all other groups, and in globus pallidus, putamen, substantia nigra and the denta

257 rally elevated (18) F-flortaucipir uptake in globus pallidus, putamen, subthalamic nucleus, midbrain,

258 ith low FA values including caudate nucleus, globus pallidus, putamen, superior temporal gyrus, and s

259 s accumbens, amygdala, caudate, hippocampus, globus pallidus, putamen, thalamus, lateral ventricles.

260 0.005), putamen (r = -0.51; P = 0.025), and globus pallidus (r = -0.47; P = 0.030).

261 0.011), putamen (r = -0.48; P = 0.022), and globus pallidus (r = -0.70; P < 0.001).

262 f GPi from the internal capsule and external globus pallidus, respectively.

263 lly significant dose-dependent relationship (globus pallidus: rho = 0.90, P = .04).

264 Projections to the globus pallidus showed solely D2-mediated presynaptic in

265 ei, including the subthalamic nucleus (STN), globus pallidus, striatum, and substantia nigra.

266 ojecting back to the same cortical areas via globus pallidus, substantia nigra, and thalamus.

267 severe degeneration in the cardinal nuclei - globus pallidus, subthalamic nucleus and substantia nigr

268 -Fos(+) neurons in the external and internal globus pallidus, subthalamic nucleus, and ventral motor

269 m and instead receiving more inputs from the globus pallidus, subthalamic nucleus, and zona incerta.

270 olinergic projection neurons in the external globus pallidus, suggesting a potential source of motor-

271 ed in the sensorimotor cortex, striatum, and globus pallidus, support the hypothesis of a circuitwide

272 tside our Holmes tremor circuit, whereas the globus pallidus target was close, consistent with publis

273 capsule, anterior commissure), gray matter (globus pallidus, thalamus), and cortices (cingulate, mot

274 atients bilaterally in the ventral striatum, globus pallidus, thalamus, amygdala and right insula.

275 creased susceptibility of the neurons of the globus pallidus, thalamus, and substantia nigra pars com

276 The SI in the globus pallidus, thalamus, dentate nucleus, and pons was

277 ead of the caudate nucleus (hCaud), putamen, globus pallidus, thalamus-and four cortical regions-rost

278 Globus pallidus-thalamus and dentate nucleus-pons SI rat

279 are distributed more caudally in the caudal globus pallidus than noncholinergic projection neurons.

280 density of direct pathway collaterals in the globus pallidus that bridge the direct pathway with the

281 in demonstrate the highest Mn content in the globus pallidus, the thalamus, and the substantia nigra

282 e stria terminalis; the caudate-putamen; the globus pallidus; the lateral septum; and the islands of

283 ing potential, which ranged from 2.42 in the globus pallidus to 8.48 in the nucleus accumbens.

284 ential values (from approximately 0.5 in the globus pallidus to approximately 3.5 in the insula) for

285 ly distinct projections through striatum and globus pallidus to EP targets within epithalamus and tha

286 discovered feedback projection from external globus pallidus to striatum is crucial for inhibitory co

287 intensity changes, signal intensity ratios (globus pallidus-to-thalamus and dentate nucleus-to-pons

288 reservation in vicinities of the cerebellum, globus pallidus, visual cortex, and parietal cortex in o

289 n, TBF was solely negatively associated with globus pallidus volume.

290 al model of subthalamic nucleus and external globus pallidus, we extend the concept of adaptive stimu

291 However, shorter T1 relaxation times in the globus pallidus were found in group 1 compared with grou

292 alamus and thalamic subnuclei, striatum, and globus pallidus were segmented using a fully automated m

293 Protein components of the globus pallidus were studied including glial fibrillary

294 ation, including the dorsal striatum and the globus pallidus, were also activated.

295 ith diabetes and MDD showed lower MTR in the globus pallidus when compared with the group with MDD.

296 l, particularly in the nucleus accumbens and globus pallidus, where the change in [(11)C]raclopride n

297 ) with glucose metabolism in the putamen and globus pallidus, which receive projections from this str

298 ers decreased in the thalamus, striatum, and globus pallidus, while iron-sensitive markers decreased

299 Moreover, low signal intensity in globus pallidus with hyperintensive streaking and low si

300 ging showed iron in the substantia nigra and globus pallidus, with a 'halo' of T1 hyperintense signal