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1 he treated group, as was thrombosis of renal glomerular capillaries.
2 at correlated with interactions with forming glomerular capillaries.
3 cular, AP activation is often limited to the glomerular capillaries.
4 tion requires continuous blood filtration by glomerular capillaries.
5 e filtration process governed by fenestrated glomerular capillaries.
6 lity to change the geometry and curvature of glomerular capillaries.
7 isruption under the high shear conditions in glomerular capillaries.
8 ration barrier and cover the outer aspect of glomerular capillaries.
9 igitating secondary extensions to enwrap the glomerular capillaries.
10  and the deposition of fibrin thrombi in the glomerular capillaries.
11 of these podocytes mimicked the formation of glomerular capillaries.
12 teoglycans, coats the luminal surface of the glomerular capillaries.
13 wing more and slow RAP fluctuations to reach glomerular capillaries.
14 g and detachment of endothelial cells of the glomerular capillaries.
15 utrophils, which were similar in diameter to glomerular capillaries, abruptly arrested following anti
16 ant) rats showed splitting and thickening of glomerular capillaries and had a longer survival rate, c
17  in neutrophil recruitment and dwell time in glomerular capillaries and in reactive oxygen species (R
18 levels of all inflammatory markers, restored glomerular capillaries and podocyte structure, and arres
19 ctor (vWF), were identified predominantly in glomerular capillaries and rarely in arterioles, but not
20 fferentiated endothelial cells in developing glomerular capillaries and reduced lumen formation in vi
21 h are preferentially recruited, dwell within glomerular capillaries, and acquire proinflammatory char
22                                   The kidney glomerular capillaries are frequent sites of immune comp
23    Cells of renin lineage residing alongside glomerular capillaries are reported to have progenitor c
24 e interstitial space and within the walls of glomerular capillaries as well as the cellular processes
25 fit the dextran sieving data represented the glomerular capillary as being perforated by small restri
26 results from immune complex precipitation in glomerular capillaries, as in some cryoglobulinaemic hum
27 merular volume, fractional mesangial volume, glomerular capillary basement membrane width, and urinar
28 uring formation of the renal glomerulus, the glomerular capillary becomes enveloped by highly special
29                                          The glomerular capillary bed seems to contribute to all subt
30 dothelium, and mesangium associated with the glomerular capillary bed to maintain filtration barrier
31 correlation was observed between retinal and glomerular capillary BM thickness (r=0.79, P=0.0001), be
32 chanism whereby mesangial cells organize the glomerular capillaries by adhering to the G domain of la
33  endothelial cells are cleared from immature glomerular capillaries by apoptosis, a process regulated
34 molecule in glomerular endothelial cells and glomerular capillary circumference.
35 lecule-1 in glomerular endothelial cells and glomerular capillary circumference.
36               In animals with renal failure, glomerular capillary collapse and tubular necrosis were
37 Human, but not murine, renal peritubular and glomerular capillaries constitutively express class II m
38              Physiologic permeability of the glomerular capillary depends on the normal structure of
39 e expression of growth factors that regulate glomerular capillary development and that abnormal capil
40 genesis of the kidney vasculature, including glomerular capillary development, arterial mural cell co
41 lium adjacent to the GBM, but convolution of glomerular capillaries did not occur.
42 r pressure, the latter eventually leading to glomerular capillary dropout (rarefaction) and further i
43  patterning, excess endothelial cells within glomerular capillaries, effaced podocytes with extremely
44 tients, viral inclusions were present in the glomerular capillary endothelia without any associated i
45 ne methyl ester (NMMA) increased TGF-beta in glomerular capillary endothelial cells (GCECs) and stimu
46 nchymal cells and on putative progenitors of glomerular capillary endothelial cells early in their re
47 Hg and led to severe loss of fenestration of glomerular capillary endothelial cells in both eNOS-defi
48                     Tie2 was demonstrated on glomerular capillary endothelial cells, particularly on
49  ECRTP/DEP-1 is expressed in anticipation of glomerular capillary endothelial recruitment during deve
50 tic eNOS(-/-) mice, even though it inhibited glomerular capillary enlargement in both.
51                                       Normal glomerular capillaries filter plasma through a basement
52 y a pathway of neutrophil recruitment within glomerular capillaries following IgG deposition that may
53  (pod-Cre), which express cre at the time of glomerular capillary formation.
54                       Detailed assessment of glomerular capillaries from diabetic D2 mice demonstrate
55 unction despite BP fluctuations and protects glomerular capillaries from hypertensive injury.
56                                          The glomerular capillaries function as the filtration barrie
57 dified serum proteins adversely affect renal glomerular capillary function, structure, and metabolism
58 ed spatial orientation of their processes on glomerular capillaries goes well in line with the role o
59 hly dynamic biomechanical environment of the glomerular capillaries greatly influences the cell biolo
60 elae of endothelial dysfunction in diabetes: glomerular capillary growth and effects on neighboring p
61 t protocol demonstrated greater reduction of glomerular capillary hydraulic pressure with OMA than wi
62 tionship between systemic blood pressure and glomerular capillary hydrostatic pressure and by decreas
63                                              Glomerular capillary hydrostatic pressure and hydrostati
64 iltration coefficient and an increase in the glomerular capillary hydrostatic pressure gradient.
65                       SGLT2 inhibitors lower glomerular capillary hypertension and hyperfiltration, t
66             Experimental studies showed that glomerular capillary hypertension and impaired sieving f
67 esangial cell stretching, mimicking in vitro glomerular capillary hypertension, enhances MCP-1 expres
68 rophy and glomerular hyperfiltration but not glomerular capillary hypertension.
69 abetes BM thickening develops in retinal and glomerular capillaries in a correlated manner.
70 ellular matrix (ECM) replacement of areas of glomerular capillaries in histologic variants of FSGS ar
71 cytes are epithelial cells that surround the glomerular capillaries in the kidney and are necessary f
72  showed accumulation of fibrin/fibrinogen in glomerular capillaries, increased numbers of glomerular
73 sses may display a preferable orientation on glomerular capillaries instead of a random distribution.
74 eased renal vasculature, particularly of the glomerular capillary knot, dysregulation of nephrin and
75 tification of key structural proteins in the glomerular capillary loop which may contribute to defect
76  duct, (2) the fleer mutation with distended glomerular capillary loops and cystic tubules, and (3) t
77 ed both histological TMA lesions (thrombi in glomerular capillary loops and small arteries, mesangiol
78 about the molecular changes occurring within glomerular capillary loops during development of disease
79 n mutant kidneys, the extent of branching of glomerular capillary loops was decreased, with capillary
80  around the circumference of human and mouse glomerular capillary loops, it co-localized only partial
81 ly increased APA staining in areas of intact glomerular capillary loops.
82 ction in vasculature density and dilation of glomerular capillary loops.
83     This study explored the question whether glomerular capillary lumen formation in vivo may involve
84 ious finding, that TGF-beta1 blockade blunts glomerular capillary lumen formation in vivo, it is prop
85  subendothelial lucency, platelet thrombi in glomerular capillary lumina).
86 biopsy showed massive pseudo-thrombi filling glomerular capillary lumina.
87                 Thus the 3D structure of the glomerular capillary network provides useful information
88 eory analysis to explore the topology of the glomerular capillary network.
89 bloodstream and bind to proteoglycans in the glomerular capillaries of kidneys, where it can react wi
90  between GEC MMP-9 expression and changes in glomerular capillary permeability.
91                     Due to their position on glomerular capillaries, podocytes are continuously count
92                                              Glomerular capillary pressure (P(GC)) was also significa
93 olecular pathways that are activated by high glomerular capillary pressure and hyperglycemia and how
94 tes may be able to respond to changes in the glomerular capillary pressure and modulate the GFR.
95 in the kidney glomerulus that are exposed to glomerular capillary pressure and possible increases in
96 ated with glomerular capillary pressure, and glomerular capillary pressure no longer correlated with
97 lic blood pressure no longer correlated with glomerular capillary pressure, and glomerular capillary
98                                 ACEI reduced glomerular capillary pressure, increased glomerular ultr
99 ve agents affect systemic blood pressure and glomerular capillary pressure.
100  densa and (b) glomerular filtration rate or glomerular capillary pressure.
101         This process develops over time into glomerular capillary rarefaction and glomerulosclerosis,
102 s due to a significantly increased number of glomerular capillary segments and capillary branch point
103 odocyte injury and depletion and collapse of glomerular capillary segments.
104 se glomerular endothelial cells and maintain glomerular capillary structure by mechanical and poorly
105                                              Glomerular capillary surface area remained stable, but t
106 se or focal, global or segmental collapse of glomerular capillaries, swelling and hypercellularity of
107                         Endothelial cells of glomerular capillaries, the podocytes wrapped around the
108  how parietal epithelial cells (PECs) invade glomerular capillaries, thereby promoting injury and kid
109  in eNOS-/- mice, especially with respect to glomerular capillary thrombosis and neutrophil infiltrat
110 ar capillaries+, endothelial C4d staining of glomerular capillaries+, transplant glomerulopathy and v
111 iated mesangial cell process invasion of the glomerular capillary tuft as an initiation mechanism of
112 order characterized by focal scarring of the glomerular capillary tuft, podocyte injury, and nephroti
113 s or increases blood flow through the entire glomerular capillary tuft.
114 duced mesangial cell process invasion of the glomerular capillary tuft.
115 yte catastrophe) and subsequent wrinkling of glomerular capillaries, tuft collapse, and periglomerula
116    Streptozotocin-induced diabetes increased glomerular capillary volume in both C57BL/6 and eNOS(-/-
117 ent membrane (GBM) is a key component of the glomerular capillary wall and is essential for kidney fi
118            In addition, they had substantial glomerular capillary wall deposits of IgG and C3, which
119 standing to emerge of how the 3 parts of the glomerular capillary wall jointly determine its function
120 circulation causes albuminuria by increasing glomerular capillary wall permeability and intraglomerul
121 glomerulitis showed ultrastructural signs of glomerular capillary wall remodeling.
122 APKD can identify focal abnormalities of the glomerular capillary wall to aid in the early diagnosis
123 ting from immune complexes formed within the glomerular capillary wall.
124  the exact locations of Ang1 and Tie2 in the glomerular capillary wall.
125 e combined properties of the three layers of glomerular capillary wall: glomerular endothelial cells
126                      Immune complexes within glomerular capillary walls cause crescentic GN (CrGN).
127 disease characterized by a thickening of the glomerular capillary walls due to immune complex deposit
128  deposits of immune complexes in situ in the glomerular capillary walls.
129 ng showed extensive linear C3 staining along glomerular capillary walls.
130          Odor-evoked functional hyperemia in glomerular capillaries was highly correlated with glutam
131  and positive C4d staining of peritubular or glomerular capillaries were present at the time of diagn
132 lomerulonephritis, NETs are generated in the glomerular capillaries, where they are short lived and m
133  ultrafiltration in the kidney occurs across glomerular capillaries, which are surrounded by epitheli
134 ration driven by the hydrostatic pressure in glomerular capillaries, which is considerably higher tha
135                        Podocytes embrace the glomerular capillaries with foot processes, which are in
136 ighly arborized interdigitating cells on the glomerular capillaries with important function for the g
137 trophils underwent prolonged interactions in glomerular capillaries, with the duration of these inter
138                     Platelets accumulated in glomerular capillaries within 4 hours of TGN before evid

 
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