ライフサイエンスコーパス: glomeruli

1 ells were seen in the tubulointerstitium and glomeruli.

2 n and abrogated macrophage infiltration into glomeruli.

3 'alb by endothelial glycocalyx disruption in glomeruli.

4 glomerulonephritis when MPO is deposited in glomeruli.

5 F1 display impaired maturation of peripheral glomeruli.

6 specific RNA sequencing analysis in isolated glomeruli.

7 or (A-P) activity wave typical of the dorsal glomeruli.

8 se type that has not been reported in dorsal glomeruli.

9 tly recruits neutrophils that deposit MPO in glomeruli.

10 d DUSP4 expression in cultured podocytes and glomeruli.

11 tubules and by parietal epithelial cells of glomeruli.

12 nd a higher percentage of globally sclerotic glomeruli.

13 conformation map from freshly isolated human glomeruli.

14 estrations leading to fibrosis in subsets of glomeruli.

15 ed glomerular injury when MPO was planted in glomeruli.

16 of odorant receptors (ORs) and antennal lobe glomeruli.

17 ing long lasting depolarization of olfactory glomeruli.

18 uclear cell infiltrates, and reduced size of glomeruli.

19 pressing endothelial cells (ECs) in affected glomeruli.

20 ) as the fourth most abundant protein in FGN glomeruli.

21 lly, manifests as crescent formation in most glomeruli.

22 ptic input from both temperature and dry-air glomeruli.

23 ept, with 94% of axons terminating in single glomeruli.

24 lized with a podocyte-specific marker in rat glomeruli.

25 egulating the position of T cells within the glomeruli.

26 ce synchronized activity of MCs at different glomeruli.

27 ce upregulate the proteasome system in their glomeruli.

28 nto TCs, but not MCs, that project to nearby glomeruli.

29 are subepithelial deposits were found in the glomeruli.

30 local interneurons within antennal lobe (AL) glomeruli.

31 and in particular to the false activation of glomeruli.

32 ient analysis of podocyte depletion in whole glomeruli.

33 tensively before entering up to six distinct glomeruli.

34 ete cortical modules known as olfactory bulb glomeruli.

35 l before they form odorant receptor-specific glomeruli.

36 ey form spherical neuropil structures called glomeruli.

37 ial activity patterns of olfactory bulb (OB) glomeruli.

38 the introduced capsules in the renal cortex glomeruli.

39 nsequence of the altered architecture of the glomeruli.

40 predicted to cleave ECM proteins in the AMR glomeruli.

41 expression was dysregulated in TrkC-KO mouse glomeruli.

42 modulatory protein linked to the ECM, in AMR glomeruli.

43 frequently bifurcate and connect to multiple glomeruli.

44 the response of MOR18-2 and its neighbouring glomeruli.

45 l epithelial cells (PECs) infiltrating cFSGS glomeruli.

46 deposition of collagen type 3 fibrils in the glomeruli.

47 eurons in spherical neuropil regions, called glomeruli.

48 erulus, and percentage of globally sclerotic glomeruli.

49 uctural plasticity only in respective target glomeruli.

50 n air) nor by the response amplitudes across glomeruli.

51 the projection neurons that innervate the OB glomeruli.

52 e or two of about 50 morphologically defined glomeruli [1-3].

53 group (66.7% vs 49.8%, P = .001), with more glomeruli (23 vs 16, P = .014).

54 promising candidate miRNAs in microdissected glomeruli a confirmation set of 20 human transplant biop

55 m body calyx on a set of secondary olfactory glomeruli, a feature that is not known from olfactory pa

56 metalloproteinase-12 started manifesting in glomeruli affected by early-stage lesions, whereas AT1 r

57 nse polarity mapped uniformly to discrete OB glomeruli, allowing us to analyze how inhibition shapes

58 This raises the question of whether the glomeruli also become functional upon transplantation.

59 h adjacent "hot," "cold," "dry," and "humid" glomeruli-an organization that allows for both unique an

60 29 years old had a mean 990,661 nonsclerotic glomeruli and 16,614 globally sclerotic glomeruli per ki

61 abundantly upregulated in hypertensive human glomeruli and animal kidney samples.

62 gher mtDNA levels compared with cells within glomeruli and collecting duct epithelial cells.

63 ation and activity by sulfenylation in mouse glomeruli and cultured podocytes.

64 ant C57BL/6J and DKD-susceptible DBA/2J (D2) glomeruli and demonstrated a significant downregulation

65 rophy and reabsorption of globally sclerotic glomeruli and hypertrophy of remaining nephrons.

66 RNA-sequencing (RNA-seq) analysis of kidney glomeruli and identified Tug1 as a differentially expres

67 oreover, transmission electron microscopy of glomeruli and immunofluorescent staining of glomerular e

68 n proteinuria presents, fibrosis of both the glomeruli and interstitium rapidly progresses, microthro

69 racterized by predominant C3 deposits in the glomeruli and is commonly the result of acquired or gene

70 amyloidosis with predominant involvement of glomeruli and medullary interstitium.

71 iched with H3K4me1 and H3K9/14ac in diabetic glomeruli and podocytes, which remained elevated despite

72 We also studied glomeruli and primary renal vascular smooth muscle cells

73 syndrome), then the disease rapidly affects glomeruli and progresses towards end stage renal failure

74 used laser-capture microdissection to obtain glomeruli and proximal tubules from 98 human needle kidn

75 the number of T cells positioned inside the glomeruli and reduced inflammation.

76 tion of the OB, reduced numbers of olfactory glomeruli and reduced OB-size without alterations in SVZ

77 posure increases the volume of the activated glomeruli and show that exposure increases M/TC number b

78 these processes controlled key molecules in glomeruli and specifically podocytes, including cytoskel

79 ot uniform, but rather heterogeneous, across glomeruli and stereotyped from animal to animal.

80 ves loss of OSN innervation of antennal lobe glomeruli and subsequent axon retraction in a dose-depen

81 mong macrophages that are recruited into the glomeruli and the damaged rat mesangial cells leads to d

82 These glomeruli and the postsynaptic targets of OSNs, includin

83 DSA) against HLA and non-HLA antigens in the glomeruli and the tubulointerstitium cause AMR while inf

84 eruli, and obtained detailed MRRs of MOR18-2 glomeruli and their neighbours.

85 colocalization of MASP-2 and C3 in both the glomeruli and tubules indicated that the lectin pathway

86 (EGFR) is widely expressed in the kidney in glomeruli and tubules, and persistent and dysregulated E

87 failure (CKD) can exert toxic effects on the glomeruli and tubulo-interstitial region.

88 rformed on laser-captured and microdissected glomeruli and tubulointerstitium identified early ECM re

89 nalysis of laser-captured and microdissected glomeruli and tubulointerstitium was performed on 30 for

90 correlation of HIF-target genes with eGFR in glomeruli and tubulointerstitium.

91 e studies revealed TBC1D8B presence in human glomeruli, and affected individual podocytes displayed a

92 The number of cores, glomeruli, and complications were reviewed in 431 CT-gui

93 ic kidneys, poorly differentiated peripheral glomeruli, and decreased proximal tubular mass in the ou

94 ides access to only approximately 25% of all glomeruli, and little is known about how the lateral bul

95 including cystic and collapsing/degenerating glomeruli, and marked disruptions in podocyte arrangemen

96 eviously unknown odorants activating MOR18-2 glomeruli, and obtained detailed MRRs of MOR18-2 glomeru

97 ed 338 genes altered in diabetes-induced DKD glomeruli, and PLK2 exhibited the most dramatic change.

98 large laminae, medullae formed from optical glomeruli, and robust mushroom bodies.

99 heterogeneous patterns of inhibition across glomeruli, and that the canonical model of global inhibi

100 r cellularity, crescent formation, sclerotic glomeruli, and tubulointerstitial injury were significan

101 performance using just a small subset of the glomeruli ( approximately 15).

102 Glomeruli are highly sophisticated filters and glomerula

103 dition, we estimate that approximately 3,500 glomeruli are present in each main olfactory bulb.

104 Olfactory bulb glomeruli are regions of neuropil that contain input and

105 In the olfactory bulb (OB), glomeruli are the functional units for odor information

106 imaging OSN axon terminals in olfactory bulb glomeruli as well as OSN cell bodies within the olfactor

107 (ORN) axons that target a left-right pair of glomeruli, as well as all the projection neurons (PNs) p

108 red for collagen synthesis, were depleted in glomeruli at early time points.

109 d adequate if the biopsy yielded at least 10 glomeruli at light microscopy, one glomerulus at immunof

110 tal day 1, and have about half the number of glomeruli at postnatal day 21.

111 hin the main olfactory bulb, the size of the glomeruli, at approximately 350 mum in diameter, are on

112 In these glomeruli, at least two of the postsynaptic dendrites or

113 Mer is constitutively expressed in the glomeruli; Axl expression is inducible in glomeruli unde

114 Lateral glomeruli became active in a dorso-ventral (D-V) sequenc

115 In glomeruli, both MLII and MLIII mice exhibited reduced le

116 ate that human pluripotent stem cell-derived glomeruli can develop an appropriate barrier function an

117 ere clearance preserved the functionality of glomeruli, cardio-protective KATP channels and adipocyte

118 ences are apparent across antennal lobe (AL) glomeruli (compact microcircuits corresponding to differ

119 or cathepsins B and C were increased in FSGS glomeruli compared with normal controls, and urinary exc

120 hers with heterogenous strength), or global (glomeruli connect to all others with equal strength).

121 with heterogeneous strength), nonselective (glomeruli connect to most others with heterogenous stren

122 ted that all PNs innervating ventroposterior glomeruli contact a protocerebral neuropil rarely target

123 On average, 79% of glomeruli contained fibrin thrombi.

124 icroscopy revealed that approximately 20% of glomeruli contained structures composed of extracellular

125 rface area and diameter were measured on all glomeruli containing a vascular pole.

126 from DIC-positive kidneys when the extent of glomeruli containing fibrin thrombi is less than 50% and

127 ransplant renal biopsies showing 100% of the glomeruli containing fibrin thrombi.

128 intraglomerular compartment (IGC), with more glomeruli containing RFP(+)CoRL and, within these glomer

129 ts due to their association with progressive glomeruli damage in disease states.

130 ldest age groups, the number of nonsclerotic glomeruli decreased by 48%, whereas cortical volume decr

131 Within the accessory olfactory bulb, the glomeruli did not appear distinct, rather forming a homo

132 wed that the DNAJB9 protein deposited in FGN glomeruli did not have any major sequence or structural

133 Compared with control glomeruli, DN, FSGS, IgAN, and MPGN glomeruli exhibited

134 ors on the periostin promoter is enriched in glomeruli during experimental GN.

135 Time-course microarray analysis of glomeruli during nephrotoxic serum nephritis revealed si

136 system is divided into processing channels (glomeruli), each receiving input from a different type o

137 es with Young's modulus near that of healthy glomeruli elicit a pro-differentiation and maturation re

138 ach mitral cell receives input from multiple glomeruli, enables integration of chemosensory stimuli o

139 Compared with amyloidosis glomeruli, FGN glomeruli exhibited a >6-fold overexpression of DNAJB9 p

140 control glomeruli, DN, FSGS, IgAN, and MPGN glomeruli exhibited differential expression of 18, 12, t

141 ompared with control or FSGS glomeruli, IgAN glomeruli exhibited downregulated expression of hsa-miR-

142 Compared with amyloidosis glomeruli, FGN glomeruli exhibited a >6-fold overexpress

143 crocapsule dose leads to carriers staying in glomeruli for at least 48 h which has consequences of bl

144 ation, pheromone induced activation, correct glomeruli formation in the accessory olfactory bulb (AOB

145 in all mature VSNs only compromises correct glomeruli formation in the posterior AOB.

146 lfactory bulb neurons in the vicinity of the glomeruli formed by axons of Gucy1b2+ sensory neurons.

147 most of the approximately 500 antennal lobe glomeruli found in wild-type ants.

148 In freshly isolated glomeruli from Akita mice, csGRP78 co-localized with the

149 ECM proteins in glomeruli from biopsy specimens of patients with FSGS no

150 gulation of essential mitochondrial genes in glomeruli from diabetic D2 mice, but not in C57BL/6J, wi

151 We isolated glomeruli from formalin-fixed and paraffin-embedded biop

152 vel of KLF15 expression in the podocytes and glomeruli from human biopsy specimens correlated with gl

153 Podocytes in glomeruli from humans with focal segmental glomeruloscle

154 rotective factors for DN using proteomics on glomeruli from individuals with extreme duration of diab

155 llent performance was observed at segmenting glomeruli from non-glomerular structure and subsequently

156 Transcriptome and proteome analysis of glomeruli from patients with FSGS revealed an underrepre

157 Moreover, microdissected glomeruli from patients with small vessel vasculitis (SV

158 Compared with controls, glomeruli from podocyte-specific beta-PIX knockout mice

159 y samples from patients with FSGS, in single glomeruli from proteinuric rats, and in podocytes underg

160 role of Ang II/TRPC6 axis in the control of glomeruli function, which is likely important for the de

161 t, the number of piriform neurons n and bulb glomeruli g scale as n~g(3/2).

162 glomerular cells from magnetic bead-purified glomeruli have identified glomerulus-infiltrating leukoc

163 Specifically, compared with control or FSGS glomeruli, IgAN glomeruli exhibited downregulated expres

164 , altered serum reactivity to DWEYS, reduced glomeruli IgG deposition, preserved kidney histology, an

165 In addition, by analyzing non-glomeruli images, the neural network identified novel hi

166 arning, we modelled local computation within glomeruli in antennal lobes with axons of projection neu

167 lomerulus and 59 and 50% connect to multiple glomeruli in larval and postmetamorphotic animals, respe

168 Here, we counted all individual glomeruli in murine kidneys and sized the capillary tuft

169 rstitium." The network detected 92.7% of all glomeruli in nephrectomy samples, with 10.4% false posit

170 We analyzed the proteomic content of glomeruli in patient biopsy specimens and detected DnaJ

171 de novo expression of connexin 43 in damaged glomeruli in patients with glomerular diseases as well a

172 med quantitative ultrastructural analyses of glomeruli in rat olfactory bulb under conditions in whic

173 nae is tightly correlated with the number of glomeruli in the antennal lobe region innervated by odor

174 We have identified two new target glomeruli in the antennal lobe, in addition to the five

175 at prevents their targeting to inappropriate glomeruli in the antennal lobe.

176 nd large wasps also have a similar number of glomeruli in the antennal lobe.

177 esponding expansion of a specific cluster of glomeruli in the antennal lobe.

178 h the growth of specific, odorant-responsive glomeruli in the antennal lobe.

179 erising molecular receptive ranges (MRRs) of glomeruli in the dorsal olfactory bulb (dOB) innervated

180 of neutrophils were detected outside of the glomeruli in the kidneys of Gsr (-/-) mice but were not

181 markably, we also found that a subset of OSN glomeruli in the lOB was highly sensitive to extranasal

182 precisely organized and sorted out onto 1800 glomeruli in the OB, from which the olfactory informatio

183 ty required for their assembly into distinct glomeruli in the olfactory bulb.

184 scence of OSN axons into approximately 3,600 glomeruli in the olfactory bulb.

185 omy (for a tumor), separately characterizing glomeruli in the superficial (subcapsular), middle, and

186 with arteriosclerosis and ischemic-appearing glomeruli in the superficial region.

187 voked OSN synaptic output into olfactory bub glomeruli in unmanipulated (gonad-intact) adult mice fro

188 patterns of suppression in only a subset of glomeruli in which such suppression could be detected, a

189 The best segmented class was "glomeruli" in both data sets (Dice coefficients, 0.95 an

190 Cav2.2 was localized in glomeruli including podocytes and in distal tubular cell

191 olfactory bulb glomerular size and number of glomeruli indicates that enhanced peripheral processing

192 This arrangement of glomeruli indicates that rather than parcellating the pr

193 ignificantly higher proportion of transplant glomeruli infiltrated with macrophages.

194 l changes, but also ameliorated podocyte and glomeruli injury in diabetic mice, which were associated

195 n 'non-sister' MCs affiliated with different glomeruli (interglomerular synchrony).

196 The total number of glomeruli is a fundamental parameter of kidney function

197 Moreover, the amount of ROBO2 in the glomeruli is also elevated in patients with membranous n

198 d increased expression of wild-type TRPC6 in glomeruli is observed in several human acquired proteinu

199 dor information coding, but inhibition among glomeruli is poorly characterized.

200 eutrophil localization, deposited MPO within glomeruli is recognized by autoreactive T cells that con

201 ng nucleic acids, whose deposition in kidney glomeruli is suspected to promote tissue injury and glom

202 d gamma), OSN axons fail to converge to form glomeruli, likely owing to contact-mediated repulsion be

203 TrkC-KO and TrkC-OE mice exhibited enlarged glomeruli, mesangial proliferation, basement membrane th

204 sclerosis and/or collapse of juxtamedullary glomeruli, microcystic tubular dilation, and tubulointer

205 his calyx region is composed of much smaller glomeruli ("microglomeruli").

206 Lateral circuit processing among activated glomeruli modulates olfactory signal transformation befo

207 ruli containing RFP(+)CoRL and, within these glomeruli, more RFP(+)CoRL.

208 levels are also significantly reduced in the glomeruli of albuminuric BTBR ob/ob mice, indicating its

209 Tmem63c is differentially expressed in glomeruli of allele-specific rat models during onset of

210 al growth factor (VEGF) signaling within the glomeruli of Alport mice is strongly elevated early on i

211 ss spectrometry was demonstrated in isolated glomeruli of Asah1(fl/fl)/Podo(Cre) mice compared with t

212 uction of MCP-1 and IL-6 was observed in the glomeruli of C/EBP-alpha knockout mice and was associate

213 stress and reduced NAPDH oxidase 4 (NOX4) in glomeruli of diabetic eNOS(-/-) mice.

214 d mTORC1 activation in the kidney cortex and glomeruli of diabetic mice and rats, respectively.

215 were also significantly up-regulated in the glomeruli of diabetic patients and mice, suggesting indu

216 Finally, in the renal glomeruli of diabetic rats, the acetylation of S6 kinase

217 lomeruli of Hmox1(-/-) rats and augmented in glomeruli of GEC(HO-1) rats.

218 lomeruli of Hmox1(-/-) rats and augmented in glomeruli of GEC(HO-1) rats.

219 Immunoglobulin eluted from the glomeruli of HCMVpp65(422-439) immunized mice showed cro

220 onstitutively expressed DAF was decreased in glomeruli of Hmox1(-/-) rats and augmented in glomeruli

221 This effect was attenuated in glomeruli of Hmox1(-/-) rats and augmented in glomeruli

222 se SNRK in glomerular endothelial cells, and glomeruli of HN patients and AngII-infused mice show red

223 We compared KMO abundance in the glomeruli of mice and humans under normal and diabetic c

224 the siRNA in the liver, intestine and kidney glomeruli of mice at siRNA doses that are at least tenfo

225 ause its expression levels are higher in the glomeruli of NTS injured mice and passive Heymann membra

226 o observed that CDK2 is downregulated in the glomeruli of obese Zucker rats before the onset of prote

227 n of KIBRA and phosphorylated YAP protein in glomeruli of patients with biopsy-proven focal segmental

228 sulin-sensitive mouse podocytes and in human glomeruli of patients with early and late-stage diabetic

229 Glomeruli of patients with FSGS lack DAF and stain posit

230 ellular traps (NETs) have been documented in glomeruli of patients with glomerulonephritis.

231 t miR-146a expression levels decrease in the glomeruli of patients with type 2 diabetes (T2D), which

232 ways in Exoc5 knockdown podocytes and in the glomeruli of podocyte-specific Exoc5 KO mice.

233 pecifically blocks TRPC5 channel activity in glomeruli of proteinuric rats.

234 as increased VWF deposition in C1q-positive glomeruli of SLE patients compared with control nephropa

235 l degradation of HIPK2, was decreased in the glomeruli of streptozotocin injected diabetic mice.

236 Renal glomeruli of the G3YR mice had significantly reduced amo

237 ly bound to structures within the developing glomeruli of the kidney, which express CS-E.

238 rs that establish excitatory synapses within glomeruli of the olfactory bulb.

239 In glomeruli of wild-type rats, the natural Hmox substrate,

240 From the primary glomeruli, olfactory projection neurons ascend to the br

241 16% and the proportion of globally sclerotic glomeruli on biopsy increased by only 15%.

242 and higher percentage of globally sclerotic glomeruli or IF/TA predicted progressive CKD.

243 in all patients with FGN but not in healthy glomeruli or in 19 types of non-FGN glomerular diseases.

244 circuit, e.g., olfactory circuits, in which glomeruli (or mitral cells) in the olfactory bulb synaps

245 in FVB/N Cd151 (-/-) compared to Cd151 (+/+) glomeruli (p < 0.05).

246 gressively decreased to 520,410 nonsclerotic glomeruli per kidney and increased to 141,714 globally s

247 and increased to 141,714 globally sclerotic glomeruli per kidney in donors 70-75 years old.

248 otic glomeruli and 16,614 globally sclerotic glomeruli per kidney, which progressively decreased to 5

249 Although uninflamed glomeruli rarely contained DCs, injury with nephrotoxic

250 Lateral inhibition between these 2 glomeruli reflects the level of attraction to the orchid

251 al connections that mediate cross talk among glomeruli, releasing GABA and DA onto sensory nerve term

252 rsely, blocking OSN synaptic output elevates glomeruli remodeling.

253 ted a significant reduction in the number of glomeruli responding to carboxylic acids-chemicals assoc

254 Ubiquitylomic analysis of mouse glomeruli revealed that podocin is ubiquitylated at two

255 ally, transcriptome analysis of presclerotic glomeruli revealed that proliferation and extracellular

256 As a result, glomeruli showed increased expression of metabolic enzym

257 tricular injected AFSC that homed within the glomeruli showed strong modulation of the VEGF activity,

258 Transcriptomic analysis of diabetic mouse glomeruli showed that cell adhesion and inflammation are

259 Studies in isolated glomeruli showed that treatment reduced inflammation and

260 arge number (about 460) of primary olfactory glomeruli, suggesting an advanced sense of smell.

261 , we then determined the total volume of the glomeruli (TGV) formed by coalescence of the fluorescent

262 ifferentiated epithelial cells in the kidney glomeruli that act as a key component of the glomerular

263 ch for assessing podocyte depletion in whole glomeruli that combines immunofluorescence, optical clea

264 rant-specific patterns of inhibition between glomeruli that could, theoretically, be tuned by experie

265 ervations in the AL and the SEZ, identifying glomeruli that may respond to human body odours or carbo

266 eq) from primary outgrowth cultures of human glomeruli that were composed mainly of podocytes and mes

267 e promoters in mesangial cells as well as in glomeruli that were purified from type I and type II dia

268 network mediating lateral inhibition between glomeruli, the functional units of early olfactory codin

269 coded by combinatorial patterns of activated glomeruli, the initial signal transformation site of the

270 of both nonsclerotic and globally sclerotic glomeruli; the total number of glomeruli was estimated f

271 be in ORNs or PNs causes overwrapping of the glomeruli their axons or dendrites target.

272 oids that possess renoprotective activity in glomeruli through their interaction with the glucocortic

273 tion images; we annotated the cortex and all glomeruli to calculate glomerular volume, cortex volume

274 s the response intensity range of individual glomeruli to increasing concentration making them more s

275 The predicted connectivity rate from glomeruli to third-order neurons can be tested experimen

276 he glomeruli; Axl expression is inducible in glomeruli under inflammatory conditions.

277 onocytes patrol both uninflamed and inflamed glomeruli using beta2 and alpha4 integrins and CX3CR1.

278 his gap, we studied serial sections of three glomeruli using electron microscopy.

279 ed amyloidosis with extensive involvement of glomeruli, vessels, and medulla.

280 lly sclerotic glomeruli; the total number of glomeruli was estimated from cortical volumexglomerular

281 Deposition of self-antigens in glomeruli was examined by staining with antibodies to ds

282 ffness spanning that of healthy and diseased glomeruli, we demonstrate that kidney podocytes show mar

283 though no significant morphologic changes in glomeruli were observed in these mice under light micros

284 emnants between damaged tubules and atubular glomeruli were observed.

285 pendently associated with fewer nonsclerotic glomeruli were older age, shorter height, family history

286 With this approach, 12 glomeruli were reconstructed at an x-y-z resolution ~ 10

287 ould be detected, and excited and suppressed glomeruli were spatially intermingled.

288 Isolated glomeruli were stimulated ex vivo with VEGFC, which redu

289 heterogeneous patterns of inhibition across glomeruli when driven by realistic sensory input pattern

290 These processes were specific to kidney glomeruli where metabolic signaling occurred through mTO

291 ir axons directly to the olfactory bulb (OB) glomeruli, where their synaptic release is subject to lo

292 ions, reduced rosette number, and dysmorphic glomeruli, whereas beta-catenin stabilization leads to i

293 sies, Nox5 was identified to be expressed in glomeruli, which appeared to be increased in diabetes.

294 pping but distinct from those represented in glomeruli, which is consistent with an extensive interpl

295 asses target dendrites to distinct olfactory glomeruli, while PNs of the same class exhibit indisting

296 In glomeruli with advanced lesions, AT1 receptor expression

297 les, the Gsalpha-deficient mice had enlarged glomeruli with mesangial expansion, injury, and FSGS at

298 ndicate chemotopy, that is, a preference for glomeruli with similar physico-chemical MRR descriptions

299 indings that demonstrated tunotopy, that is, glomeruli with similar tuning curves tend to be located

300 ured as the percentage of globally sclerotic glomeruli) with age, obesity, diabetes, smoking, kidney