ライフサイエンスコーパス: glomerulitis

1 re interstitial inflammation, tubulitis, and glomerulitis.

2 or tubulitis, interstitial inflammation, and glomerulitis.

3 less numerous CD3+ cells was found in TG and glomerulitis.

4 ar to but not higher than cases of g2 and g3 glomerulitis.

5 trophils in PTC, 65% versus 9%; neutrophilic glomerulitis, 55% versus 4%; neutrophilic tubulitis, 55%

6 associated with capillaritis in the biopsy (glomerulitis, 6.1% vs. 32%, P=0.003; peritubular capilla

7 at progressed to rejection had more frequent glomerulitis (7 of 18 versus 3 of 47, P = 0.003) and Ban

8 lls, predominantly macrophages manifested as glomerulitis and capillaritis.Throughout the course of A

9 Glomerulitis and detectable posttransplantation donor-sp

10 ular ABMR activity correlated with increased glomerulitis and donor-specific antibody.

11 circulation inflammation was prevalent, with glomerulitis and peritubular capillaritis found in 60.0%

12 001) and Banff inflammation scores including glomerulitis and peritubular capillaritis were lower on

13 01), and Banff inflammation scores including glomerulitis and peritubular capillaritis were lower on

14 to assess the reproducibility of transplant glomerulitis and to prospectively investigate the pathog

15 nine, whose allograft biopsy confirmed acute glomerulitis and vascular rejection.

16 ) infiltrate, tubulitis, endothelialitis and glomerulitis, and anti-donor CTL reactivity in vitro.

17 transplant glomerulopathy lesions, and lower glomerulitis, but similar levels of peritubular capillar

18 Inter-observer reproducibility of transplant glomerulitis can be improved by using more stringent his

19 inflammation (MI; defined by the addition of glomerulitis (g) and peritubular capillaritis (ptc) scor

20 Glomerulitis (g) and peritubular capillaritis (ptc) scor

21 Renal transplant glomerulitis (G) is associated with acute antibody-media

22 nt in mean scores for acute Banff components glomerulitis (g), C4d, g+ peritubular capillaritis (ptc)

23 ity index was determined as the sum of Banff glomerulitis (g), peritubular capillaritis (ptc), arteri

24 tudied, comparing one group with significant glomerulitis (G, n=28) with those with no glomerulitis (

25 body (DSA), but MVI at AMR thresholds (Banff glomerulitis [g] + peritubular capillaritis [ptc] score

26 compartments, intraglomerular leukocytes in glomerulitis group consisted largely of monocytes.

27 Patients with glomerulitis had high levels of IL-6 and IL-1beta secret

28 ignificance of moderate-to-severe transplant glomerulitis in acute rejection.

29 TxGN was preceded by glomerulitis in more than 90% of cases, with a median ti

30 The glomerulitis, interstitial inflammation, and peritubular

31 Microcirculation inflammation, particularly glomerulitis, irrespective of C4d, is associated with a

32 Acute transplant glomerulitis is a unique lesion in renal allografts, the

33 Transplant glomerulitis is an active form of glomerular injury asso

34 Glomerulitis is strongly associated with increased risk

35 and chronic antibody-mediated rejection with glomerulitis, microthrombosis, microaneurysms, glomerula

36 without glomerulitis (n=21), and transplant glomerulitis (n=18).

37 flamed (borderline changes or above) without glomerulitis (n=21), and transplant glomerulitis (n=18).

38 nt glomerulitis (G, n=28) with those with no glomerulitis (NG, n=35).

39 The presence of glomerulitis or chronic interstitial fibrosis (g and ci

40 al hemorrhage (OR 13.2), and the presence of glomerulitis (OR 3.7) (all P < 0.05).

41 teritis (OR=0.5, 95% CI=0.2-1.2, P=0.11) and glomerulitis (OR=0.9, 95% CI=0.4-2.1, P=0.8) were not.

42 HLA Abs positively correlated with increased glomerulitis (P=0.002), microvascular inflammation (P=0.

43 iopsies and dichotomized 202 MVI >= 2 (Banff glomerulitis + peritubular capillaritis >= 2) samples by

44 r de novo DSA had a higher incidence of MVI (glomerulitis + peritubular capillaritis >= 2) than patie

45 ity, as well as microvascular injury scores (glomerulitis + peritubular capillaritis), were less in t

46 rrent ABMR criteria, including capillaritis, glomerulitis, peritubular capillary C4d deposition, and

47 with indices of microvascular inflammation (glomerulitis, peritubular capillary infiltrates; P value

48 with indices of microvascular inflammation (glomerulitis, peritubular capillary infiltrates; p-value

49 lomerular filtration rate at time of biopsy, glomerulitis, rituximab, diabetes, v score, allograft gl

50 y-mediated changes with significantly higher glomerulitis scores and numerically higher C4d staining

51 Biopsies with glomerulitis showed ultrastructural signs of glomerular

52 igher inter-observer agreement for detecting glomerulitis than that of the current Banff schema.

53 ction (OR=4.9, 95% CI=1.1-20.8, P=0.03), and glomerulitis was associated with the development of tran

54 Glomerulitis was associated with worse graft survival (8

55 Glomerulitis was independently associated with the risk