ライフサイエンスコーパス: gluconate

1 lectivity sequence of Cl(-) > I(-) > Br(-) > gluconate(-).

2 n is severely limited by large anions (i.e., gluconate).

3 complex combinations thereof (l-arabinose, d-gluconate).

4 ility sequence: SCN- > I- > Br- > Cl- > F- > gluconate-.

5 ing of babies >=1.5 kg with 2% chlorhexidine gluconate.

6 unable to grow in minimal medium containing gluconate.

7 al Na(+) with NMDG and by internal F(-) with gluconate.

8 ronate but grows normally on glucuronate and gluconate.

9 a hypertonic solution in which the anion was gluconate.

10 aspartate increased quantal size similar to gluconate.

11 ng cells was SCN(-) > I(-) > Br(-) > Cl(-) > gluconate.

12 paracellular pathway is limited by the anion gluconate.

13 e activity, but there was less activity with gluconate.

14 tracers of Zn in the form of sulfate and/or gluconate.

15 that were still able to oxidize glucose into gluconate.

16 SO42- were similar and about half of that in gluconate.

17 with a measured apparent Km of 6 microM for gluconate.

18 arge intestine is defined by the presence of gluconate.

19 calcium lactate pentahydrate instead of zinc gluconate.

20 tients (26 cluster periods) to chlorhexidine gluconate.

21 y intraperitoneal injection of chlorhexidine gluconate.

22 loz1Delta cells accumulate higher levels of gluconate.

23 skin cleansing protocol using chlorhexidine gluconate.

24 hydrolysed by TM0413 (IolN) to form 5-keto-l-gluconate.

25 ntly 6 hours after administration of calcium gluconate.

26 - (1.7) > Br- (1.2) > Cl- (1.0) > F- (0.7) > gluconate (0.18).

27 y assigned to no iron (control) or to ferric gluconate 125 mg intravenously with eight consecutive he

28 Administration of ferric gluconate (125 mg for eight treatments) is superior to n

29 e randomly assigned to receive either ferric gluconate 187.5 mg intravenously (IV) every 3 weeks, ora

30 eks), a calcium-enriched diet alone (calcium gluconate 2 g/L in drinking water), both vitamin D suppl

31 , one alpha-hydroxyglutarate racemase, two D-gluconate 2-epimerases, and one short-chain aliphatic al

32 red the safety and efficacy of chlorhexidine gluconate 2.5 mg chip (CHX chips) as an adjunctive treat

33 nt stereo-specific reduction of 2,5-diketo-d-gluconate (2,5-DKG) to 2-keto-l-gulonate, a precursor in

34 zes stereospecific reduction of 2,5-diketo-D-gluconate (2,5-DKG) to 2-keto-L-gulonate.

35 dextran was 3.6 (95% CI, 2.4-5.4); for iron gluconate, 2.0 (95% CI 1.2, 3.5); and for ferumoxytol, 2

36 /mL, respectively; and with 2% chlorhexidine gluconate, 2.1 (2.0-2.3), 1.8 (1.5-2.0), and 1.7 (1.5-1.

37 3, chloride 98, phosphate 1, acetate 28, and gluconate 23).

38 One tablet of ferrous gluconate (37.5 mg of elemental iron) daily or no iron f

39 nate, modestly induced by very low levels of gluconate (4 microM), and partially catabolite repressed

40 Volunteers used 118 mL of chlorhexidine gluconate, 4%, for each shower.

41 hat includes 118 mL of aqueous chlorhexidine gluconate, 4%, per shower; a minimum of 2 sequential sho

42 g the preadmission shower with chlorhexidine gluconate, 4%, resulting in maximal, persistent skin ant

43 olunteers were randomized to 2 chlorhexidine gluconate, 4%, showering groups (2 vs 3 showers), contai

44 Preadmission showers using chlorhexidine gluconate, 4%.

45 ing a precise dose (volume) of chlorhexidine gluconate, 4%; duration (number of showers); and timing

46 The zinc-supplemented group received zinc gluconate (45 mg elemental Zn/d) orally for 12 mo.

47 nate ((4R)-KDGal) over (4S)-2-keto-3-deoxy-D-gluconate ((4S)-KDGlu), with AbHpaI*Zn(2+) displaying th

48 The L-idonate 5-dehydrogenase and 5-keto-D-gluconate 5-reductase reactions were characterized both

49 encodes L-idonate 5-dehydrogenase, 5-keto-D-gluconate 5-reductase, an L-idonate transporter, and an

50 ive pentose phosphate pathway, gnd (encoding gluconate 6-phosphate [6-P] dehydrogenase) or zwf (encod

51 e complexed with the competitive inhibitor D-gluconate 6-phosphate by X-ray crystallography at 2.5 A

52 The location of the bound D-gluconate 6-phosphate inhibitor leads to the identificat

53 ctive site ligands, 5-phosphoarabinonate and gluconate 6-phosphate.

54 zation was the redistribution of 6-phospho-D-gluconate (6-PG) between the Entner-Doudoroff pathway an

55 nd showed that the Crc-regulated edd mutant (gluconate-6-phosphate dehydratase) had similar gluconate

56 ternal solutions containing CH(3)SO(3)(-) or gluconate, a replacement of the Ca(2+) with Mg(2+) reduc

57 Balanced solutions (gluconate/acetate-buffered solution and lactate-buffered

58 ndomized with 178, 171, and 167 allocated to gluconate/acetate-buffered solution, lactate-buffered so

59 Enrolled children were 1:1:1 allocated to gluconate/acetate-buffered solution, lactate-buffered so

60 Patients rinsed with 0.12% chlorhexidine gluconate after debridement, and twice daily, for 2 week

61 upplementations with (t68)ZnSO(4) or (t70)Zn-gluconate alone and in combination (1:1 molar ratio) wer

62 Replacement of Cl- with gluconate also increased the potency of ATP as an induce

63 al mouthrinses containing 0.2% chlorhexidine gluconate, an herbal mouthwash, and water in reducing th

64 oach involving a direct pyrolysis of ferrous gluconate and a following removal of free iron, but prov

65 ng of GntR to the operators is eliminated by gluconate and also by 6-phosphogluconate at a 10-fold-hi

66 , calcium chloride, calcium lactate, calcium gluconate and calcium lactobionate, on the physico-chemi

67 E. faecalis strain OG1RF that phosphorylates gluconate and contains the genes OG1RF_12399 to OG1RF_12

68 eads to the induction of genes involved with gluconate and formate metabolism and represses genes req

69 e logarithmic phase, and for cometabolism of gluconate and glucose.

70 P1 controls eda induction on gluconate and is regulated by GntR.

71 ive, while expression of gntKU is induced by gluconate and is subject to fourfold glucose catabolite

72 ose, and ribose, whereas E. coli MG1655 used gluconate and N-acetylneuraminic acid.

73 sent in the cocoa infusions as Ni(2+) and Ni-gluconate and Ni-citrate complexes.

74 The effects of sodium gluconate and other sodium salts on the hydration behavi

75 Escherichia coli is specifically induced by gluconate and repressed via catabolite repression.

76 029 to restore normal growth on mannitol and gluconate and restored Pgl activity.

77 ivity of catheters coated with chlorhexidine gluconate and silver sulfadiazine (P < .01).

78 ocycline and rifampin and with chlorhexidine gluconate and silver sulfadiazine were evaluated.

79 days for catheters coated with chlorhexidine gluconate and silver sulfadiazine.

80 r the substrate analogue 6-phospho-2-deoxy-D-gluconate and suggest targets for anti-parasite drug des

81 ranscription specifically in the presence of gluconate and that E. faecalis strains lacking, or harbo

82 , these results implicate the utilization of gluconate and the Entner-Doudoroff pathway as important

83 f antifungal agents (cinnamon bark oil, zinc gluconate and trans-ferulic acid) in oil-in-water emulsi

84 differentially expressed genes for growth on gluconate and under salt and magnesium stress.

85 GntP is not induced by gluconate, and despite being located adjacent to genes i

86 ran IV iron products combined (iron sucrose, gluconate, and ferumoxytol) (95% CI, 20.0-29.5 per 100,0

87 impermeants (sorbitol, raffinose, trehalose, gluconate, and polyethylene glycol-20k (PEG-20k).

88 Accumulation of gluconate- and Ca2+ in the system over time correlated w

89 athing all patients daily with chlorhexidine gluconate; and healthcare-worker education and adherence

90 Gluconate appeared to be a major carbon source used by E

91 servatively with topical 0.12% chlorhexidine gluconate application.

92 skin surface concentrations of chlorhexidine gluconate applied during preoperative showering.

93 > SO4(2-) approximately HCO3- approximately gluconate- approximately aspartate- approximately cyclam

94 e in the ED pathway grows poorly not only on gluconate as a sole carbon source but on a number of oth

95 n defined media with d-gluconate or 2-keto-d-gluconate as a sole carbon source, revealing that glucon

96 suggest that the ability of C. jejuni to use gluconate as an electron donor via GADH activity is an i

97 awake) containing 13.3 mg of zinc from zinc gluconate as long as they had cold symptoms.

98 ource and at 50% of the wild-type rate using gluconate as the carbon source.

99 a selectivity sequence of Br- >= I- > Cl- > gluconate = aspartate.

100 ] and French fries (40 mEq K) with potassium gluconate at the same doses when added to a basal diet t

101 Using a Cs(+)-gluconate-based internal solution, the leptin-activated

102 ntion components such as daily chlorhexidine gluconate bathing of all patients and hand-hygiene educa

103 and personnel cohorting; daily chlorhexidine gluconate baths; dedicating equipment to be used solely

104 enase, catalyzes the oxidation of glucose to gluconate, but has been shown to have activity with a br

105 CaC), tricalcium phosphate (CaP) and calcium gluconate (CaG).

106 Soluble calcium salts, such as calcium gluconate, calcium acetate and CaCl2, had greater effect

107 With an HSV-2 isolate, 50 and 15 mM zinc gluconate caused 30% inactivation and 5 and 1 mM caused

108 ot CF, epithelia, replacing mucosal Cl- with gluconate caused intracellular pH (pHi) to increase, and

109 intraperitoneal injections of chlorhexidine gluconate (CG) in mice with type I pro-collagen promoter

110 oneal MCs to myofibroblasts in chlorhexidine gluconate (CG)-induced fibrosis compared with that of ph

111 Chlorhexidine gluconate (CHG) and mupirocin are widely used to decolon

112 d tested for susceptibility to chlorhexidine gluconate (CHG) by microtiter dilution; mupirocin suscep

113 ssess whether daily bathing in chlorhexidine gluconate (CHG) compared with standard bathing practices

114 5 days and to bathe daily with chlorhexidine-gluconate (CHG) for up to 5 days before their operations

115 Chlorhexidine gluconate (CHG) is an antiseptic that is widely used in

116 pecies oral biofilms following chlorhexidine gluconate (CHX) and CHX with surface modifiers (CHX-Plus

117 tigated following treatment by chlorhexidine gluconate (CHX), iodine-potassium iodide (IPI) and Sodiu

118 The mean (SD) composite chlorhexidine gluconate concentrations were significantly higher (P <

119 ed with discontinuation of the chlorhexidine gluconate-containing dressings, local wound care, and al

120 tage-clamp recordings with KCl- or potassium gluconate-containing electrodes, bath-applied NA increas

121 ZHER2:V2 was labeled with (188)Re using a gluconate-containing kit.

122 Infusion of sodium gluconate-containing solution but not gluconate-free Pla

123 nt with cardiac glycosides in the hypertonic gluconate-containing solutions hitherto reported to emph

124 ume and in vitro concentrations of GM within gluconate-containing solutions of infused Plasma-Lyte.

125 hibian skeletal muscle fibres studied in the gluconate-containing solutions previously reported to em

126 bian muscle fibres studied in the hypertonic gluconate-containing solutions that were hitherto report

127 tant for colonization, including L-fucose, D-gluconate, D-glucuronate, N-acetyl-D-glucosamine, D-mann

128 mutants, and the activity of the Fe-S enzyme gluconate dehydratase is diminished in the suf mutant du

129 the presence of Mg2+ and the 2-keto-3-keto-d-gluconate dehydration product; the structure of the cata

130 proteins, Cj0414 and Cj0415, orthologous to gluconate dehydrogenase (GADH) from Pectobacterium cypri

131 ype but not cj0415 mutant bacteria exhibited gluconate-dependent respiration.

132 f deuterium 3 relative to deuterium 2 in the gluconate derivative as quantitated by (2)H NMR was show

133 Cl(-) approximately Br(-) > I(-) > acetate > gluconate) different from that of control oocytes.

134 that replacement of extracellular Cl(-) with gluconate(-) diminishes the inward tail current (Cl(-) e

135 th either soap and water or 2% chlorhexidine gluconate eliminated 1.5 to 2.0 log10 CFUs/mL of B atrop

136 Chloride replacement with sulfate or gluconate enhanced the efflux of aspartate, glutamate, G

137 90% of the consumed sugar was converted into gluconate, entering central carbon metabolism as 6-phosp

138 ctures of lactate racemase holoprotein and D-gluconate epimerase apoprotein, to identify key residues

139 hypothesize that this PTS permits growth in gluconate, facilitates E. faecalis intestinal colonizati

140 y KCl-filled, as compared with KCH3SO3- or K-gluconate-filled, electrodes.

141 anion selectivity sequence, I- > Br- > Cl- > gluconate, followed Eisenman's sequence I.

142 d >98% by treatment in vitro with 50 mM zinc gluconate for 2 h and nine were inactivated >97% by trea

143 was given an oral dose of 45 mg zinc/d as a gluconate for 6 mo.

144 sed the safety and efficacy of chlorhexidine gluconate for cutaneous antisepsis and silver alginate-i

145 mportant for growth on low concentrations of gluconate, for entry into the logarithmic phase, and for

146 e inorganic (ZnSo(4)) and even organic (Zinc gluconate) forms tested which has 15.6% and 21.7% respec

147 sodium gluconate-containing solution but not gluconate-free Plasma-Lyte resulted in positive serum GM

148 Gluconate (Gluc(-)) is a structural and functional repre

149 w that Eda synthesis is induced by growth on gluconate, glucuronate, or methyl-beta-D-glucuronide; ph

150 In the presence of the impermeant anion gluconate, glutamate pulses activated smaller currents p

151 zinc may be an effective treatment, and zinc gluconate glycine (ZGG) lozenges have been shown to redu

152 nal mutation resulted in the partial loss of gluconate (gnt) and xylose (xyl) operon catabolite repre

153 more serious adverse events than the ferric gluconate group (incidence rate ratio = 1.73, P = 0.041)

154 e end of observation, patients in the ferric gluconate group required significantly less epoetin than

155 erritin levels remained higher in the ferric gluconate group than in the control group (P = 0.062, P

156 esponding metabolic lesions on colonization: gluconate > N-acetylglucosamine > N-acetylneuraminic aci

157 was calcium chloride>calcium lactate>calcium gluconate>calcium lactobionate.

158 tion of Cl- by the larger (impermeant) anion gluconate had no effect on the reversal potential of SV

159 ng bath Na+ with NMDG+ or perfusate Cl- with gluconate- had no effect.

160 gly alkaline solutions containing Ca(2+) and gluconate have been studied.

161 patients each to receive 0.2% chlorhexidine gluconate, herbal mouthwash, and water, respectively, as

162 cts of three test agents, 0.2% chlorhexidine gluconate, honey mouthwash, and saline, against six oral

163 sible reduction of 5-ketogluconate to form D-gluconate; IdnK catalyzes an ATP-dependent phosphorylati

164 extension designed to investigate how ferric gluconate impacted epoetin dosage after DRIVE.

165 Chlorhexidine gluconate-impregnated dressings have become widely adopt

166 ange of this adverse effect of chlorhexidine gluconate-impregnated dressings in critically ill patien

167 der-recognized complication of chlorhexidine gluconate-impregnated dressings.

168 oup), a standard catheter plus chlorhexidine-gluconate-impregnated sponge (chlorhexidine-gluconate-im

169 ence was observed between the chlorhexidine- gluconate-impregnated sponge group and the standard grou

170 -gluconate-impregnated sponge (chlorhexidine-gluconate-impregnated sponge group), or an Oligon cathet

171 the standard-group, 150 in the chlorhexidine-gluconate-impregnated sponge group, and 159 in the Oligo

172 ard catheters, 21 (14%) in the chlorhexidine-gluconate-impregnated sponge group, and 25 (15.7%) in th

173 ard catheters, six (4%) in the chlorhexidine-gluconate-impregnated sponge group, and seven (4.4%) in

174 tant contact dermatitis due to chlorhexidine gluconate-impregnated transparent dressings.

175 nfections than antisepsis with chlorhexidine gluconate in alcohol.

176 e metabolism in general, and E. faecalis PTS-gluconate in particular, during inflammation may identif

177 evaluate the efficacy of intravenous ferric gluconate in such patients.

178 external Ba2+ or by replacement of potassium gluconate in the electrode solution with caesium acetate

179 Zinc gluconate in the form and dosage studied significantly r

180 RpoN (sigma(54)), accumulated high levels of gluconate in the medium.

181 tropic anion thiocyanate was substituted for gluconate in the whole-cell recording pipette, consisten

182 supply of a readily metabolized sugar, i.e., gluconate, in the animal's drinking water, the competiti

183 tes were also recruited to the chlorhexidine gluconate-induced fibrotic area upon their transplantati

184 In the chlorhexidine gluconate-induced peritoneal fibrosis model, AAM s worse

185 ences in the regulatory regions of all known gluconate-inducible genes, and these seven putative gnt

186 t appears on a Northern blot to be a single, gluconate-inducible, 1.42-kb gntT transcript.

187 -100 mV due to the lowered outward current (gluconate(-) influx) at membrane potential of 100 mV.

188 can be inhibited by zinc, we found that zinc gluconate inhibited potassium efflux from RBC exposed to

189 erms of solute (saccharide)-cosolute (sodium gluconate) interactions.

190 Then, 10 ZD rats were treated with zinc gluconate intragastrically and switched to ZS diet; the

191 cer and a repressor of gntT expression since gluconate is a catabolite-repressing sugar.

192 Although chlorhexidine gluconate is a known cause of contact dermatitis, the ph

193 y for catabolism of L-idonic acid in which D-gluconate is an intermediate.

194 Thus, gluconate is both an inducer and a repressor of gntT exp

195 ultrasonic unit, and that 0.2% chlorhexidine gluconate is more effective than herbal mouthwash.

196 otassium methylsulphate (KMeth) or potassium gluconate (KGluc).

197 ranscript (kdgK1) levels of 2-keto-3-deoxy-D-gluconate kinase (KDGK), a key enzyme of haloarchaeal gl

198 In this shunt glucose dehydrogenase and gluconate kinase catalyze the two-step conversion of glu

199 es, it is 45% identical to a second putative gluconate kinase from E. coli,gntV.

200 gntU, which code for a regulatory protein, a gluconate kinase, and a gluconate transporter, respectiv

201 y: the idnK gene, encoding a thermosensitive gluconate kinase, is monocistronic and transcribed diver

202 ogenase that acts in a pathway with the Idn1 gluconate kinase.

203 sider four different carbon sources glucose, gluconate, lactate, and glycerol.

204 uconate-6-phosphate dehydratase) had similar gluconate levels as the rpoN mutant.

205 We show that the altered gluconate levels in loz1Delta cells result from increase

206 f recombinant Gcd1 in vitro and by measuring gluconate levels in strains lacking enzymes of the gluco

207 .009 and 0.011 vs. 0.006 mM, P = 0.036), and gluconate levels were also significantly different betwe

208 es accumulated and secreted large amounts of gluconate, likely derived from labile 6-phosphogluconola

209 patient was treated with intravenous calcium gluconate, magnesium and oral vitamin D3.

210 In conclusion, ferric gluconate maintains hemoglobin and allows lower epoetin

211 Since the edd gene is involved only in gluconate metabolism via the Entner-Doudoroff pathway, t

212 Three genes involved in gluconate metabolism, gntR, gntK, and gntU, which code f

213 (EGTA) and the potassium salts of aspartate, gluconate, methylsulfate and monobasic phosphate increas

214 that gntT is maximally induced by 500 microM gluconate, modestly induced by very low levels of glucon

215 l sugar unit consisting of a 2-amino-2-deoxy-gluconate moiety in place of glucosamine.

216 t in B and C, but an unusual 2-amino-2-deoxy-gluconate moiety is found in D-1 and E.

217 Sodium gluconate (NaGlu) and NaCl thresholds did not differ, im

218 nalysis of GntU indicates an apparent Km for gluconate of 212 microM, indicating that this is a low-a

219 ther the possible beneficial effects of zinc gluconate on cold symptoms outweigh the possible adverse

220 ospitals must maintain intravenous quinidine gluconate on formulary because it is the only drug avail

221 Replacement of Cl(-) by gluconate or 2-(N-morpholino)ethanesulfonic acid decreas

222 Neither strain grew in defined media with d-gluconate or 2-keto-d-gluconate as a sole carbon source,

223 x mutants were found to be unable to oxidize gluconate or 2-ketogluconate, resulting in an inability

224 'impermeant' anion, such as SO42-, CH3SO3-, gluconate or glutamate, greatly reduced the calcium-depe

225 The incorporation of zinc gluconate or trans-ferulic acid, independently of the co

226 With an HSV-1 isolate, 50 mM zinc gluconate or zinc lactate caused 100% inactivation, 15 m

227 nge of 6.1 to 7.6 since inactivation by zinc gluconate or zinc lactate in that pH range was 99.7 to 1

228 3 ml of sterile saline, 0.12% chlorhexidine gluconate, or 0.1% phosphate-buffered chlorine dioxide m

229 ashing with soap and water, 2% chlorhexidine gluconate, or chlorine-containing towels reduced the amo

230 se oxidoreductase, an enzyme in the sorbitol-gluconate pathway.

231 replaced by gluconate (permeability ratio P(gluconate)/PCl = 0.17).

232 urrent when the external Cl- was replaced by gluconate (permeability ratio P(gluconate)/PCl = 0.17).

233 In contrast, changes in selectivity made gluconate permeant in L46P EAAT2, and nonstationary nois

234 gntP encodes a high-affinity gluconate permease, suggesting that the distinct niche i

235 ermeable to chloride (PCl/PNa = 0.5) but not gluconate (Pgluc/PNa = 0.01) ions.

236 cZ (but not crc) mutant also shared the high-gluconate phenotype.

237 were randomly assigned to Ca/Mg (1g calcium gluconate plus 1g magnesium sulfate pre- and post-oxalip

238 intaining normothermia, use of chlorhexidine gluconate plus alcohol-based skin preparation agents, de

239 aining 61% ethyl alcohol, a 2% chlorhexidine gluconate preparation, and an antibacterial microfiber t

240 ages, substitution of extracellular Cl- with gluconate produced a 10-fold increase in the rate and ex

241 ol intermediate to yield the 2-keto-3-keto-d-gluconate product with the observed retention of configu

242 phic DNA (RAPD) type-specific differences in gluconate production, which were associated significantl

243 At low salinity, by-products (mainly gluconate, pyruvate, and acetate) accumulate extracellul

244 acid dehydrogenase) and b0207 (2,5-diketo-D-gluconate reductase B), is assigned to 15 of those react

245 The chlorhexidine gluconate rinse had the lowest MICs compared with honey

246 ll courses of treatment with sodium antimony gluconate (SAG).

247 on-gamma (IFN-gamma; n = 9), sodium antimony gluconate (SAG; n = 8), or amphotericin B lipid complex

248 ne-2,2'-disulfonic acid (DIDS)-sensitive and gluconate-sensitive Cl(-) channels.

249 ate levels in strains lacking enzymes of the gluconate shunt we demonstrate that Gcd1 encodes a novel

250 that can facilitate glucose breakdown is the gluconate shunt.

251 The safety and efficacy of chlorhexidine gluconate, silver alginate, and antibiotic-coated cathet

252 effect by measuring responses to NaCl and Na-gluconate (small and large anion sodium salts, respectiv

253 hird larger than those in a CH(3)SO(3)(-) or gluconate solution, whereas the values in the CH(3)SO(3)

254 whereas the values in the CH(3)SO(3)(-) and gluconate solutions had no statistically significant dif

255 0.50, 1.00 and 1.50)molkg(-1) aqueous sodium gluconate solutions over a temperature range of (288.15-

256 Administrations of IV iron dextran, gluconate, sucrose, or ferumoxytol as reported in outpat

257 , 40, and 60 mEq K/d consumed as a potassium gluconate supplement or as unfried potato or 40 mEq K fr

258 m is as high from potatoes as from potassium gluconate supplements.

259 microg/cm2) concentrations of chlorhexidine gluconate that are sufficient to inhibit or kill gram-po

260 activity, converting d-gluconate to 2-keto-d-gluconate, that was higher at 42 degrees C than at 37 de

261 eletion strains are grown in the presence of gluconate, there is a twofold decrease in gntT expressio

262 176 demonstrated GADH activity, converting d-gluconate to 2-keto-d-gluconate, that was higher at 42 d

263 e, which is followed by further oxidation of gluconate to as yet unknown chromogenic compounds.

264 urth step involves epimerization of 5-keto-l-gluconate to d-tagaturonate by TM0416 (IolO).

265 In patients with CAA, addition of IV ferric gluconate to darbepoetin failed to provide additional be

266 alyzes an ATP-dependent phosphorylation of D-gluconate to form 6-phosphogluconate, which is metaboliz

267 perior solubilizing potential of copper (II) gluconate to form a complex with gemcitabine at copper:g

268 By oxidizing glucose to gluconate to improve both ionization yield and fragmenta

269 onstrated the efficacy of intravenous ferric gluconate to improve hemoglobin levels in anemic hemodia

270 ented mutant E. coli strains with defects in gluconate transport and directed the formation of a high

271 sence of two systems in Escherichia coli for gluconate transport and phosphorylation is puzzling.

272 rm that gntK and gntU, together with another gluconate transport gene, gntT, constitute the GntI syst

273 nd directed the formation of a high-affinity gluconate transporter with a measured apparent Km of 6 m

274 egulatory protein, a gluconate kinase, and a gluconate transporter, respectively, were cloned from Es

275 Escherichia coli has four gluconate transporters, GntP, GntU, GntT, and IdnT, whic

276 predicted amino acid sequence similarity to gluconate transporters.

277 Chlorhexidine gluconate treatment induced disappearance of CD11b(High)

278 u kinetic studies reveal that the rates of 6-gluconate turnover are indistinguishable in samples from

279 She has been on supplemental calcium gluconate twice daily.

280 ) was replaced by either methanesulfonate or gluconate two nonpermeable anions.

281 e oxidase that generates the 2-amino-2-deoxy-gluconate unit from a glucosamine-containing precursor i

282 t gene, gntT, constitute the GntI system for gluconate utilization, under control of the gntR gene pr

283 smic (TRAP) transporter that is required for gluconate utilization.

284 smid pSymA has previously been implicated in gluconate utilization.

285 x-coupled interconversion of L-idonate and D-gluconate via the intermediate 5-ketogluconate.

286 We demonstrated that sodium gluconate was the factor causing false-positive galactom

287 ted with hypertonic solution in which sodium gluconate was the major constituent, which substantially

288 nate as a sole carbon source, revealing that gluconate was used as an electron donor rather than as a

289 Chlorhexidine gluconate was used for FMD.

290 -glucamine (NMDG) and of Cl- with sulfate or gluconate was used to evaluate the contribution that the

291 tion of 1% sodium alginate plus 0.3% calcium gluconate, was administered by selective injection throu

292 decontamination protocol using chlorhexidine gluconate washcloths and intranasal antiseptic ointment

293 ing substrates such as glycerol, glucose, or gluconate were abundant.

294 Skin surface concentrations of chlorhexidine gluconate were analyzed using colorimetric assay at 5 se

295 e to utilize glucuronate, galacturonate, and gluconate) were constructed by insertional mutagenesis.

296 D-gluconate which is primarily catabolized via the Entner-

297 lasma glucose was enzymatically converted to gluconate, which displays fully resolved deuterium 2 and

298 is initiated by the oxidation of glucose to gluconate, which is followed by further oxidation of glu

299 s-enediol(ate) intermediate than 6-phospho-D-gluconate, which was used in a previously reported cryst

300 n) treatments of selected isolates with zinc gluconate, zinc lactate, zinc acetate, or zinc sulfate y