ライフサイエンスコーパス: glucosamine

1 lying galactose yielding a terminal N-acetyl glucosamine.

2 a functional competition between glucose and glucosamine.

3 on product, UDP-(3-O-(R-3-hydroxymyristoyl))-glucosamine.

4 the polysaccharide poly beta-1,6-N-acetyl-d-glucosamine.

5 sphate, the intracellular form of N-acetyl-D-glucosamine.

6 UDP-ManNAc3NAcA starting from UDP-N-acetyl-d-glucosamine.

7 was assessed in a blocking study with folate glucosamine.

8 ition of iduronic acid and the N position of glucosamine.

9 ) microparticles, oligosaccharide chitin, or glucosamine.

10 state and liver cells, suggesting release of glucosamine.

11 aminoquinoline and naphthalimide moieties by glucosamine.

12 to the inhibition of Th1 differentiation by glucosamine.

13 nce the exchange rate of the nearby N-acetyl glucosamines.

14 human articular cartilage were treated with glucosamine (0.1- 10 mM).

15 essential for bacterial growth, converting D-glucosamine 1-phosphate into UDP-GlcNAc via acetylation

16 leophilicity of the attacking amino group of glucosamine 1-phosphate.

17 lated murine working hearts with [U-(13)C(6)]glucosamine (1, 10, 50, or 100 mum), which bypasses the

18 N-acetyl-glucosamine-1-phosphate uridyltransferase (GlmU), a bifu

19 zyme catalyzes hydrolysis of N-acetyl-beta-d-glucosamine-(1-->4)-1,6-anhydro-N-acetyl-beta-d-muramyl-

20 two activator muropeptides, N-acetyl-beta-d-glucosamine-(1-->4)-1,6-anhydro-N-acetyl-beta-d-muramyl-

21 trations were associated with regular use of glucosamine (17%, 95% confidence interval (CI): 7, 26),

22 thyl-(2-fluoroethyl)-1H-[1,2,3]triazole-4-yl)glucosamine ((18)F-NFTG) annotates glycogenesis in cance

23 ine-phosphatidylinositol (glucosamine-PI) or glucosamine 2-O-methyl inositol octadecyl phosphate, red

24 First, the sulfate analogue, d-glucosamine 2-sulfate, acted as a non-transported agonis

25 eta-d-muramyl-peptide (1) to N-acetyl-beta-d-glucosamine (2) and 1,6-anhydro-N-acetyl-beta-d-muramyl-

26 g that CsaA is the functional UDP-N-acetyl-D-glucosamine-2-epimerase and CsaB the functional poly-Man

27 -C are thought to encode the UDP-N-acetyl-D-glucosamine-2-epimerase, poly-ManNAc-1-phosphate-transfe

28 s directly regulated by heparan sulfate (HS) glucosamine 3-O-sulfotransferase 3B1 (HS3ST3B1).

29 al arylsulfatase G (ARSG) is the long-sought glucosamine-3-O-sulfatase required to complete the degra

30 th the peracetylated 4-fluorinated analog of glucosamine (4-F-GlcNAc) elicits anti-skin inflammatory

31 oma-bearing mice with peracetylated 4-fluoro-glucosamine (4-F-GlcNAc), a metabolic inhibitor of N-ace

32 UGlcNAcDH encodes a UDP-N-acetyl-glucosamine 6-dehydrogenase, converting UDP-N-acetylgluc

33 er cell lines results in 30-50% reduction in glucosamine 6-O-sulfate levels in HS, impairing HB-EGF-d

34 lays high levels of HS sequences that harbor glucosamine 6-O-sulfates when compared with normal ovari

35 e that heparin-induced leukocytosis requires glucosamine 6-O-sulfation and is caused by blockade of L

36 e provide genetic and chemical evidence that glucosamine 6-phosphate N-acetyltransferase (Gna1), a ke

37 e wild-type enzyme, alone or in complex with glucosamine 6-phosphate, are also consistent with a hexa

38 the hexameric form in the presence of cyclic glucosamine 6-phosphate, together with the decrease of t

39 in the ubiquitous metabolites glycolate and glucosamine 6-phosphate.

40 g glucose 6-phosphate, MondoA can also sense glucosamine 6-phosphate.

41 ice markedly overexpress the heparan sulfate glucosamine-6-O-endosulfatase-2 (SULF2), an enzyme that

42 disrupt HSPG structure: the heparan sulfate glucosamine-6-O-endosulfatase-2 (Sulf2).

43 By sensing the levels of the intermediate glucosamine-6-phosphate (GlcN6P) and self-adjusting the

44 The d-glucosamine-6-phosphate (GlcN6P) cofactor has been propo

45 Binding of glucosamine-6-phosphate (GlcN6P) uncovers the latent sel

46 th the assistance of the metabolite cofactor glucosamine-6-phosphate (GlcN6P), whose amino group is p

47 Two analogues of glucosamine-6-phosphate (GlcN6P, 1) and five of glucosam

48 tes, including phenethylamine, agmatine, and glucosamine-6-phosphate (P < 0.05), while F affected the

49 re thus far that utilizes a small molecule - glucosamine-6-phosphate - to participate directly in rea

50 ccur by an acid-base mechanism involving the glucosamine-6-phosphate cofactor and G40 residue.

51 ), phosphofructokinase (PfkB, but not PfkA), glucosamine-6-phosphate deaminase (NagB), and adenylate

52 The amino-terminal cysteine of glucosamine-6-phosphate synthase (GlmS) acts as a nucleo

53 ed ordered bi-bi mechanism, and performs the glucosamine-6-phosphate synthesis much more slowly than

54 gC is relieved in the presence of N-acetyl-D-glucosamine-6-phosphate, the intracellular form of N-ace

55 monstrate here that 6-azido-6-deoxy-N-acetyl-glucosamine (6AzGlcNAc) is a specific MCR for O-GlcNAcyl

56 galactosamine derivatives, N-thioglycolyl-D-glucosamine (7, C-4 epimer of 1), and alpha-O-benzyl 2-a

57 ptidoglycan is largely N-deacetylated on its glucosamine (93% of muropeptides) through the activity o

58 is also shown to be the first characterized glucosamine ABC transporter.

59 sis root and that SPINDLY (SPY), an O-linked glucosamine acetyltransferase, regulates cortex prolifer

60 sferase (LpxA) and UDP-3-O-(R-3-hydroxyacyl)-glucosamine acyltransferase (LpxD) catalyze the first an

61 ne acyltransferase (LpxA) and UDP-3-O-(acyl)-glucosamine acyltransferase (LpxD) constitute the essent

62 ynthesis driven by HAS2 was less affected by glucosamine addition, and HAS3 was not affected at all.

63 y and reduced uridine diphosphate-N-acetyl-D-glucosamine, along with decreased O- and N-linked protei

64 response to glucose are mimicked by N-acetyl glucosamine, an intermediate of the hexosamine biosynthe

65 ctively expose the 6-OH group in N-protected glucosamine analogues, which provided another route to c

66 95% CI: 0.72, 0.97; P for trend = 0.009) for glucosamine and 0.83 (95% CI: 0.69, 1.00; P for trend =

67 NMR characterization of the amine groups of glucosamine and 3-O-sulfoglucosamine in aqueous solution

68 Glucosamine and allosamine with 3-azido substitutions we

69 recommends against their use (including all glucosamine and chondroitin formulations).

70 ort-term acetaminophen, pharmaceutical grade glucosamine and chondroitin sulfate are recommended by E

71 These results suggest that glucosamine and chondroitin supplements are associated w

72 the nonvitamin-nonmineral supplements, only glucosamine and chondroitin were associated with total m

73 S), consisting of beta-1,4-linked N-acetyl-d-glucosamine and d-glucosamine units, possess diverse bio

74 nd sodium acetate as well as by N-acetylated glucosamine and galactosamine (GlcNAc and GalNAc) and gl

75 lation with C(2)-N-substituted benzylidene D-glucosamine and galactosamine trichloroacetimidates.

76 inear polysaccharide of alternating N-acetyl-glucosamine and glucuronic acid residues, is ubiquitousl

77 a linear sequence, consisting of alternating glucosamine and hexuronic acid building blocks.

78 CAM), 1-deoxy-1-aminomannopyranoside (DAMP), glucosamine and low molecular weight chitosan bonded to

79 o-glucuronic acid and propargyluted N-acetyl glucosamine and N-acetyl galactosamine derivative, respe

80 (D)-Glucosamine and other nutritional supplements have emerg

81 id A consists of two phosphorylated N-acetyl glucosamine and several acyl chains that are directly li

82 Glucosamine and thiamet-G also inhibited SOCE and were a

83 exhibited MS/MS fragmentations identical to glucosamine and those originating form aldohexoses showe

84 ted polysaccharide consisting of alternating glucosamine and uronic acid monosaccharide residues.

85 Use of glucosamine and use of chondroitin were each associated

86 Viosamine, together with rhamnose, (N-acetyl)glucosamine, and glucose, was found as a major component

87 orted sugars, including glucose, xylose, and glucosamine, and this substrate-induced expression can b

88 e, glyceraldehyde, erythrose, a heptose, and glucosamine are also demonstrated.

89 ing the oral bioavailability of N-acetyl-(d)-glucosamine as its putative bioactive phosphate form wer

90 Numerous biomolecules possess alpha-D-glucosamine as structural component.

91 ontaining amide and urea derivatives using D-glucosamine as the starting material.

92 of HA, UDP-Glucuronic acid and UDP-N-Acetyl-Glucosamine, as well as hyaluronic acid synthase which f

93 ed enzyme with exceptional specificity for d-glucosamine at its -1 subsite, thus preferring chitosan

94 nt probe for real-time detection of cellular glucosamine at micromolar level in living Caco-2 cells i

95 ase (NAT) Orf11*/Dbv8 through N-acylation on glucosamine at the central residue of Tei/A40926 pseudoa

96 zed for compositional analysis, 3-O-sulfated glucosamine at the reducing ends appears to be susceptib

97 fically degrades HS chains with 3-O-sulfated glucosamine at the reducing-end.

98 We describe metabolically inert l-glucosamine-based glycosylated antitumor ether lipids (L

99 This novel d-glucosamine-based inclusion compound has been synthesize

100 bacteria contain lipopolysaccharide (LPS), a glucosamine-based phospholipid, in the outer leaflet of

101 3 and O-4 phosphate prodrugs of N-acetyl-(d)-glucosamine bearing a 4-methoxy phenyl group and differe

102 lucose deprivation was blocked completely by glucosamine, but not by inhibition of OGA with 2-acetami

103 we show that the N-sulfate or 6-O-sulfate of glucosamine, but not the 2-O-sulfate of iduronate within

104 cose hexoses, allose, 3-O-methylglucose, and glucosamine by accumulating in the nucleus and activatin

105 reasing the cellular content of UDP-N-acetyl glucosamine by approximately 10-fold with 1 mm glucosami

106 ctive of this study was to determine whether glucosamine can activate autophagy.

107 of UV-B with photosensitizers fructosazine, glucosamine caramel and riboflavin enhanced the UV-B eff

108 rradiation and the combinational effect with glucosamine caramel, fructosazine and riboflavin on the

109 nally protected derivatives of carba-alpha-D-glucosamine, carba-alpha-D-mannose, carba-alpha-D-mannur

110 ves the linkage of glucuronic acid linked to glucosamine carrying 6-O-sulfo groups.

111 glucuronic acid (or iduronic acid) linked to glucosamine carrying various sulfo groups.

112 -uptake channel EcChiP for processing by the glucosamine catabolic pathway.

113 es of 13 vitamin and mineral supplements and glucosamine, chondroitin, saw palmetto, Ginko biloba, ga

114 avages, as well as cross-ring and inter-ring glucosamine cleavages, compared to CID and IRMPD, becaus

115 Here we describe a glucosamine-coated NISV, for blood-brain barrier GLUT1 t

116 EV monoclonal antibody Hu1A3B-7 delivered in glucosamine-coated vesicles and had improved survival an

117 In vivo, glucosamine-coated vesicles are superior to uncoated or

118 A five-step transformation of D-glucosamine, commencing with indium-mediated Barbier rea

119 thesized and characterized cypate and cypate-glucosamine conjugate (cy-2-glu), and investigated the p

120 are implicated in the perception of N-acetyl glucosamine-containing compounds, some of which are impo

121 Accurate quantification of glucosamine content in chitin and chitosan with differen

122 ent transfer of a fatty acyl moiety to a UDP-glucosamine core ring.

123 the NIR fluorescently labeled glucose analog glucosamine (cypate-glucosamine) has applications in pre

124 UDP-3-O-((R)-3-hydroxymyristoyl)-N-glucosamine deacetylase (LpxC) is as an attractive targe

125 lation (PA): the sequence of beta-1,4-linked glucosamine (deacetylated/D) and N-acetylglucosamine (ac

126 a generated highlights the huge potential of glucosamine-decorated NISV as a drug delivery platform w

127 is altered glycoform revealed an increase in glucosamine deposition.

128 It was observed that the inclusion of a d-glucosamine derivative into the hydrophobic cavity of be

129 Two urea-based receptors containing a glucosamine derivative were synthesized and investigated

130 the chemoselective enzymatic re-acylation of glucosamine derivatives, which can introduce new stable

131 x10(4) and 1.45x10(4)M(-)(1), along with the glucosamine detection limits of 1.06 and 0.29microM are

132 In these lipid A samples the glucosamine disaccharide characteristic for enterobacter

133 The glucosamine disaccharide-as a backbone surrogate of the

134 transfers a C14:0 to the 2'-position of the glucosamine disaccharide.

135 characterized biofilm factors poly-N-acetyl glucosamine, fibronectin-binding proteins, or autolytic

136 Mechanistically, we identify the glucosamine:fructose-6-phosphate amidotransferase (GFPT)

137 hase synthesis with immobilised L-thyroxine, glucosamine, fumonisin B2 or biotin as template, can dem

138 We engineered a novel polymer, glucosamine-functionalized polyisobutylene-maleic acid,

139 ed disaccharide D-glucuronic acid-acetylated glucosamine (GlcA-GlcNAc).

140 s hydrolysed with Alcalase and glycated with glucosamine (GlcN) at moderate temperatures (37/50 degre

141 mized and validated for the determination of glucosamine (GlcN) hydrolyzed from chitin in insect mate

142 vealed that ArnT is required for addition of glucosamine (GlcN) to B. bronchiseptica lipid A.

143 old water fish skin gelatin hydrolysates and glucosamine (GlcN) via transglutaminase (TGase), as well

144 the 1- and 4'-phosphates by the addition of glucosamine (GlcN), whereas 18-323 cannot, and 2) the C3

145 GLUT2 can transport the amino sugar glucosamine (GlcN), which could increase substrate for t

146 tructural components such as N-unsubstituted glucosamine (GlcN).

147 A mixture of glucosamine (GlcN, 15% w/v) and different amino acids in

148 cosamine-6-phosphate (GlcN6P, 1) and five of glucosamine (GlcN, 2) were prepared for evaluation as ca

149 Glucosamine (GlcN, 5% w/v) was incubated in either phosp

150 sed that the high-affinity ligand N-acetyl-d-glucosamine (GlcNAc) binds in the collectin CRD calcium

151 parameters for the deacetylation of N-acetyl-glucosamine (GlcNAc) by MshB, and the results from these

152 n the glycans D-mannose (Man) and D-N-acetyl glucosamine (GlcNAc) by the enzyme variants could then b

153 smic proteins by the monosaccharide N-acetyl-glucosamine (GlcNAc) continues to emerge as an important

154 r, wheat germ agglutinin-detectable N-acetyl-glucosamine (GlcNAc) epitopes were not identified when t

155 The monosaccharide N-acetyl-d-glucosamine (GlcNAc) is an abundant building block in na

156 yzed oxidation of isopropyl N-acetyl-alpha-d-glucosamine (GlcNAc) is used to prepare the rare sugars

157 modify host protein substrates with N-acetyl glucosamine (GlcNAc) on arginine residues.

158 of the group A carbohydrate, N-acetyl-beta-D-glucosamine (GlcNAc), and heart valve endothelium, lamin

159 (PGM3) catalyzes the conversion of N-acetyl-glucosamine (GlcNAc)-6-phosphate into GlcNAc-1-phosphate

160 ling of B cells reactive with the N-acetyl-D-glucosamine (GlcNAc)-containing Lancefield group A carbo

161 n and its T cell-binding partner, N-acetyl-D-glucosamine (GlcNAc).

162 charides of D-glucuronic acid and N-acetyl-D-glucosamine (GlcNAc).

163 e (GalNAc) in a beta-1,3 linkage to N-acetyl glucosamine (GlcNAc).

164 rom the F17 pilus that recognizes n-acetyl-d-glucosamine (GlcNAc).

165 in, a polymer of beta-1, 4-linked N-acetyl-D-glucosamine (GlcNAc).

166 an sulfate by approximately 35%), N-acetyl-d-glucosamine (GlcNAc)/GalNAc containing glycans recognize

167 cetyglucosamine (GlcNAz) and N-(4-pentynoyl)-glucosamine (GlcNAl) into cell-surface glycans and secre

168 ive proteomics with 6AzGlcNAc, N-azidoacetyl-glucosamine (GlcNAz), and N-azidoacetyl-galactosamine (G

169 sulfation of the 3-OH position of N-sulfated glucosamine (GlcNS) is the most beneficial modification

170 O-linked-N-acetyl-glucosamine glycosylation (O-GlcNAcylation) of the serin

171 Glucosamine has immunomodulatory effects on autoimmune d

172 y labeled glucose analog glucosamine (cypate-glucosamine) has applications in preclinical imaging, th

173 le synthesis requires the precursor N-acetyl-glucosamine; however, capsule is synthesized during post

174 Four hexosamine monomer derivatives-glucosamine hydrochloride, glucosamine sulfate, galactos

175 We demonstrate that glucosamine impedes Th1, Th2, and iTreg but promotes Th1

176 ho-2-aminoglucolactam (31) was prepared from glucosamine in a 13-step synthesis, which included a lat

177 Actomyosin was conjugated with glucose or glucosamine in a liquid system at moderate temperatures

178 ifically removes the 6-O sulphate group from glucosamine in highly sulfated regions of HS chains.

179 , which perform sulfation at 6-O position in glucosamine in HS, impact ovarian cancer angiogenesis th

180 d motivate future studies on the efficacy of glucosamine in modifying aging-related cellular changes

181 erent peptidoglycans and to chitin, and that glucosamine in the glycan chains is the minimal binding

182 ucosamine by approximately 10-fold with 1 mm glucosamine in the growth medium.

183 locked by a nonlabeled FR-beta ligand folate glucosamine in vivo.

184 ost amino acids, similar ones were found for glucosamine, indicating that this amino sugar is release

185 ily produce mycothiol (MSH or acetylcysteine-glucosamine-inositol, AcCys-GlcN-Ins) to protect the org

186 After being conjugated with the glucosamine, insulin can efficiently bind to RBC membran

187 Taken together, our results indicate that glucosamine interferes with N-glycosylation of CD25, and

188 These findings suggest that glucosamine is an effective autophagy activator and shou

189 Glucosamine is an important constituent of the heterogen

190 ial growth when the availability of N-acetyl-glucosamine is limited.

191 panel of Le(x) analogues in which N-acetyl-d-glucosamine, l-fucose, or d-galactose (D-Gal) are replac

192 mmercially available amino sugars, including glucosamine, mannosamine, galactosamine, and muramic aci

193 These effects suggest that glucosamine may be an important modulator of T cell diff

194 This was associated with glucosamine-mediated inhibition of the Akt/FoxO3/mammali

195 Interestingly, excess glucose rescued this glucosamine-mediated regulation, suggesting a functional

196 However, the mechanism(s) through which glucosamine modulates different T cell subsets and disea

197 Glucosamine modulates molecular targets of the autophagy

198 nto 6 different hexasaccharides in which the glucosamine moieties are either acetylated (GlcNAc) or m

199 ugh, modifies its lipid A by the addition of glucosamine moieties that promote TLR4 activation in hum

200 ther 0.1 or 0.25 degrees of substitution per glucosamine monomer.

201 ry, the binding stoichiometry of 2 and 1 for glucosamine monosaccharide (GlcN) and disaccharide (GlcN

202 cations consisting of an N-linked N-acetyl-D-glucosamine monosaccharide (N-GlcNAc).

203 of later-in-life MetS included citric acid, glucosamine, myoinositol, and proline (P < 0.03).

204 es annotated as UDP-3-O-[3-hydroxymyristoyl] glucosamine N-acyltransferase genes (la0512 and la4326 [

205 lso characterized a third enzyme, PgdB, as a glucosamine N-deacetylase.

206 hat the signal for position 2 of trisulfated glucosamine [N-, 3-O-, and 6-O-sulfated] (A*) is bifurca

207 s observed with other sugars like N-acetyl-D-glucosamine, N-acetyl-D-galactosamine, D-glucose and D-g

208 only enzyme that removes O-linked N-acetyl-d-glucosamine (O-GlcNAc) from target proteins.

209 ondrial proteins by O-linked beta-N-acetyl-D-glucosamine (O-GlcNAc) has been shown to regulate over 3

210 eased deposition of O-linked beta-N-acetyl-d-glucosamine (O-GlcNAc) in cardiac proteins are a hallmar

211 iseases can be modified by O-linked N-acetyl-glucosamine (O-GlcNAc) in vivo.

212 beta-O-N-acetyl-D-glucosamine (O-GlcNAc) is a post-translational modificat

213 n O-GlcNAc regulation.O-linked beta-N-acetyl glucosamine (O-GlcNAc) is an important protein modificat

214 The O-linked beta-N-acetyl glucosamine (O-GlcNAc) modification dynamically regulate

215 the cell surface, the O-linked beta-N-acetyl glucosamine (O-GlcNAc) modification is a simple intracel

216 NleB functions as a translocated N-acetyl-D-glucosamine (O-GlcNAc) transferase that modifies GAPDH.

217 The effect of glucosamine on T helper cell differentiation was similar

218 ocation of a single sulfate at either C-6 of glucosamine or C-2 of uronic acid in the reducing disacc

219 be reversed by addition of excess glucose or glucosamine or exogenous HA to the culture medium.

220 manner by increasing O-GlcNAc synthesis with glucosamine or inhibiting O-GlcNAcase with thiamet-G.

221 rast, infusion of the glucokinase inhibitors glucosamine or mannoheptulose worsened glucose tolerance

222 ysaccharide adhesin poly-beta-1,6-N-acetyl-D-glucosamine (PGA) by binding to the pgaABCD mRNA leader,

223 RapZ contributes to the control of glucosamine phosphate biogenesis by selectively presenti

224 Expression of HBP enzyme glucosamine-phosphate N-acetyltransferase 1 (GNPNAT1) is

225 ursors, LpxK binds almost exclusively to the glucosamine/phosphate moieties of the lipid molecule.

226 etic glycosylphosphatidylinositol analogues, glucosamine-phosphatidylinositol (glucosamine-PI) or glu

227 Glucosamine-PI caused the displacement of PrP(C) from li

228 In addition, ScGT1 cells treated with glucosamine-PI did not transmit infection following intr

229 Treatment with glucosamine-PI increased the cholesterol content of ScGT

230 We propose that treatment with glucosamine-PI modifies local micro-environments that co

231 The effect of glucosamine-PI on PrP(Sc) formation was also reversed by

232 analogues, glucosamine-phosphatidylinositol (glucosamine-PI) or glucosamine 2-O-methyl inositol octad

233 e exopolysaccharide poly-beta-1,6-N-acetyl-d-glucosamine (PNAG) by the extracellular protein IcaB is

234 e exopolysaccharide poly-beta-1,6-N-acetyl-D-glucosamine (PNAG) by the periplasmic protein PgaB is re

235 Poly-beta-1,6-N-acetyl-D-glucosamine (PNAG) is an exopolysaccharide produced by a

236 face polysaccharide poly-beta-(1-6)-N-acetyl-glucosamine (PNAG) mediates biofilm formation by some ba

237 nt deacetylation on poly-beta-1,6-N-acetyl-d-glucosamine (PNAG) oligomers, supporting previous immuno

238 A beta-(1-->6)-linked poly-N-acetyl-d-glucosamine (PNAG) surface capsule is synthesized by fou

239 s cell surface polysaccharide, poly-N-acetyl glucosamine (PNAG).

240 face polysaccharide poly-N-acetyl-beta-(1-6)-glucosamine (PNAG).

241 ntigen, a beta-(1-->6)-polymer of N-acetyl-D-glucosamine (PNAG).

242 psular polysaccharide (CP) and poly-N-acetyl glucosamine (PNAG).

243 two enzymes: uridine diphosphate-N-acetyl-D-glucosamine:polypeptidyltransferase (OGT) and O-GlcNAcas

244 a series of novel O-6 phosphate N-acetyl (d)-glucosamine prodrugs aimed at improving the oral bioavai

245 6-O-Sulfation at the glucosamine residue contributes to a wide range of biolo

246 The glucosamine residue of heparan sulfate can carry sulfo g

247 Furthermore, a preexisting N-sulfo glucosamine residue prevents the action of NDST-1 at the

248 asaccharides with 3-O-sulfo group-containing glucosamine residues at their reducing ends.

249 ate the 6-sulfate groups from the internal d-glucosamine residues in the highly sulfated subdomains o

250 f heparan sulfate oligosaccharides that have glucosamine residues modified by different patterns of N

251 Enzymatic cleavage between the two N-acetyl glucosamine residues of the chitobiose core of N-glycans

252 lfation of uronic acids and 6-O-sulfation of glucosamine residues, we genetically ablated heparan sul

253 y depend on the distribution of 6-O-sulfated glucosamine residues, which is generated by glucosaminyl

254 gh detection of the NH resonances of N-sulfo-glucosamine residues.

255 ing to the product with a cluster of N-sulfo glucosamine residues.

256 at attaches beta-O-GlcNAc (beta-O-N-acetyl-D-glucosamine) residues to S. aureus WTAs.

257 complex with beta-1,6-(GlcNAc)6, GlcNAc, and glucosamine reveal a unique binding mode suitable for in

258 fter transfer of the fatty acyl group to the glucosamine ring using the thiol-specific labeling reage

259 High-dose glucosamine significantly decreased Glut1 N-glycosylatio

260 nomer derivatives-glucosamine hydrochloride, glucosamine sulfate, galactosamine hydrochloride, and ma

261 s) GlmY and GlmZ activate the translation of glucosamine synthase (GlmS) in E. coli K-12, and in EHEC

262 A linear alpha-(1,4)-glucosamine tetrasaccharide was assembled to prove the u

263 saccharide is a hexaacylated disaccharide of glucosamine that is phosphorylated at the 1 and 4' posit

264 accharide is a hexa-acylated disaccharide of glucosamine that makes up the outer monolayer of the out

265 rticular the ability of fructose to generate glucosamine, the amino acid-metal complexes were heated

266 o N-phenyl carbamate (GalPUGNAc), N-acetyl-D-glucosamine-thiazoline (NGT), and N-acetyl-D-galactosami

267 pxQ in E. coli converts much of the proximal glucosamine to 2-amino-2-deoxygluconate.

268 Administration of glucosamine to GFP-LC3-transgenic mice markedly activate

269 hanced, the ability of both high glucose and glucosamine to induce insulin resistance.

270 that modifies host proteins with N-acetyl-d-glucosamine to inhibit antibacterial and inflammatory ho

271 alogs of thymidine, uridine, methionine, and glucosamine to label nascent synthesis of DNA, RNA, prot

272 glycosides such as 6-azido-6-deoxy-d-glucose/glucosamine to lead to beta-d-galactopyranosyl-(1->3)-d-

273 revealed the involvement of a novel N-acetyl glucosamine transporter and an alpha/beta-fold hydrolase

274 Finally, glucosamine treatment inhibited Th1 cells in vivo, prolo

275 Glucosamine treatment of chondrocytes activated autophag

276 Glucosamine treatment, which bypasses the rate-limiting

277 Glucosamine treatment, which increases UDP-GlcNAc availa

278 s in autophagy in response to starvation and glucosamine treatment.

279 A increased after O-GlcNAcylation induced by glucosamine treatments or by inhibiting O-GlcNAc transfe

280 ogue data for uridine diphosphate N-acetyl-D-glucosamine (UDP-GlcNAc) in human prostate cancer LnCaP-

281 o occur by 4,6-dehydration of UDP-N-acetyl-d-glucosamine (UDP-GlcNAc) to UDP-2-acetamido-2,6-dideoxy-

282 e linkage between a GlcA unit and an N-sulfo glucosamine unit carrying either a 3-O-sulfo or a 6-O-su

283 eaves the linkage of a GlcA unit and N-sulfo glucosamine unit with a 2-O-sulfated GlcA residue, not a

284 e sulfate group at the 6-O position of the d-glucosamine unit with the sulfonamide motif effectively

285 fers a sulfo group to the 3-OH position of a glucosamine unit.

286 y increased N-sulfation and 6-O-sulfation of glucosamine units in response to the decrease in 2-O-sul

287 ger glycans, the N-acetylamino groups of the glucosamine units were converted to imides to prevent fo

288 beta-1,4-linked N-acetyl-d-glucosamine and d-glucosamine units, possess diverse bioactivities that ca

289 tly regulates several chitin- and N-acetyl-D-glucosamine-utilization genes that are co-regulated duri

290 OS sialylation, increasing abundance of LA d-glucosamine versus 2,3-diamino-2,3-dideoxy-d-glucose, an

291 In vitro, we show glucosamine vesicle transcytosis across the blood-brain

292 Glucosamine was found to be more effective for glycation

293 limit of detection of 5 nM for DABSYL-tagged glucosamine was obtained using long injections (to give

294 s, the induction of TRIB3 by high glucose or glucosamine was reversible upon removal of these substra

295 The increased leptin levels induced by glucosamine were susceptible to the inhibition by iron,

296 ression was also substantially stimulated by glucosamine, which bypasses GFAT, accompanied by a decre

297 fated iduronic acid linked to 6-O-sulfated N-glucosamine, which contains a free amine at position 2,

298 intramolecular anomeric protection (iMAP) of glucosamine, which facilitates concise transformation of

299 he alpha-anomeric glycoside of L-cysteinyl-D-glucosamine with L-malic acid, is a major low-molecular-

300 lent selective fluorescent responses towards glucosamine with the enhancement of fluorescence quantum