ライフサイエンスコーパス: glucose transporter 1

1 ulin-independent glucose transporter GLUT-1 (glucose transporter-1).

2 ls of vascular endothelial growth factor and glucose transporter 1.

3 the synthesis of the ubiquitously expressed glucose transporter 1.

4 density (MVD) (CD31 and CD105/endoglin), and glucose transporter 1.

5 18)F]FDG uptake and macrophage expression of glucose transporter 1.

6 of resulting glycoconjugates, where GLUT1 is glucose transporter 1.

7 ch as for VEGF, phosphoglycerate kinase, and glucose transporter-1.

8 ed upregulation of glycolytic pathway genes, glucose transporter 1-4 (Glut1-4), phosphoglycerate kina

9 Microarray experiments revealed that glucose transporters 1, 8, and 10 were expressed in MCF-

10 ecific loss of HIF1alpha or its target gene, glucose transporter 1, ameliorated epidermal, immune, va

11 Immunohistochemical Ki-67 and glucose transporter 1 analysis was used to evaluate tumo

12 sion of marker proteins such as facilitative glucose transporter 1 and claudin-5 in freshly isolated

13 cell lines leads to increased expression of glucose transporter 1 and consequent increased cellular

14 imens, mRNA levels and protein expression of glucose transporter 1 and glucose transporter 3 (GLUT-1

15 Furthermore, the expression of both glucose transporter 1 and hexokinase 2, the first enzyme

16 sion of hypoxia-inducible factor 1-alpha and glucose transporter 1 and increased glucose uptake.

17 he functional level, we show that removal of Glucose transporter 1 and Lactate dehydrogenase A gene a

18 l death and that pharmacologic inhibition of glucose transporter 1 and PRC1 synergistically promoted

19 Mechanistically, glucose uptake via GLUT (glucose transporter)-1 and enhanced glycolysis in neutro

20 on of glucose transporters (sodium-dependent glucose transporter-1 and glucose transporter-2) and swe

21 rom the small intestine via sodium-dependent glucose transporter-1 and glucose transporter-2, which m

22 Low serum glucose, downregulation of glucose transporter-1 and glucose-6-phosphatase mRNA, an

23 of other hypoxia-inducible genes, including glucose transporter-1 and insulin-like growth factor-bin

24 Glucose transporter-1 and Ki-67 were negative in the end

25 unohistochemical staining revealed increased glucose transporter-1 and norepinephrine transporter exp

26 tilization accompanies the downregulation of glucose transporter-1 and poly (ADP-ribose) polymerase c

27 at cells in the invasive edges expressed the glucose transporter-1 and the sodium-hydrogen exchanger-

28 Surprisingly, the expression of glucose transporters 1 and 4 and insulin receptor substr

29 lts in more abundant membrane association of glucose transporters 1 and 4, which enhances glucose upt

30 endothelial markers (von Willebrand factor, glucose transporter-1) and robust expression of tight an

31 ecreased vascular endothelial growth factor, glucose transporter 1, and erythropoietin expression.

32 encoding vascular endothelial growth factor, glucose transporter 1, and erythropoietin were up-regula

33 ith upregulation of the glucose transporter, glucose transporter 1, and glycolytic genes, hk1 and pdk

34 he expression of the HIF-1alpha-target gene, glucose transporter 1, and report that HIF-1alpha promot

35 ression of hypoxia-inducible factor 1 alpha, glucose transporter 1, and vascular endothelial growth f

36 expression of the HIF-1alpha targets VEGF-A, glucose transporter-1, and lactate dehydrogenase A.

37 The localization of uncoupling protein-1, glucose transporter-1, and norepinephrine transporter wa

38 s vascular endothelial growth factor (VEGF), glucose transporter-1, and pyruvate dehydrogenase kinase

39 We discovered that a glucose transporter 1- and pyruvate kinase-dependent glu

40 E-19 cells were labeled with MCT1, MCT4, and glucose transporter-1 antibodies.

41 le proteomics approaches, we have identified glucose transporter-1 as the receptor for dematin and ad

42 ines by HIV-1 led to increased expression of glucose transporter 1, associated with increased transpo

43 with a modest reduction in the expression of glucose transporter 1, but not pyruvate kinase, in the A

44 Profibrotic up-regulation of glucose transporter 1 by TGF-beta involves activation of

45 Although glucose transporter 1, claudin-3, and plasmalemma vesicu

46 kers of proliferation (K(i)-67) and hypoxia (glucose transporter 1) defined metabolic signaling in th

47 /IGF-1 signaling, decreased INS receptor and glucose transporter 1 densities, and changes in the meta

48 n CD46-costimulated T cells and identify the glucose transporter 1 encoding transcript SLC2A1 as a ta

49 Because homologues of dematin, adducin, and glucose transporter-1 exist in many non-erythroid cells,

50 r analysis revealed that cicaprost decreased glucose transporter 1 expression and glucose uptake and

51 y regulators in energy substrate metabolism (glucose transporter 1, glucose transporter 4, pyruvate d

52 nd expression of intestinal sodium-dependent glucose transporter-1, glucose transporter-2, and sweet

53 atients, duodenal levels of sodium-dependent glucose transporter-1, glucose transporter-2, and T1R2 t

54 ligation and puncture mice sodium-dependent glucose transporter-1, glucose transporter-2, and T1R2 t

55 man) and relative levels of sodium-dependent glucose transporter-1, glucose transporter-2, and taste

56 nterference (RNAi), to determine the role of glucose transporter 1 (GLUT-1) and GLUT-3 in L-14C-DHA t

57 searched until July 2012 by using the terms glucose transporter 1 (GLUT-1) deficiency syndrome, gluc

58 sults were supported by significantly higher glucose transporter 1 (Glut-1) expression of CD4+ cells

59 U binding studies revealed that, for HTLV-1, glucose transporter 1 (GLUT-1) functions at a postbindin

60 The observation that overexpression of glucose transporter 1 (GLUT-1) in mesangial cells could

61 ascular endothelial growth factor (VEGF) and glucose transporter 1 (Glut-1) is markedly reduced.

62 or-activated receptor gamma (PPAR-gamma) and glucose transporter 1 (GLUT-1) levels in human brain mic

63 has a bona fide candidate cellular receptor, glucose transporter 1 (glut-1), been identified.

64 HIF-1alpha, and its downstream proteins, glucose transporter 1 (GLUT-1), erythropoietin (EPO), an

65 s in placental homogenates and expression of glucose transporter 1 (GLUT-1), taurine transporter (TAU

66 smooth muscle actin (SMA), D2-40, CD34, and glucose transporter 1 (GLUT-1).

67 Glucose transporter-1 (Glut-1) and hexokinase II (HKII)

68 synthesized and covalently attached to anti-glucose transporter-1 (GLUT-1) antibodies via carbodiimi

69 es using adherent cell lines have shown that glucose transporter-1 (GLUT-1) can function as a recepto

70 nd to relate this to physiologic hypoxia and glucose transporter-1 (GLUT-1) expression.

71 scular endothelial growth factor (VEGF), and glucose transporter-1 (Glut-1) is mediated in part by a

72 The expression of glucose transporter-1 (Glut-1), hexokinase-1 and -2 (Hk-

73 ascular endothelial growth factor (VEGF) and glucose transporter-1 (Glut-1).

74 (DHA), enters mitochondria via facilitative glucose transporter 1 (Glut1) and accumulates mitochondr

75 ins, including the insulin receptor (CD220), glucose transporter 1 (GLUT1) and fatty acid translocase

76 , at E19.5 labyrinth trophoblast had reduced glucose transporter 1 (GLUT1) and glucocorticoid recepto

77 hypoglycemia-induced gene expression of both glucose transporter 1 (Glut1) and inhibitor of kappa bet

78 n-responsive vesicular pool of intracellular glucose transporter 1 (GLUT1) and transferrin receptors

79 e factor 1 alpha and its downstream targets, glucose transporter 1 (GLUT1) and vascular endothelial g

80 esis was demonstrated by immunostaining with glucose transporter 1 (GLUT1) antibody, in viable vessel

81 rs-2 does regulate the surface expression of glucose transporter 1 (Glut1) as assessed by flow cytome

82 We examined whether glucose transporter 1 (GLUT1) deficiency causes common i

83 To test this hypothesis, we evaluated glucose transporter 1 (Glut1) expression and glucose upt

84 y hypoxia-inducible factor (HIF)-1 alpha and glucose transporter 1 (Glut1) expression for proinflamma

85 Immunohistochemical FAP and glucose transporter 1 (GLUT1) expression of stromal and

86 TATA sequence, whereas the promoter for the glucose transporter 1 (Glut1) gene contains a TATA eleme

87 ingle-nucleotide polymorphisms (SNPs) in the glucose transporter 1 (GLUT1) gene to the uptake of (18)

88 Glucose transporter 1 (GLUT1) immunohistochemistry was p

89 Computational simulations of glucose transporter 1 (GLUT1) inhibition in the model ac

90 we show that expression of the facilitative glucose transporter 1 (GLUT1) is induced by TGF-beta in

91 A significant increase in the glucose transporter 1 (GLUT1) level, as well as in the g

92 tant increases in glucose import and surface glucose transporter 1 (GLUT1) levels, leading to elevate

93 ough independent mechanisms, and we identify glucose transporter 1 (GLUT1) messenger RNA as a direct

94 Glucose transporter 1 (Glut1) of murine HSC was disrupte

95 The glucose transporter 1 (GLUT1) protein is underexpressed

96 +) T cells in CLL have reduced intracellular glucose transporter 1 (GLUT1) reserves, and have an alte

97 tas of HF-fed animals, protein expression of glucose transporter 1 (GLUT1) was increased 5-fold, and

98 nd lipolysis (17.7%); the mRNA expression of glucose transporter 1 (GLUT1) was upregulated but glucos

99 The glucose transporter 1 (GLUT1) with high expression in ca

100 , with or without concurrent deletion of the glucose transporter 1 (Glut1), also known as solute carr

101 RNA expression, and decreases in HXK1, PFK1, glucose transporter 1 (GLUT1), and GLUT3 mRNA expression

102 depleted LICs by reducing the expression of glucose transporter 1 (Glut1), compromising glucose flux

103 Astrocytes express glucose transporter 1 (GLUT1), considered their primary

104 tion of EVs, specifically those carrying the glucose transporter 1 (GLUT1), promotes glucose uptake i

105 In contrast, follower cells highly express glucose transporter 1 (GLUT1), which sustains an elevate

106 into cells by membrane transporters such as glucose transporter 1 (GLUT1).

107 sion increases glucose uptake by stimulating glucose transporter 1 (GLUT1).

108 ficacy of PDT in cancer cells overexpressing glucose transporter 1 (GLUT1).

109 transcriptional co-activator p300 as well as glucose transporter 1 (Glut1).

110 g glucose uptake via selective expression of glucose transporter 1 (Glut1).

111 novo serine-glycine biosynthetic pathway and glucose transporter 1 (GLUT1).

112 Here, we show that a subset of glucose transporter 1 (GLUT1/SLC2A1) co-endocytoses with

113 parameters were compared with expressions of glucose transporter-1 (GLUT1) and hexokinase-2 measured

114 A structure has been proposed for glucose transporter-1 (GLUT1) based upon homology modeli

115 glycemia of maternal diabetes suppresses the glucose transporter-1 (GLUT1) facilitative glucose trans

116 uction in the endothelium by deletion of the glucose transporter-1 (GLUT1) in mice results in loss of

117 r of NF-kappaB-kinase beta (IKKbeta) induced glucose transporter-1 (GLUT1) membrane trafficking in bo

118 e and paucity of its translated product, the glucose transporter-1 (Glut1) protein, disrupt brain fun

119 a proliferative arteriopathy associated with glucose transporter-1 (Glut1) up-regulation and a glycol

120 d glucose uptake through the upregulation of glucose transporter-1 (Glut1), lactate secretion and ind

121 ntial proteome analysis, we identify SLC2A1 (glucose transporter 1, GLUT1) as a downstream target of

122 s human leukocyte antigen-antigen D-related, glucose transporter 1, granzyme B, and the short-lived e

123 lization of HIF-1alpha and an enhancement in glucose transporter-1, hexokinase-2, and monocarboxylate

124 energy, leading to increased levels of human glucose transporter 1 (hGLUT1).

125 Da protein-interacting protein 3) and GLUT1 (glucose transporter 1); (ii) secretion of pre-formed IL-

126 d higher expression levels of an HIF target, glucose transporter 1, in the brain.

127 to meet its increased bioenergetic demands; glucose transporter-1 is up-regulated, basolateral gluco

128 s correlated by flow cytometric detection of glucose transporter 1 levels in mucosal CD4(+) T lymphoc

129 was compared with histologic scores and with glucose transporter 1 levels in mucosal immune cells by

130 ally included perturbed vascular endothelial glucose transporter-1 localization.

131 ecent structural studies suggest that GLUT1 (glucose transporter 1)-mediated sugar transport is media

132 ivity were markedly increased as a result of glucose transporter 1-mediated glucose influx that drive

133 RC2 to drive glycolysis and lipogenesis, and glucose transporter 1-mediated glucose metabolism promot

134 Furthermore, the glucose transporter-1 mRNA, which is also stabilized by

135 etabolism and increases the transcription of Glucose-transporter-1 mRNA, and of Hexokinase and Pyruva

136 Importantly, inhibitors of the glucose transporter 1 or glutaminase in vivo attenuated

137 rol, or white blood counts, nor reduce day 2 glucose transporter 1 or myeloperoxidase expression in t

138 e infusion was also negated by knocking down glucose transporter 1 or pyruvate kinase in the NTS.

139 Glucose transporter 1 or pyruvate kinase lentiviral-medi

140 2 and the cerebrovascular-selective proteins glucose transporter 1, permeability-glycoprotein, and la

141 ze, non-signet ring cell carcinoma type, and glucose transporter 1-positive expression on immunohisto

142 of high SUVmax, with an increased number of glucose transporter-1-positive cells.

143 absorbed into intestine cells via the sodium glucose transporter 1 (SGLT-1) and glucose transporter 2

144 Sodium-glucose transporter 1 (SGLT1) has been proposed as this

145 tration (20%) of the nonmetabolizable sodium-glucose transporter 1 (SGLT1) substrate, methyl-alpha-D-

146 usly proposed alternative transducer, sodium-glucose transporter 1 (SGLT1).

147 hypoxia-inducible factor 1alpha (HIF1alpha), glucose transporter 1 (SLC2A1; also known as GLUT1), and

148 Proof-of-concept microCT and glucose transporter-1 staining served as morphologic val

149 This, in turn, selectively induces glucose transporter 1 surface localization and glucose u

150 annexin V-depleted neutrophils, we show that glucose transporter-1 translocation is impaired in neutr

151 VHL function, we found that high basal VEGF, glucose transporter-1, urokinase-type plasminogen activa

152 or BRAF genes, we found that GLUT1, encoding glucose transporter-1, was one of three genes consistent

153 dly enrich a 12-transmembrane helix protein, glucose transporter 1 with antibodies (GLUT1 complex), b