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1 he liver stage are lethal to patients with a glucose-6-phosphate dehydrogenase deficiency.
2 t Asian ovalocytosis and two common forms of glucose-6-phosphate dehydrogenase deficiency.
3 lpha-thalassemia, 0.3%; ABO group, 0.3%; and glucose-6-phosphate dehydrogenase deficiency, 0.5%) and
4 re both contraindicated for individuals with glucose-6-phosphate dehydrogenase deficiency and in preg
6 5 mg/kg/d for 14 days) without screening for glucose-6-phosphate dehydrogenase deficiency and were fo
8 of pooled severe malaria data reported that glucose-6-phosphate dehydrogenase deficiency (G6PDd) was
9 acute haemolytic anaemia in individuals with glucose-6-phosphate dehydrogenase deficiency (G6PDd).
10 uinolines cause dose-dependent haemolysis in glucose-6-phosphate dehydrogenase deficiency (G6PDd).
12 on in Israel, we found that individuals with glucose-6-phosphate dehydrogenase deficiency had an incr
13 se, fetal hemoglobin, alpha-thalassemia, and glucose-6-phosphate dehydrogenase deficiency had no effe
14 [95% confidence interval {CI}, 0.52-0.90]), glucose-6-phosphate dehydrogenase deficiency in female c
15 tors for neurotoxicity, such as prematurity, glucose-6-phosphate dehydrogenase deficiency, or hypoxia
17 lasmic PICD, the phenotypes of patients with glucose-6-phosphate dehydrogenase deficiency suggest tha
19 No correlation with sickle cell trait or glucose-6-phosphate-dehydrogenase deficiency was observe