ライフサイエンスコーパス: glucosuria

1 eneral feature of patients with diabetes and glucosuria.

2 ar glucose transport, resulting in increased glucosuria.

3 ke was entirely explained by the increase in glucosuria.

4 cohort of 60 individuals with familial renal glucosuria.

5 nuria or proteinuria but was associated with glucosuria (1.00 glucosuria vs. 0.19 no glucosuria; P =

6 , as well as in patients with familial renal glucosuria, a condition that is considered a natural mod

7 We investigated glucosuria and aminoaciduria in patients with HNF-1alpha

8 t mice exhibit a renal Fanconi syndrome with glucosuria and generalized aminoaciduria in addition to

9 Dapagliflozin treatment induced glucosuria and markedly lowered fasting plasma glucose.

10 ment of salt-induced hypertension by causing glucosuria and natriuresis without changes in the Renin-

11 um-glucose cotransporter-2 inhibitors induce glucosuria and sodium excretion with nephroprotective ef

12 e in urine output and a 500-fold increase in glucosuria, as well as compensatory increases in feeding

13 The HNF-1alpha patients had glucosuria at lower glycemic control (as shown by HbA1c)

14 ubjects with IFG and NFG produces comparable glucosuria but lowers the plasma glucose concentration a

15 SGLT2 (SLC5A2) associate with familial renal glucosuria, but the role of SGLT2 in the kidney is incom

16 ia and effectively normalized hyperglycemia, glucosuria, glucose intolerance, and insulin resistance

17 glycemic effect was associated with a robust glucosuria (> 8 g/dL) observed in nondiabetic mice.

18 t works specifically on the kidney to induce glucosuria improves muscle insulin sensitivity.

19 me with juvenile cataracts, microcornea, and glucosuria in a single family.

20 nM), and 101 (72 nM, 6.7 nM, 0.8 nM) induced glucosuria in nondiabetic rats.

21 There was mild glucosuria in some animals.

22 se uptake (sum of tissue glucose uptake plus glucosuria) increased in both T2D and NGT following DAPA

23 ion overrides the osmotic diuretic effect of glucosuria induced by dapagliflozin treatment.

24 However, glucosuria induction following SGLT2 inhibition is assoc

25 Thus, glucosuria is not part of the MCT12 mutation syndrome.

26 ening for hypophosphatemia, proteinuria, and glucosuria is recommended for HBV-LT recipients on tenof

27 ical entities defined by the urine dipstick: glucosuria, ketonuria, proteinuria, hematuria and urine

28 Empagliflozin caused 50 +/- 4 and 45 +/- 4 g glucosuria on day 2 in subjects with IFG and NFG, respec

29 with glucosuria (1.00 glucosuria vs. 0.19 no glucosuria; P = 0.002).

30 the eye phenotype, poor correlation with the glucosuria phenotype did not support a pathogenic role o

31 in the family that segregated with the renal glucosuria phenotype.

32 RC2 alone develop a Fanconi-like syndrome of glucosuria, phosphaturia, aminoaciduria, low molecular w

33 Sglt2(-/-) mice had glucosuria, polyuria, and increased food and fluid intak

34 drugs is entirely dependent on the amount of glucosuria produced, it is important to understand why S

35 Significant decrease in severe glucosuria, proteinuria, blood creatinine, urea and adva

36 These include three independent glucosuria variants at SLC5A2, the gene encoding the sod

37 ria but was associated with glucosuria (1.00 glucosuria vs. 0.19 no glucosuria; P = 0.002).

38 ects with IFG and NFG, respectively, and the glucosuria was maintained for 2 weeks in both groups.

39 showed significantly more aminoaciduria when glucosuria was present compared with when it was absent

40 Diabetic mice also had glucosuria, which may enhance bacterial replication in t

41 jection of glucose induced hyperglycemia and glucosuria with increased GFR in mice.

42 be ameliorated in animal models by inducing glucosuria with renal glucose transport inhibitors.