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1 sed genomic map EcoMap7) near gadB (encoding glutamate decarboxylase).
2 ivity for gamma-aminobutyric acid (GABA) and glutamate decarboxylase.
3 e mice generated Abs against host insulin or glutamate decarboxylase.
4 caused increased levels of the GadA and GadB glutamate decarboxylases.
5 hat cancer cells with aberrant expression of glutamate decarboxylase 1 (GAD1) rewire glutamine metabo
6 setting caused by epigenetic upregulation of glutamate decarboxylase 1 (GAD1), a regulator of the GAB
7 modulin-dependent kinase alpha [CaMKIIa] and glutamate decarboxylase 1 [GAD1] promoters, respectively
8 ng glutamate and GABA, glutaminase (Gls) and glutamate decarboxylase 1 and 2 (Gad1 and Gad2).
9 3W showed deficits in the induction of GAD1 (glutamate decarboxylase 1) protein expression.
10 otein 1, synaptosomal-associated protein 29, glutamate decarboxylase 1, metabotropic glutamate recept
11  Knockout of the major GABA synthesis enzyme Glutamate Decarboxylase 2 (GAD2) increases the aperture
12 icular glutamate transporter 2 (SLC17A6) and glutamate decarboxylase 2 (GAD2), but these transcripts
13 n of the gene for the GABA synthetic enzyme, glutamate decarboxylase 65 (GAD(65)), reduce basal corti
14 tion of gamma-aminobutyric acid synthesis by glutamate decarboxylase 65 (GAD65) in response to high i
15  gamma-aminobutyric acid-synthesizing enzyme glutamate decarboxylase 65 (GAD65) to presynaptic cluste
16  expression leads to decreased expression of glutamate decarboxylase 65 (GAD65), the enzyme required
17 nciples of PIP design to the T1D autoantigen glutamate decarboxylase 65 (GAD65).
18 T1D based on the T1D-associated auto-antigen glutamate decarboxylase 65 (GAD65).
19                            Autoantibodies to glutamate decarboxylase 65 (GAD65Ab) are commonly believ
20  amacrine cells (82%), was immunoreactive to glutamate decarboxylase 65, but no Per1 : :GFP(+) amacri
21 whether messenger RNA interference targeting glutamate decarboxylase 65/67 (GAD65/67) gene expression
22  gluco-inhibitory y-aminobutyric acid (GABA; glutamate decarboxylase(65/67)-ir-positive) neurons exhi
23 ter-1 and ATP-alpha, but increased levels of glutamate decarboxylase-65 and glutamine synthetase in P
24 r genes that may be related to regulation of glutamate decarboxylase 67 (GAD(67)), a marker for this
25 ynaptic density protein 95 (PSD95) and lower glutamate decarboxylase 67 (GAD67) expression in GluD1 K
26     The temporal and spatial distribution of glutamate decarboxylase 67 (GAD67) mRNA-containing neuro
27 ization with the interneuron-specific enzyme glutamate decarboxylase 67 (Gad67), but not other cell-t
28 al nAChR subunit mRNAs are co-expressed with glutamate decarboxylase 67 (GAD67), the marker for GABAe
29                                           3) Glutamate decarboxylase 67, vesicular GABA transporter,
30 rain-derived neurotrophic factor (BDNF), and glutamate decarboxylase-67 (GAD67).
31 f ARC neurons coexpress orexin receptors and glutamate decarboxylase-67 and are excited by orexin.
32 n-10 (IL-10), IL1 receptor 1, BDNF, NGF, and glutamate decarboxylase-67 in vitro using hypothalamic a
33 immunoreactive contacts with ChAT-, PV-, and glutamate decarboxylase-67-positive neurons that project
34 lecule to be a substrate of Escherichia coli glutamate decarboxylase, a pyridoxal 5'-phosphate-depend
35  of the GDAR system by direct measurement of glutamate decarboxylase activity and acid survival.
36 s is believed to be initiated by a change in glutamate decarboxylase activity, but the underlying mec
37 s suggest that GadC (XasA) participates in a glutamate decarboxylase alkalinization cycle to protect
38 is located downstream of gadA, which encodes glutamate decarboxylase, an enzyme involved in acid resi
39 ry gene gadE resulted in very high levels of glutamate decarboxylase and almost complete protection a
40 ion of GadE removes most pH control over the glutamate decarboxylase and antiporter genes.
41                                 GadAB encode glutamate decarboxylase and protect E. coli from the tox
42      Transcription of the genes encoding the glutamate decarboxylases and the substrate-product antip
43 aminobutyric acid (GABA)ergic markers (GABA, glutamate decarboxylase) and to peptides and calcium bin
44 ase), bNOS (brain-type nitric oxidase), GAD (glutamate decarboxylase), and glial markers, and occasio
45 uble staining studies (using SMI-32 and anti-glutamate decarboxylase antibodies, both markers of cort
46 related to carbon isotope discrimination and glutamate decarboxylase associated with foliar nitrogen
47                             For example, the glutamate decarboxylase beta hexamer (~317 kDa) exhibits
48  regulates two genes that encode isoforms of glutamate decarboxylase critical to this system, but add
49 ticularly observed the downregulation of the glutamate decarboxylase encoding genes GAD1 and GAD2, as
50                             GAD1 encodes the glutamate decarboxylase enzyme GAD67, a critical actor o
51 d2 mRNA in POMC neurons, as these encode the glutamate decarboxylase enzymes GAD67 and GAD65, respect
52  GABA shunt pathway-related genes, including glutamate decarboxylase, GABA transaminase, and succinic
53 nsity of both the 65- and 67-kDa isoforms of glutamate decarboxylase (GAD(65) and GAD(67)) -immunorea
54 een shown to increase pallidal expression of glutamate decarboxylase (GAD(67) isoform) mRNA.
55 f mRNA encoding the 67-kilodalton isoform of glutamate decarboxylase (GAD(67)), an enzyme for GABA sy
56 then expressed three TCR specific for either glutamate decarboxylase (GAD) 206-220 or GAD 524-538 or
57  CRPC, via phosphorylation and activation of glutamate decarboxylase (GAD) 65.
58 rch 18(th), 2023 for studies reporting GABA, glutamate decarboxylase (GAD) 65/67, GABA(A), GABA(B,) a
59 ypersensitivity induced by stress due to its glutamate decarboxylase (GAD) activity.
60 ine whether CGRP-containing neurons also had glutamate decarboxylase (GAD) and other markers for GABA
61 of evidence are presented to indicate that l-glutamate decarboxylase (GAD) can become membrane-associ
62              The human neuroendocrine enzyme glutamate decarboxylase (GAD) catalyses the synthesis of
63                                              Glutamate decarboxylase (Gad) enzyme, converts glutamate
64 amatergic signaling becomes predominant when glutamate decarboxylase (GAD) function is compromised.
65          In GABAergic neurons [identified by glutamate decarboxylase (GAD) immunocytochemistry], foca
66 zes, which were colocalized with clusters of glutamate decarboxylase (GAD) immunoreactivity at rates
67 eads to persistent reductions in hippocampal glutamate decarboxylase (GAD) interneuron numbers withou
68    Two distinct cDNA clones encoding for the glutamate decarboxylase (GAD) isoenzymes GAD1 and GAD2 f
69                                   The enzyme glutamate decarboxylase (GAD) produces the neurotransmit
70 a microti possesses a potentially functional glutamate decarboxylase (GAD) system involved in extreme
71 n essential transcriptional activator of the glutamate decarboxylase (GAD) system, the most efficient
72 isiae homologue of the GABA-producing enzyme glutamate decarboxylase (GAD) that is required for norma
73 emistry against tyrosine hydroxylase (TH) or glutamate decarboxylase (GAD) to systematically compare
74 on of an immunodominant epitope derived from glutamate decarboxylase (GAD) was observed regardless of
75 lin-dependent diabetes mellitus autoantigen, glutamate decarboxylase (GAD), was tested.
76 ll bodies were identified by the presence of glutamate decarboxylase (GAD)-67 mRNA or glycine transpo
77 receptors were found to be co-localized with glutamate decarboxylase (GAD)-positive neurons (approxim
78 as expressing GABA, and its synthetic enzyme glutamate decarboxylase (GAD).
79 ressed or stimulated hypoglycemia-associated glutamate decarboxylase (GAD)1 and GAD2 mRNA expression
80                                     In vivo, glutamate decarboxylase (GAD, the enzyme which synthesiz
81 the cloned yhiX gene increased production of glutamate decarboxylases (GAD) and activated the transcr
82 ma aminobutyric acid(GABA) is synthesized by glutamate decarboxylase(GAD) in beta-cells.
83  mouse and human genes coding for the 67 kDa glutamate decarboxylase (Gad1) also contain binding site
84 scription factor, which we report to bind to glutamate decarboxylase (Gad1), which encodes GAD67, a r
85 ate transporters (vglut1, vglut2.1, vglut3), glutamate decarboxylases (gad1, gad2), and choline acety
86      Autoantibodies to the 65-kDa isoform of glutamate decarboxylase GAD65 (GAD65Ab) are strong candi
87  the conventional GABA-synthesizing enzymes, glutamate decarboxylases GAD65 and GAD67.
88 n localizing the two GABA-producing forms of glutamate decarboxylase (GAD65 and GAD67) in the normal
89          Islet antigen (IA)-2, IA-2beta, and glutamate decarboxylase (GAD65) are major autoantigens i
90 ated that a spontaneous Th1 response against glutamate decarboxylase (GAD65) arises in NOD mice at fo
91 -cell responses to the beta-cell autoantigen glutamate decarboxylase (GAD65), induced an active form
92  mice had no spontaneous responses to 65-kDa glutamate decarboxylase (GAD65), its immunodominant pept
93 nal activity-regulated pentraxin (Narp), and glutamate decarboxylase (GAD65).
94 h corresponding reductions in this region of glutamate decarboxylase (GAD65/67) and markers of dendri
95 earliest times of detection for two forms of glutamate decarboxylase (GAD67 and GAD65) in the embryon
96 -related genes such as the 67 kDa isoform of glutamate decarboxylase (GAD67) and parvalbumin (PV), ap
97 protein levels of tyrosine hydroxylase (TH), glutamate decarboxylase (GAD67), and vesicular glutamate
98 orescence studies using antibodies to TH and glutamate decarboxylase (GAD67), the synthetic enzyme fo
99 xpression of BB2 receptor mRNA together with glutamate decarboxylase (GAD67).
100 els for Lhx6, parvalbumin, somatostatin, and glutamate decarboxylase (GAD67; the principal enzyme in
101             The larger isoform of the enzyme glutamate decarboxylase, GAD67, synthesizes >90% of basa
102 esistance system encompasses two isoforms of glutamate decarboxylase (gadA and gadB) and a putative g
103 tection at pH 2.5, one of two genes encoding glutamate decarboxylase (gadA or gadB), and the gene enc
104 smid with a 3.1-kb insert that contained the glutamate decarboxylase (gadA) and D-alanine racemase (a
105                            Autoantibodies to glutamate decarboxylase (GADA) are widely used in the pr
106 and autoantibody responses to insulin (IAA), glutamate decarboxylase (GADA), IA-2, IA-2beta, and ZnT8
107 y-negative on the basis of existing markers [glutamate decarboxylase (GADA), protein tyrosine phospha
108 ed at high pH, but only in anaerobiosis, was glutamate decarboxylase (GadA).
109  efficient and most studied uses isozymes of glutamate decarboxylase (GadA/GadB) to consume intracell
110 n the acid challenge media and relies on two glutamate decarboxylases (GadA and B) combined with a pu
111              Not only were the two genes for glutamate decarboxylases (gadA and gadB) and the gene fo
112              As expected, the genes encoding glutamate decarboxylase, gadA and gadB, were significant
113 ly development, and GABA is generated by the glutamate decarboxylase, GadB, during growth and in earl
114                   RipI associates with plant glutamate decarboxylases (GADs) to promote the accumulat
115 om the amino acid glutamate by the action of glutamate decarboxylases (GADs).
116       A high copy number plasmid bearing the glutamate decarboxylase gene (GAD1) increases resistance
117 ow that UNC-30 directly regulates the unc-25/glutamate decarboxylase gene that encodes the enzyme for
118 ing the expression and activity of three tea glutamate decarboxylase genes (CsGAD1, 2, and 3), and th
119 aminase under anaerobiosis; in addition, the glutamate decarboxylase genes gadA and gadB were induced
120 major autoantigens are established (insulin, glutamate decarboxylase, IA2, and zinc transporter-8), b
121                                          All glutamate decarboxylase-immunoreactive (i.e., gamma-amin
122 n the mammalian brain, is synthesized by two glutamate decarboxylase isoforms, GAD65 and GAD67.
123  intracellular consumption of protons by the glutamate decarboxylase isozymes GadA and GadB.
124      However, survival at pH 2 required both glutamate decarboxylase isozymes.
125            Pharmacological block of GlyT2 or glutamate decarboxylase led to rapid and complete rundow
126 e-specific transcription of mouse and cattle glutamate decarboxylase-like protein 1 (GADL1) and the b
127 nd part of the promoter of the gene encoding glutamate decarboxylase-like protein 1 (GADL1) in 94 pat
128 ults suggest that three distinct moieties of glutamate decarboxylase localize to membrane compartment
129 mma-aminobutyric acid-synthesizing enzyme, l-glutamate decarboxylase (MGAD), is regulated by the vesi
130 t acid stress by increasing the synthesis of glutamate decarboxylase, presumably by increasing the le
131                              AGBL1 encodes a glutamate decarboxylase previously identified in serial
132 lt, more glutamate becomes accessible to the glutamate decarboxylase reaction to yield gamma-aminobut
133 subpopulation of neurons; immunostaining for glutamate decarboxylase revealed the responding neurons
134 ansporter GlyT2 and the intracellular enzyme glutamate decarboxylase supply the majority of glycine a
135  showed that this defect is due to decreased glutamate decarboxylase synthesis, probably caused by el
136 21a-AR-Ss, by inserting Shigella glutaminase-glutamate decarboxylase systems coexpressed with S. sonn
137                                              Glutamate decarboxylase, the product of the gadA gene, g
138 amus in the mutant larvae, and expression of glutamate decarboxylase was reduced throughout the brain
139                          Only one of the two glutamate decarboxylases was needed for protection at pH
140 aminobutyric acid (GABA)-synthesizing enzyme glutamate decarboxylase, which is present in inhibitory

 
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