ライフサイエンスコーパス: gluten-free diet

1 way for enzyme therapy as an adjunct to the gluten free diet.

2 al diagnosis may need more time to adjust to gluten free diet.

3 en and in treated CD adults (T-CD, n = 6) on gluten free diet.

4 c alternative other than the observance of a gluten-free diet.

5 lth-related quality of life despite a strict gluten-free diet.

6 ective treatment for this disease is a total gluten-free diet.

7 treated coeliac disease and the effects of a gluten-free diet.

8 The current treatment is a strict gluten-free diet.

9 s or their presenting clinical features on a gluten-free diet.

10 stionnaire while undergoing treatment with a gluten-free diet.

11 rds the development of an alternative to the gluten-free diet.

12 merging in celiac patients unresponsive to a gluten-free diet.

13 pain improved after diagnosis and initiated gluten-free diet.

14 liac disease usually resolve with a lifelong gluten-free diet.

15 estigational therapeutic alternatives to the gluten-free diet.

16 d therapeutic interventions alternative to a gluten-free diet.

17 aseline levels following implementation of a gluten-free diet.

18 rsist or recur despite strict adherence to a gluten-free diet.

19 containing, gluten-free diet vs. a naturally gluten-free diet.

20 n in persons with celiac disease following a gluten-free diet.

21 pation, both of which were responsive to the gluten-free diet.

22 nsive to oral pancreatic enzyme therapy or a gluten-free diet.

23 ded by early diagnosis and compliance with a gluten-free diet.

24 hea in these patients after treatment with a gluten-free diet.

25 776 (1.4%) participants reported following a gluten-free diet.

26 ed disease or those poorly responsive to the gluten-free diet.

27 nd histologic improvements after 1 year of a gluten-free diet.

28 se to gluten and treatment is adherence to a gluten-free diet.

29 et of patients with celiac disease despite a gluten-free diet.

30 able for the condition except for a lifelong gluten-free diet.

31 CI: 1.42, 1.77) were more likely to follow a gluten-free diet.

32 disease is adherence to a strict, lifelong, gluten-free diet.

33 ic changes, despite attempted adherence to a gluten-free diet.

34 racteristics in association with following a gluten-free diet.

35 h care provider diagnosis and adherence to a gluten-free diet.

36 accine in patients with coeliac disease on a gluten-free diet.

37 isorders is based on a lifelong, and strict, gluten-free diet.

38 years who had coeliac disease and were on a gluten-free diet.

39 treatment is strict adherence to a life-long gluten-free diet.

40 l and histological features and adherence to gluten-free diet.

41 ls from patients who had been on a long-term gluten-free diet.

42 t diagnosis and after at least one year on a gluten-free diet.

43 gliadinases may provide an alternative to a gluten-free diet.

44 e aimed to be a potential alternative to the gluten-free diet.

45 tients with CD and monitoring adherence to a gluten-free diet.

46 gnancy each can be reduced by adherence to a gluten-free diet.

47 ass) circulating in CD patients' sera before gluten-free diet.

48 and histologic markers of improvement on the gluten-free diet.

49 th celiac disease who improved with a strict gluten-free diet.

50 nding celiac disease that was managed with a gluten-free diet.

51 40 celiac disease patients after 6 months of gluten-free diet.

52 rexate restored the patients' sensitivity to gluten-free diets.

53 who consume gluten, but not from patients on gluten-free diets.

54 l children develop celiac disease or require gluten-free diets.

55 eir symptoms improve when they are placed on gluten-free diets.

56 th 30 of 52 patients who did not adhere to a gluten-free diet (58%) (P < .0001).

57 according to their subsequent responses to a gluten-free diet, 7 of the 17 who were responsive to glu

58 45 of 148 patients who adhered strictly to a gluten-free diet (98%) had reduced symptoms, compared wi

59 Greater self-reported gluten-free diet adherence significantly predicted a rec

60 Lack of profile differences in gluten-free diet adherence suggests that adjunctive diet

61 and sleep, but not clinical characteristics, gluten-free diet adherence, or QOL.

62 the relationship between those patterns and gluten-free diet adherence, psychiatric symptoms, and va

63 nt may facilitate standardized evaluation of gluten-free diet adherence.

64 on scale, the SF-12, and the Biagi scale for gluten-free diet adherence.

65 ealth; (b) Celiac Dietary Adherence Test for gluten-free diet adherence; (c) PROMIS-29, SF-36, and Ce

66 th celiac disease who no longer responded to gluten-free diets after they developed T-cell LGL, with

67 indings, one year following institution of a gluten free diet, also suggest that ulcerative jejunitis

68 tivity reflecting a strict compliance to the gluten-free diet and a good clinical response to gluten

69 cantly correlated with the low compliance to gluten-free diet and a mild clinical response.

70 odenal histology, comorbidities, response to gluten-free diet and complications were evaluated.

71 While typically managed by gluten-free diet and dapsone, treatment of DH refractory

72 th aspects, circumscribed limitations of the gluten-free diet and diagnostics helping to define celia

73 Clinico-pathological features heal on a gluten-free diet and relapse when gluten is reintroduced

74 ensure strict adherence to treatment with a gluten-free diet and the ensuing clinical and histologic

75 clearly are needed to assess the efficacy of gluten-free diet and to address the underlying mechanism

76 ash resolves when the patient is placed on a gluten-free diet and/or dapsone.

77 t signs and symptoms who do not respond to a gluten-free diet, and for whom no etiology of enteropath

78 s and/or signs of disease activity despite a gluten-free diet, and the more severe forms of refractor

79 ional strategies (elimination of cow's milk, gluten-free diets, and 4-food elimination diet) were als

80 dds to the evidence supporting oat safety in gluten-free diets, and we perform mapping-by-sequencing

81 of the current standard of care, which is a gluten-free diet; and that we lack objective markers of

82 ogenesis, novel therapies alternative to the gluten-free diet are currently in advanced phase of deve

83 Gluten-free diets are now readily achievable with approp

84 collected before and after treatment with a gluten-free diet as well as biopsies from patients with

85 with celiac disease (35 untreated and 5 on a gluten-free diet) as well as 18 subjects with confirmed

86 5 patients with treated celiac disease (on a gluten-free diet), as well as 43 individuals without cel

87 ive serologic findings found initiation of a gluten-free diet associated with small improvement in ga

88 Patients' symptoms improved on a gluten-free diet at 12- to 18-month follow-up.

89 ies from individuals with CeD on a long-term gluten-free diet before and after a 6-week gluten challe

90 ectable in biopsy samples from patients on a gluten-free diet but expanded rapidly and specifically a

91 oeliac patients that initially improved on a gluten-free diet but then relapsed despite a strict diet

92 fore, AN-PEP should not be used to replace a gluten free diet, but rather to support digestion of occ

93 After treatment of celiac sprue with a gluten-free diet, chronic diarrhea persists in a substan

94 Gluten-free diet, claimed to be helpful in treating the

95 celiac disease in the context of an everyday gluten-free diet containing daily up to 2 g gluten.

96 Many patients following a gluten-free diet continue to have symptoms and have smal

97 coeliac patients who normally responded to a gluten-free diet (controls) were collected among 11 Ital

98 A gluten-free diet currently remains the only treatment op

99 n gastrointestinal symptoms compared with no gluten-free diet (difference less than 1 point on a scal

100 Although both conditions are treated with a gluten-free diet, distinguishing between celiac disease

101 However, compliance with the gluten-free diet, especially in the United States, is ex

102 In celiac patients on a gluten-free diet, exposure to gluten induces the appeara

103 enetic variants were associated with being a gluten-free diet follower.

104 better cardiovascular risk profile than non-gluten-free diet followers but poorer self-reported heal

105 Gluten-free diet followers have a better cardiovascular

106 Gluten-free diet followers were more likely to be women,

107 The number of gluten-free diet followers without celiac disease (CD) i

108 gluten challenge, participants adherent to a gluten-free diet for 12 months or more with normal or ne

109 disease is based on a positive response to a gluten-free diet for a limited period, followed by the r

110 bers of individuals are empirically trying a gluten-free diet for a variety of signs and symptoms.

111 Participants reported following a gluten-free diet for at least 1 year before the study be

112 nosis of coeliac disease, and adherence to a gluten-free diet for at least 12 months before screening

113 c sprue (59 women and 19 men) treated with a gluten-free diet for at least 12 months were surveyed ab

114 ctive to or replacing a burdensome life-long gluten-free diet for coeliac disease.

115 years old; 17 male), who had been on strict gluten-free diets for at least 3 months, were given a 3-

116 lenge of 59 individuals on a self-instituted gluten-free diet, for whom celiac disease had been exclu

117 evices for an efficient, simple and accurate gluten free diet (GFD) monitoring as well as therapy fol

118 m the latter two groups after treatment with gluten free diet (GFD) or exclusive enteral nutrition (E

119 Adherence to a gluten-free diet (GFD) and food insecurity (FI) may infl

120 opsy-proven CD who were being treated with a gluten-free diet (GFD) for > or =2 y.

121 A strict gluten-free diet (GFD) for life is the only treatment fo

122 actively sought as alternative therapy to a gluten-free diet (GFD) for patients with coeliac disease

123 es (a low-FODMAP diet) in combination with a gluten-free diet (GFD) has been proposed to reduce the s

124 illous atrophy despite strict adherence to a gluten-free diet (GFD) have non-responsive celiac diseas

125 To assess the effectiveness of a Gluten-Free Diet (GFD) in seven adult female screening-d

126 objective evidence regarding the impact of a Gluten-Free Diet (GFD) in the large proportion of asympt

127 ess patients' knowledge about gluten and the gluten-free diet (GFD) is lacking.

128 A gluten-free diet (GFD) is the only effective treatment f

129 A gluten-free diet (GFD) is the only effective treatment f

130 deficiencies, while individuals following a gluten-free diet (GFD) may lack essential nutrients.

131 The treatment of celiac disease (CeD) with gluten-free diet (GFD) normalizes gut inflammation and d

132 A symptomatic response to a gluten-free diet (GFD) occurred in between 7% and 93% of

133 ing married and having longer adherence to a gluten-free diet (GFD) were independently associated wit

134 growing number of individuals adhering to a gluten-free diet (GFD) without exclusion of celiac disea

135 The only effective treatment is a gluten-free diet (GFD), but adherence can be challenging

136 tients with celiac disease should maintain a gluten-free diet (GFD), excluding wheat, rye, and barley

137 Six DH patients were studied while on a gluten-free diet (GFD), whereas four continued on a norm

138 se symptoms and prevent complications is the gluten-free diet (GFD), which is challenging to maintain

139 symptoms are common despite treatment with a gluten-free diet (GFD), yet no approved or proven nondie

140 an impressive increase in popularity of the gluten-free diet (GFD)-now the most trendy alimentary ha

141 esult of disease and/or its treatment with a gluten-free diet (GFD).

142 er active celiac disease (ACD) or those on a gluten-free diet (GFD).

143 s a confirmatory test before initiation of a gluten-free diet (GFD).

144 and the only available treatment is a strict gluten-free diet (GFD).

145 (CeD) is inaccurate in patients consuming a gluten-free diet (GFD).

146 eatment of celiac disease (CD) is a lifelong gluten-free diet (GFD).

147 ease patients both at diagnosis and while on gluten-free diet (GFD).

148 ain treatment for Celiac Disease (CD) is the gluten-free diet (GFD).

149 isease (CD) at diagnosis before starting the Gluten-free diet (GFD).

150 coming a fraction of individuals following a gluten-free diet (GFD).

151 tment for celiac disease (CeD) is a lifelong gluten-free diet (GFD).

152 y are commonly used to monitor patients on a gluten-free diet (GFD).

153 h endomysial antibodies (EmA) benefit from a gluten-free diet (GFD).

154 bowel syndrome (IBS-D) could benefit from a gluten-free diet (GFD).

155 ease in individuals who have self-prescribed gluten-free diets (GFD) or have atypical presentations.

156 and nutrient absorption to the level of the gluten-free diet group.

157 Difficulties adhering to a gluten-free diet have led to the development of non-diet

158 Poor adherence to a gluten-free diet, HLA-DQ2 homozygosity, and late diagnos

159 mins, that has been recommended as part of a gluten-free diet; however, few experimental data support

160 he main therapeutic intervention for CD is a gluten-free diet; however, novel nondietary agents are u

161 Treatment involves a gluten-free diet; however, owing to the high psychosocia

162 ent for coeliac disease is a lifelong strict gluten-free diet; however, the diet is restrictive and g

163 Introduction of a gluten-free diet improves coeliac patients' quality of l

164 Initiated treatment with a gluten-free diet improves the situation for celiac disea

165 as a therapy for MS that is analogous to the gluten-free diet in coeliac disease.

166 Studies of gluten-free diet in patients with gluten sensitivity and

167 ore, it allowed monitoring of adherence to a gluten-free diet in patients.

168 mptoms before and after the institution of a gluten-free diet in the patients, all of whom had been g

169 ciated with prevalence of celiac disease and gluten-free diet in the United States.

170 in antibodies of IgG class after 6 months of gluten-free diet; in contrast, 16/40 (40%) of celiac pat

171 isorders, and they should remain on a strict gluten-free diet indefinitely.

172 tion of clinical manifestations follows if a gluten-free diet is adopted.

173 Although a gluten-free diet is an effective treatment in most indiv

174 A gluten-free diet is an effective treatment, but complian

175 In addition, a gluten-free diet is highly burdensome and can profoundly

176 ening for celiac disease must occur before a gluten-free diet is implemented, since once a patient in

177 nal histology before and after introducing a gluten-free diet is not yet established.

178 Although a strict gluten-free diet is the current treatment for celiac dis

179 A gluten-free diet is the mainstay of safe and effective t

180 A gluten-free diet is the only available treatment for cel

181 A gluten-free diet is the only means to manage coeliac dis

182 A strict, lifelong gluten-free diet is the only treatment.

183 A strict gluten-free diet is the sole cure for these ailments.

184 up children with CD after prescription of a gluten-free diet is unclear.

185 med to investigate the effects of low FODMAP-gluten free diet (LF-GFD) on clinical symptoms, intestin

186 Despite adherence to a gluten-free diet, many patients remain symptomatic and s

187 th poor prognosis, but strict adherence to a gluten-free diet may prevent its occurrence.

188 ure, omega-3 fatty acid supplementation, and gluten-free diet, may have additional benefits, as do po

189 New approaches to making the gluten-free diet more palatable are being studied.

190 After diagnosis, a strict gluten-free diet must be followed, which in most cases w

191 A gluten-free diet normalizes liver enzymes and histologic

192 zes a review of scientific studies using the gluten-free diet, nutritional risk factors, controversia

193 s designed to characterize the effect of the gluten-free diet on anti-gliadin antibodies of IgG class

194 Our aim was to investigate the effect of a gluten-free diet on celiac disease related symptoms, hea

195 We examined the effect of a gluten-free diet on gastrointestinal symptoms in a cohor

196 ed celiac disease should immediately start a gluten-free diet or be monitored on their regular diet.

197 n = 81; villous atrophy despite a adhering a gluten-free diet), patients with untreated CeD (n = 82)

198 With a gluten-free diet, patients have substantial and rapid im

199 ive CeD compared with remission disease from gluten-free diets, predominantly in lamina propria mesen

200 d that most patients with IBS improve with a gluten-free diet, randomized controlled trials have yiel

201 port screening based on high compliance to a gluten-free diet, rapid progression from symptom-free co

202 Loss of response to a gluten-free diet (refractory sprue) and ulcerative jejun

203 Treatment options other than the restrictive gluten-free diet remain limited.

204 Adherence to a gluten-free diet remains paramount as the recognition of

205 of coeliac disease, early introduction of a gluten-free diet remains the cornerstone of treatment.

206 in our understanding of celiac disease, the gluten-free diet remains the only current viable therapy

207 Strict adherence to a gluten-free diet remains the only way to prevent EATL.

208 le achieving negative serology with a strict gluten-free diet should be the goal.

209 scopy, 1 year and 4 years following a strict gluten free diet showed endoscopic and histological norm

210 CD diagnosis and the introduction of a gluten-free diet substantially improved the quality of l

211 mplemented, since once a patient initiates a gluten-free diet, testing for celiac disease is no longe

212 Because the symptoms of CS respond to a gluten-free diet, testing for CS in IBS may prevent year

213 Therapy is a gluten-free diet that requires avoidance of wheat, rye,

214 Despite his compliance to the gluten-free diet the symptoms persisted and we eventuall

215 Despite adhering to a gluten-free diet (the only management option available t

216 ter COVID-19 can impact on CeD treatment and gluten-free dieting, the only available therapy for CeD.

217 o endoscopic evaluation after 1-3 years on a gluten-free diet to evaluate improvements in villous atr

218 red for screening for gluten sensitivity and gluten-free diet trials.

219 disease that do not improve despite a strict gluten-free diet (type A cases) and previously diagnosed

220 On a gluten-free diet, visits to primary care decreased signi

221 s choosing a strict wheat starch-containing, gluten-free diet vs. a naturally gluten-free diet.

222 Reasons for following a gluten-free diet warrant further investigation.

223 alth-related characteristics and following a gluten-free diet was investigated in 124,447 women and m

224 wide association study (GWAS) of following a gluten-free diet was performed.

225 peutic approaches beyond the lifelong strict gluten-free diet, which poses a burden to patients and o

226 ct is particularly important for people on a gluten-free diet who often represent mineral deficiencie

227 ring phenomenon with the administration of a gluten-free diet with or without dapsone were observed.

228 : A man in his 80s with DH not controlled by gluten-free diet (with poor adherence), dapsone, and con

229 celiac disease was defined as adherence to a gluten-free diet without a diagnosis of celiac disease.

230 in diagnosis to be made in the context of a gluten-free diet without intestinal biopsy.