戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 BALB/c mice with 1-O-octadecyl-2-O-benzyl-sn-glyceryl-3-phospho-RVn (60 mg/kg orally, once daily for
2         By tracking the kinetics of [(13)C(3)glyceryl] and [(13)C(2)acyl] labeling, we observed two d
3 which is specifically esterified to the sn-2 glyceryl carbon of phosphatidylglycerol.
4 doglycan and the lipopeptide tri-palmitoyl-S-glyceryl-Cys-Ser-(Lys)(4) that occurred earlier and was
5 conjugated to the TLR2 agonist dipalmitoyl-S-glyceryl cysteine stimulated CD4 T lymphocyte IFN-gamma
6 polysaccharide, Poly[I:C], or tripalmitoyl-S-glyceryl cysteine) in the presence or absence of epineph
7 osinic:polycytidylic acid, or tripalmitoyl-S-glyceryl cysteine-induced reductions in transendothelial
8 embrane signal transducer for tripalmitoyl-S-glyceryl-cysteine (Pam(3)Cys)-modified lipoproteins, inc
9  recombinant OspA or OspB, or tripalmitoyl-S-glyceryl-cysteine had little or no effect on expression
10 e stability of DNA duplexes bearing isolated glyceryl cytosine residues has also been investigated.
11 ppended with templates incorporating atactic glyceryl cytosine residues.
12                                      Atactic glyceryl cytosine units are found to impede, but not to
13  that the C20BAS derivative has a short sn-1 glyceryl diether C20H40 transmembrane alkyl chain and tw
14 gether with estimates of endogenous [(12)C(3)glyceryl] dilution, showed that each biosynthetically ac
15  specifically catalyzes the formation of 1,2-glyceryl dinitrate and nitrite from GTN, leading to prod
16 se 2 (mtALDH) catalyzes the formation of 1,2-glyceryl dinitrate and nitrite from nitroglycerin (glyce
17  thionitrate intermediate and release of 1,2-glyceryl dinitrate.
18 2(S)-Hydroxyeicosa-5,8,10,14-tetraenoic acid glyceryl ester (12-HETE-G) was produced following additi
19 r release of 15-hydroxyeicosatetraenoic acid glyceryl ester (15-HETE-G), establishing that lipoxygena
20 , to quantitatively analyze changes in those glyceryl ester molecular species containing identical fa
21 poxygenase (12-LOX) oxygenated 2-AG to the 2-glyceryl ester of 12(S)-hydroperoxyeicosa-5,8,10,14-tetr
22                           We report that the glyceryl ester of PGE(2), PGE(2)-G, triggers rapid, conc
23                                    Thus, the glyceryl ester of prostaglandin E2, PGE2-G, mobilizes Ca
24 iding 15(S)-hydroperoxyeicosatetraenoic acid glyceryl ester.
25 thod for the quantification of prostaglandin glyceryl esters (PG-Gs), a newly discovered class of eic
26 ), to prostaglandins (PGs) and prostaglandin glyceryl esters (PG-Gs), respectively.
27 caused release of 2-AG and production of the glyceryl esters of PGE2 and PGI2 over 60 min.
28       Its turnover leads to the formation of glyceryl esters of prostaglandins (PG-Gs), a subset of w
29 thanolamide to prostaglandins, prostaglandin glyceryl esters, and prostaglandin ethanolamides, respec
30 cs include the following: lytic reactions of glyceryl esters, including hydrolysis, aminolysis, and t
31 scribe the synthesis and evaluation of alkyl glyceryl ether phosphodiesters of GS-441524 (RVn), lysop
32                    The compound DAGE (DiAcyl Glyceryl Ether, 1-stearyl-2,3-dioleoyl glycerol), presen
33  stereoselective formation of 1'-(Z)-alkenyl glyceryl ethers.
34 g, UDP from a hydrolyzed donor, and an alpha-glyceryl-GlcNAc product formed in situ.
35 ece, an enantiopure isopropylidene-protected glyceryl glycidyl ether diastereomer, was accomplished i
36 nce) involving an enantio- and diastereopure glyceryl glycidyl ether key C6-building blocks from whic
37  caryophilli PG2982, was observed to utilize glyceryl glyphosate as a sole phosphorus source.
38 that hydrolyzes phosphonate esters including glyceryl glyphosate to glyphosate and glycerol was clone
39 hytotoxicity resulted from the hydrolysis of glyceryl glyphosate to glyphosate and subsequent inhibit
40                            The hydrolysis of glyceryl glyphosate to glyphosate by a phosphonate ester
41 hereas its phosphonate monoester derivative, glyceryl glyphosate, is approximately 50-fold less activ
42  the pehA gene were killed when treated with glyceryl glyphosate.
43 promoter and treating the mature plants with glyceryl glyphosate.
44                                     [(13)C(3)glyceryl] labeling of DAG and PC, together with estimate
45       The use of solid support bearing diDMT-glyceryl-linker permitted convenient synthesis of immuno
46                        Vitamin C donates the glyceryl moiety to 5mC with concurrent formation of glyo
47  acyl transfer from the sn-2 position of the glyceryl moiety to the amino group on the N-terminal cys
48 homologue and catalyses the conjugation of a glyceryl moiety to the methyl group of 5mC through a car
49 cenoic acid in the sn-2 position of its core glyceryl moiety.
50 rger (approximately 10-fold) than in neutral glyceryl monooleate (GMO) membranes.
51                                 Particularly glyceryl monooleate, in association with phosphatidylcho
52 ediated dilatation/Deltadilation response to glyceryl nitrate (95% CI 0.13-2.11).
53 pain, nonpungent analogs such as olvanil and glyceryl nonivamide (GLNVA) were synthesized to enhance
54          Evidence suggests that both D and L glyceryl nucleoside configurations at a given template p
55 ned sunflower oils, which 1% w/w addition of glyceryl oleate was determined as the optimal.
56 ne or more alkyl- (C2-C12), branched alkyl- (glyceryl or aminobutyryl-propanediol), or ethyleneglycol
57  (GGGP synthase) catalyzes alkylation of (S)-glyceryl phosphate [(S)-GP] by geranylgeranyl diphosphat
58 aining lysolecithin (LL), monoolein (MG), or glyceryl polyethyleneglycol ricinoleate (SYN), or their
59 glyceryl prostaglandins, some of which (e.g. glyceryl prostaglandin E(2) and glyceryl prostaglandin I
60  which (e.g. glyceryl prostaglandin E(2) and glyceryl prostaglandin I(2)) exhibit interesting biologi
61                      The sterically hindered glyceryl prostaglandin was the poorest substrate examine
62                                              Glyceryl prostaglandins (PG-Gs) are generated by the oxy
63 gical activities of 2-arachidonylglycerol or glyceryl prostaglandins in humans.
64 of COX-2 oxygenation of endocannabinoids are glyceryl prostaglandins, some of which (e.g. glyceryl pr
65 monstrated by analysis of prostaglandins and glyceryl prostaglandins.
66 ends of medium chain triglyceride (MCT) oil, glyceryl stearate (GS) or hydrogenated palm oil (HPO) as
67          Oleogels containing 0.1% betaC, 20% glyceryl stearate (GS), with lecithin (L) or hydrogenate
68 erred to acyl groups plus fatty acids, or to glyceryl structures.
69  dietary lipid tracer-stable isotope-labeled glyceryl tri(oleate-1,2,3,7,8-(13)C(5)) ((13)C(15)-TO)-w
70 ar rats by the administration of cerulein or glyceryl tri-linoleate (GTL).
71                                              Glyceryl tri-oleate but not OAEE resulted in 70% mortali
72                The triglyceride of OA (i.e., glyceryl tri-oleate) or OAEE was injected into the pancr
73 , by increasing systemic acetate levels with glyceryl triacetate supplementation, we do not observe a
74  disperse nanoparticles of PGT (propoxylated glyceryl triacylate) that contain a glaucoma drug timolo
75                                              Glyceryl tributyrate was used for the in situ selective
76 by intraductal injection of the triglyceride glyceryl trilinoleate alone or with the chemically disti
77 necrosis milieu was simulated in vitro using glyceryl trilinoleate because linoleic acid is increased
78 e acid requirements to effectively hydrolyze glyceryl trilinoleate were studied using exogenous or ov
79                                  Intraductal glyceryl trilinoleate with or without the lipase inhibit
80 ydroxytoluene (BHT) on the biodegradation of glyceryl trilinoleate, a model vegetable oil highly susc
81           Both lipase inhibitors reduced the glyceryl trilinoleate-induced increase in serum lipase,
82 observed for the silica-induced pyrolysis of glyceryl trimyristate and real waste, rapeseed meals.
83 onduit artery selectivity similar to that of glyceryl trinitrate (0.013-4.4 nmol/min) over resistance
84 f albuterol (5 microg/min, DeltaRI(ALB)) and glyceryl trinitrate (5 microg/min, DeltaRI(GTN)).
85 ependent dilation, EDD) and after sublingual glyceryl trinitrate (causing endothelium-independent dil
86 .01% (P=0.036) with no change in response to glyceryl trinitrate (endothelial-independent dilatation)
87 ers of each sex, we assessed FMD response to glyceryl trinitrate (GTN) at baseline and at 8 hours and
88                                 The NO donor glyceryl trinitrate (GTN) caused an increase in cGMP in
89 donor drugs isosorbide mononitrate (ISMN) or glyceryl trinitrate (GTN) for stable angina.
90                  Nitric oxide donors such as glyceryl trinitrate (GTN) have been shown to cause a rev
91 n each group, a migraine was induced using a glyceryl trinitrate (GTN) infusion.
92                                  Infusion of glyceryl trinitrate (GTN) into patients with migraine pr
93     Therefore, we investigated the effect of glyceryl trinitrate (GTN) on the efficacy of cell engraf
94                                 The NO donor glyceryl trinitrate (GTN) provokes delayed migraine atta
95                   Albuterol (salbutamol) and glyceryl trinitrate (GTN) were administered locally by b
96                               The effects of glyceryl trinitrate (GTN), amlodipine, and atenolol were
97                                        Using glyceryl trinitrate (GTN), an agonist of nitric oxide (N
98 ing pacing and after intragraft injection of glyceryl trinitrate (GTN).
99 h after infusing the NO donor nitroglycerin [glyceryl trinitrate (GTN)] to migraineurs.
100 ent) and response to 50 microg of sublingual glyceryl trinitrate (GTN, endothelium independent) were
101 abolism of the cornerstone nitrovasodilator, glyceryl trinitrate (GTN; 0.1-100 mg/kg), in the rat in
102                                      We used glyceryl trinitrate (nitroglycerin) to trigger premonito
103 educed responses to bradykinin (P=0.005) and glyceryl trinitrate (P=0.006).
104 achidonic acid: 0.2, 2, and 20 nmol/min; and glyceryl trinitrate 1, 2, and 4 pmol/min) were construct
105 mediated dilatation [FMD]) and -independent (glyceryl trinitrate [GTN]) changes in brachial artery ar
106 yl dinitrate and nitrite from nitroglycerin (glyceryl trinitrate [GTN]) within mitochondria, leading
107  The metabolism of nitrovasodilators such as glyceryl trinitrate and nitroprusside provides the activ
108 P=0.045) after adjusting for the response to glyceryl trinitrate and was independent of conventional
109     In addition, exogenous acetylcholine and glyceryl trinitrate both increase arterial distensibilit
110    There was no significant change in FMD or glyceryl trinitrate during the placebo phase.
111    Sublingual administration of the NO donor glyceryl trinitrate normalized platelet VASP phosphoryla
112 rently available nitric oxide donors such as glyceryl trinitrate or isosorbide mononitrate could be u
113  been short-term; a new topical, easy-to-use glyceryl trinitrate preparation has been shown to improv
114 inistration of the nitric oxide (NO) mimetic glyceryl trinitrate prevented altered uteroplacental per
115                Infusion of acetylcholine and glyceryl trinitrate reduced PWV significantly, by 6+/-4%
116                      Responses to sublingual glyceryl trinitrate were unchanged.
117 , D-S-nitrosocysteine, S-nitrosoglutathione, glyceryl trinitrate, and sodium nitroprusside produced m
118 ular mechanisms through which nitroglycerin (glyceryl trinitrate, GTN) elicits nitric oxide (NO)-base
119 atalyzes the bioactivation of nitroglycerin (glyceryl trinitrate, GTN) in blood vessels, resulting in
120                               Nitroglycerin (glyceryl trinitrate, GTN), originally manufactured by Al
121 thysmography with acetylcholine, bradykinin, glyceryl trinitrate, norepinephrine, and l-NG-monomethyl
122                         In in vitro studies, glyceryl trinitrate, sodium nitroprusside, and 3'-morpho
123 ts, intravenous infusion of three NO donors, glyceryl trinitrate, sodium nitroprusside, or 3'-morphol
124 ry flow-mediated (endothelium-dependent) and glyceryl trinitrate-induced (endothelium-independent) di
125                                 In contrast, glyceryl trinitrate-induced dilatation was similar in th
126 15) and lower FMD percentage (P = 0.026) and glyceryl trinitrate-induced dilation (P = 0.012) than he
127  pattern was identical in migraineurs during glyceryl trinitrate-induced migraine attacks as well as
128 ix control subjects at rest and during acute glyceryl trinitrate-induced migraine attacks using posit
129 ins tight for dihydroergotamine during acute glyceryl trinitrate-induced migraine attacks.
130 pothalamus, seen in the premonitory phase of glyceryl trinitrate-triggered migraine attacks can expla
131 ere was no significant change in response to glyceryl trinitrate.
132 sting or reducing anginal episodes or use of glyceryl trinitrate.
133 (BK) and the endothelium-independent dilator glyceryl-trinitrate (GTN).
134 harged on the same day with diclofenac, 0.2% glyceryl-trinitrate ointment, lactulose, a telephone num
135 id to 9,10-chlorohydrins in chlorine-treated glyceryl trioleate and produce was much lower, reflectin
136 eeding the fly mutant one of the components, glyceryl trioleate oil, blocked the accumulation of exce
137 wed higher affinity towards soybean oil than glyceryl-trioleate.
138 rmed from the model compounds palmitic acid, glyceryl tripalmitate and 1,3-dipalmitoyl-2-oleoylglycer

 
Page Top