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1 ntal as well as supraspinal, are exclusively glycinergic.
2  and receive synapses that are predominantly glycinergic.
3 eir fate but is downregulated in the related glycinergic AC subtype.
4 holine acetyltransferase (ChAT)-positive and glycinergic AC subtypes were unaffected.
5 n properties to these morphological types of glycinergic ACs in the mouse retina.
6 /medium-field GABAergic ACs and narrow-field glycinergic ACs, which mediate lateral and vertical inte
7 oked excitation are initially facilitated by glycinergic activity due, at least in part, to glycinerg
8  and female) to target expression of ChR2 to glycinergic afferents in the ICC and made whole-cell rec
9 lycine feedback to the bipolar cell and this glycinergic amacrine cell is suppressed by GABAergic ama
10 ct glycinergic inhibition, indicating that a glycinergic amacrine cell, most likely the AII amacrine
11                                 We find that glycinergic amacrine cells innervating RBCs receive exci
12 de of low-frequency signals was regulated by glycinergic amacrine cells, while GABAergic inhibition r
13 nas, and it is present in both GABAergic and glycinergic amacrine cells.
14 inhibitory connections between GABAergic and glycinergic amacrine cells.
15 expanded by gap junction connections between glycinergic amacrine cells.
16 tinal precursors toward the fates of non-AII glycinergic amacrine, type 2 OFF-cone bipolar and horizo
17 essary and sufficient for specifying non-AII glycinergic amacrine, type 2 OFF-cone bipolar and horizo
18 nes are generated on average 2-3 days before glycinergic amacrines.
19                In total 14 glutamatergic, 22 glycinergic and 2 GABAergic/glycinergic interneurons wer
20                   A significant reduction of glycinergic and GABAergic ACs with a substantial increas
21 ge recurrence depends on opposing effects of glycinergic and GABAergic inhibition and can be explaine
22 microM) evoked increases in the frequency of glycinergic and GABAergic miniature inhibitory postsynap
23 , beta-methylene ATP (100 microM), increased glycinergic and GABAergic mIPSC neurotransmission to car
24 edulla, retrogradely-labeled, glutamatergic, glycinergic and GABAergic neurons were found in the vent
25 e 5'-triphosphate (ATP) modulated inhibitory glycinergic and GABAergic neurotransmission to cardiac v
26 ing protein gephyrin to mammalian inhibitory glycinergic and GABAergic postsynaptic membranes in nerv
27                      Terminal projections of glycinergic and glutamatergic cells were found within mo
28                                              Glycinergic and glutamatergic flocculus target neurons (
29     This correlated with LSO mRNA levels for glycinergic and glutamatergic ionotropic receptor subuni
30 n this paper, we show that in zebrafish most glycinergic and many GABAergic spinal interneurons expre
31 2-expressing neurons were non-GABAergic, non-glycinergic and predominantly catecholaminergic (54%).
32 eously knocked-down, many neurons lose their glycinergic and/or GABAergic characteristics, but they d
33 clerosis mouse model, we measured GABAergic, glycinergic, and cholinergic immunoreactive terminals on
34 nsferase were used as markers for GABAergic, glycinergic, and cholinergic terminals, respectively.
35 the two main amacrine subsets--GABAergic and glycinergic--and further subdivided these groups into sm
36        In vagotomized rats, GABA(A)ergic and glycinergic antagonists had little, if any, effect on rh
37 , we found that application of GABAergic and glycinergic antagonists in the BotC caused opposing effe
38                          Cartwheel cells are glycinergic auditory interneurons which fire Na(+)- and
39 cells include selected rhythmogenic neurons (glycinergic Botzinger neurons) but not RTN chemoreceptor
40 a heterogeneous collection of E-AUG neurons (glycinergic Botzinger neurons, possibly facial motor and
41  it was clear that a significant fraction of glycinergic boutons corelease GABA in the ICC.
42  show that microglia control the strength of glycinergic but not GABAergic synapses via modulation of
43 ng beta1 receptors prevented the increase in glycinergic, but not GABAergic, IPSCs in CVNs.
44 ndings suggest that the alpha3 GlyRs mediate glycinergic cannabinoid-induced suppression of chronic p
45 s known about the potential and mechanism of glycinergic cannabinoids for chronic pain treatment.
46                                          The glycinergic cell populations in the brain of the lesser
47 iological tools, we revealed a population of glycinergic cells in the VCN distinct from the D-stellat
48                                           No glycinergic cells were observed in the cerebellum.
49                             These multipolar glycinergic cells were smaller in soma size and dendriti
50 35.5% were GABA negative, and most contacted glycinergic cells.
51      The cell-type-specific targeting of the glycinergic circuits further supports the proposed physi
52                 The results demonstrate that glycinergic circuits within the retina can produce sacca
53 ncluding subsets of cells in cholinergic and glycinergic circuits.
54                          The slow and potent glycinergic component dominates the inhibitory conductan
55 ry phenotype, including the slow kinetics of glycinergic components.
56                            Importantly, this glycinergic conductance is greatly enhanced in a strychn
57 ptic silencing and the strengthening of GABA/glycinergic connections that normally occur with maturat
58  at the lamina II/III border are under tonic glycinergic control and receive synapses that are predom
59       Unlike OFF BSGCs, which receive strong glycinergic crossover inhibition from the ON pathway, th
60 tically released glycine also enhanced tonic glycinergic currents and resulted in decreased parvalbum
61 ree of four patients also profoundly reduced glycinergic currents compared with control Fab-IgG.
62 nterneurons, showing that synaptic and tonic glycinergic currents dominate, blocking neuronal or glia
63 auditory brainstem, coreleased GABAergic and glycinergic currents in the midbrain are strikingly simi
64 erved a significant reduction in spontaneous glycinergic currents induced in spinal motor neurons.
65 eased excitability was replicated when tonic glycinergic currents were increased by electrically acti
66 trast, we observed substantial reductions in glycinergic currents with all four patient IgG preparati
67 or glial glycine transporters enhances tonic glycinergic currents, and these manipulations reduce exc
68 g receptors underlie both synaptic and tonic glycinergic currents.
69  reveal a potential biophysical mechanism of glycinergic dis-inhibition and suggest that post-transla
70  Despite the importance of this mechanism of glycinergic dis-inhibition associated with dysfunctional
71 ycinergic activity due, at least in part, to glycinergic disinhibition of GAD67 cells.
72 Cs receive additional excitation mediated by glycinergic disinhibition.
73                                          The glycinergic enhancement of neurotransmitter release in e
74 ession of sst2a with particular reference to glycinergic/expiratory neurons in the Botzinger Complex
75 annels was demonstrated by administration of glycinergic factors.
76                                              Glycinergic fastigial neurons make functional projection
77 cate that a diversity of mechanisms underlie glycinergic feedback inhibition onto RBCs, yet they high
78                                              Glycinergic feedback signaling depends strongly, althoug
79                  We found that GABAergic and glycinergic fibers ascending from the pontine reticular
80 ed to target ChR2 expression specifically to glycinergic fibers ascending from the VNLL, allowing for
81 hole-cell recordings in vitro while exciting glycinergic fibers with light.
82 ct subpopulations of pre-BotC glutamatergic, glycinergic, GABAergic, and glycine-GABA coexpressing in
83                We additionally observed that glycinergic/GABAergic activity in NA was usually depolar
84               We find that the vIRt contains glycinergic/GABAergic cells that rhythmically inhibit vi
85 ectively suppresses excitatory synapses, but glycinergic/GABAergic inputs onto CWCs are not affected.
86 ough descending projections to motor-related glycinergic/GABAergic neurons in the spinal cord and ven
87 t to have fast kinetics and be predominantly glycinergic in mammals.
88 or olive (LSO) in the auditory brainstem are glycinergic in maturity, but also GABAergic and glutamat
89                                              Glycinergic inhibition also varied across BC class.
90 macrine cells by pharmacologically isolating glycinergic inhibition and evoking feedback IPSCs in a s
91 ich triggers postsynaptic changes leading to glycinergic inhibition and only then is balanced excitat
92        That such local circuits are gated by glycinergic inhibition and that A- and C-fibers low thre
93  we show that both phasic and tonic forms of glycinergic inhibition are mediated by heteromeric alpha
94            We further show that the onset of glycinergic inhibition depends upon the maturation of C-
95            In an auditory brainstem nucleus, glycinergic inhibition features fast decay kinetics, the
96 ther highlighted by the presence of abnormal glycinergic inhibition in many pathophysiological states
97             We show an absence of functional glycinergic inhibition in newborn dorsal horn circuits:
98  model of ALS allow the detection of altered glycinergic inhibition in spinal microcircuits.
99 us solitarius and caudal VLM) unmasked tonic glycinergic inhibition in the RVLM.
100    We compare and discuss possible roles for glycinergic inhibition in the two cell types.
101 ulation, excitation is suppressed and direct glycinergic inhibition is increased in HS-GCs, whereas f
102  of increased synaptic activity in the RVLM, glycinergic inhibition is recruited to strengthen sympat
103                     We furthermore show that glycinergic inhibition may be an important contributor t
104 /or disinhibition of the CVLM unmasked tonic glycinergic inhibition of the RVLM.
105 transporter, regulates the strength of tonic glycinergic inhibition of these glycine-dominant neurons
106 , whereas the efficacy of both GABAergic and glycinergic inhibition onto the same population was comp
107                   Our findings indicate that glycinergic inhibition provides critical control of exci
108 her these data suggest both phasic and tonic glycinergic inhibition regulate the output of PV+ INs an
109                       Futhermore, this tonic glycinergic inhibition remains strong as the mice mature
110 d GABAergic inhibition to 53%; and increased glycinergic inhibition to 47%.
111 urons, glutamatergic drive was reduced while glycinergic inhibition was potentiated.
112 y lamina II interneurons while GABAergic and glycinergic inhibition were reduced.
113 eus, the bushy and T-stellate cells, receive glycinergic inhibition with different synaptic conductan
114               Dim light evoked GABAergic and glycinergic inhibition with rapid kinetics and a large s
115 tion from ON to OFF pathways required intact glycinergic inhibition, indicating that a glycinergic am
116 ays the maturation of both GlyR subunits and glycinergic inhibition, maintaining dorsal neurons in a
117 ion of GABAergic inhibition and emergence of glycinergic inhibition.
118                                              Glycinergic inhibitory control emerges in the second pos
119 plexia could potentially result from reduced glycinergic inhibitory efficacy.
120 monstrating an essential role for SLC7A10 in glycinergic inhibitory function in the central nervous s
121 dexmedetomidine decreases both GABAergic and glycinergic inhibitory input to cardiac vagal neurons, w
122                                              Glycinergic inhibitory inputs to the MSO, which tune the
123 , a large and diverse group of GABAergic and glycinergic inhibitory interneurons.
124 at ablating, silencing, or activating spinal glycinergic inhibitory neurons with viral vectors all ha
125             Accumulating evidence shows that glycinergic inhibitory neurotransmission in the spinal c
126 sed by mutations that impair the efficacy of glycinergic inhibitory neurotransmission.
127                                         This glycinergic inhibitory S-cone amacrine cell is ideally p
128 ibution corresponding with high densities of glycinergic inhibitory synapses.
129 ion is because of the selective targeting of glycinergic inhibitory synaptic inputs to ON-DSGCs.
130                                Dysfunctional glycinergic inhibitory transmission underlies the debili
131 atergic (excitatory) processes laterally and glycinergic (inhibitory) processes medially.
132                              Conversely, the glycinergic, inhibitory input properties remained unaffe
133                        Motoneurons lost most glycinergic innervation by 16 weeks of age (end-stage) w
134 ALS mouse model reported a selective loss of glycinergic innervation in cultured MNs, suggestive of a
135               Our previous study showed that glycinergic innervation of spinal motoneurons is deficie
136                                 Furthermore, glycinergic innervation of the SOC is maintained without
137                                 Reduction of glycinergic innervation preceded mitochondrial swelling
138                                    When this glycinergic input is blocked, both cell types respond si
139 hat spinal endogenous NO enhances inhibitory glycinergic input to dorsal horn neurons through sGC-cGM
140  unlike AII cells, A8 amacrine cells provide glycinergic input to ON A-type ganglion cells.
141 tion selective responses without significant glycinergic inputs for mediating monosynaptic crossover
142 ts from VNLL, INLL, or MSO and low-frequency glycinergic inputs from VNLL or INLL.
143 arises through convergence of high-frequency glycinergic inputs from VNLL, INLL, or MSO and low-frequ
144 Instead, facilitation may depend entirely on glycinergic inputs that are presumed to be inhibitory.
145  that this saccadic suppression results from glycinergic inputs that are specific to ON-DSGCs and are
146 neurodegeneration, we analyzed the GABAergic/glycinergic inputs to E17.5 fetal MNs from SOD1(G93A) (S
147                    Large ON pathway-mediated glycinergic inputs to ON and OFF BSGCs also showed surro
148                              We propose that glycinergic inputs tuned to two distinct spectral elemen
149  and depends on low and high frequency-tuned glycinergic inputs.
150 while almost all radial cells also possessed glycinergic inputs.
151 cording to study preBotC EGFP-labeled, i.e., glycinergic, inspiratory-modulated neurons with pacemake
152 inergic regulation of motoneuron function or glycinergic interneuron degeneration contributes to moto
153 eal the activity pattern of four commissural glycinergic interneuron types during escape, swimming an
154  mutant sod1 fish first exhibited the HSR in glycinergic interneurons at 24 hours postfertilization (
155  cells (also called tuberculoventral cells), glycinergic interneurons thought to provide on- or near-
156 lutamatergic, 22 glycinergic and 2 GABAergic/glycinergic interneurons were retrieved.
157 ent terminals, but not that on GABAergic and glycinergic interneurons, in the spinal cord is reduced
158 renergic silencing of spontaneous spiking in glycinergic interneurons.
159                        The results show that glycinergic interplexiform cells activate bipolar cell d
160  effects of the synaptic connections between glycinergic interplexiform cells, photoreceptors and bip
161 t drug, tropisetron, significantly increases glycinergic IPSC decay times without causing motor side
162                 Interestingly, SOD GABAergic/glycinergic IPSCs and evoked GABA(A)R-currents exhibited
163                                 We show that glycinergic IPSCs are present in all cells.
164 yclase abolished the effect of L-arginine on glycinergic IPSCs but not on evoked monosynaptic EPSCs.
165  neurons increased the frequency of isolated glycinergic IPSCs by 27 +/- 8% (p = 0.003, n = 26) and a
166                         We propose that slow glycinergic IPSCs may provide an inhibitory tone, settin
167                                              Glycinergic IPSCs were evoked by threshold stimulation o
168 and NMDA receptor (NMDAR)-mediated EPSCs and glycinergic IPSCs.
169                                 Furthermore, glycinergic ipsilateral vestibular inhibitory inputs are
170 he averaged current densities of spontaneous glycinergic miniature IPSCs (mIPSCs) were significantly
171 -release of GABA and glutamate from immature glycinergic MNTB terminals.
172 of MNTB principal neurons, colocalizing with glycinergic nerve endings to mediate fast, phasic IPSCs.
173 s are facilitated, rather than inhibited, by glycinergic network activity.
174 nction of Pax2/8 in specifying GABAergic and glycinergic neuronal fates is much broader than was prev
175                                GABAergic and glycinergic neurones are known components of these circu
176 ic, gamma-aminobutyric acid (GABA)ergic, and glycinergic neurons (detected in transgenic mice express
177                     This work establishes CN glycinergic neurons as a significant source of inhibitio
178 ry sequences to study for the first time the glycinergic neurons in the brain of an adult teleost.
179 nd this view by showing that inhibitory GABA-glycinergic neurons of the cerebellar nuclei (CN) projec
180 ervated by Purkinje cells, glutamatergic and glycinergic neurons projected to the contralateral and i
181 vely expressing green fluorescent protein in glycinergic neurons to demonstrate that many premotor ou
182 aminobutyric acid (GABA) as their output and glycinergic neurons, along with the pronounced loss of c
183 st of a mix of glutamatergic, GABAergic, and glycinergic neurons, which can drive both excitatory and
184 chondrial proteins was seen in GABAergic and glycinergic neurons; however, cholinergic neurons indica
185         Alterations in GlyT2 activity modify glycinergic neurotransmission and may underlie several n
186 porter GlyT2 plays a fundamental role in the glycinergic neurotransmission by recycling the neurotran
187  to selectively and genetically disrupt GABA/glycinergic neurotransmission from PZ neurons.
188 pies capable of increasing inhibitory spinal glycinergic neurotransmission hold in providing new and
189 aimed to determine the physiological role of glycinergic neurotransmission in baroreflex function, id
190 ne transport that may affect the function of glycinergic neurotransmission in vivo.
191                                              Glycinergic neurotransmission is a major inhibitory infl
192    In the vertebrate central nervous system, glycinergic neurotransmission is regulated by the action
193 e describe the effect of genetic deletion of glycinergic neurotransmission on single MN structure and
194                   Disruption of PZ GABAergic/glycinergic neurotransmission resulted in sustained incr
195  modified after pharmacological reduction of glycinergic neurotransmission such that embryos produced
196    CIHH induced an increase in GABAergic and glycinergic neurotransmission to CVNs in NA and DMNX, re
197 on of GABAergic transmission while enhancing glycinergic neurotransmission to CVNs in NA.
198 y glutamatergic and inhibitory GABAergic and glycinergic neurotransmission to parasympathetic cardiac
199 uman glycine receptor autoantibodies disrupt glycinergic neurotransmission, and also suggest that the
200 or several postsynaptic proteins involved in glycinergic neurotransmission, including the glycine rec
201 erekplexia is caused by defective inhibitory glycinergic neurotransmission.
202 e in the physiological control of inhibitory glycinergic neurotransmission.
203 es of effect are known, and all three affect glycinergic neurotransmission.
204 ibodies, were observed to disrupt profoundly glycinergic neurotransmission.
205  children and usually involves dysfunctional glycinergic neurotransmission.
206 nvolved adaptations in the glutamatergic and glycinergic neurotransmitter systems.
207    Both arise postnatally and one is neither glycinergic nor GABAergic (nGnG).
208 al cerebellar (fastigial) nuclei are in fact glycinergic, not glutamatergic as previously thought.
209 gic ACs (gammaACs) and motif C2 narrow field glycinergic ON ACs (GACs).
210 ing the first three postnatal weeks, whereas glycinergic ones exhibited age-dependent changes compara
211 rthermore, additional experiments yielded no glycinergic or cholinergic positive cells in the IC, and
212                 We found decreases in either glycinergic or GABAergic inhibition to the medial nucleu
213 ergic neurotransmission but had no effect on glycinergic or glutamatergic pathways to cardiac vagal n
214                                   We defined glycinergic pacemaker neurons as those preBotC EGFP neur
215 port the presence of a population of preBotC glycinergic pacemaker neurons.
216 ng from the spinal cord to reveal descending glycinergic pathways.
217 m a large heterogeneous population with GABA/glycinergic phenotypes, distinct from GABAergic olive-pr
218 d to gamma-aminobutyric acid (GABA)ergic and glycinergic postsynapses.
219                  Measurements of spontaneous glycinergic postsynaptic currents and GlyR immunolabelin
220 reduced in Slc7a10-null mice and spontaneous glycinergic postsynaptic currents in motor neurons show
221   These findings emphasize the importance of glycinergic postsynaptic inhibition in motor neurons and
222                                            A glycinergic postsynaptic response was not found although
223 point, using transgenic mice with negligible glycinergic postsynaptic responses, we show that this de
224 dorsal nucleus (SLD), which in turn activate glycinergic pre-motor neurons in the spinal cord and/or
225                  We investigated the role of glycinergic preBotC neurons in respiratory rhythmogenesi
226                             We conclude that glycinergic preBotC neurons modulate inspiratory pattern
227  or Archaerhodopsin (Arch) were expressed in glycinergic preBotC neurons of glycine transporter 2 (Gl
228  population of preBotC inspiratory-modulated glycinergic, presumably inhibitory, pacemaker neurons th
229 synaptic inhibition was blocked by GABAergic/glycinergic receptor antagonists.
230            We quantified the distribution of glycinergic receptor dynamics using fluorescence recover
231 ne-tuned through heterogeneous GABAergic and glycinergic receptor ratios expressed at individual syna
232 he presence of blockers of glutamatergic and glycinergic receptor-mediated transmission application o
233                       Block of GABAergic and glycinergic receptors does not attenuate or modify L ver
234 eactive interneurons contain GABAA ergic and glycinergic receptors.
235 hus, either the selective loss of inhibitory glycinergic regulation of motoneuron function or glycine
236 duction of external inhibition (in this case glycinergic), saccadic oscillations and limb tremor were
237 be a novel strategy to modulate GABA- and/or glycinergic signaling for therapeutic benefit.
238 ty and learning and memory, and link altered glycinergic signaling to social and cognitive impairment
239  of protein kinase G blocked the increase in glycinergic sIPSCs by the cGMP analog 8-bromo-cGMP.
240 cific AII amacrine cell, the connectivity of glycinergic small-field amacrine cells has not been inve
241                                       In the glycinergic sound localization pathway from the medial n
242 nsmission are prominent in a developing GABA/glycinergic sound-localization pathway.
243         Comparing the effects of caffeine on glycinergic spontaneous and evoked IPSCs indicates that
244 NAP significantly increased the frequency of glycinergic spontaneous and miniature inhibitory postsyn
245 ic spontaneous IPSCs and mIPSCs and those of glycinergic spontaneous IPSCs and mIPSCs did not differ
246 hibitory effect of baclofen on GABAergic and glycinergic spontaneous IPSCs and mIPSCs was not signifi
247 the trapezoid body to lateral superior olive glycinergic synapse, and the basket/stellate cell-Purkin
248 nterneurons in the retina that exhibit large glycinergic synapses at their dendritic lobular appendag
249 n late during development ablates alpha1beta glycinergic synapses but spares GABAergic synapses.
250 egulate the activity-dependent plasticity of glycinergic synapses by tuning the GlyR diffusion trap.
251 rod to OFF cone bipolar cell synapses and/or glycinergic synapses from AII amacrine cells to OFF gang
252                          Although functional glycinergic synapses have not been identified in the hip
253  peripheral inflammation in vivo potentiates glycinergic synapses on dorsal horn neurons, suggesting
254                     We found an exception at glycinergic synapses on granule cells of the rat dorsal
255  superficial dorsal horn of the spinal cord, glycinergic synapses on inhibitory GABAergic neurons exh
256                                              Glycinergic synapses play a central role in motor contro
257      The AII amacrine cell also makes direct glycinergic synapses with certain RGCs, but it is not we
258                        GACs selectively make glycinergic synapses with uniformity detectors (UDs) and
259 aling, as spillover of glutamate onto nearby glycinergic synapses would permit rapid crosstalk betwee
260 s-of-function mutations in proteins found at glycinergic synapses, most commonly in the alpha1 subuni
261    This circuitry, mediated by GABAergic and glycinergic synapses, provides expiratory inhibition tha
262 the main source of releasable transmitter at glycinergic synapses.
263 ere have been no reports of LTP at mammalian glycinergic synapses.
264 ubunits or in other proteins associated with glycinergic synapses.
265 ts were increased by electrically activating glycinergic synapses.
266  cell terminals and sign-inverting chemical (glycinergic) synapses to modulate OFF cone cell bipolar
267          These findings suggest that central glycinergic synaptic activity plays a vital role in regu
268 ings from cultured rat spinal motor neurons, glycinergic synaptic currents were almost completely abo
269                     Inhibitory GABAergic and glycinergic synaptic currents were also significantly in
270 ters, and depleting terminals of GABA slowed glycinergic synaptic currents.
271                                              Glycinergic synaptic inhibition is part of acoustic info
272 MOC neurons receive inhibitory GABAergic and glycinergic synaptic inputs.
273 lutamatergic, and/or inhibitory GABAergic or glycinergic synaptic neurotransmission to cardiac vagal
274 rrents recorded from KI mice showed that the glycinergic synaptic transmission had normal properties,
275 be mimicked by pharmacologically blocking of glycinergic synaptic transmission on age-matched wildtyp
276 ostsynaptic heteromeric GlyRs (which mediate glycinergic synaptic transmission), because heteromeric
277  antagonists of glutamatergic, GABAergic, or glycinergic synaptic transmission.
278 c GlyT2 drastically impairs the refilling of glycinergic synaptic vesicles and severely disrupts neur
279 hasic excitatory (glutamatergic)/inhibitory (glycinergic) synaptic drive was recorded in thoracic loc
280 lasticity of the gamma-aminobutyric acid and glycinergic system by targeting KCC2 may be a tenable me
281                  Comparison of the zebrafish glycinergic system with the glycinergic systems of other
282  reveal a complex and divergent evolution of glycinergic systems in the major groups of fishes.
283 of the zebrafish glycinergic system with the glycinergic systems of other adult vertebrates reveals s
284 f a longer persistence of GABA in SOD1(G93A) glycinergic terminals in cultured and ex vivo spinal sli
285 less precise and the shift from a mixed GABA/glycinergic to a purely glycinergic transmission before
286 deg, but throughout the retina, the ratio of glycinergic to gamma-aminobutyric acid (GABA)ergic to am
287 ypothesized that agents capable of enhancing glycinergic tone within the dorsal horn could obtund noc
288 ft from a mixed GABA/glycinergic to a purely glycinergic transmission before hearing onset did not oc
289            Thus, coreleased GABA accelerates glycinergic transmission by acting directly on glycine r
290 lling postural muscles that are inhibited by glycinergic transmission during REM sleep, hypoglossal m
291 nstrating for the first time a role for fast glycinergic transmission in the avian auditory brainstem
292 lysis of inhibitory synaptic currents showed glycinergic transmission is the dominant form of phasic
293                              Fast inhibitory glycinergic transmission occurs in spinal cord, brainste
294 , and are mediated by combined GABAergic and glycinergic transmission.
295 d Tcf4, expressed by most GABAergic and most glycinergic types, respectively.
296         Considerable data support a role for glycinergic ventromedial medulla neurons in the mediatio
297          We then investigated aspects of the glycinergic versus GABAergic current components to probe
298 eep-active neurons are inhibitory (GABAergic/glycinergic, VGAT-positive) in nature.
299 excitability disorders and may extend to the glycinergic visual system.
300  the LSO reportedly shifts from GABAergic to glycinergic within the first three postnatal weeks.

 
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