戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ates GP and moves it toward the SR, favoring glycogenolysis.
2  of an elevation of both gluconeogenesis and glycogenolysis.
3 l glucose production and glucagon-stimulated glycogenolysis.
4 edly increasing HGP as a result of increased glycogenolysis.
5 hrough glucose cycling, gluconeogenesis, and glycogenolysis.
6 zes the terminal step in gluconeogenesis and glycogenolysis.
7 on was solely attributable to an increase in glycogenolysis.
8 m to limit lactate accumulation during rapid glycogenolysis.
9 cesses like glycolysis, gluconeogenesis, and glycogenolysis.
10 ownregulated genes related to glycolysis and glycogenolysis.
11 ta-oxidation of fatty acids, glycolysis, and glycogenolysis.
12 alyses the final step of gluconeogenesis and glycogenolysis.
13 c equilibrium between glycogen synthesis and glycogenolysis.
14 accelerated gluconeogenesis, and decelerated glycogenolysis.
15 n from glycogen in the cascade activation of glycogenolysis.
16 flux, and gluconeogenesis without increasing glycogenolysis.
17  equivalent estimates of gluconeogenesis and glycogenolysis.
18 oma cells by suppressing gluconeogenesis and glycogenolysis.
19  (alphabetagammadelta)(4) complex, regulates glycogenolysis.
20 (by approximately 75%), gluconeogenesis, and glycogenolysis.
21 limited oxidative capacity caused by blocked glycogenolysis.
22 ter GP because of a compensatory decrease in glycogenolysis.
23 ate by promoting hepatic gluconeogenesis and glycogenolysis.
24 pendent pathway causing inhibition of GP and glycogenolysis.
25 urce of blood glucose is gluconeogenesis and glycogenolysis.
26 irrhotic patients because of a lower rate of glycogenolysis (0.63 +/- 0.23 vs. 1.22 +/- 0.23 mg.kg-1.
27 p was due to a marked suppression of hepatic glycogenolysis (0.7 +/- 0.1 versus 4.1 +/- 0.6 mg/kg.min
28 36%) was from gluconeogenesis, 1.40 was from glycogenolysis, 0.30 was retained in glycogen via UDP-gl
29 oneogenesis from the TCA cycle (67.3+/-5.6), glycogenolysis (1.0+/-0.8), pyruvate cycling (154.4+/-43
30 lucagon-stimulated cAMP production (55%) and glycogenolysis (27%) in human hepatocytes.
31                             We conclude that glycogenolysis accounts for the majority of EGP during t
32 that this M6P originates from glycogen, with glycogenolysis activated by the kinase domain of the str
33  insulin from inhibiting gluconeogenesis and glycogenolysis and activating glycolysis.
34                                              Glycogenolysis and astrocytic lactate transporters are a
35    After a 4-h fast, glucose production from glycogenolysis and conversion of glycerol to glucose rem
36 hanism by which insulin inhibits net hepatic glycogenolysis and endogenous glucose production in huma
37 uccessfully determined that gluconeogenesis, glycogenolysis and fatty acid oxidation were active in b
38  Furthermore, pharmacological attenuation of glycogenolysis and functional depletion of glycogen both
39                                              Glycogenolysis and gluconeogenesis are sensitive to nutr
40                               HNPs inhibited glycogenolysis and gluconeogenesis in isolated hepatocyt
41 genes encoding the rate-limiting enzymes for glycogenolysis and gluconeogenesis, including liver glyc
42  glycogenesis and the release of glucose via glycogenolysis and gluconeogenesis.
43 Glc-6-P to glucose in the terminal stages of glycogenolysis and gluconeogenesis.
44 possible mechanism for the direct linkage of glycogenolysis and glycolysis in skeletal muscle.
45 ; this adaptation results in slower rates of glycogenolysis and lactic acid accumulation in muscle du
46 n ischaemic exercise ATP is supplied only by glycogenolysis and net splitting of phosphocreatine (PCr
47                                              Glycogenolysis and PEP-gluconeogenesis (2.1 +/- 0.3 mg/k
48 organic phosphate (Pi) levels, which impacts glycogenolysis and proton buffering, and in intracellula
49 e that energy is supplied in milliseconds by glycogenolysis and that between contractions, glycogenes
50 gen metabolizing enzymes, the enhancement of glycogenolysis, and a dramatic decrease in cellular glyc
51 n increase in endogenous glucose production, glycogenolysis, and gluconeogenesis from phosphoenolpyru
52 hepatic glucose production, gluconeogenesis, glycogenolysis, and glucose cycling.
53  of appearance, production, gluconeogenesis, glycogenolysis, and hexoneogenesis were measured by usin
54 nd metabolic (endogenous glucose production, glycogenolysis, and lipolysis) responses were increased
55 ncluding glycolysis, pentose phosphate (PP), glycogenolysis, and polyols to translate the glucose met
56 tractility, platelet aggregation, lipolysis, glycogenolysis, and smooth muscle contraction.
57 s revealed that genes related to glycolysis, glycogenolysis, and the pentose phosphate pathway (PPP)
58 ects of leptin on GP, on gluconeogenesis, on glycogenolysis, and/or on the hepatic expression of the
59 hydrogen at carbon 2 of glucose produced via glycogenolysis are estimated from the enrichments to be
60 15 mm glucose, AdCMV-P46 treatment activated glycogenolysis, as indicated by a 50% reduction in glyco
61 ay also have an important role in inhibiting glycogenolysis at rest as well as improving the efficien
62 portant regulatory role in the inhibition of glycogenolysis at rest.
63 arlier exhaustion of glycogen stores, slowed glycogenolysis before complete glycogen depletion, and/o
64 aining induces adaptations that downregulate glycogenolysis before there is an increase in functional
65                    Therefore, glycolysis and glycogenolysis behave independently in vascular smooth m
66 ates of total glucose production and hepatic glycogenolysis but similar rates of gluconeogenesis comp
67 r by the decreased inhibition of hepatic net glycogenolysis by hyperglycemia (3.3 +/- 0.8 and 1.1 +/-
68 vels within the liver secondary to increased glycogenolysis caused by systemic hypoglycemia.
69 nzyme activity and that this interferes with glycogenolysis causing increased levels of glycogen in h
70 bit glycogen phosphorylase, in turn blocking glycogenolysis causing the massive liver in Mauriac dise
71 d reduced glycogen accumulation and enhanced glycogenolysis compared with their respective controls,
72 creased rates of gluconeogenesis and perhaps glycogenolysis contribute to hepatic insulin resistance.
73  +/- 9% of total hepatic glucose output with glycogenolysis contributing the remainder.
74  metabolic (glucose kinetics, lipolysis, and glycogenolysis) counterregulatory responses.
75                        This kinetic model of glycogenolysis, coupled to creatine kinase and adenylate
76 ic gluconeogenesis increased by 70%, and net glycogenolysis declined by 20%.
77  of Molecular Cell, Zhou et al.(1) show that glycogenolysis-derived glucose-1-phosphate enhances gluc
78                                This channels glycogenolysis-derived glucose-6-phosphate into the pent
79                 This may occur via increased glycogenolysis during beta1-AR stimulation, facilitating
80 has been used to measure gluconeogenesis and glycogenolysis during hyperinsulinemic-euglycemic clamps
81 e quantified the contribution of net hepatic glycogenolysis during insulin-induced hypoglycemia in 10
82                    Moreover, G6PC or another glycogenolysis enzyme-liver glycogen phosphorylase (PYGL
83                                              Glycogenolysis, essentially 0 in +/+ rats after a 24-h f
84                         Rates of net hepatic glycogenolysis, estimated by 13C nuclear magnetic resona
85 phofructokinase together with ATP demand and glycogenolysis exert the highest control on the glycolyt
86  The data do not support the hypothesis that glycogenolysis follows Michealis-Menten kinetics with an
87 haustion of glycogen stores but by depressed glycogenolysis from the onset of ischemia.
88                                  Conversely, glycogenolysis (GL) increased from 41.9% at 16 hours to
89 luconeogenesis (GNG) (determined with 2H2O), glycogenolysis (GL), and endogenous glucose production (
90  (EGP) via changes in gluconeogenesis (GNG), glycogenolysis (GL), or both, we measured GNG (with (2)H
91 ose production secondary to an inhibition of glycogenolysis, gluconeogenesis, and glucose-6-phosphata
92 ardial glycogen and the rates of glycolysis, glycogenolysis, glucose utilization, and glycolytic ATP
93  glucose production (EGP) occurs via hepatic glycogenolysis (GLY) and gluconeogenesis (GNG) and plays
94 creased while both GP and calculated hepatic glycogenolysis (GLY) decreased.
95 age diseases resulting from known defects in glycogenolysis, glycolysis, and glycogen synthesis that
96 pd 1 is capable of blocking glucagon-induced glycogenolysis in a dosage-dependent manner.
97                   In contrast, LamA-mediated glycogenolysis in amoebae deprives the natural host from
98 ed hepatic lipogenesis, gluconeogenesis, and glycogenolysis in an AHR-dependent manner.
99 otal endogenous glucose production) rates of glycogenolysis in both the nondiabetic and diabetic subj
100                                              Glycogenolysis in glycogen-replete hearts perfused with
101 ally, CP-91149 inhibited glucagon-stimulated glycogenolysis in isolated rat hepatocytes (P < 0.05 at
102                            PST also triggers glycogenolysis in liver and reduces glucose uptake in ad
103 In addition, Cpd 1 blocked glucagon-mediated glycogenolysis in primary human hepatocytes.
104 Phosphorylase kinase, a regulatory enzyme of glycogenolysis in skeletal muscle, is a hexadecameric ol
105 phabetagammadelta)(4) complex that regulates glycogenolysis in skeletal muscle.
106 in glycemia by promoting gluconeogenesis and glycogenolysis in target tissues.
107 reas glucagon stimulates gluconeogenesis and glycogenolysis in the liver.
108 ree-enzyme complex, a novel net reaction for glycogenolysis in the vicinity of the sarcoplasmic retic
109 glucose lowering resulted from inhibition of glycogenolysis in vivo.
110  for the final stages of gluconeogenesis and glycogenolysis, in which glucose-6-phosphate (G6P) is hy
111  hepatic glucose production due to increased glycogenolysis, indicating hepatic insulin resistance; t
112 lucose production, and they demonstrate that glycogenolysis inhibitors may be useful in the treatment
113                        In the final steps of glycogenolysis, intracellular glucose 6-phosphate (Glc-6
114  During the early stages of fasting, hepatic glycogenolysis is a primary energy source.
115            These results indicate that liver glycogenolysis is acutely sensitive to small changes in
116              The ability to fuel neurons via glycogenolysis is believed to be an important function o
117        The final step of gluconeogenesis and glycogenolysis is catalyzed by the glucose-6-phosphatase
118             It is concluded that the rate of glycogenolysis is determined by the content of glycogen
119 osphatase (G6PC) catalyzing the last step of glycogenolysis is frequently downregulated to augment gl
120 diminish glucose release from the liver when glycogenolysis is not needed.
121 o blood glucose, and the duration of reduced glycogenolysis is short-lived after relaxation of energy
122 ate that after an overnight fast, basal HGP (glycogenolysis) is highly sensitive to the hepatic sinus
123 is (glycerol, nonesterified fatty acid), and glycogenolysis (lactate) were also reduced during day 2
124 inborn error of metabolism where a defect in glycogenolysis leads to the inability to break down glyc
125 to glucose production of gluconeogenesis and glycogenolysis, measured by labeling of blood glucose by
126 eas in protocol II inhibition of net hepatic glycogenolysis occurred exclusively through the activati
127                    Inhibition of net hepatic glycogenolysis occurred in both protocols I and II compa
128                    Inhibition of net hepatic glycogenolysis occurred in protocol I mostly due to decr
129                         Since suppression of glycogenolysis occurred without a decrease in UDP-glucos
130 ed with its inhibitory effect on net hepatic glycogenolysis, occurred within 30 min, and was associat
131 nphosphorylated form, "GPb," which catalyzes glycogenolysis only in the presence of appropriate allos
132 the terminal step of the gluconeogenesis and glycogenolysis pathways.
133  hyperglycemia, per se, inhibits net hepatic glycogenolysis primarily through inhibition of glycogen
134 perinsulinemia, per se, inhibits net hepatic glycogenolysis primarily through stimulation of glycogen
135                                 In contrast, glycogenolysis rates were markedly increased, suggesting
136 umol/ min per gram wet weight [P < .001] for glycogenolysis, respectively, at 2.5 minutes of ischemia
137  were abolished, and a marked suppression of glycogenolysis resulted in decreased GP.
138      Targeted inhibition of the G3P shuttle, glycogenolysis, serine biosynthesis, and mitochondrial r
139 esponse to a 'second hit' via the astrocytic glycogenolysis signaling pathway.
140  whereas it decreased fluxes associated with glycogenolysis, TCA cycle, fatty acid oxidation and elec
141 with reduced proglucagon gene expression and glycogenolysis that result from pancreatic islet cell de
142 ase (ANOVA; P < 0.05) in the contribution of glycogenolysis to EGP (4.7 +/- 1.7 vs. 3.4 +/- 1.2 vs. -
143 are required to suppress the contribution of glycogenolysis to EGP in healthy nondiabetic humans.
144 with the contribution of gluconeogenesis and glycogenolysis to EGP.
145 n response to hormonal signaling, fine-tunes glycogenolysis to fulfill energetic and metabolic requir
146   As a result, PTG-G(L) expression permitted glycogenolysis under 5 mm glucose conditions that was pr
147 sess rates of hepatic glycogen synthesis and glycogenolysis under euglycemic (approximately 5 mmol/l)
148            The estimated initial increase in glycogenolysis was approximately 1.7 and 2.3 mg/kg x min
149                           This lower rate of glycogenolysis was associated with lower hepatic glycoge
150                           On the other hand, glycogenolysis was equally suppressed in both groups.
151  (G6Pase) gene expression, however net liver glycogenolysis was impaired in C/EBPbeta-/- mice.
152 periments were designed to determine whether glycogenolysis was influenced by the glycogen concentrat
153    Importantly, the reduction in the rate of glycogenolysis was larger and out of proportion to the r
154 h groups receiving ICV leptin, while hepatic glycogenolysis was markedly suppressed (0.7 +/- 0.3 and
155 se and urea production, gluconeogenesis, and glycogenolysis were calculated using stable isotope meth
156  0.01); however, rates of glucose uptake and glycogenolysis were similar between the two groups.
157  analysis indicated both gluconeogenesis and glycogenolysis were suppressed in Snell dwarfs.
158 ree methods of measuring gluconeogenesis and glycogenolysis were used: 1) the hepatic arteriovenous d
159 effect on NHGO, caused by the suppression of glycogenolysis, while an equal increment in arterial ins
160 arlier mathematical model of skeletal muscle glycogenolysis with pH-dependent enzyme kinetics and rea

 
Page Top