ライフサイエンスコーパス: gonadotropin-releasing hormone

1 ultimately drive the pulsatile secretion of gonadotropin-releasing hormone.

2 l basis for a putative role of sbGnRH as the gonadotropin-releasing hormone.

3 to provide an episodic, excitatory drive to gonadotropin-releasing hormone 1 (GnRH) neurons, the syn

4 nit, which is transcriptionally regulated by gonadotropin-releasing hormone 1 (GNRH).

5 Gonadotropin-releasing hormone 1 (GnRH1) from the brain

6 thalamic neurons that synthesize and release gonadotropin-releasing hormone 1 (GnRH1), but the precis

7 Pulsatile secretion of gonadotropin-releasing hormone-1 (GnRH-1) is essential f

8 cystokinin (CCK) in the developing olfactory-gonadotropin-releasing hormone-1 (GnRH-1) neuroendocrine

9 Gonadotropin-releasing hormone-1 (GnRH-1) neurons migrat

10 During mammalian development, gonadotropin-releasing-hormone-1 neurons (GnRH-1ns) migr

11 asic ovarian sex hormone fluctuation using a gonadotropin-releasing hormone agonist (GnRHa) and evalu

12 rted previously that after 1-year follow up, gonadotropin-releasing hormone agonist (GnRHa) did not p

13 by surgical castration and those who receive gonadotropin-releasing hormone agonist (GnRHa) therapy.

14 nditions: ovarian suppression induced by the gonadotropin-releasing hormone agonist leuprolide acetat

15 We administered the gonadotropin-releasing hormone agonist leuprolide acetat

16 umulation increased after treatment with the gonadotropin-releasing hormone agonist leuprolide, which

17 Gonadotropin-releasing hormone agonist therapy is associ

18 over study, 12 healthy, young males received gonadotropin-releasing hormone agonist treatment 1 month

19 nditions: ovarian suppression induced by the gonadotropin-releasing hormone agonist, leuprolide aceta

20 al women, or premenopausal women receiving a gonadotropin-releasing hormone agonist, with estrogen re

21 nmetastatic disease, intervention group with gonadotropin-releasing hormone agonist-based ADT, contro

22 The authors examined the effects of gonadotropin-releasing hormone agonist-induced ovarian s

23 othersome side effects than treatment with a gonadotropin-releasing hormone agonist.

24 efore and after treatment with a long-acting gonadotropin-releasing hormone agonist.

25 ntraindication to tamoxifen may be offered a gonadotropin-releasing hormone agonist/antagonist plus a

26 production was achieved with the use of the gonadotropin-releasing-hormone agonist triptorelin, ooph

27 Gonadotropin-releasing hormone agonists (GnRHa) have bee

28 Puberty suppression by gonadotropin-releasing hormone agonists (GnRHa), such as

29 herapies (OR, 4.04 [95% CI, 1.88-8.69]), and gonadotropin-releasing hormone agonists (OR, 1.93 [95% C

30 androgen deprivation therapy in the form of gonadotropin-releasing hormone agonists and newer antago

31 Gonadotropin-releasing hormone agonists decrease bone mi

32 Gonadotropin-releasing hormone agonists given with chemo

33 y a combined androgen blockade consisting of gonadotropin-releasing hormone agonists with oral antian

34 er treatments such as surgical oophorectomy, gonadotropin-releasing hormone agonists, chemotherapy-in

35 rized into 1 of 6 mutually exclusive groups: gonadotropin-releasing hormone agonists, oral antiandrog

36 ess of depot leuprolide acetate, a synthetic gonadotropin-releasing hormone analog (GnRH-a), for prot

37 of glucocorticoid replacement, 19.2%; and of gonadotropin-releasing hormone analog therapy, 34.2%.

38 ne [n = 33 835], gonadotropins [n = 57 136], gonadotropin-releasing hormone analogs [n = 38 653], hum

39 Gonadotropin-releasing hormone analogue triptorelin, bil

40 rel-releasing intrauterine system (LNG-IUD), gonadotropin-releasing hormone analogues (GnRHa; nafarel

41 Gonadotropin-releasing hormone analogues and laparoscopi

42 n suppression (castration via orchiectomy or gonadotropin-releasing hormone analogues) suppresses cir

43 Gonadotropin-releasing hormone analogues, danazol, and d

44 ikely to be mediated by reduced secretion of gonadotropin-releasing hormone and our results support t

45 Moreover, ERbeta2 is coexpressed in gonadotropin-releasing hormone and oxytocin neurons.

46 onsiveness to the endogenous natural ligand, gonadotropin releasing hormone, and an agonist that is s

47 Gonadotropin releasing hormone antagonism successfully r

48 o the antral stage in both immature mice and gonadotropin releasing hormone antagonist-treated adult

49 differences in T through administration of a gonadotropin releasing hormone antagonist.

50 (ii) a single subcutaneous injection of the gonadotropin-releasing hormone antagonist (GnRH-A), acyl

51 Elagolix, an oral gonadotropin-releasing hormone antagonist resulting in r

52 le-stimulating hormone and administration of gonadotropin-releasing hormone antagonist to prevent pre

53 of short-term castration (1 month) using the gonadotropin-releasing hormone antagonist, Acyline, vers

54 he efficacy and safety of relugolix, an oral gonadotropin-releasing hormone antagonist, as compared w

55 been found for other classes of drugs (e.g., gonadotropin-releasing hormone antagonists, neurokinin r

56 levels are increased somewhat by exposure to gonadotropin-releasing hormone but are not necessarily l

57 e likely to be factors in the suppression of gonadotropin releasing hormone (GnRH) activity.

58 17beta-estradiol (E2) regulating release of gonadotropin releasing hormone (GnRH) and luteinizing ho

59 tress index, CSI), women self administered a gonadotropin releasing hormone (GnRH) antagonist for 16

60 zed by absent puberty and infertility due to gonadotropin releasing hormone (GnRH) deficiency, which

61 ntrolled in part by the pulsatile release of gonadotropin releasing hormone (GnRH) from the hypothala

62 Release of gonadotropin releasing hormone (GnRH) from the medial ba

63 stone deacetylases (HDACs) in the control of gonadotropin releasing hormone (GnRH) neuronal developme

64 odulate the central driver of fertility: the gonadotropin releasing hormone (GnRH) neuronal system.

65 gy balance and season, time the awakening of gonadotropin releasing hormone (GnRH) neurons at the ons

66 onadotropic hypogonadism to study effects of gonadotropin releasing hormone (GnRH) neurons on neurona

67 tory receptor (ORNs), vomeronasal (VRNs) and gonadotropin releasing hormone (GnRH) neurons.

68 Gonadotropin releasing hormone (GnRH) plays an important

69 e associated with regulation by activins and gonadotropin releasing hormone (GnRH).

70 acetate, a synthetic nonapeptide analogue of gonadotropin-releasing hormone (GnRH or LHRH), is the ac

71 Gonadotropin-releasing hormone (GnRH) acts at gonadotrop

72 a potent and very long acting antagonist of gonadotropin-releasing hormone (GnRH) after subcutaneous

73 mone but apparently normal responsiveness to gonadotropin-releasing hormone (GnRH) agonist and antago

74 Androgen deprivation therapy with a gonadotropin-releasing hormone (GnRH) agonist is associa

75 Gonadotropin-releasing hormone (GnRH) agonists (e.g., tr

76 bjected to 26 ART treatment cycles receiving gonadotropin-releasing hormone (GnRH) agonists and recom

77 Gonadotropin-releasing hormone (GnRH) agonists are assoc

78 Gonadotropin-releasing hormone (GnRH) agonists decrease

79 Gonadotropin-releasing hormone (GnRH) agonists decrease

80 Studies of the use of gonadotropin-releasing hormone (GnRH) agonists to protec

81 n were on antiandrogens (AA), 26,959 were on gonadotropin-releasing hormone (GnRH) agonists, and 3,74

82 ective randomized trial evaluated the use of gonadotropin-releasing hormone (GnRH) analog triptorelin

83 Because gonadotropin-releasing hormone (GnRH) analogs constitute

84 Women treated with gonadotropin-releasing hormone (GnRH) analogs may develo

85 estrogens, finasteride, spironolactone, and gonadotropin-releasing hormone (GnRH) analogs.

86 Both gonadotropin-releasing hormone (GnRH) and activins, memb

87 ted with oocyte meiosis, TGF-beta signaling, gonadotropin-releasing hormone (GnRH) and epidermal grow

88 In vertebrates, gonadotropin-releasing hormone (GnRH) and gonadotropin-i

89 and controls the synthesis and/or release of gonadotropin-releasing hormone (GnRH) and gonadotropins.

90 ons are thought to regulate the secretion of gonadotropin-releasing hormone (GnRH) and thus coordinat

91 Elagolix, an oral, nonpeptide, gonadotropin-releasing hormone (GnRH) antagonist, produc

92 Neurons that produce gonadotropin-releasing hormone (GnRH) are the final comm

93 amining the distribution of cells containing gonadotropin-releasing hormone (GnRH) as well as estroge

94 Dysfunction of the gonadotropin-releasing hormone (GnRH) axis causes a rang

95 male rat preoptic area (POA), containing the gonadotropin-releasing hormone (GnRH) cell bodies, treat

96 ypothalamic-pituitary-ovarian axis, in which gonadotropin-releasing hormone (GnRH) controls the relea

97 Isolated gonadotropin-releasing hormone (GnRH) deficiency caused

98 othalamic amenorrhea is a reversible form of gonadotropin-releasing hormone (GnRH) deficiency commonl

99 Gonadotropin-releasing hormone (GnRH) deficiency in the

100 Congenital gonadotropin-releasing hormone (GnRH) deficiency manifes

101 Neurons that produce gonadotropin-releasing hormone (GnRH) drive the reproduc

102 In mice, gonadotropin-releasing hormone (GnRH) expression is limi

103 Individuals with an inherited deficiency in gonadotropin-releasing hormone (GnRH) have impaired sexu

104 action on puberty by stimulating release of gonadotropin-releasing hormone (GnRH) in the hypothalamu

105 levated levels of transcripts for the salmon gonadotropin-releasing hormone (GnRH) in the male telenc

106 Administration of gonadotropin-releasing hormone (GnRH) induces a surge of

107 that FOXO1 repressed basal transcription and gonadotropin-releasing hormone (GnRH) induction of both

108 amic median eminence (ME) where they release gonadotropin-releasing hormone (GnRH) into a specialized

109 Gonadotropin-releasing hormone (GnRH) is a key regulator

110 Gonadotropin-releasing hormone (GnRH) is a trophic pepti

111 Gonadotropin-releasing hormone (GnRH) is a trophic pepti

112 propriate tissue-specific gene expression of gonadotropin-releasing hormone (GnRH) is critical for pu

113 In vertebrates, gonadotropin-releasing hormone (GnRH) is essential to th

114 Gonadotropin-releasing hormone (GnRH) is exclusively exp

115 Gonadotropin-releasing hormone (GnRH) is found in a wide

116 Gonadotropin-releasing hormone (GnRH) is released in a p

117 Pulsatile release of gonadotropin-releasing hormone (GnRH) is required for fe

118 The hypothalamic neuropeptide gonadotropin-releasing hormone (GnRH) is secreted in a p

119 Gonadotropin-releasing hormone (GnRH) is secreted in bri

120 Gonadotropin-releasing hormone (GnRH) is secreted in bri

121 Gonadotropin-releasing hormone (GnRH) is the central reg

122 Gonadotropin-releasing hormone (GnRH) is the central reg

123 Gonadotropin-releasing hormone (GnRH) is the central reg

124 The gonadotropin-releasing hormone (GnRH) is the master regu

125 The gonadotropin-releasing hormone (GnRH) is the master regu

126 posed to pulsatile, delivery of hypothalamic gonadotropin-releasing hormone (GnRH) leads to a marked

127 We have previously shown that gonadotropin-releasing hormone (GnRH) ligand-independent

128 The gonadotropin-releasing hormone (GnRH) neuron is the pivo

129 An attractive model is the gonadotropin-releasing hormone (GnRH) neuron system, mas

130 (RFRP-3) neurons have been shown to inhibit gonadotropin-releasing hormone (GnRH) neuronal activity

131 a profound influence on the activity of the gonadotropin-releasing hormone (GnRH) neuronal network c

132 isspeptin is a key regulator of hypothalamic gonadotropin-releasing hormone (GnRH) neurons and is ess

133 Estradiol feedback regulates gonadotropin-releasing hormone (GnRH) neurons and subseq

134 diol alters both the intrinsic properties of gonadotropin-releasing hormone (GnRH) neurons and synapt

135 Gonadotropin-releasing hormone (GnRH) neurons are born i

136 Gonadotropin-releasing hormone (GnRH) neurons are critic

137 Gonadotropin-releasing hormone (GnRH) neurons are neuroe

138 Gonadotropin-releasing hormone (GnRH) neurons are neuroe

139 Gonadotropin-releasing hormone (GnRH) neurons are the ce

140 Gonadotropin-releasing hormone (GnRH) neurons are the ce

141 Gonadotropin-releasing hormone (GnRH) neurons are the fi

142 Gonadotropin-releasing hormone (GnRH) neurons are the fi

143 The gonadotropin-releasing hormone (GnRH) neurons are the ke

144 Gonadotropin-releasing hormone (GnRH) neurons are the pr

145 Central output of gonadotropin-releasing hormone (GnRH) neurons controls f

146 sspeptin neurons act cooperatively to excite gonadotropin-releasing hormone (GnRH) neurons during pos

147 Gonadotropin-releasing hormone (GnRH) neurons exhibit bu

148 al axis is dependent on correct migration of gonadotropin-releasing hormone (GnRH) neurons from the n

149 oductive functioning in mammals depends upon gonadotropin-releasing hormone (GnRH) neurons generating

150 in mice for controlling the activity of the gonadotropin-releasing hormone (GnRH) neurons in vivo to

151 Hypothalamic gonadotropin-releasing hormone (GnRH) neurons integrate

152 ut from the suprachiasmatic nucleus (SCN) to gonadotropin-releasing hormone (GnRH) neurons is critica

153 Secretion from gonadotropin-releasing hormone (GnRH) neurons is necessa

154 Gonadotropin-releasing hormone (GnRH) neurons migrate fr

155 Gonadotropin-releasing hormone (GnRH) neurons originate

156 Gonadotropin-releasing hormone (GnRH) neurons produce th

157 Gonadotropin-releasing hormone (GnRH) neurons regulate p

158 Gonadotropin-releasing hormone (GnRH) neurons represent

159 n, rostral projections from the AVPV contact gonadotropin-releasing hormone (GnRH) neurons surroundin

160 onstructions and electrophysiology, that the gonadotropin-releasing hormone (GnRH) neurons that contr

161 rculating estradiol on proestrus to activate gonadotropin-releasing hormone (GnRH) neurons that, in t

162 estradiol (E2) regulates the activity of the gonadotropin-releasing hormone (GnRH) neurons through bo

163 nisms through which estradiol (E2) regulates gonadotropin-releasing hormone (GnRH) neurons to control

164 s how nerve terminal Ca(2+) is controlled in gonadotropin-releasing hormone (GnRH) neurons via action

165 peptin regulates reproduction by stimulating gonadotropin-releasing hormone (GnRH) neurons via the ki

166 Gonadotropin-releasing hormone (GnRH) neurons, a small n

167 mice lacking necdin have reduced numbers of gonadotropin-releasing hormone (GnRH) neurons, but the m

168 eactive fibres also abut the preoptic-septal gonadotropin-releasing hormone (GnRH) neurons, suggestin

169 ne hormone secretion, especially that of the gonadotropin-releasing hormone (GnRH) neurons, via neura

170 arcuate nucleus (Arc) provide tonic drive to gonadotropin-releasing hormone (GnRH) neurons, which in

171 o a developmental defect in the migration of gonadotropin-releasing hormone (GnRH) neurons.

172 The brain regulates fertility through gonadotropin-releasing hormone (GnRH) neurons.

173 l pathways contain cell bodies and fibers of gonadotropin-releasing hormone (GnRH) neurons.

174 ARH)), which drive the pulsatile activity of gonadotropin-releasing hormone (GnRH) neurons.

175 TEMENT The brain regulates fertility through gonadotropin-releasing hormone (GnRH) neurons.

176 f reproduction by acting on gonadotropes and gonadotropin-releasing hormone (GnRH) neurons.

177 icating abnormal steroid hormone feedback to gonadotropin-releasing hormone (GnRH) neurons.

178 , is enriched in cilia projecting from mouse gonadotropin-releasing hormone (GnRH) neurons.

179 o adulthood and depends upon the activity of gonadotropin-releasing hormone (GnRH) neurons.

180 yclicity, including long-acting analogues of gonadotropin-releasing hormone (GnRH) or oestradiol (adm

181 Most cells are immunoreactive for either gonadotropin-releasing hormone (GnRH) or RF-amide-like p

182 Gonadotropin-releasing hormone (GnRH) plays a central ro

183 Hypothalamic gonadotropin-releasing hormone (GnRH) plays a critical r

184 ty of kisspeptin-10 to elicit the release of gonadotropin-releasing hormone (GnRH) precociously, and

185 genes are expressed under the control of the gonadotropin-releasing hormone (GnRH) promoter has made

186 lth and wellbeing.SIGNIFICANCE STATEMENT The gonadotropin-releasing hormone (GnRH) pulse generator co

187 Fertility critically depends on the gonadotropin-releasing hormone (GnRH) pulse generator, a

188 Convergent evolution of the gonadotropin-releasing hormone (GnRH) receptor (GnRHR) p

189 Efforts to develop orally available gonadotropin-releasing hormone (GnRH) receptor antagonis

190 The gonadotropin-releasing hormone (GnRH) receptor is a drug

191 The human gonadotropin-releasing hormone (GnRH) receptor is evolut

192 Gonadotropin-releasing hormone (GnRH) receptor mutants f

193 o and caused cell death in cells bearing the gonadotropin-releasing hormone (GnRH) receptor, we could

194 ion as a measure of information transfer via gonadotropin-releasing hormone (GnRH) receptors (GnRHR)

195 Activation of gonadotropin-releasing hormone (GnRH) receptors inhibits

196 have specialized adaptations associated with gonadotropin-releasing hormone (GnRH) regulation to opti

197 ement during lethargus, by signaling through gonadotropin-releasing hormone (GnRH) related receptors.

198 NMDA and kisspeptins can stimulate gonadotropin-releasing hormone (GnRH) release after peri

199 ance and obesity are associated with reduced gonadotropin-releasing hormone (GnRH) release and infert

200 t the release of kisspeptin, which modulates gonadotropin-releasing hormone (GnRH) release from GnRH

201 Pulsatile gonadotropin-releasing hormone (GnRH) release is critica

202 A robust surge of gonadotropin-releasing hormone (GnRH) release triggers t

203 ing hormone, the latter reflecting increased gonadotropin-releasing hormone (GnRH) release.

204 important in modulating the tonic pulsatile gonadotropin-releasing hormone (GnRH) release.

205 of reproduction by brain-secreted pulses of gonadotropin-releasing hormone (GnRH) represents a longs

206 Neurons that produce gonadotropin-releasing hormone (GnRH) reside in the basa

207 otropic hypogonadism (IHH) due to defects of gonadotropin-releasing hormone (GnRH) secretion and/or a

208 Ovulation is initiated by a surge of gonadotropin-releasing hormone (GnRH) secretion by the b

209 suggests that MKRN3 is acting as a brake on gonadotropin-releasing hormone (GnRH) secretion during c

210 uberty is orchestrated by an augmentation of gonadotropin-releasing hormone (GnRH) secretion from a f

211 Acquisition of a mature pattern of gonadotropin-releasing hormone (GnRH) secretion from the

212 Gonadotropin-releasing hormone (GnRH) secretion is regul

213 in the regulation of pubertal and adulthood gonadotropin-releasing hormone (GnRH) secretion, and mut

214 wn to be key components in the regulation of gonadotropin-releasing hormone (GnRH) secretion.

215 try regulating estrogen negative feedback on gonadotropin-releasing hormone (GnRH) secretion.

216 uctive development and for the regulation of gonadotropin-releasing hormone (GnRH) secretion.

217 plicated in the neuroendocrine regulation of gonadotropin-releasing hormone (GnRH) secretion.

218 mammalian reproduction as key regulators of gonadotropin-releasing hormone (GnRH) secretion.

219 ic hypogonadism had a measurable response to gonadotropin-releasing hormone (GnRH) stimulation, sugge

220 Crz belongs to gonadotropin-releasing hormone (GnRH) superfamily, imply

221 ic focus was on ligands and receptors of the gonadotropin-releasing hormone (GnRH) superfamily.

222 A robust gonadotropin-releasing hormone (GnRH) surge is a prerequ

223 lmann syndrome is an inherited deficiency of gonadotropin-releasing hormone (GnRH) that is characteri

224 rmal puberty promotes the central release of gonadotropin-releasing hormone (GnRH) that, in turn, lea

225 revealed that IKK-beta and NF-kappaB inhibit gonadotropin-releasing hormone (GnRH) to mediate ageing-

226 The binding of hypothalamic gonadotropin-releasing hormone (GnRH) to the pituitary G

227 itary gonadotropin hormones are regulated by gonadotropin-releasing hormone (GnRH) via MAPK signaling

228 duce ciliated neurons that express genes for gonadotropin-releasing hormone (GnRH), a G-protein-coupl

229 ) designed to express an antibody that binds gonadotropin-releasing hormone (GnRH), a master regulato

230 and negative modulators drives secretion of gonadotropin-releasing hormone (GnRH), a neuropeptide th

231 (MCH), thyrotropin-releasing hormone (TRH), gonadotropin-releasing hormone (GnRH), and kisspeptin.

232 brain controls fertility through release of gonadotropin-releasing hormone (GnRH), but the mechanism

233 under the control of pulsatile hypothalamic gonadotropin-releasing hormone (GnRH), is essential for

234 Pubertal onset, initiated by pulsatile gonadotropin-releasing hormone (GnRH), only occurs in a

235 pite of the fact that hypothalamic levels of gonadotropin-releasing hormone (GnRH), pituitary release

236 sprouting in hypothalamic neurons secreting gonadotropin-releasing hormone (GnRH), the neuropeptide

237 blished that hypothalamic neurons, including gonadotropin-releasing hormone (GnRH), VP, OT, beta-endo

238 The decapeptide gonadotropin-releasing hormone (GnRH), which regulates r

239 ndent upon the appropriate neurosecretion of gonadotropin-releasing hormone (GnRH), yet the endogenou

240 Gonadotropin-releasing hormone (GnRH)-expressing neurons

241 Mammalian reproduction requires gonadotropin-releasing hormone (GnRH)-mediated signaling

242 lamic-pituitary-gonadal axis is dependent on gonadotropin-releasing hormone (GNRH)-stimulated synthes

243 ted as an increase in pulsatile secretion of gonadotropin-releasing hormone (GnRH).

244 neural network that drives the secretion of gonadotropin-releasing hormone (GnRH).

245 or action of the master reproductive hormone gonadotropin-releasing hormone (GnRH).

246 etic disorders associated with deficiency of gonadotropin-releasing hormone (GnRH).

247 genital defect in the secretion or action of gonadotropin-releasing hormone (GnRH).

248 in the pituitary gonadotrope by hypothalamic gonadotropin-releasing hormone (GnRH).

249 ontrols reproduction in mammals by secreting gonadotropin-releasing hormone (GnRH).

250 n mammals by secreting the 'master molecule' gonadotropin-releasing hormone (GnRH).

251 They secrete gonadotropin-releasing hormone (GnRH-1), communicate wit

252 rmone-releasing hormone (LHRH, also known as gonadotropin-releasing hormone [GnRH]), a key neurohormo

253 e reproduction requires pulsatile release of gonadotropin-releasing hormone (GnRH1) from the hypothal

254 In mammals, the receptor of the neuropeptide gonadotropin-releasing hormone (GnRHR) is unique among t

255 basis of selectivity of naturally occurring gonadotropin-releasing hormones (GnRHs) from different s

256 Gonadotropin-releasing hormone-I (GnRH-I) cells are loca

257 r fibers were observed in close proximity to gonadotropin-releasing hormone-I, dopamine, beta-endorph

258 ing hormone-I, dopamine, beta-endorphin, and gonadotropin-releasing hormone-II neurons in the preopti

259 a neuropeptide homologous to the vertebrate gonadotropin-releasing hormone, is downregulated as work

260 ty and infertility as a result of defects in gonadotropin-releasing hormone neuron development or fun

261 st gonadotropin responses, suggesting normal gonadotropin-releasing hormone neuronal and gonadotrope

262 ive control, including direct innervation of gonadotropin releasing hormone neurons.

263 se neurons also precede the emergence of the gonadotropin-releasing hormone neurons and ensheathing g

264 lamic nucleus and send axonal projections to gonadotropin-releasing hormone neurons and regulate repr

265 In immortalized immature gonadotropin-releasing hormone neurons endogenously expr

266 Notably, TRPC1 suppressed the migration of gonadotropin-releasing hormone neurons without affecting

267 Z formed appositions with orexin neurons and gonadotropin-releasing hormone neurons, two cell populat

268 sspeptin neurons relay estradiol feedback to gonadotropin-releasing hormone neurons, which regulate t

269 ive Ca(2+) imaging in the nerve terminals of gonadotropin-releasing hormone neurons.

270 ed hypothalamic cells and immortalized GnRH (gonadotropin-releasing hormone) neurons (GT1-7 cells) tr

271 g hormone, growth hormone-releasing hormone, gonadotropin-releasing hormone, oxytocin, somatostatin,

272 cant relation between the number of doses of gonadotropin-releasing hormone received during the 12 mo

273 Pharmacoperones of the gonadotropin releasing hormone receptor (GnRHR) have eff

274 ere designed and synthesized as potent human gonadotropin releasing hormone receptor antagonists.

275 The gonadotropin releasing hormone receptor, because of its

276 in-conjugated gold nanorods (gGNRs) promotes gonadotropin releasing hormone receptor-mediated interna

277 eptide agonists and antagonists of the human gonadotropin-releasing hormone receptor (GnRH-R) are wid

278 of a series thienopyrimidinediones with the gonadotropin-releasing hormone receptor (GnRH-R).

279 targeting of tumor cells overexpressing the gonadotropin-releasing hormone receptor (GnRH-R).

280 Long-term suppression of the gonadotropin-releasing hormone receptor (GnRHR) is an im

281 on in pedigree 1 and a compound heterozygous gonadotropin-releasing hormone receptor (GNRHR) mutation

282 icipate in partitioning mutant conformers of gonadotropin-releasing hormone receptor (GnRHR), a G pro

283 mines bearing a butyric acid as potent human gonadotropin-releasing hormone receptor (hGnRH-R) antago

284 oral contraceptives/antagonization of human gonadotropin-releasing hormone receptor [hGnRH-R] activi

285 s capable of rescuing the activity of mutant gonadotropin-releasing hormone receptor or GnRHR which,

286 he C-terminal domains of either GluR1 or the gonadotropin-releasing hormone receptor permits efficien

287 y of several potent antagonists of the human gonadotropin-releasing hormone receptor.

288 regulation (reduced pituitary sensitivity to gonadotropin-releasing hormone), reduced conception rate

289 oter activity, highlighting a role of SET in gonadotropin-releasing hormone regulation of gene expres

290 from the Kiss1 promoter disrupted pulsatile gonadotropin-releasing hormone release, delayed puberty

291 Three prepro-gonadotropin-releasing hormones, seabream GnRH (sbGnRH),

292 Gene expression of salmon gonadotropin-releasing hormone (sGnRH) and NR1 increased

293 ns, suggesting sensitization of the NKB-NK3R-gonadotropin-releasing hormone signaling pathway under m

294 nsgenic mice, and is released in response to gonadotropin-releasing hormone, similar to LH.

295 lular: oxytocin, vasopressin; parvicellular: gonadotropin-releasing hormone, somatostatin, thyrotropi

296 androgen receptors, and by the hypothalamic gonadotropin-releasing hormone through activation of PKA

297 olic, and other factors control secretion of gonadotropin-releasing hormone to determine initiation o

298 uires the pulsatile, coordinated delivery of gonadotropin-releasing hormone to the pituitary and the

299 In the BPD mouse model, gonadotropin-releasing hormone was increased in females

300 uctive function requires timely secretion of gonadotropin-releasing hormone, which is controlled by a