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1 al potent treatment for antibiotic-resistant gonococcal infection.
2 roteins was observed to occur in response to gonococcal infection.
3 ent, a physiologically relevant state during gonococcal infection.
4 aneous bacterial peritonitis, and outpatient gonococcal infection.
5 al to be effective oral therapeutics against gonococcal infection.
6 erapeutic and prophylactic compounds against gonococcal infection.
7 ted a protective effect against experimental gonococcal infection.
8 play a role in tubal scarring in response to gonococcal infection.
9 vaccines provided partial protection against gonococcal infection.
10 immune responses in a mouse model of genital gonococcal infection.
11 sion of IgD(+)CD27(+) B cells in response to gonococcal infection.
12 oluble 17beta-estradiol to promote long-term gonococcal infection.
13 rations in host innate responses may enhance gonococcal infection.
14 ffects of H(2)O(2)-producing lactobacilli on gonococcal infection.
15 lain species-specific restriction of natural gonococcal infection.
16 mens from female subjects with uncomplicated gonococcal infection.
17  a key arm of innate immune defenses against gonococcal infection.
18 ge of nucleic acid amplification testing for gonococcal infection.
19 the human host, the only known reservoir for gonococcal infection.
20 BV), often without concomitant chlamydial or gonococcal infection.
21  implications for understanding asymptomatic gonococcal infection.
22 gnificantly to the reduction in the rates of gonococcal infection.
23  samples from individuals with no history of gonococcal infection.
24 ls of IL-6 and IL-8 cytokines in response to gonococcal infection.
25 e by LCR following therapy for uncomplicated gonococcal infection.
26 ated in the pathogenesis of various forms of gonococcal infection.
27 standard that defines AZI susceptibility for gonococcal infections.
28 s to present a challenge to the treatment of gonococcal infections.
29 (14.8% of the 608) had a total of 112 repeat gonococcal infections.
30 gical memory is not induced by uncomplicated gonococcal infections.
31 reatment is associated with subsequent AZMRS gonococcal infections.
32 dence, and the cumulative number of incident gonococcal infections.
33  96.9 to 100% using LCA for the detection of gonococcal infections.
34 et for beta-lactam antibiotics used to treat gonococcal infections.
35 G, which for over 40 years was used to treat gonococcal infections.
36 rkers, a continuing problem for treatment of gonococcal infections.
37 arm of the innate immune system that combats gonococcal infections.
38 nd a collection of strains from disseminated gonococcal infections.
39 arynx, possibly modulating meningococcal and gonococcal infections.
40 ent in the prevalence of both chlamydial and gonococcal infections.
41 ases ranging from urethritis to disseminated gonococcal infections.
42       Positive test result for chlamydial or gonococcal infections.
43 o = 0.50; 95% CI, 0.28 to 0.88; P = .02) and gonococcal infections (48 vs 54 participants, respective
44  invasive meningococcal B (MenB) disease and gonococcal infections 5 years after the program introduc
45                        For identification of gonococcal infections, all assays performed similarly (P
46 ltaABR dOMVs can protect mice from ascending gonococcal infection and (2) vaccination route influence
47  proven to be an efficient means of studying gonococcal infection and focusing vaccine development.
48 hat DeltaABR dOMVs protect against ascending gonococcal infection and that cellular and functional an
49 gulated tbpA and tbpB genes are expressed in gonococcal infection and that male subjects with mucosal
50 mportant tool for epidemiological studies of gonococcal infection and transmission.
51 a FSWs, we evaluated factors associated with gonococcal infection and with gonococcal antimicrobial r
52 and public health strategy for management of gonococcal infections and antimicrobial resistance.
53      Rapid point-of-care tests that diagnose gonococcal infections and identify susceptibility to ant
54 ceiving eculizumab on their risk for serious gonococcal infections and perform screening for sexually
55 her insights into the species specificity of gonococcal infections and proof-of-concept of a novel th
56 ere calculated separately for chlamydial and gonococcal infections and were stratified by assay and p
57 ulation aged 18 to 35 years has an untreated gonococcal infection, and 3.0% (SE, 0.8%) is estimated t
58 ialoglycoprotein receptor increase following gonococcal infection, and, like the primary cells, the i
59 h benefits in settings where the majority of gonococcal infections are ciprofloxacin susceptible.
60                               Chlamydial and gonococcal infections are important causes of pelvic inf
61 ensitive for the detection of chlamydial and gonococcal infection at the rectal site than is culture.
62 ay have resulted in decreased chlamydial and gonococcal infections at the population level.
63 ral barriers to screening for chlamydial and gonococcal infections, but most test samples are obtaine
64                                              Gonococcal infections cause significant morbidity, parti
65                                  Symptomatic gonococcal infection, caused by the pathogen Neisseria g
66                                  Symptomatic gonococcal infection, caused exclusively by the human-sp
67              The gel group had 116 diagnosed gonococcal infections, chlamydial infections, or both fo
68 ined from female subjects with uncomplicated gonococcal infection corroborated our in vitro findings
69 ent of a C1q-dependent experimental model of gonococcal infection created an opportunity to evaluate
70 for simultaneous detection of chlamydial and gonococcal infections demonstrated superior sensitivity
71  specific vaginal bacteria and chlamydial or gonococcal infection detected by strand displacement ass
72                                 Disseminated gonococcal infection (DGI) and pelvic inflammatory disea
73 n increase in reported cases of disseminated gonococcal infection (DGI).
74 Neisseria gonorrhoeae can cause disseminated gonococcal infection (DGI).
75 the isolates from patients with disseminated gonococcal infection (DGI).
76 uce the serious complication of disseminated gonococcal infection (DGI).
77                                 Disseminated gonococcal infections (DGIs) are thought to be uncommon;
78                    Also, previous history of gonococcal infection did not increase antibody levels in
79                                              Gonococcal infection did not induce evident immunity to
80                                              Gonococcal infections do not elicit protective immunity,
81 n with the same serovar can occur, and prior gonococcal infection does not alter the Ig response upon
82  of participants with a laboratory-confirmed gonococcal infection during the 36-month follow-up.
83 nfection and that male subjects with mucosal gonococcal infections exhibit antibodies to these protei
84 of ocular prophylaxis, transmission rates of gonococcal infection from mother to newborn are 30% to 5
85 tal model that may mimic the transmission of gonococcal infection from mother to the fetus during pre
86 ariable pathophysiology of meningococcal and gonococcal infections given that after an initial exposu
87                 Screening for chlamydial and gonococcal infection has been strongly recommended for a
88         Here, we demonstrate that women with gonococcal infections have levels of sialidases present
89 ted bacterial growth and was able to treat a gonococcal infection in a human cervical epithelial cell
90 the contributions of MisR and MisS (CpxA) to gonococcal infection in a murine model of cervicovaginal
91 cific antibiotic susceptibility profile of a gonococcal infection in a patient.
92 d genes are expressed in vivo during mucosal gonococcal infection in men, which suggests that this or
93  are invaded by Neisseria gonorrhoeae during gonococcal infection in men.
94  not constitutive activation is required for gonococcal infection in mice.
95 erefore the potential for spread, (b) use of gonococcal infection in the animal model system to study
96                                   The RR for gonococcal infection in the gel group vs the condom grou
97  and Prevention STD treatment guidelines for gonococcal infections in adolescents and adults.
98  for identifying and treating chlamydial and gonococcal infections in adolescents and young adults wh
99            VE for 2-dose vaccination against gonococcal infections in adolescents was 39.1% (95% CI,
100  protection against the first and subsequent gonococcal infections in adolescents was observed up to
101 the evidence on screening for chlamydial and gonococcal infections in asymptomatic patients from stud
102 ies for screening and testing chlamydial and gonococcal infections in individuals aged 15 to 21 years
103 he prevalence of asymptomatic chlamydial and gonococcal infections in male and female military popula
104  specificity of polymerase chain reaction to gonococcal infections in men was 90.4%.
105 rnative treatment regimen, and management of gonococcal infections in persons with severe cephalospor
106 e regarding the prevalence of chlamydial and gonococcal infections in the general young adult populat
107            The estimated number of untreated gonococcal infections in the population (9241; SE, 2441)
108                               Chlamydial and gonococcal infections in women are usually asymptomatic
109 ions in men; and 55.6%, 91.3%, and 84.9% for gonococcal infections in women.
110 n vivo would require the presence of a mixed gonococcal infection, in which an individual is concurre
111                                              Gonococcal infection induced a significant increase in s
112                              Initiation of a gonococcal infection involves attachment of Neisseria go
113                 The role of autolysis during gonococcal infection is not known, but possible advantag
114                             During and after gonococcal infection, local and systemic antigonococcal
115 thout new effective antibiotics, untreatable gonococcal infections loom as a real possibility.
116 is cytokine has no previous association with gonococcal infection nor pelvic inflammatory disease and
117                                     Although gonococcal infection of B cells produced small amounts o
118 mphenicol demonstrated that the Opa-mediated gonococcal infection of Chang cells required bacterial p
119 gested a role for the cervical epithelium in gonococcal infection of females; however, the nature of
120  that an intact MisRS system is required for gonococcal infection of mice.
121 ansferrin, nonhemoglobin iron sources during gonococcal infection of the female genital tract.
122                                              Gonococcal infection of the male urogenital tract has be
123 infection of females; however, the nature of gonococcal infection of the normal uterine cervix remain
124 irectly influences B cells, we observed that gonococcal infection prolonged viability of primary huma
125                                         Most gonococcal infections remain localized to the genital tr
126  Neisseria gonorrhoeae to cause disseminated gonococcal infection requires that such strains resist t
127 incidence rates, and cumulative incidence of gonococcal infection, resistance dynamics (time to 5% pr
128 were phenotypically similar and responded to gonococcal infection similarly to primary cells.
129                                              Gonococcal infection studies and electron microscopy sho
130                                          For gonococcal infection, the overall prevalence per 100 pop
131               In a laboratory model of mixed gonococcal infections, the por type of one strain could
132 tudy, we examined the immune response during gonococcal infection to the individual transferrin bindi
133 resistance and minimise the number of annual gonococcal infections (under baseline model conditions,
134                The VE against the subsequent gonococcal infection was 27.0% (adjusted hazard ratio, 0
135        In this work, a cell culture model of gonococcal infection was adapted to examine the effects
136                    By multivariate analysis, gonococcal infection was associated with sex with a new
137                    VE against the subsequent gonococcal infection was measured using a Cox regression
138                              Specificity for gonococcal infections was >/= 99.8%.
139 ence of coinfection with both chlamydial and gonococcal infections was 0.030% (95% CI, 0.18%-0.49%).
140      Testing for asymptomatic chlamydial and gonococcal infections was highly accurate at most anatom
141 regulated genes in vivo during uncomplicated gonococcal infection, we examined gene expression profil
142 A microarrays and a tissue culture model for gonococcal infection, we examined global changes in gene
143      To better understand the role of Opa in gonococcal infections, we created and characterized a de
144 imilar individuals with laboratory-confirmed gonococcal infection were compared before, during, and a
145 inal pH, positive whiff test, and concurrent gonococcal infection were positively associated with TV
146                                           No gonococcal infections were detected.
147 es were evaluated, and 281 chlamydial and 69 gonococcal infections were identified.
148                                   Cumulative gonococcal infections were reduced by a median of 14.5%
149          Eighteen men had 20 incident rectal gonococcal infections, which persisted for 2-23 weeks; 3
150             However, among men with urethral gonococcal infections who attended one sexually transmit
151         We developed a transmission model of gonococcal infection with multiple resistant strains to
152                             The unsialylated gonococcal infections, with a median incubation time of
153 genic diversity and allow the persistence of gonococcal infection within the human population.

 
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