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1 of NMO lesions in mice made neutrophilic by granulocyte colony stimulating factor.
2 duce proinflammatory genes, such as CCL2 and granulocyte colony-stimulating factor.
3 ing recombinant zebrafish erythropoietin and granulocyte colony-stimulating factor.
4 obilization induced by a CXCR4 antagonist or granulocyte colony-stimulating factor.
5 nia in G6PC3 deficiency and responds well to granulocyte colony-stimulating factor.
6 th increased levels of circulating IL-17 and granulocyte colony-stimulating factor.
7 ecreted protein Bv8, which is upregulated by granulocyte colony-stimulating factor.
8 (ethylene glycol) modified recombinant human granulocyte-colony stimulating factor.
9 nors, and is synergistic in combination with granulocyte-colony stimulating factor.
10 den but was augmented by coadministration of granulocyte-colony stimulating factor.
11 usion, tumor necrosis factor inhibitors, and granulocyte colony-stimulating factors.
12 of up to two 28-day cycles); or intravenous granulocyte colony-stimulating factor (300 mug/m(2) per
13 tarabine 2,000 mg/m(2)/dose on days 1-5; and granulocyte-colony stimulating factor 5 ug/kg/dose, days
14 3 ligand, interleukin-3, interleukin-6, and granulocyte colony-stimulating factor (5 GFs) either alo
16 in systemic inflammatory markers, including granulocyte colony-stimulating factor (adjusted OR 2.8 [
17 nd quantitatively more HSPC from the BM than granulocyte colony-stimulating factor alone, including i
19 bute to an infectious phenotype such as anti-granulocyte colony stimulating factor and anti-IFN-alpha
21 the SC group, CD34+ cells were mobilized by granulocyte colony-stimulating factor and collected via
22 n 1beta, monocyte chemotactic protein 1, and granulocyte colony-stimulating factor and decreased leve
23 ssor cells, which release elevated levels of granulocyte colony-stimulating factor and granulocyte ma
24 CCL1, CCL2, CCL3, CCL5), and growth factors (granulocyte colony-stimulating factor and granulocyte-ma
25 Within 4 days, bone marrow cells cultured in granulocyte colony-stimulating factor and granulocyte-ma
26 l severity (e.g. decreased responsiveness to granulocyte colony-stimulating factor and increased leuk
27 ficient) mice, they maintained expression of granulocyte colony-stimulating factor and leukemia inhib
29 tion prevented CXCL12 induction and improved granulocyte-colony stimulating factor and ischemia-induc
30 e primed macrophages in adulthood and raised granulocyte-colony stimulating factor and neutrophil cou
31 l expressed and presumably secreted), G-CSF (granulocyte-colony-stimulating factor) and MMP2 (matrix
32 d increased BAL fluid IL-1alpha, IL-6, IL-8, granulocyte colony-stimulating factor, and GM-CSF levels
33 luid myeloperoxidase, IL-8, IL-1alpha, IL-6, granulocyte colony-stimulating factor, and GM-CSF levels
34 1983) or ADE versus fludarabine, cytarabine, granulocyte colony-stimulating factor, and idarubicin (F
35 , and etoposide; or fludarabine, cytarabine, granulocyte colony-stimulating factor, and idarubicin.
36 select interleukins, growth factors such as granulocyte colony-stimulating factor, and l-ferritin, h
37 reduced concentrations of interleukin-1beta, granulocyte colony-stimulating factor, and matrix metall
38 CCR2 and CX3CR1 up-regulation, whereas CCL1, granulocyte colony-stimulating factor, and MIP1alpha wer
39 ") CLAG-M (cladribine, high-dose cytarabine, granulocyte colony-stimulating factor, and mitoxantrone)
40 h significantly higher IL-1beta, IL-6, IL-8, granulocyte colony-stimulating factor, and monocyte chem
41 , soluble vascular cell adhesion molecule-1, granulocyte colony-stimulating factor, and soluble Fas.
42 ls produced higher levels of interleukin-10, granulocyte colony-stimulating factor, and transforming
43 icin 120 mg/m(2) and ifosfamide 9 g/m(2) and granulocyte colony-stimulating factor (arm A) or to rece
44 a levels of interleukin-1alpha and -beta and granulocyte-colony stimulating factor as well as increas
45 atory protein 1alpha, interleukin 1beta, and granulocyte colony-stimulating factor, as well as interl
46 on of the knob-domain of Ab-coil with bovine granulocyte colony-stimulating factor (bGCSF) results in
47 and many were prevented by interleukin-1 or granulocyte colony-stimulating factor blockade, revealin
50 locyte-macrophage colony-stimulating factor, granulocyte colony-stimulating factor, CCL2, and CCL5, w
51 O mice had decreased concentrations of IL-6, granulocyte colony stimulating factor, chemokine CXC lig
52 pheral blood CD34(+) cells were mobilized by granulocyte colony stimulating factor, collected via aph
53 fts were mobilized with cyclophosphamide and granulocyte colony-stimulating factor, collected by peri
54 tely linked to innate immune cell expansion (granulocyte colony-stimulating factor), cytotoxicity (in
55 a monocyte differentiation program in human granulocyte colony-stimulating factor-dependent neutroph
56 ta show that hematopoietic stress, including granulocyte colony-stimulating factor, do not increase t
57 mune illness, and stroke in donors receiving granulocyte colony-stimulating factor during this period
58 otein-1, monocyte chemotactic protein-3, and granulocyte-colony stimulating factor during the acute p
60 d fluorouracil 750 mg/m2 IV over 5 days with granulocyte colony-stimulating factor, every 3 weeks).
62 mly assigned to fludarabine, cytarabine, and granulocyte colony-stimulating factor (FLAG) or to FLAG
63 nsivist-led service, meropenem, and adjuvant granulocyte colony-stimulating factor for confirmed meli
64 l transplantation, with a five-day course of granulocyte colony stimulating factor (G-CSF) as the mos
66 gation of BSA, beta2-microglobulin (beta2m), granulocyte colony stimulating factor (G-CSF), and three
67 d neutrophil-activating protein 78 (ENA-78), granulocyte colony stimulating factor (G-CSF), granulocy
68 Eighteen molecules (interleukine (IL)-1beta, granulocyte colony stimulating factor (G.CSF), IL-13, IL
70 with CSF3R truncation mutations; therapeutic granulocyte colony-stimulating factor (G-CSF) administra
71 to the treatment when forced into cycle via granulocyte colony-stimulating factor (G-CSF) administra
72 the stabilization of the therapeutic protein granulocyte colony-stimulating factor (G-CSF) against st
73 ersely, the myeloproliferation was driven by granulocyte colony-stimulating factor (G-CSF) and admini
75 phil expansion and migration by antagonizing granulocyte colony-stimulating factor (G-CSF) and chemok
76 yte transfusions from donors stimulated with granulocyte colony-stimulating factor (G-CSF) and dexame
78 sed body weight, and increased production of granulocyte colony-stimulating factor (G-CSF) and IL-1be
79 -response genes in mucosal RNA and increased granulocyte colony-stimulating factor (G-CSF) and IP-10
80 o myeloid lineage specification, we compared granulocyte colony-stimulating factor (G-CSF) and macrop
81 Rgamma) produced severely reduced amounts of granulocyte colony-stimulating factor (G-CSF) and of nit
82 AE, in response to systemic up-regulation of granulocyte colony-stimulating factor (G-CSF) and the EL
83 stigates the potential protective effects of granulocyte colony-stimulating factor (G-CSF) and underl
84 or neutrophil response despite high doses of granulocyte colony-stimulating factor (G-CSF) are at gre
85 independence 1 (Gfi1) and the growth factor granulocyte colony-stimulating factor (G-CSF) are indivi
86 CAIX is indispensable for the production of granulocyte colony-stimulating factor (G-CSF) by hypoxic
87 ith the clinically relevant mobilizing agent granulocyte colony-stimulating factor (G-CSF) caused rap
88 ), along with local interleukin (IL)-12, and granulocyte colony-stimulating factor (G-CSF) concentrat
89 , MIP-1beta, and IL-15 and semen eotaxin and granulocyte colony-stimulating factor (G-CSF) concentrat
90 ation of bone marrow-derived stem cells with granulocyte colony-stimulating factor (G-CSF) could prom
91 is was 0.4 x 10(9)/L, and median ANC without granulocyte colony-stimulating factor (G-CSF) during fol
93 Finally, the concurrent administration of granulocyte colony-stimulating factor (G-CSF) enhanced R
95 marrow into peripheral blood by the cytokine granulocyte colony-stimulating factor (G-CSF) has become
99 d the safety and efficacy of plerixafor with granulocyte colony-stimulating factor (G-CSF) in mobiliz
101 ne recommended primary prophylaxis (PP) with granulocyte colony-stimulating factor (G-CSF) instead of
104 stem/progenitor cell (HSPC) mobilization by granulocyte colony-stimulating factor (G-CSF) is mediate
107 ocyte chemoattractive protein 1 (MCP-1), and granulocyte colony-stimulating factor (G-CSF) levels in
111 antithymocyte globulin (ATG) plus pegylated granulocyte colony-stimulating factor (G-CSF) preserves
112 Investigations in this model revealed that granulocyte colony-stimulating factor (G-CSF) produced b
113 was to describe the effect of antibiotic and granulocyte colony-stimulating factor (G-CSF) prophylaxi
115 who were administered T-BEP received primary granulocyte colony-stimulating factor (G-CSF) prophylaxi
117 opoietic counterpart, 2 designs bound to the granulocyte colony-stimulating factor (G-CSF) receptor a
118 ed downregulation of the Csf3r gene, reduced granulocyte colony-stimulating factor (G-CSF) receptor l
119 1 (LEF-1), which plays a definitive role in granulocyte colony-stimulating factor (G-CSF) receptor-t
120 of-function mutations in CSF3R, encoding the granulocyte colony-stimulating factor (G-CSF) receptor.
121 elopoiesis prevented HSPC mobilization after granulocyte colony-stimulating factor (G-CSF) stimulatio
122 es the risk of myelotoxicity and may require granulocyte colony-stimulating factor (G-CSF) support.
123 greatest differential was with the cytokine granulocyte colony-stimulating factor (G-CSF) that cause
124 BM) to the blood circulation by the cytokine granulocyte colony-stimulating factor (G-CSF) through co
125 rleukin-17 (IL-17) induced the expression of granulocyte colony-stimulating factor (G-CSF) through nu
126 lls for transplantation, use of the cytokine granulocyte colony-stimulating factor (G-CSF) to mobiliz
127 mononuclear cells (BMMC) and the ability of granulocyte colony-stimulating factor (G-CSF) to mobiliz
128 vances in the therapeutic use of recombinant granulocyte colony-stimulating factor (G-CSF) to promote
129 ulate MTA1 expression in neuronal cells, and granulocyte colony-stimulating factor (G-CSF) was chosen
130 erleukin 1 receptor antagonist (IL-1Ra), and granulocyte colony-stimulating factor (G-CSF) were raise
131 ls were randomized to receive plerixafor and granulocyte colony-stimulating factor (G-CSF), agents kn
132 er levels of cytokines/chemokines, including granulocyte colony-stimulating factor (G-CSF), and enhan
133 ony formation, decreased in vivo response to granulocyte colony-stimulating factor (G-CSF), and impai
134 a), tumor necrosis factor alpha (TNF-alpha), granulocyte colony-stimulating factor (G-CSF), and inter
137 lassemic patients mobilized with hydroxyurea+granulocyte colony-stimulating factor (G-CSF), G-CSF, Pl
138 interleukin 1beta (IL-1beta), IL-6, IL-17A, granulocyte colony-stimulating factor (G-CSF), granulocy
140 15, tumor necrosis factor alpha (TNF-alpha), granulocyte colony-stimulating factor (G-CSF), interfero
141 LCs expressed significantly higher levels of granulocyte colony-stimulating factor (G-CSF), interleuk
143 IL-15), IL-18, gamma interferon (IFN-gamma), granulocyte colony-stimulating factor (G-CSF), monocyte
144 Tlr4 and Myd88 and are the primary source of granulocyte colony-stimulating factor (G-CSF), the key g
147 ophil count and their priming is mediated by granulocyte colony-stimulating factor (G-CSF), which acc
148 ts with WHIM syndrome are often treated with granulocyte colony-stimulating factor (G-CSF), which can
149 cally, TLR4 and NOD1 synergistically induced granulocyte colony-stimulating factor (G-CSF), which was
150 4+ peripheral-blood stem cells, mobilized by granulocyte colony-stimulating factor (G-CSF), which wer
153 Functional studies demonstrated elevated granulocyte colony-stimulating factor (G-CSF)-induced pr
154 ately 35% at 1 year after transplantation of granulocyte colony-stimulating factor (G-CSF)-mobilized
155 nse to damaged cells, promoting an excessive granulocyte colony-stimulating factor (G-CSF)-regulated
156 capitulates a Gfi1 loss-of-function block to granulocyte colony-stimulating factor (G-CSF)-stimulated
157 opulation that appears in the circulation of granulocyte colony-stimulating factor (G-CSF)-treated do
162 MIP-2)/CXCL2, IP-10/CXCL10, MIP-1alpha/CCL3, granulocyte colony-stimulating factor (G-CSF)/CSF3, CXCL
163 increases in IL-1alpha/beta, IL-6/IL-12(p40)/granulocyte colony-stimulating factor (G-CSF)/keratinocy
164 ve mice but can be induced by treatment with granulocyte colony-stimulating factor (G-CSF/Csf3) or by
166 he combination of stem cell factor (SCF) and granulocyte-colony stimulating factor (G-CSF) (SCF+G-CSF
167 ies have found that the neuroactive cytokine granulocyte-colony stimulating factor (G-CSF) alters coc
168 he neuroprotective effects of the cytokines, granulocyte-colony stimulating factor (G-CSF) and stem c
169 Previous work from our group has identified granulocyte-colony stimulating factor (G-CSF) as a neuro
172 by a novel mechanism in which tumor-derived granulocyte-colony stimulating factor (G-CSF) directs ex
173 few published cases, there is evidence that granulocyte-colony stimulating factor (G-CSF) in patient
175 ence coverage was obtained by reducing human granulocyte-colony stimulating factor (G-CSF) prior to M
179 of four drugs: antithymocyte globulin (ATG), granulocyte-colony stimulating factor (G-CSF), a dipepti
180 her key inflammatory factors including IL-8, granulocyte-colony stimulating factor (G-CSF), IL-33, IL
181 he metastatic tumors we examined overexpress granulocyte-colony stimulating factor (G-CSF), which exp
182 kaemia (AML) samples, and downregulated upon granulocyte-colony stimulating factor (G-CSF)- mediated
183 ncouraging outcomes after transplantation of granulocyte-colony stimulating factor (G-CSF)- primed un
184 transfer of transplant (tx), tumor (tm), and granulocyte-colony stimulating factor (g-csf)-expanded M
187 aly in adulthood due to the up-regulation of granulocyte-colony stimulating factor (G-CSF); these eff
188 is, we identify the proinflammatory cytokine granulocyte-colony-stimulating factor (G-CSF or Csf-3) a
189 -term repopulating capacity as well as hyper granulocyte-colony-stimulating factor (G-CSF)-induced mo
190 ]) and differences (interleukin-6 [IL-6] and granulocyte colony-stimulating factor [G-CSF]) elicited
191 okines (interferon gamma, interleukin 6, and granulocyte colony-stimulating factor [G-CSF]) were high
192 lammatory mediators (including CXCL1, CXCL2, granulocyte colony-stimulating factor [G-CSF], interleuk
193 models: (i) three different ligands based on granulocyte colony-stimulating factor GCSF homo-dimeric
194 al use of zebrafish Tpo, erythropoietin, and granulocyte colony stimulating factor (Gcsf) allowed the
195 ignificant controversy surrounds the role of granulocyte colony stimulating factor (GCSF) in levamiso
196 stered with Flt3 ligand (FL) (days 1-10) and granulocyte colony-stimulating factor (GCSF) (days 4-10)
199 GSD-Ib patients with incomplete response to granulocyte colony-stimulating factor (GCSF) treatment d
200 89) were randomized to 1) ATG and pegylated granulocyte colony-stimulating factor (GCSF), 2) ATG alo
201 nterleukin (IL)-4, IL-6, IL-8, IL-10, IL-15, granulocyte colony-stimulating factor (GCSF), MCP-1, tum
202 lin (ATG), as well as the mobilization agent granulocyte colony-stimulating factor (GCSF), to reverse
203 (6S,7S,8S)-1a and (6R,7S,8S)-1b inhibited granulocyte colony-stimulating factor (GCSF)-stimulated
206 nterleukin-2, IFN alfa-2b (IFN-alpha-2b) and granulocyte colony-stimulating factor given every 21 day
207 erleukin-17, basic fibroblast growth factor, granulocyte colony-stimulating factor, granulocyte macro
208 ulating levels of IL-1beta, IL-1alpha, IL-6, granulocyte-colony stimulating factor, granulocyte-macro
209 emonstrate that a post-BMT regimen of either granulocyte-colony stimulating factor, growth hormone, p
212 and exhibited significantly lower levels of granulocyte colony stimulating factor, IL-8, macrophage
213 of machrophage colony stimulating factor and granulocyte colony stimulating factor in splenic macroph
215 gG deposits and the pathophysiologic role of granulocyte colony-stimulating factor in exacerbation of
216 CCL2/monocyte chemoattractant protein 1, and granulocyte colony-stimulating factor in lung homogenate
217 2 and 8 of a 21-day cycle with prophylactic granulocyte colony-stimulating factor in the heavily pre
218 ed in diabetic mice lacking the receptor for granulocyte colony-stimulating factor in their marrow-de
219 f serum cytokines showed increased levels of granulocyte colony-stimulating factor in transgenic anim
220 r stem cell mobilization in combination with granulocyte-colony stimulating factor in patients with n
221 dvent of hematopoietic cytokines (especially granulocyte colony-stimulating factor) in shortening the
225 mobilized more circulating neutrophils after granulocyte colony-stimulating factor infusion compared
226 sis demonstrated significant upregulation of granulocyte colony-stimulating factor, interferon gamma,
227 d plasma levels of cytokines (interleukin 6, granulocyte colony-stimulating factor, interleukin 1 rec
228 e elevated in the posttransplantation phase: granulocyte colony-stimulating factor, interleukin-8, ti
229 eraction chromatography of recombinant human granulocyte colony stimulating factor is demonstrated.
231 A-EX) mice had less IL-6, interleukin 12p40, granulocyte colony-stimulating factor, keratinococyte-de
232 te proinflammatory cytokines (interleukin-6, granulocyte colony-stimulating factor, keratinocyte chem
233 rotein-1, macrophage inflammatory protein-2, granulocyte colony-stimulating factor, keratinocyte chem
234 rrelated with the production of 5 cytokines (granulocyte colony-stimulating factor, keratinocyte-deri
235 for 5 days on days 10-14; two cycles), with granulocyte-colony stimulating factor (lenograstim) supp
236 he treatment diminished circulating IL-6 and granulocyte colony-stimulating factor levels and limited
237 locyte-macrophage colony-stimulating factor, granulocyte colony-stimulating factor, macrophage colony
238 ith no prophylactic use of recombinant human granulocyte-colony stimulating factor mandated in this g
239 are (either fludarabine plus cytarabine plus granulocyte colony-stimulating factor, mitoxantrone plus
240 nsplantation (HCT), either marrow (n = 4) or granulocyte-colony stimulating factor mobilized peripher
241 D161(hi) cells from umbilical cord blood and granulocyte colony stimulating factor-mobilized leukaphe
242 e globulin (ATG) followed by the infusion of granulocyte colony-stimulating factor-mobilized grafts.
243 nditioned with 2-Gy TBI and given autologous granulocyte colony-stimulating factor-mobilized leukocyt
244 Following the infusion of PMF and normal granulocyte colony-stimulating factor-mobilized peripher
245 increasing use of mismatched, unrelated, and granulocyte colony-stimulating factor-mobilized peripher
246 tation of primary functional human MEPs from granulocyte colony-stimulating factor-mobilized peripher
247 ct of a set of immune cells contained within granulocyte colony-stimulating factor-mobilized peripher
248 er serum levels of interleukin (IL)-6, IL-8, granulocyte colony-stimulating factor, monocyte chemoatt
249 ecreased interferon-inducible protein 10 and granulocyte colony-stimulating factor more than did nore
251 ary vaccine recipients had maximal levels of granulocyte-colony-stimulating factor on days 6-7 after
252 iesis-stimulating agents in combination with granulocyte colony-stimulating factor or all-trans-retin
255 sly every 3 weeks, plus prednisone daily and granulocyte colony-stimulating factor) or the other andr
257 0.002), IL-6 (P = 0.002), IL-8 (P = 0.026), granulocyte colony-stimulating factor (P = 0.002), and m
259 ene glycol) (PEG), such as recombinant human granulocyte-colony stimulating factor (PEGylated rhG-CSF
260 lone, patients receiving EPO with or without granulocyte colony-stimulating factor plus SC had improv
261 fety of erythropoietin (EPO) with or without granulocyte colony-stimulating factor plus supportive ca
264 tors for erythropoietin, thrombopoietin, and granulocyte colony-stimulating factor, provided an expla
265 -derived CD34(+) cells mobilized in blood by granulocyte colony-stimulating factor, purified WAT-CD34
266 demonstrate that activating mutations in the granulocyte colony stimulating factor receptor (CSF3R),
268 hosphatase-1 (SHP-1)-dependent inhibition of granulocyte colony-stimulating factor receptor (G-CSFR)
269 companied by mutations in CSF3R encoding the granulocyte colony-stimulating factor receptor (G-CSFR)
271 s and monocytes is primarily governed by the granulocyte colony-stimulating factor receptor family (C
272 me high frequency recurrent mutations in the granulocyte colony-stimulating factor receptor gene CSF3
273 cytokine receptors (erythropoietin receptor, granulocyte colony-stimulating factor receptor, and MPL)
274 of type I and II cytokine receptors, except granulocyte colony-stimulating factor receptor, which su
275 in the secretory pathway and is required for granulocyte colony-stimulating factor receptor-mediated
276 ed high-frequency oncogenic mutations in the granulocyte-colony stimulating factor receptor (CSF3R) i
277 ause the erythropoietin, thrombopoietin, and granulocyte colony-stimulating factor receptors are all
280 s Stat3-NF-kappaB cross-talk, the release of granulocyte colony-stimulating factor, soluble intercell
281 ments with primary keratinocytes showed that granulocyte colony-stimulating factor stimulated Ras act
282 3-deficient cells showed hypersensitivity to granulocyte-colony stimulating factor, suggesting enhanc
283 aily oral cyclophosphamide administered with granulocyte colony-stimulating factor support for 15 wee
288 n antibacterial cytokine response comprising granulocyte colony-stimulating factor, tumor necrosis fa
289 d trial, eligible patients were treated with granulocyte colony-stimulating factor, underwent an aphe
290 osis, neutrophil count, type of peg-IFN, and granulocyte colony-stimulating factor use, none of these
291 terferon-gamma, tumor necrosis factor-alpha, granulocyte colony-stimulating factor, vascular-endothel
292 y every 3 weeks, 10 mg daily prednisone, and granulocyte colony-stimulating factor) versus abirateron
295 d to support erythroid colony formation, and granulocyte colony-stimulating factor was required to su
298 protein levels of tumor necrosis factor and granulocyte colony-stimulating factor were rapidly upreg
299 serum levels of IFN-gamma, IL-12, IL-6, and granulocyte colony-stimulating factor were significantly
300 , and inflammatory cytokines (interleukin-8, granulocyte colony-stimulating factor) were elevated in