ライフサイエンスコーパス: growth inhibition

1 -PD-1 Abs, each of which resulted in partial growth inhibition.

2 n vitro DHFR activity assay and A. baumannii growth inhibition.

3 ed cellular efficacy, caspase induction, and growth inhibition.

4 produce paradoxical responses leading to PC growth inhibition.

5 photosynthetic carbon assimilation rate, and growth inhibition.

6 l growth through unilateral photomorphogenic growth inhibition.

7 de MEK/ERK activity can paradoxically induce growth inhibition.

8 correlation between BRC1 expression and bud growth inhibition.

9 and provides insight into the origins of ice-growth inhibition.

10 degrees is absent and slow H2 efflux causes growth inhibition.

11 ne regulator (agr) alleles in the absence of growth inhibition.

12 ownregulation of EGFR protein and tumor cell growth inhibition.

13 specific antibodies led to significant tumor growth inhibition.

14 del-dependent tumor cell apoptosis and tumor growth inhibition.

15 d-type p53, leading to cell cycle arrest and growth inhibition.

16 more activated, cytotoxic, and resistant to growth inhibition.

17 assicaceae and has also been associated with growth inhibition.

18 ative breast cancer (TNBC) resulted in tumor growth inhibition.

19 athogens is often accompanied by significant growth inhibition.

20 actate production in vitro, followed by cell growth inhibition.

21 in Ewing sarcoma cells produced significant growth inhibition.

22 e effects and produce cell and proliferation growth inhibition.

23 d decreases sensitivity to cytokinin-induced growth inhibition.

24 ants are hypersensitive to osmotic stress in growth inhibition.

25 of the GSC critical genes, leading to tumor growth inhibition.

26 aft tumors resulted in PD-L1-dependent tumor growth inhibition.

27 t exhibits high levels of defense and strong growth inhibition.

28 to MeJA- and COR-induced root and hypocotyl growth inhibition.

29 using a HUWE1 knockdown model showed similar growth inhibition.

30 creased cancer cell proliferation, and tumor growth inhibition.

31 ysfunctional phenotype, culminating in tumor growth inhibition.

32 n of dapF(Ct) allowed the mutant to overcome growth inhibition.

33 where loss of PRDM10 results in severe cell growth inhibition.

34 -induced antitumor immune response and tumor growth inhibition.

35 sulfoximine (BSO) leads to synergistic cell growth inhibition.

36 de antibodies that override MAG-based neuron growth inhibition.

37 ary and sufficient for the reversion of root growth inhibition.

38 ile LuxS alleviates the B. fragilis-mediated growth inhibition.

39 , p53/LKB1 and p73/LKB1 axes in breast tumor growth-inhibition.

40 altered sensitivity to germination- and root growth-inhibition.

41 ts potent cytostatic properties (mean of 50% growth inhibition = 0.2 mum) in almost all cell lines of

42 litaxel antitumor activity (percentage tumor growth inhibition, 67.5%) in a xenograft syngeneic non-s

43 eria, but disruption of both bonds abrogates growth inhibition activity.

44 ypic readouts beyond classical antibacterial growth inhibition adopting a combined multiparametric hi

45 Regarding ZOL, we found a powerful growth inhibition after enforced miR-34a expression, whi

46 n CD8 and PD-L1-dependent tumor rejection or growth inhibition and a reduction in myeloid cells endog

47 h the TWIST1 inhibitor, harmine, resulted in growth inhibition and apoptosis in EGFR-mutant NSCLC.

48 bitor Ex527 greatly enhances MK-1775-induced growth inhibition and apoptosis in human lung cancer cel

49 CDK7 inhibitor THZ1 synergized in producing growth inhibition and apoptosis of human TNBC cells.

50 eases genomic instability, resulting in cell growth inhibition and apoptosis.

51 thin 1 hour of exposure, resulting in robust growth inhibition and apoptosis.

52 circulatory networks, requiring coordinated growth inhibition and arterial-venous specification.

53 Growth inhibition and cold-acclimation strategies help p

54 aphylococcus aureus (6.1 x 10(5) CFU/mg) via growth inhibition and cytoplasmic membrane damage.

55 potent inhibitor providing a balance between growth inhibition and cytotoxicity as well, which offer

56 with anti-Ang2 and radiation improved tumor growth inhibition and extended the survival of mice with

57 ng antibodies with radiation increases tumor growth inhibition and extends the survival of tumor-bear

58 Apple replant disease (ARD) leads to growth inhibition and fruit yield reduction in replanted

59 45 are more susceptible to IFNgamma-mediated growth inhibition and have reduced virulence in mice.

60 ed rates of protein synthesis, which lead to growth inhibition and hypersensitivity to osmotic stress

61 It was most effective for melanoma in growth inhibition and in preventing tumor formation in m

62 d exhibited the highest potency both in cell growth inhibition and in suppressing beta-cell death.

63 Similarly, in vivo DOX release, tumour growth inhibition and mice survival rates were influence

64 8 partially rescued SHMT1 loss-induced tumor growth inhibition and migration.

65 tentiates plant responses to JA such as root-growth inhibition and MYC-regulated gene expression.

66 Cytokinin-induced growth inhibition and osmotic stress hypersensitivity we

67 e P rhoeas SI response, including S-specific growth inhibition and PCD of root cells.

68 pression of membrane proteins often leads to growth inhibition and perturbs central metabolism and th

69 systems are important factors implicated in growth inhibition and plasmid maintenance.

70 f cognate PrsS and PrpS triggers pollen tube growth inhibition and programmed cell death (PCD) of sel

71 and gemcitabine significantly enhanced tumor growth inhibition and progression-free survival in an ag

72 tolerated and resulted in significant tumor growth inhibition and prolonged overall survival in both

73 (177)Lu-NM600 resulted in significant tumor growth inhibition and prolonged overall survival in mice

74 This results in tumour growth inhibition and radiosensitization despite the use

75 become partially insensitive to NO-dependent growth inhibition and re-orientation responses.

76 y and CDK4/6 inhibitors induces irreversible growth inhibition and senescence in vitro, and diminishe

77 tested range, all the extracts showed embryo growth inhibition and skeleton malformation activities w

78 This HS-PD method was applied to algal growth inhibition and springtail lethality tests with te

79 lon cancer cells, enhancing TGFbeta-mediated growth inhibition and stress-induced apoptosis.

80 Growth inhibition and suppression of phenylpropanoid met

81 f p38-p53 signaling, thereby contributing to growth inhibition and suppression of stress-induced apop

82 Both the tumor growth inhibition and survival were significantly improv

83 esults show a link between the observed leaf growth inhibition and the expression of specific cell cy

84 and sensing by PKR and MDA5; this results in growth inhibition and tumour inflammation, respectively.

85 ntimicrobial activity, rescues bacteria cell growth inhibition, and blocks induced cell permeabilizat

86 (LAZ1H1), causes vacuole morphology defects, growth inhibition, and constitutive activation of BR sig

87 in that binds AtRALF1, is essential for root growth inhibition, and has no role in AtRALF1 alkaliniza

88 reorientation, clustering, membrane damage, growth inhibition, and in the extreme case of hydrocarbo

89 d region (3'UTR) and 5'UTR, leading to tumor growth inhibition, and sensitizes NPC tumors to radiatio

90 ed growth of CC, whereas SC was resistant to growth inhibition, and this was coupled to increased tyr

91 of cancer cell in a quiescent state, such as growth inhibition, apoptosis, G0/G1 arrest and chemoresi

92 MMAE-based NDC doubled the duration of tumor growth inhibition as compared to cisplatin.

93 iple regression analysis of dose versus root growth inhibition, as well as saturation binding kinetic

94 B. pertussis was measured using B. pertussis growth inhibition assay (BGIA).

95 parasites as judged by an in vitro parasite Growth Inhibition Assay (GIA).

96 e present an optimised ex vivo mycobacterial growth inhibition assay (MGIA) to assess the ability of

97 We implemented an ex vivo mycobacterial growth inhibition assay (MGIA) to discriminate vaccine r

98 Interestingly, a real-time cell growth inhibition assay demonstrated that a single dose

99 er alone or in combinations in P. falciparum growth inhibition assay to determine Bliss' and Loewe's

100 ssis was measured using Bordetella pertussis growth inhibition assay.

101 nterest in functional in vitro mycobacterial growth inhibition assays (MGIAs), which provide a measur

102 Multicolor flow cytometry, cell sorting and growth inhibition assays were employed to compare the fr

103 in Tanzania, Senegal, and Mali were used in growth inhibition assays with transgenic parasite lines.

104 teractions between jazQ and phyB reveal that growth inhibition associated with enhanced anti-insect r

105 hey further establish an association between growth inhibition at high levels of defense and dysregul

106 Fraxini-induced growth inhibition (at a concentration of 1.25 mug/ml) wa

107 differences following exposure to CS, namely growth inhibition, augmented biofilm formation, increase

108 This mechanism is naturally robust to static growth inhibition but also allows us to "reprogram" cell

109 namic range by >70-fold, and mitigating host growth inhibition by >20%.

110 3)H]PMX and [(3)H]C2 transport and decreased growth inhibition by C1 and C2, and to a lesser extent b

111 dietary ellagic acid did not alter the tumor growth inhibition by docetaxel of xenografted 22Rv1 cell

112 c sensitizes enzalutamide-resistant cells to growth inhibition by enzalutamide.

113 nylation, and sensitized RAS-mutant cells to growth inhibition by FTI.

114 Tumor growth inhibition by full thickness excisional wounds wa

115 Thus, growth inhibition by individual drugs and combinations w

116 DeltaTgist mutants were more susceptible to growth inhibition by murine and human macrophages activa

117 ed tumor growth in vivo, and prevented tumor growth inhibition by PMPs.

118 RMP2, a cytosolic protein implicated in axon growth inhibition by Semaphorin/Plexin complexes.

119 Not only subjected to growth inhibition by spermine, the pauA2 mutant became m

120 hysical chemistry of the synergistic crystal growth inhibition by two inhibitors.

121 contributes to CD8(+) T cell-mediated tumor growth inhibition by unleashing inflammasome activation.

122 e growth at a level on par with the level of growth inhibition by vancomycin.

123 error induction, is the major cause of cell growth inhibition by viomycin.

124 Moreover, we have identified that cell growth inhibition can be achieved by expressing moderate

125 rradiation provided the most efficient tumor growth inhibition capability without severe systemic tox

126 Further investigation revealed that the growth inhibition caused by DDX24 depletion is independe

127 on of ABCF3 and OAS1B in bacteria alleviated growth inhibition caused by expression of OAS1B alone, a

128 Contact-dependent growth inhibition (CDI) is a mechanism by which bacteria

129 Contact-dependent growth inhibition (CDI) is a mechanism of inter-cellular

130 Contact-dependent growth inhibition (CDI) is a phenomenon defined by the d

131 Contact-dependent growth inhibition (CDI) is a wide-spread mechanism of in

132 Contact-dependent growth inhibition (CDI) is a widespread mechanism of bac

133 Contact-dependent growth inhibition (CDI) is an important mechanism of int

134 , we identified a specific contact-dependent growth inhibition (CDI) system enriched among highly vir

135 tive bacterial species use contact-dependent growth inhibition (CDI) systems to compete with neighbor

136 Bacterial contact-dependent growth inhibition (CDI) systems use a type Vb secretion

137 In CME and growth inhibition cell-based assays, the data obtained w

138 Measures of growth inhibition, cell cycle analysis, morphologic asse

139 Marked tumor growth inhibition compared to controls was observed, wit

140 Marked tumor growth inhibition compared with controls was observed, w

141 INOX experienced significantly greater tumor growth inhibition compared with i.v. FOLFIRINOX.

142 tants, the wild-type showed significant root growth inhibition compared with the fer mutant.

143 2-bisbenzimidazole combination had a similar growth inhibition curve to that of norfloxacin in E.coli

144 The mechanism for growth inhibition does not, however, involve canonical r

145 K studies and demonstrated significant tumor growth inhibition efficacy in mouse flank xenograft mode

146 Synergistic therapies show a higher tumor growth inhibition efficiency at a lower X-ray dose than

147 We performed extensive growth inhibition experiments, and we discovered that ce

148 umor cells and is able to drive potent tumor growth inhibition following intratumoral or intravenous

149 ed in 1alpha,25(OH)2D3 and MART-10 meditated growth inhibition for CCA as knockdown of NGAL decreased

150 Root growth inhibition from phosphate (Pi) deficiency is trig

151 ing the early stages of melanoma, poor tumor growth inhibition has been observed in more advanced mel

152 y gene regulator (agr) alleles in absence of growth inhibition (IC(50) values: 1-64 mug/mL).

153 D4(+) or CD8(+) T lymphocytes abolished this growth inhibition, identifying it as an immune-mediated

154 raft studies, compound 41 demonstrated tumor growth inhibition in both p53 wildtype and p53 mutant ca

155 cromolar range, whereas measurable bacterial growth inhibition in broth medium required >10-fold high

156 Growth inhibition in cAS cells was closely related to th

157 er, and type 2 diabetes syndrome, as well as growth inhibition in children.

158 wth inhibition) of 90% without any mortality growth inhibition in comparison to reported leads.

159 optosis, which resulted in significant tumor growth inhibition in CRC mouse models that express high

160 of caspase-1 and caspase-11 are required for growth inhibition in different cell types.

161 1 deficiency-associated loss of cell contact growth inhibition in endothelial cells and a potential t

162 sted at 25-500 mug/mL, showed up to 50% cell growth inhibition in four selected tumoral cell lines.

163 Optimization of BETi using growth inhibition in fulvestrant-resistant (MCF-7:CFR) c

164 d p16(INK4a) promoter repression and induced growth inhibition in HCT116 cells.

165 eceptor subtypes in regulating signaling and growth inhibition in human airway smooth muscle.

166 ally, RT53-killed B16F10 cells induced tumor growth inhibition in immunocompetent mice following a re

167 on toward the M1 phenotype, and led to tumor growth inhibition in MC38 and PANC02 syngeneic tumor mod

168 The nanovaccine led to potent tumour growth inhibition in melanoma, colon cancer and human pa

169 Solid tumor-growth inhibition in mice was increased when CAR T cells

170 t inhibitor causes apoptosis and synergistic growth inhibition in multiple EGFR tyrosine kinase inhib

171 the adaptive transition and led to prolonged growth inhibition in multiple patient-derived cell lines

172 azole 4-carboxamide ribonucleotide (ZMP) and growth inhibition in NCI-H460 and MDA-MB-231met2 cancer

173 ervical cancer cells and caused potent tumor growth inhibition in orthotopic mouse model.

174 ion in different tissue types and subsequent growth inhibition in plants.

175 key downstream target, and demonstrate that growth inhibition in PRDM10-deficient mESCs is in part m

176 imilarly alleviates the cellular defects and growth inhibition in ref8, suggesting that the growth re

177 SHP2 inhibition has demonstrated tumor growth inhibition in RTK-activated cancers in preclinica

178 iltration associated with tumor necrosis and growth inhibition in syngeneic murine allograft tumors.

179 eover, it showed high anticancer activity by growth inhibition in the MDA-MB-231 breast cancer cell l

180 he new compounds (2d, 2j, 2k, and 2n) showed growth inhibition in the XTT dye assay.

181 g large amounts of siderophore suffered less growth inhibition in toxic copper.

182 istently correlated with the extent of tumor growth inhibition in tumor xenograft models.

183 ehydrogenase complex, caused a profound cell growth inhibition in tumour cells harbouring KRAS mutati

184 ith AKT inhibitors causes synergistic tumour growth inhibition in vitro and in vivo.

185 d photodynamic therapy demonstrated complete growth inhibition in vitro of 2 multidrug-resistant MRSA

186 Growth inhibition in vitro was likely dependent on L. mu

187 ession of PUMA and BIM, led to apoptosis and growth inhibition in vitro, and tumor regression in vivo

188 esence of known AMPs and effectiveness of Bd growth inhibition in vitro.

189 cancer cells in culture, and enhances tumour growth inhibition in vivo.

190 in different models, and also leads to tumor growth inhibition in vivo.

191 prodrug molecules and shows effective tumor growth inhibition in vivo.

192 impact in vitro but results in a remarkable growth inhibition in vivo.

193 f MEK1/2, HDAC3, and G9a sustains PDAC tumor growth inhibition in vivo.

194 ll lines promoted tumor cell death and tumor growth inhibition in xenograft mouse models.

195 ivo effecting c-Myc downregulation and tumor growth inhibition in xenograft studies.

196 , resulting in mitochondrial dysfunction and growth inhibition, in an miR-24-dependent manner.

197 In contrast to the broad range of cell growth inhibition induced by DbBi, the antiproliferative

198 ctivation reduced premature catagen and hair growth inhibition induced by oxidative stress (H2O2 or m

199 In addition, the growth inhibition induced by Pcz could be rescued by exo

200 ibition exhibited the most potent effects on growth inhibition, induction of apoptosis, and delay of

201 The mechanism of unacylated ghrelin-mediated growth inhibition involves activation of Galphai and sup

202 cterial growth, it is generally assumed that growth inhibition is also their primary ecological funct

203 ium smegmatis, suggesting that V-58-mediated growth inhibition is due to interference with specific M

204 though beneficial for plant survival, active growth inhibition is often undesirable for crop producti

205 n vivo studies show that the extent of tumor growth inhibition is similar when mice are treated with

206 bodies, measured as serum-mediated bacterial growth inhibition, is better after wP than aP vaccinatio

207 tuents adopt distinct mechanisms of haematin growth inhibition, kink blocking and step pinning(12,13)

208 owth zone leads to dysproteostasis, skeletal growth inhibition, malformation, and chondrodysplasia, b

209 stress triggers various responses, including growth inhibition, mediated by the plant hormone abscisi

210 In vitro studies demonstrate that Ct growth inhibition occurs by interferon-gamma (IFN-gamma)

211 and enhancing the PDT efficacy with a tumor growth inhibition of 96.0%.

212 1B molecules are sufficient to induce strong growth inhibition of a microtubule plus end in vitro.

213 of tankyrase, revealing the compound induced growth inhibition of a number of tumor derived cell line

214 tivity and on its ability to rescue the cold-growth inhibition of a Saccharomyces cerevisiae tgs1Delt

215 d that SNI-1 enhances chemotherapy-dependent growth inhibition of a variety of cancer cells, includin

216 HP1BP3 inhibition led to preferential growth inhibition of ALT cells, which was accompanied by

217 while aged Ti3C2Tx membrane showed over 99% growth inhibition of both bacteria under same conditions

218 The growth inhibition of both drugs were mediated by inducti

219 ilic molecule in the series showing the best growth inhibition of both leukemia and solid tumor cell

220 Complete growth inhibition of both MRSA strains was demonstrated

221 DS-6051b induces dramatic growth inhibition of both wild type and G2032R mutant RO

222 hanced LSD1 inhibitor-induced senescence and growth inhibition of cancer cells in vitro and in mice.

223 We show that knockdown of Nrf2 caused modest growth inhibition of cells growing in two-dimension, whi

224 ons in traditional medicine was screened for growth inhibition of CRAB.

225 contact- and Esx secretion pathway-dependent growth inhibition of diverse Firmicute species.

226 destruction of the cell wall, and eventually growth inhibition of E. coli K-12.

227 of a biocide in situ, which resulted in the growth inhibition of E. coli, with as little as 3 min of

228 Moreover, tumor growth inhibition of E.G-7 tumors was closely correlated

229 nversely, overexpression of FabI rescued the growth inhibition of Escherichia coli by 6-OH-BDE-47, va

230 ef8, suggesting that the function of MYB4 in growth inhibition of lignin-modified plants is likely to

231 geneic orthotopic animal models, and induces growth inhibition of low-passage human PDAC cells in a s

232 , leading to suppression of Akt activity and growth inhibition of lung cancer in vitro and in vivo.

233 ly member inhibitors resulted in synergistic growth inhibition of MDA-MB453 cells, implicating Src as

234 Functional analyses encompassing in vitro growth inhibition of MRSA, and in vivo protection studie

235 or NSAH is within twofold of gemcitabine for growth inhibition of multiple cancer cell lines, while d

236 e novel AKT3 variant resulted in significant growth inhibition of multiple head and neck cell lines,

237 APLNR is involved in ATRA-induced growth inhibition of nasopharyngeal carcinoma and may su

238 , knockdown of APLNR diminished ATRA-induced growth inhibition of NPC cells.

239 nin deficiency, as gir1 has no effect on the growth inhibition of other lignin mutants that show the

240 In agreement, we demonstrate TspA-dependent growth inhibition of RN6390 by strain COL in the zebrafi

241 eractive effects and concentration dependent growth inhibition of S. acutus.

242 D complex of the HIPPO pathway, resulting in growth inhibition of several neoplasias.

243 produce functioning CDI systems that mediate growth inhibition of sister cells lacking the cognate im

244 ted corepressor protein KAP1 all resulted in growth inhibition of TBX2-expressing breast cancer cells

245 +) near the bacteria cell wall that leads to growth inhibition of the bacteria.

246 cell lines and was able to induce 60% tumor growth inhibition of the CW22Rv1 in vivo xenograft model

247 Additionally, 1 showed strong growth inhibition of the marine fungus Dendryphiella sal

248 Root toxicity assays revealed increased root growth inhibition of the mutants if cultivated in the pr

249 iazoles and leucine linker to obtain maximal growth inhibition of the parasite.

250 26a was able to induce an 80% tumor growth inhibition of xenografts derived from the enzalut

251 ant in vivo anticancer efficacies with tumor growth inhibitions of 45.8% (po, 150 mg/kg) and 62.6% (i

252 hich were screened for effects on tumor cell growth, inhibition of tubulin polymerization, and induct

253 er (TNBC) syngeneic models with a TGI (tumor growth inhibition) of 90% without any mortality growth i

254 >/=25 muM As, reflected in stronger seedling growth inhibition on agar plates.

255 TMNPs showed enhanced penetration and growth inhibition on MCs.

256 acteria upon cell-cell contact, resulting in growth inhibition or death unless a specific immunity pr

257 asm of a neighboring bacterium, resulting in growth inhibition or death unless the recipient bacteriu

258 uggested to function in Al(3+)-mediated root growth inhibition: our data suggest that if Ulva Al(3+)

259 using both sorafenib and MEAN enhanced tumor growth inhibition over monotherapy with either agent.

260 igh specific activity induced superior tumor growth inhibition (P = 0.021, n = 5/group) without subac

261 neuroblastoma cells resulted in significant growth inhibition (P = 2.6 x 10(-2) to 3.4 x 10(-5)) and

262 The growth inhibition potentials of both 0.06%w La(NO3)3 and

263 ant ref8, and report the identification of a GROWTH INHIBITION RELIEVED 1 (GIR1) gene from a suppress

264 e affinity of cell binding and ensuing tumor growth inhibition reveal the linker length to be a bidir

265 To corroborate the growth inhibition role of SVP2 in kiwifruit development

266 ty-related readouts, including flg22-induced growth inhibition, ROS production and callose deposition

267 ime (2 h for our biosensor vs. 24 h-10 d for growth inhibition test) and the data are consistent with

268 Tumor growth inhibition (TGI) following treatment with RXDX-10

269 ent FLT3 exhibited more robust apoptosis and growth inhibition than did Y969 phosphorylation-deficien

270 Lactobacillus crispatus demonstrated less growth inhibition than Escherichia coli.

271 K-562 cell line) did not result in selective growth inhibition; this is similar to the findings repor

272 deacetylation of Miro1 as a mediator of axon growth inhibition through decreased mitochondrial transp

273 s and in stress by antagonizing SA-dependent growth inhibition through UPR modulation.

274 Antibiotic screens typically rely on growth inhibition to characterize compound bioactivity-a

275 immune system to antiprogestin-induced tumor growth inhibition using a hormone-dependent breast cance

276 eened a set of predicted essential genes for growth inhibition using CRISPR-interference (CRISPRi) kn

277 th in vivo efficacy outcomes that ~90% tumor growth inhibition was achieved by Dox-loaded HER2 recept

278 About 99% growth inhibition was achieved if amp-resistant E. coli

279 Growth inhibition was associated with increased numbers

280 We predicted that parasite-growth inhibition was associated with platelet consumpti

281 ivity of g6pd6 mutant plants to PAMP-induced growth inhibition was complemented by a phosphomimetic b

282 BMP4-mediated growth inhibition was dependent on type I receptor activ

283 ft models as a single agent; SY-1365-induced growth inhibition was enhanced in combination with the B

284 Growth inhibition was observed following treatment with

285 Similar growth inhibition was observed in syngeneic GL261 GBM (p

286 Importantly, growth inhibition was reversed when the compounds were r

287 In xenografts, a significant tumor growth inhibition was seen after twice-weekly intravenou

288 a significantly lower nitrogen content and a growth inhibition were observed, with a concomitant incr

289 pe II PRMT, produces synergistic cancer cell growth inhibition when combined with GSK3368715.

290 kinase (MEK) inhibitors yielded synergistic growth inhibition when combined with the EGFR inhibitors

291 types of the corresponding knockouts include growth inhibition when exposed to ethanol or other short

292 sponse to single agent with lack of enhanced growth inhibition when the two agents were combined.

293 over, both cell types manifested synergistic growth inhibition when treated with chloroquine plus the

294 ain inhibitor I-BET151 resulted in selective growth inhibition, whereas treatment of leukaemia cells

295 Setdb1 ablated ES cells exhibit severe growth inhibition, which is not rescued by exogenous Nan

296 ic conditions that provide significant tumor growth inhibition with acceptable host body weight reduc

297 Significant tumor growth inhibition with brontictuzumab was observed exclu

298 s whose inhibition creates synergistic tumor growth inhibition with gemcitabine (Gem), a first- or se

299 th mainly MET phosphorylation, profound cell growth inhibition with induction of apoptosis was observ

300 ing kiwifruit development SVP2 has a role in growth inhibition, with high-chill kiwifruit Actinidia d