ライフサイエンスコーパス: growth phase

1 y phase of ENSO is more predictable than the growth phase.

2 July 2014, when the outbreak entered a rapid growth phase.

3 enous jasmonates across the plant vegetative growth phase.

4 by increase in beta-sheet content during the growth phase.

5 terial growth, but not during the stationary growth phase.

6 ion-collapsing alkaline pH of the stationary growth phase.

7 ensity data for cycles near the onset of the growth phase.

8 for this process, but can enhance the later growth phase.

9 acterized by a lag phase followed by a rapid growth phase.

10 models, which involve an initial exponential growth phase.

11 with the lack of Ace expression in the early growth phase.

12 isms to the drug is independent of bacterial growth phase.

13 ere age, site, Breslow thickness, and radial growth phase.

14 rization phase, an intermediate phase, and a growth phase.

15 criptional program that depend mainly on the growth phase.

16 and reaches its maximum at late exponential growth phase.

17 ly higher cell quotas in the mid-exponential growth phase.

18 ty near neutral pH and during the stationary-growth phase.

19 mpact of infection occurring during the host growth phase.

20 ted and responsive to both growth medium and growth phase.

21 ylakoids when cells were in post-exponential growth phase.

22 itial seed concentration to the onset of the growth phase.

23 smegmatis is selectively modulated with the growth phase.

24 cripts were most abundant at the exponential growth phase.

25 hyma, and vascular tissues) at their maximal growth phase.

26 nt and wild-type (WT) strains in exponential growth phase.

27 cause of a shortage of ribosomes during this growth phase.

28 rature raised it; radiation impact varied by growth phase.

29 ulture is detectable only in the exponential growth phase.

30 n the resting pool and more committed in the growth phase.

31 eping close to 100% viability throughout the growth phase.

32 ination efficiency seems to be unaffected by growth phase.

33 ximal expression observed during exponential-growth phase.

34 ch as that seen during the early exponential growth phase.

35 of the tetQ-rteA-rteB operon is affected by growth phase.

36 bundance of VOCs was influenced by bacterial growth phase.

37 nvert into protofibrils that elongate in the growth phase.

38 by RteA did not make excision independent of growth phase.

39 conditions made NBU1 excision independent of growth phase.

40 ganic solvent, octanol and in the stationary growth phase.

41 y, thereby suppressing the subsequent fibril growth phase.

42 teria consume nutrients and enter stationary growth phase.

43 into ordered structures that then enter the growth phase.

44 utions to obtain a sequential nucleation and growth phase.

45 n as compared to cultures in the exponential growth phase.

46 zed to the inner bulge during the hair cycle growth phase.

47 rains of varying serotypes in the stationary growth phase.

48 es are translationally induced at transition growth phase.

49 BB0449 protein levels are low during various growth phases.

50 SELEX targeted bacterial cells at different growth phases.

51 pylori strains, genetic loci, and bacterial growth phases.

52 species-level and to discriminate bacterial growth phases.

53 syn amyloid formation affecting both lag and growth phases.

54 depth and its evolution during the different growth phases.

55 ant occurred at middle, late, and stationary growth phases.

56 merases were constitutively expressed in all growth phases.

57 ulator (TcdC) allows toxin production at all growth phases.

58 es in M. pneumoniae during distinct in vitro growth phases.

59 es in cells grown to mid to late exponential growth phases.

60 n response to environmental changes, such as growth phases.

61 ess phenotypes in logarithmic and stationary growth phases.

62 chromosome during exponential and stationary growth phases.

63 During stationary growth phase, 285-GHz continuous wave EPR measurements s

64 After the first exponential growth phase, a short lag phase of nongrowth is observed

65 xamine differential gene expression in these growth phases, a microarray was constructed based on the

66 ep-wise transition to latent and overt tumor growth phases, a process that is preceded by recruitment

67 g phase at the proximal poly(A) site, but as growth phase advances, it extends to the distal site.

68 wth on CO and H(2) results in an accelerated growth phase, after which a downcycle is observed in syn

69 ia also illustrate that an extended pubertal growth phase allows very considerable height recovery, e

70 ls early insulin hypersecretion and a robust growth phase along with hyperexpression of related genes

71 Independence of growth phase also occurred when rteA and rteB were place

72 pression is somewhat constant throughout the growth phases, although it gradually decreases as cells

73 cular mechanism responsible for the end of a growth phase, an event called catastrophe, is still not

74 either Drosha or Dicer during an established growth phase (anagen) caused failure of hair follicles t

75 ed for hair follicle development and for the growth phase (anagen) of postnatal follicles.

76 hair follicle repetitively progresses from a growth phase (anagen) to a rapid apoptosis-driven involu

77 delayed entry and progression into the hair growth phase (anagen).

78 viduals, with an increased proportion in the growth phase, anagen.

79 iently, as indicated by a longer logarithmic growth phase and a higher final cell density.

80 enes so that regulation is synchronized with growth phase and cellular energy status.

81 Depending on growth phase and culture conditions, cardiolipin (CL) ma

82 a continuous increase in activity during the growth phase and decreased during the stationary phase.

83 extent of this cyclization also varies with growth phase and electron acceptor or donor limitation.

84 s to regulate gene expression in response to growth phase and environmental change.

85 mental isolates changed with P availability, growth phase and genotype, with P availability having th

86 eculation represents the initial ventricular growth phase and is necessary for embryonic survival.

87 The effects of salts on the growth phase and lag phase of IAPP amyloid formation are

88 l lines WM793 and especially WM239 (vertical growth phase and metastatic cells, respectively) overexp

89 e expression of virulence genes according to growth phase and nutritional status.

90 expression of 60 genes independently of the growth phase and of 122 genes in a growth phase-dependen

91 val after ampicillin exposure throughout its growth phase and protected the population against exposu

92 data showed a transition between the initial growth phase and stable-state phase that, in the case of

93 Amount of 7-heptadecene varied with growth phase and temperature and was strictly dependent

94 riation in their binding profiles across the growth phase and the genome-scale nature of their impact

95 lity of yeast during the initial exponential growth phase and throughout fermentation.

96 and protects sigma(S) during the exponential growth phase and thus enables rapid gene activation by s

97 ice exhibit enhanced resting and abbreviated growth phases and are delayed in response to tissue-rege

98 ibutions showed population differences among growth phases and community differences among lakes that

99 normal cell replacement cycle throughout all growth phases and do not undergo spontaneous involution.

100 In woody species, growth phases and dormancy follow one another consecutiv

101 herichia coli) over time through lag and log growth phases and following antibiotic administration an

102 -3 cross-linkages predominate throughout all growth phases and the ratio of 4-3/3-3 linkages does not

103 show that production of BoNTs depends on the growth-phase and is under the control of positive and ne

104 ved for preaggregated medin species from the growth-phase and rarely for lag-phase species.

105 , Spd-sr37 (80 nt; strongly expressed in all growth phases), and CcnA (93 nt; induced by competence s

106 al levels of 2.5 muM in the late logarithmic growth phase, and both wild-type and pigmentation (pgm)

107 ary-state markers are present throughout the growth phase, and increase in frequency with cell densit

108 to eradicate persisters, MRSA in stationary growth phase, and showed significant clearance of intrac

109 lla larvae that was strain, infectious dose, growth phase, and T4SS dependent.

110 genome is modified in response to change in growth phase, and that 5% (68 genes) of the genome is te

111 es with a few more DP cells can re-enter the growth phase, and those that do exploit an intrinsic mec

112 , Spitzoid or Non-Spitzoid histology, radial growth phase, and vascular invasion.

113 rly flowering time, a prolonged reproductive growth phase, and, thereby, increased seed yield suggest

114 utrient iron concentrations, carbon sources, growth phases, and O(2) concentrations to better underst

115 fluxes corresponding to the mid-exponential growth phase are elucidated for seven single gene deleti

116 a that are expressed during the necrotrophic growth phase, as well as programmed cell death mediated

117 ate enhanced body and muscle size during the growth phase associated with elevated IGF1 levels.

118 om bacteria in the mid- and late-exponential growth phases at 28 degrees C and 37 degrees C correlate

119 opose that the murine iBAT has two different growth phases between birth and weaning: increase of BAs

120 By univariable analysis, age, growth phase, Breslow thickness, ulceration, mitotic rat

121 ssful in prototroph yeast during exponential growth phase but not in stationary phase.

122 o be present in cotyledons during their main growth phase, but not later.

123 MCSP expression in a radial growth phase cell line also promotes an epithelial-to-me

124 ss the plasma membrane whereas in stationary growth phase cells Ca(2+) influx from intracellular and

125 d accumulation of myosin II in the cortex of growth-phase cells.

126 We show that cells harvested in exponential growth phase consistently display mixtures of 2-fold and

127 early a century ago, the molecular basis for growth phase control of speB gene expression remains unk

128 drate regulation of lctO as a key element of growth phase control.

129 rential gene expression, morphology, and the growth-phase dependence of Hg transformations suggests t

130 nical strain of S. pyogenes and relieved its growth phase dependency.

131 of LspA2 but not LspA1 was shown to be both growth phase dependent and affected by the presence of f

132 his pattern of transcription was mirrored by growth phase dependent expression of the K1 capsule.

133 erogeneity of cap expression or the strictly growth phase dependent expression.

134 The growth phase dependent transcription was regulated by In

135 Expression of sarZ was growth phase dependent with maximal expression in the ea

136 The expression of covR is growth phase dependent, with maximal expression observed

137 xpression is also heterogeneous and strongly growth-phase dependent.

138 mutations on gene expression was analyzed in growth phase-dependent conditions using C medium, report

139 antibiotic exposure, and it also displays a growth phase-dependent derepression of the mexAB-oprM op

140 However, CP synthesis remains growth phase-dependent even when transcription is render

141 that the T2S pathway is characterized by the growth phase-dependent expression of genes encoding carg

142 nteracts with the cofactor LacD.1 to control growth phase-dependent expression of genes, including sp

143 This growth phase-dependent gene expression is controlled tra

144 axis is determinative for regulation of the growth phase-dependent gene expression.

145 Growth phase-dependent gene regulation has recently been

146 Bvg(-) phase-locked mutants, indicating that growth phase-dependent gene regulation in B. bronchisept

147 analysis revealed and qRT-PCR confirmed that growth phase-dependent gene regulation occurred in both

148 ative real-time PCR (qRT-PCR) confirmed that growth phase-dependent gene regulation occurs in B. bron

149 iseptica-like ancestor, we hypothesized that growth phase-dependent gene regulation would also occur

150 olymerase sigma initiation factors regulates growth phase-dependent gene transcription.

151 anning hours to weeks, we establish distinct growth phase-dependent hierarchies of polymerase mutant

152 e III secretion system (TTSS) genes led to a growth phase-dependent increase in a TTSS-dependent func

153 B0693) as a negative regulator that controls growth phase-dependent induction of rpoS and bosR in Bb.

154 Our data identify CPK28 as a growth phase-dependent key negative regulator of distinc

155 of several JA metabolites were elevated in a growth phase-dependent manner in cpk28, and accumulation

156 In addition, PepO protein was secreted in a growth phase-dependent manner to the culture supernatant

157 coli that nanoRNAs prime transcription in a growth phase-dependent manner, resulting in alterations

158 ated by the PhoB/R two-component system in a growth phase-dependent manner, which is coordinated with

159 unt of Pah1p; enzyme abundance declined in a growth phase-dependent manner.

160 DnaB is truncated at its C-terminus in a growth phase-dependent manner.

161 ly of the growth phase and of 122 genes in a growth phase-dependent manner.

162 Production of H(2)O(2) follows a growth phase-dependent pattern that mimics that of many

163 dative phosphorylation is a primary cause of growth phase-dependent persistence to quinolone antibiot

164 ClpXP is a major regulator of growth phase-dependent proteins, and these results sugge

165 , we show that RNase BN itself is subject to growth phase-dependent regulation, because both rbn mRNA

166 In this study, we examined the growth phase-dependent speB mRNA level and decay using q

167 ughout V. cholerae growth, whereas there was growth phase-dependent transcriptional activity of genes

168 lycogen accumulation were carbon source- and growth phase-dependent, and were repressed by glucose.

169 protein radiolabeling that LLO synthesis was growth phase-dependent.

170 NAs and formation of the L1 and L2 loops are growth phase-dependent.

171 f atcRS during late-stage growth, indicating growth-phase-dependent expression.

172 BCP-deficient B. cenocepacia exhibit a growth-phase-dependent hypersensitivity to oxidative kil

173 er and slowly yields to the EA1 S layer in a growth-phase-dependent manner.

174 amount and type of recombination events in a growth-phase-dependent manner.

175 e with iron transport and the correlation of growth-phase-dependent morphology with MeHg production a

176 iably distinguish between the nucleation and growth phases, despite extensive and diverse attempts.

177 developmental switch out of an initial "pro-growth" phase during which muscle arms grow out and form

178 itory phase between the attack phase and the growth phase, during which the bdelloplast (the invaded

179 Moreover, RteA is not required for the growth phase effect, because a mutant form of RteB that

180 four-electrode configuration along all cell growth phases, enabling determination of relevant cell g

181 factor with IHF playing a role in regulating growth phase expression.

182 g the transcriptome to degrade RNAs encoding growth-phase factors and, thus, support the maturation p

183 ctivation, quiescent naive T cells undergo a growth phase followed by massive clonal expansion and di

184 at cell cultures have reached an appropriate growth phase for addition of an agent to induce protein

185 on natural killer T cells helped to launch a growth phase for this field of research.

186 m forest procedure selected 6 variables (not growth phase) for inclusion in the logistic model and no

187 Substrates consumed in the second growth phase (glutamate, proline, and associated organic

188 eat stress presents a temporal pattern among growth phases (GPs) and vary interannually.

189 haping the Drosophila wing during its larval growth phase has been limited, impeding our ability to u

190 Plant extracts and their different growth phases have been manipulated for the fabrication

191 In the subsequent growth phase, hierarchical agglomeration could be a domi

192 ontrolled virulence genes at the exponential growth phase; however, mutations of RocA but not mutatio

193 Vertical growth phase human melanoma cells show higher oxygen con

194 he pathogen's early (day 2 to 3) exponential-growth phase in an experiment involving synchronized inf

195 omic DNA methylation levels as a function of growth phase in Escherichia coli.

196 We further identify a growth phase in which the C- ribosome remains active, wh

197 teobacteria, it is ubiquitous throughout all growth phases in S. aureus The biological significance o

198 us, and its increased production during late growth phases indicates that c-di-AMP controls processes

199 underlie the mechanism of antibiotic- and/or growth phase-induced lysis for other important Gram-posi

200 ported - and that the effect varies by taxa, growth phase, isotope label and applied protocol.

201 the crossover time (t(c)), the slope of the growth phase (k(g)), and the arrest time (t(a)).

202 f this kinase in stasis, while in stationary growth phase, LinDYRK1(-/-) parasites had important defe

203 In logarithmic growth phase, LinDYRK1(-/-) parasites proliferated bette

204 Paradoxically, the growth phase-mediated inductions of PAH1 and phosphatida

205 mental and cellular state, including strain, growth phase, medium, oxygen level, antibiotic and carbo

206 utilization of glutamine compared to radial growth phase melanoma cells.

207 in normal immortal melanocytes, early radial growth phase melanoma, and metastatic melanoma cells.

208 t forced expression of OPN in early vertical-growth-phase melanoma cells dramatically increased their

209 Overexpression of OPN in vertical-growth-phase melanoma cells induced down-regulation of C

210 c melanoma cell lines, derived from vertical growth phase (MelJuSo) and metastatic (SKMel28) melanoma

211 mperature (35 degrees C or 40 degrees C) and growth phase (mid-exponential or stationary phase).

212 otein-DNA complexes formed by Fis, the major growth phase nucleoid protein, have a markedly different

213 ly, consistent with aestivation, whereas the growth phase of both A. gambiae s.s. and A. arabiensis l

214 ile influenced by compound glycosylation and growth phase of cultured cells.

215 veloping on hard substrates and report a new growth phase of MDCK II cells on soft gels.

216 The growth phase of OEC photoassembly shows an H/D solvent i

217 ining characteristics showed variations with growth phase of the bacteria.

218 es that enabled direct identification of the growth phase of the bacteria.

219 ants/mug DNA) was obtained at the stationary growth phase of the bacterium (OD 6.0) using 25 ng of pl

220 at binucleate cells are delayed in the first growth phase of the cell cycle (G1) and undergo interpha

221 nuclei/10(6) myocyte nuclei, P<0.05) and the growth phase of the cell cycle (Ki67; 410+/-82 to 1261+/

222 logical evidence of myocytes re-entering the growth phase of the cell cycle and increases in the numb

223 g to this institution during the exponential growth phase of the COVID-19 outbreak in New Orleans (Ma

224 r they originate from skin in the resting or growth phase of the hair cycle or skin with beta-catenin

225 n of the NMR spectra clearly depended on the growth phase of the host cells.

226 t for V. cholerae PBP1a pathway mutants, the growth phase of the inoculum is a key modulator of infec

227 riptional program, which in turn extends the growth phase of the thymus and enhances thymic output; m

228 ured in different time points throughout the growth phases of 4469 Scottish Blackface sheep and weath

229 c aspects of metabolome reprogramming in the growth phases of Colletotrichum spp as determined by nut

230 rations above the lower CC (CC(Elongation)), growth phases of individual filaments can occur transien

231 ression of ldtMt2 is dominant throughout the growth phases of M. tuberculosis.

232 yzing the metabolic changes accompanying the growth phases of medically treated AAA.

233 In contrast, cells in stationary growth phase or cells treated with a protonophore causin

234 ABC mutant lacks detectible CL regardless of growth phase or growth conditions.

235 gulation of speB transcription at stationary growth phase or in subcutaneous infection of mice.

236 RNA polymerase, is expressed independent of growth phase or nutrient stress in culture, and is proce

237 odification, or degradation depending on the growth phase or presence of environmental stressors.

238 rapid and synchronous cell divisions without growth phases or cell cycle checkpoints.

239 expression in radial growth phase, vertical growth phase, or metastatic cell lines causes sustained

240 e found to increase in number throughout the growth phases, particularly in the stationary phase.

241 an uncoupling of lctO transcription from its growth phase pattern.

242 hafniense strain Y51 was affected by neither growth phase, pceA transcription, or translation, nor by

243 The distinct LinDYRK1(-/-) growth phase phenotypes were mirrored by the distinct Li

244 ssion changes between biofilm and stationary growth-phase planktonic cultures.

245 ntiation in which exponential and stationary growth phases play key biological roles.

246 S exposure on the surface of late stationary growth phase promastigotes from patients with LCL, compa

247 macrophage interaction with late stationary growth phase promastigotes in which PS was blocked by an

248 expressed on the surface of early stationary growth phase promastigotes.

249 The EPS produced simultaneously with the growth phase reached a maximum of 22 g/L after attaining

250 the first indication that RNase BN itself is growth phase-regulated.

251 We studied the role of the growth-phase-regulated outer membrane protein OpnS in ho

252 e distinct mechanisms and that disruption of growth phase regulation alters transcriptional patterns

253 stal promoter indicated that it accounts for growth-phase regulation and DNA damage inducibility.

254 Cells in exponential growth phase released 220 exosomes/cell in culture mediu

255 ession during the logarithmic and stationary growth phases respectively.

256 ring carbon availability or in fueling later growth phases, respectively, has been proposed.

257 low phosphate and this increase prolongs the growth phase, resulting in the development of long root

258 nsition of melanoma from non-invasive radial growth phase (RGP) to invasive and metastatically compet

259 During both growth phases, several genes carried in O-islands were a

260 ystem, and a poorly defined post-exponential growth phase signal independent of Agr.

261 genetic stability approximately tenfold and growth phase-specific productivity approximately fourfol

262 Our findings identify StpA as a novel growth phase-specific regulator that plays an important

263 es C), to probe for temperature-specific and growth phase-specific transcriptomes.

264 f cobalt, nickel, and sodium showed distinct growth-phase-specific patterns.

265 Following the initial growth phase, spheroids were treated with auristatin as

266 Regulation by StpA varied with growth phase; StpA controlled sigma(38) levels at mid-ex

267 l conditions such as antibiotic exposure and growth phase, suggesting that observed shifts in the pha

268 gene in a codY mutant during the exponential-growth phase, suggesting that the quorum-sensing system,

269 of uptake was no longer documented after the growth phases, suggesting a pattern of cyclic metabolic

270 cally homogenous, whereas in the exponential growth phase, the average wall stiffness increased, with

271 (E(h)), the degree of which is dependent on growth phase, the microbial taxa, and their physiology.

272 ssive phenotype and the postexponential (PE) growth phase, the pathogens express virulence factors, b

273 ition if initial infection occurs during the growth phase then an additional "invasion zone" can exis

274 reorganization of the oligomers, whereas the growth phase ultimately results in the formation of fibr

275 essed during late exponential and stationary growth phase under normal growth conditions, whereas the

276 chicken cecum and in two different in vitro growth phases using strand-specific RNAseq.

277 ed levels of Ace surface expression at early growth phase versus those of wild-type OG1RF.

278 results show that MCSP expression in radial growth phase, vertical growth phase, or metastatic cell

279 year old female skin melanoma at a vertical growth phase (VGP) in the primary melanoma site, (2) WM7

280 vasive and metastatically competent vertical growth phase (VGP) is a major step in tumor progression,

281 2) WM793 cells established from the vertical growth phase (VGP) of a primary skin melanoma lesion, (3

282 of melanoma cells originating from vertical growth phase (VGP), and skin and lung metastasis, respec

283 -factorial experimental design we found that growth phase, voltage, and resistance all significantly

284 of the LCL strain during the late stationary growth phase was highly expressed on the surface of earl

285 changes in response to temperature shift and growth phase was the induction of known B. quintana viru

286 produced during the late stage of stationary growth phase, was discovered and purified from the cultu

287 isms coupling virulence factor expression to growth phase, we investigated the molecular basis for H(

288 d to the light-dark cycle at the exponential growth phase, we repeatedly observed that the mean swimm

289 ant strain during logarithmic and stationary growth phases, we determined that PknK regulates the exp

290 ogen removal rates during the initial linear growth phase were 17 and 122 mg.L(-1).d(-1), respectivel

291 ss section of the solutes during the fibrils growth phase were thus recovered.

292 VOCs emitted over the course of the growth phases were collected from the headspace above th

293 speB transcript level during the exponential growth phase, whereas provision of only the amino-termin

294 growth pattern and exhibited only the first growth phase, which is marked by the consumption of aspa

295 normally exhibits a lag phase followed by a growth phase, which leads to amyloid fibrils.

296 and declined sharply during mid-to-late log growth phases, which was in direct contrast to other myc

297 ntracellular bacteria are in the exponential growth phase with a reduced replication rate and biochem

298 is disabled, consistent with the exponential growth phase with an infinite replicative capacity.

299 r dioxide, and nitrogen dioxide in different growth phases with clinically assessed pubertal stage at

300 d melanoma cells originating from the radial growth phase (WM35) and from lung metastasis (A375-P).