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1 ine and the adrenergic neuron-blocking agent guanethidine.
2 reatment with the adrenergic neuron blocker, guanethidine (20 mg/kg), also abolished the PVN-mediated
3 sions of the mesentery and by application of guanethidine (3 microM) to the stimulation and recording
4      In contrast, neither splanchnectomy nor guanethidine (5 mg kg(-1)) had an effect.
5               Pretreatment with subcutaneous guanethidine (5 mg/kg) and propranolol (1 mg/kg), but no
6 oxamine (6-20 microM) and in the presence of guanethidine (5 microM), EFS of perivascular sensorimoto
7 rivative, displays the same pH dependence as guanethidine, a heptamethyleniminoethyl- guanidine deriv
8 as inhibited by 1 mum tetrodotoxin and 5 mum guanethidine, although not by the 1 mum of the CGRP rece
9     These events are stimulated by NE and by guanethidine, an hNET substrate, and they are blocked by
10 ing NET is metaiodobenzylguanidine (MIBG), a guanethidine analog of norepinephrine.
11 HDA] treatment) and long-term sympathectomy (guanethidine and 8 days after 6-OHDA) on the distributio
12                 In the presence of atropine, guanethidine and propranolol, electrical field stimulati
13  88.6+/-10.9% (n=5) and 80.4+/-2.2% (n=5) by guanethidine and propranolol, respectively.
14 TX, C6 and L-NAME, but not by VIP antiserum, guanethidine and splanchnicotomy.
15 s blocked by tetrodotoxin, nitro-L-arginine, guanethidine, and beta2-adrenoceptor antagonists.
16 were antagonized by postganglionic action of guanethidine, but not by phentolamine or suramin.
17                                              Guanethidine completely suppressed the heat hyperalgesia
18 he release of sympathetic neurotransmitters (guanethidine or reserpine) inhibited capsaicin-evoked iC
19  with atropine or hexamethonium but not with guanethidine or vagotomy.
20 tive intestinal polypeptide (VIP) antiserum, guanethidine, or splanchnicotomy.
21                                              Guanethidine pretreatment attenuated myenteric but not D
22  of sympathetic fiber function with systemic guanethidine significantly decreased the pain-related be
23 ale Sprague-Dawley rats were pretreated with guanethidine sulfate (40 mg/kg daily for 5 weeks) or veh
24 cted 50% with noradrenaline and treated with guanethidine (to inhibit sympathetic neurotransmission),